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1 es and deconjugates Nedd8/Rub1 from cullins (deneddylation).
2 ddylation) and deactivated by NEDD8 removal (deneddylation).
3 ed with substrate are normally refractory to deneddylation.
4 losome regulates CDT2 stability through CUL4 deneddylation.
5 process and ubiquitin chain elongation after deneddylation.
6  alpha stabilization independent of Cullin 2 deneddylation.
7 nt flowers without an obvious defect in CUL1 deneddylation.
8  indicating that CSN2 is essential in cullin deneddylation.
9      We conclude that the CSN and its cullin deneddylation activity are required to sustain postembry
10 IM2 exhibit reduced nuclear accumulation and deneddylation activity of the CSN toward the cullin 1 an
11  While there was no obvious influence on CSN deneddylation activity, the increase in CSN subunits and
12  suggesting that these functions require the deneddylation activity.
13 ed conformational changes are retained after deneddylation, allowing both initiation of the ubiquitin
14 ith an F-box-Skp1 complex markedly inhibited deneddylation, although the magnitude varied considerabl
15 RL complexes are controlled by cycles of CRL deneddylation and CAND1 binding.
16 ng with the ubiquitin-proteasome pathway via deneddylation and phosphorylation.
17 mpaired CSN holocomplex formation and cullin deneddylation and resulted in decreases in F-box protein
18                   Although CIF causes cullin deneddylation and the generation of free NEDD8 Q40E, inh
19               The RING domain is crucial for deneddylation, and works in part through conformational
20     This inhibition is independent of cullin deneddylation but mediated by the CSN-associated deubiqu
21 otes substrate adaptor release following SCF deneddylation by the COP9 signalosome (CSN).
22 Upon consumption of substrate and subsequent deneddylation, CSN can remain stably bound to the CRL an
23  and demonstrate that the cullin neddylation-deneddylation cycle is not only required to activate CRL
24  Together, these mechanisms enable a dynamic deneddylation-disassembly cycle that promotes rapid remo
25 ivo, raising the question of how neddylation/deneddylation exerts its effects.
26 quitin ligases, where neddylation as well as deneddylation, facilitated by the protease activity of t
27 It promotes cleavage of the Nedd8 conjugate (deneddylation) from the cullin component of SCF ubiquiti
28 sought to determine the role of CSN-mediated deneddylation in UPS function and postnatal cardiac deve
29  complexes known to regulate neddylation and deneddylation, including the COP9 signalosome, Nub1, and
30 provide evidence that cullin neddylation and deneddylation is highly dynamic, that its equilibrium ca
31  whereas selectively promoting CSN5-mediated deNEDDylation may be beneficial in all stages of atheros
32 ects of csn mutants and monitored the cullin deneddylation/neddylation ratio during embryonic and ear
33                                              Deneddylation of both SCF(Fbw7) and SCF(Skp2-Cks1) was f
34 -1) deneddylation, we found a dose-dependent deneddylation of Cul-1 by Ado receptor stimulation predo
35 unit of the COP9 signalosome responsible for deneddylation of Cul-1, partially reversed HPC-mediated
36 do and suppresses NF-kappaB activity through deneddylation of Cul-1.
37 ates cell cycle at the G1 phase by promoting deneddylation of Cul1.
38 r (TLR) and reactive oxygen species-mediated deneddylation of Cul3, which is essential for Cul3/Keap1
39 on inhibitor MLN4924 prominently induces the deneddylation of Cullin-1.
40 s ubiquitin-dependent protein degradation by deneddylation of cullin-based ubiquitin ligases and, the
41 tent with NEDD8 deamidation causing enhanced deneddylation of cullins by the CSN.
42 ied by subunit 5 (CSN5/Jab1), resides in the deneddylation of the CRLs that is the hydrolysis of the
43 ochemical activity intrinsic to the complex, deneddylation of the Cullin subunits from Cullin-RING ub
44 nalysis indicates that NEDP1 mutants perturb deNEDDylation of the tumour suppressor p53.
45  besides CSN and the role of neddylation and deneddylation of their substrates.
46   Csn8/CSN plays an essential role in cullin deneddylation, UPS-mediated degradation of a subset of p
47                                        Cul-1 deneddylation was evident in a murine model of HPC and l
48 bition of NF-kappaB through cullin-1 (Cul-1) deneddylation, we found a dose-dependent deneddylation o
49                                     However, deneddylation, which removes NEDD8 from cullin, does not

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