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1 disorder distinct from chronic inflammatory demyelinating polyneuropathy.
2 lain-Barre syndrome and chronic inflammatory demyelinating polyneuropathy.
3 rse myelitis, and 1 had chronic inflammatory demyelinating polyneuropathy.
4 anti-myelin-associated glycoprotein antibody demyelinating polyneuropathy.
5 llain-Barre syndrome or chronic inflammatory demyelinating polyneuropathy.
6 %) with newly diagnosed chronic inflammatory demyelinating polyneuropathy.
7 opathy as distinct from chronic inflammatory demyelinating polyneuropathy.
8 within the spectrum of chronic inflammatory demyelinating polyneuropathy.
9 ssociation between CD and acute inflammatory demyelinating polyneuropathy (0.8; 0.3-2.1; P = .68).
12 ropathy and variants of chronic inflammatory demyelinating polyneuropathy, account for a proportion o
13 pathologic criteria, with acute inflammatory demyelinating polyneuropathy (AIDP) and acute motor axon
14 hina occurs in two forms: acute inflammatory demyelinating polyneuropathy (AIDP) and acute motor axon
15 ts in early stages of the acute inflammatory demyelinating polyneuropathy (AIDP) pattern of Guillain-
17 ived from patients with chronic inflammatory demyelinating polyneuropathy, an autoimmune disease of t
19 tion indicated for a patient presenting with demyelinating polyneuropathy and concurrent papilledema.
21 uillain-Barre syndrome, chronic inflammatory demyelinating polyneuropathy, and multifocal motor neuro
23 a multifocal variant of chronic inflammatory demyelinating polyneuropathy but that multifocal motor n
24 inclusion-body myositis, paraproteinemic IgM demyelinating polyneuropathy, certain intractable childh
25 arre syndrome (GBS) and chronic inflammatory demyelinating polyneuropathy (CIDP) are conditions that
27 thirds of patients with chronic inflammatory demyelinating polyneuropathy (CIDP) need long-term intra
28 and nine patients with chronic inflammatory demyelinating polyneuropathy (CIDP) were compared with 1
29 opsies of patients with chronic inflammatory demyelinating polyneuropathy (CIDP), CIDP associated wit
30 of nerve hypertrophy in chronic inflammatory demyelinating polyneuropathy (CIDP), magnetic resonance
35 autoimmunity resembling chronic inflammatory demyelinating polyneuropathy develops spontaneously in N
36 e (GBS), especially the classic inflammatory demyelinating polyneuropathy form, seems to involve lymp
37 ence of CMT disease and chronic inflammatory demyelinating polyneuropathy indicate that the associati
39 uillain-Barre syndrome, chronic inflammatory demyelinating polyneuropathy, multifocal motor neuropath
40 uillain-Barre syndrome, chronic inflammatory demyelinating polyneuropathy, multifocal motor neuropath
41 e immunopathogenesis of chronic inflammatory demyelinating polyneuropathy remain still fragmentary an
44 were consistent with the acute inflammatory demyelinating polyneuropathy subtype of the Guillain-Bar
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