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1  paracrine factors that stimulate melanocyte dendricity.
2 ing contribute to PGE(2)-mediated melanocyte dendricity.
3 rradiation, a potent stimulus for melanocyte dendricity.
4 a (PKCzeta) activity abrogated LPC-dependent dendricity.
5 e to sPLA2-X activity, stimulates melanocyte dendricity.
6 late both tyrosinase activity and melanocyte dendricity.
7 ns that upon activation stimulate melanocyte dendricity and activity of tyrosinase, a key enzyme in m
8             MLO-Y4 cells show an increase in dendricity and elongation of dendrites in response to sh
9 days 1-5 and comparably increased melanocyte dendricity and epidermal melanin content.
10 ary changes through modulation of melanocyte dendricity and melanin synthesis.
11 diated skin, an increase in melanocyte size, dendricity, and number, as well as increased pigment in
12 CRH-R2, variably induced cell melanogenesis, dendricity, and proliferation.
13 via its ability to upregulate melanogenesis, dendricity, and proliferation.
14 levels, gp100 protein expression, melanocyte dendricity, and the number gp100+ HF melanocytes, were a
15        Keratinocytes control cell growth and dendricity, as well as expression of melanoma-associated
16 induces significant melanocyte spreading and dendricity in human melanocytes.
17        POMC also increased melanogenesis and dendricity in human pigment cells.
18 teins Rac and Rho link hormone signaling and dendricity in melanocytes.
19        Downstream mediators of LPC-dependent dendricity include Rac and Rho.
20             Factors that increase melanocyte dendricity increase melanosome transfer, and are importa
21 or agonists resulted in increased melanocyte dendricity, indicating that both EP1 and EP3 receptor si
22 vated cAMP in human melanocytes showing that dendricity observed in response to PGE(2) and PGF(2alpha
23 T inhibition decreased the number, size, and dendricity of melanocytes.
24 2) and PGF(2alpha) that stimulate melanocyte dendricity through EP1, EP3, and FP receptors.
25 guidance, stimulates melanocyte adhesion and dendricity through opposing actions of beta1-integrin an

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