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1 2 diabetes), and a prospective cohort study (dental caries).
2 ponses to the levy on obesity, diabetes, and dental caries.
3 treptococcus mutans is prominently linked to dental caries.
4 nd its essential role in the pathogenesis of dental caries.
5 ssociation between PAD and periodontitis and dental caries.
6 ng to active and passive vaccination against dental caries.
7 ry teeth that are particularly vulnerable to dental caries.
8 e biofilm and the primary causative agent of dental caries.
9 is a widely recognized etiological agent of dental caries.
10 ey organisms associated with the etiology of dental caries.
11 uller understanding of the genetic risks for dental caries.
12 There are many determinants of children's dental caries.
13 er of the oral plaque and is associated with dental caries.
14 expression and activation were increased in dental caries.
15 een a focus for immunization studies against dental caries.
16 gent for dentin-pulp complex regeneration in dental caries.
17 rimary etiological agent in the formation of dental caries.
18 he pathogenesis of S. mutans associated with dental caries.
19 iding further insight into the prevention of dental caries.
20 juice was less likely to be associated with dental caries.
21 rming oral pathogen commonly associated with dental caries.
22 le that diet and sealants have in preventing dental caries.
23 is an effective agent for the prevention of dental caries.
24 S. mutans plays a key role in dental caries.
25 h mutans streptococci and the development of dental caries.
26 view of potential new diagnostic methods for dental caries.
27 rapy was not identified as a risk factor for dental caries.
28 ent of new technologies for the detection of dental caries.
29 tans, the primary etiological agent of human dental caries.
30 s as well as new insights in the etiology of dental caries.
31 red to be the major etiologic agent of human dental caries.
32 mutans, one of the major causative agents of dental caries.
33 response in this experimental rat model for dental caries.
34 smoking may be a modifiable risk factor for dental caries.
35 s and species not previously associated with dental caries.
36 ecisely study the microbiota associated with dental caries.
37 nactivate GTF, a factor in the production of dental caries.
38 construct resulted in protective effects on dental caries.
39 wn to induce protection against experimental dental caries.
40 i demineralize the tooth surface and lead to dental caries.
41 cariogenicity of S. mutans in a rat model of dental caries.
42 que pH, which can inhibit the development of dental caries.
43 ral ecological approaches toward controlling dental caries.
44 teractions occur among major determinants of dental caries.
45 ccus mutans, an oral bacterium implicated in dental caries.
46 ructed for use in the replacement therapy of dental caries.
47 hogens and commensals in the pathogenesis of dental caries.
48 tooth surfaces and subsequent development of dental caries.
49 itro, and can protect rats from experimental dental caries.
50 prevalent infectious disease in pediatrics: dental caries.
51 ead in the environment and the prevalence of dental caries.
52 uced cariogenicity in vivo in a rat model of dental caries.
53 ability to characterize fully the impact of dental caries.
54 disease and metabolism of sugar alcohols in dental caries.
55 in genes lacking known or plausible roles in dental caries.
56 ociation between amount of sugars intake and dental caries.
57 dered the principal cariogenic bacterium for dental caries.
58 , hypertension, obesity-related cancers, and dental caries.
59 ptococcus mutans, a causative agent of human dental caries.
60 between hair disorders and susceptibility to dental caries.
61 1T) and KRT75(E337K), are prone to increased dental caries.
62 proved nor precluded their associations with dental caries.
63 -kingdom interaction for the pathogenesis of dental caries.
64 ting against demineralization of teeth (i.e. dental caries), a highly prevalent infectious disease as
65 is considered the primary etiologic agent of dental caries, a global health problem that affects 60 t
66 ococcus mutans is a major etiologic agent of dental caries, a prevalent worldwide infectious disease
71 constructs resulted in significantly reduced dental caries after infection with S. sobrinus compared
72 h Sjogren's syndrome have elevated levels of dental caries, along with the loss of many teeth, early
73 we examined variability in susceptibility to dental caries among children and adolescents in rural Ap
74 is warranted to determine if disparities in dental caries among women are due to differences in pari
75 was an association between HIV infection and dental caries among women enrolled in the Women's Intera
78 parts, Indigenous persons had more untreated dental caries and missing teeth, fewer teeth that had be
79 tion between the amount of sugars intake and dental caries and on the effect of restricting sugars in
80 ptococcus mutans is the etiological agent of dental caries and one of the many bacterial species impl
81 acy of hygiene-therapists when screening for dental caries and periodontal disease in regularly atten
87 oral microbes contribute to the formation of dental caries and periodontitis; however, there is littl
89 s and confounders assessed were governorate, dental caries and restorations, and obesity by waist cir
90 nts with hyposalivation are at high risk for dental caries and thus require aggressive oral hygiene r
92 entification of the bacteria associated with dental caries and with dental health in a subset of 204
93 ns is the primary etiological agent of human dental caries and, at times, of infective endocarditis.
94 of bacteria associated with the formation of dental caries, and attempts to identify some areas of or
95 crobiology and on foodstuff as it relates to dental caries, and for fostering the careers of many dis
96 streptococci are major etiological agents of dental caries, and several of their secreted products co
97 es into resin for Ca-PO(4) release to combat dental caries, and to investigate the filler level effec
99 countries, universal health problems such as dental caries, anemia, ophthalmologic conditions, and hy
100 tified include the misconception of defining dental caries as a tooth or surface-level condition vers
101 old increase in presentation and severity of dental caries as compared with wild-type control mice.
104 igated the differential effects of MA use on dental caries by comparing the patterns of decayed, miss
106 Fluoridation causes an obvious reduction of dental caries by interference with cariogenic streptococ
108 ing the enzyme that actively participates in dental caries by taking advantage of a Fenton reaction w
109 eses regarding the role of oral ureolysis in dental caries, calculus formation, and periodontal disea
111 sponses which are potentially protective for dental caries can be induced to peptide-based GTF vaccin
113 y the S. mutans 59-kDa GBP (GBP59) to affect dental caries caused by experimental infection with S. m
115 ion of S. mutans and can reduce the level of dental caries caused by this cariogenic streptococcus.
116 t demonstrates activity against the dominant dental caries-causing pathogen as well as the first lysi
117 mutans, the primary causative agent of human dental caries, contains a single copy of the gene encodi
120 intraclass correlation coefficient (ICC) for dental caries data at tooth and surface level, 2) to pro
123 ammatory mediators, including pericoronitis, dental caries, dentate status, root remnants, and gingiv
125 lts worldwide; however, the risk factors for dental caries do not necessarily exert their effects uni
127 nicity groups were disparate with respect to dental caries; effort is needed to treat active caries c
128 rimary etiological agent in the formation of dental caries, encodes at least 18 TetR regulators.
132 and LPO) with plausible biological roles in dental caries exhibited suggestive evidence for associat
133 endance behaviors; those would lead to lower dental caries experience and better self-reported oral h
134 idate genes for evidence of association with dental caries experience in 13 race- and age-stratified
135 ionship between oral arginine metabolism and dental caries experience in children, we measured ADS ac
136 attern; they also had a significantly higher dental caries experience in the primary dentition than d
137 were used to generate two common indices of dental caries experience which were tested for associati
138 ne source of information on past and present dental caries experience; however, important limitations
139 randomized or controlled clinical trials on dental caries, fluorides, sealants, and "restorative" ca
140 The first genome-wide association study of dental caries focused on primary teeth in children aged
141 dietary carbohydrates caused an increase in dental caries following the development of agriculture,
143 calcium phosphate surfaces, the mechanism of dental caries formation and the effects of fluoride on t
144 utans, a major oral pathogen responsible for dental caries formation, possesses a variety of mechanis
147 ugar-related feeding practices on changes in dental caries from early childhood to young adulthood.
148 s considered "optimal" for the prevention of dental caries (from 0.7 to 1.2 mg/L) appears to have no
154 benefit of fluoridation in the prevention of dental caries has been overwhelmingly substantiated, the
155 th loss, the relationship between parity and dental caries has not been extensively investigated.
158 tans, the primary etiological agent of human dental caries, has developed multiple mechanisms to colo
159 Streptococcus mutans, a primary agent of dental caries, has three (p)ppGpp synthases: RelA, which
160 ed genome-wide association scans (GWASs) for dental caries in a population-based cohort of 12 000 His
168 cal effectiveness of FS and FV in preventing dental caries in first permanent molars (FPMs) in 6- to
169 efficient carbohydrate for the promotion of dental caries in humans, and the primary caries pathogen
170 cus mutans, the principal etiologic agent of dental caries in humans, possesses a variety of virulenc
175 lear is how the distribution and severity of dental caries in MA users differ from the general popula
178 ks by young children is a risk indicator for dental caries in the primary dentition and should be dis
180 the United States, included an assessment of dental caries in US children and adolescents and provide
181 ofilms by 3F1 was able to effectively reduce dental caries in vivo without affecting the overall oral
183 ention of Streptococcus mutans infection and dental caries include mucosal immunization, which result
188 tive GTF and demonstrated protection against dental caries infection after immunization, implying tha
196 l, behavioral, and community determinants of dental caries is based on cross-sectional analyses.
208 lth Organization (WHO) stated that globally, dental caries is the most important oral condition.
210 occus mutans, a principal causative agent of dental caries, is considered to be the most cariogenic a
211 se, an inflammatory disease mainly caused by dental caries, is one of the most prevalent infectious d
214 model that specified that parity influences dental caries levels through dental care, psycho- social
215 s mutans, a major etiological agent of human dental caries, lives primarily on the tooth surface in b
218 from nine adults who have remained free from dental caries (mean age = 59.2; Decayed Missing Filled S
219 Relative to controls, the extent of molar dental caries measured on day 78 was also significantly
221 s reported the absolute amount of sugars and dental caries, measured as prevalence, incidence, or sev
223 The high rates and distinctive patterns of dental caries observed could be used 1) to alert dentist
224 to explain the comparatively high levels of dental caries observed in the inner cities of the United
225 isk of lead exposure to be 13.5% and 9.6% of dental caries occurring in 5- to 17-year-olds exposed to
226 evel was associated with an elevated risk of dental caries (odds ratio, 1.8; 95% confidence interval,
227 ed a significant genetic association between dental caries of the anterior mandibular teeth and LYZL2
228 munized groups had significant reductions in dental caries on both smooth and occlusal surfaces compa
229 determine the impact of enamel fluorosis and dental caries on oral health-related quality of life (OH
234 ften cited as the main bacterial pathogen in dental caries, particularly in early-childhood caries (E
236 nduced protein-encoding mRNA (irvA) from the dental caries pathogen Streptococcus mutans directly mod
238 ctional significance of these GTF domains in dental caries pathogenesis and present coimmunization as
240 osyltransferase (GTF), an enzyme involved in dental caries pathogenesis, was used as a test antigen.
241 raphic characteristics, smoking, tooth loss, dental caries, periodontal status, and OHRQoL in adults
242 ypertensive medication; smoking; tooth loss; dental caries; periodontal status (bleeding on probing,
244 e-wide association studies (GWAS) of 5 novel dental caries phenotypes (developed by clustering the pe
245 tans, the primary etiological agent of human dental caries, possesses at least two fructose phosphotr
246 High and upward sugar consumers had higher dental caries prevalence and mean DMFT in all cohort wav
248 ws produce conflicting conclusions regarding dental caries-preventive effects of water fluoridation i
249 cus mutans, the principal causative agent of dental caries, produces four glucan-binding proteins (Gb
250 cus mutans, the primary etiological agent of dental caries, produces several activities that promote
251 t with a significant genetic contribution to dental caries progression and severity in both emerging
252 role of genetic and environmental factors on dental caries progression in young children was determin
254 aque ureolytic activity and the incidence of dental caries, recombinant, ureolytic strains of Strepto
255 GGY and AND-injected groups had significant dental caries reductions compared with sham-injected gro
257 n before their third birthday and subsequent dental caries-related treatment (CRT) and CRT payment.
262 occus mutans, a principal causative agent of dental caries, secretes antimicrobial peptides known as
263 provided the opportunity for differences in dental caries status to be viewed by a ge, sex, race, an
264 a, Actinobacillus actinomycetemcomitans) and dental caries (Streptococcus mutans and S. sobrinus).
266 This study reinforces the complexity of dental caries, suggesting that numerous loci, mostly hav
268 identify genetic factors that contribute to dental caries susceptibility, either alone or in combina
269 ldhood caries (ECC) is a preventable form of dental caries that affects very young children, particul
271 vary IgA responses and in protection against dental caries, the finding that the GLU polypeptide alon
272 fracture toughness that protects teeth from dental caries, the most common chronic disease worldwide
273 Streptococcus mutans, the causative agent of dental caries, the most prevalent childhood disease.
275 d the first genome-wide association scan for dental caries to identify associated genetic loci and no
276 In conclusion, this was the largest GWAS of dental caries, to date and was the first to target Hispa
279 Streptococcus mutans is the leading cause of dental caries (tooth decay) worldwide and is considered
282 rpose was to evaluate the natural history of dental caries using a standardized, visually based syste
286 plus 16-immunized groups, and the extent of dental caries was also significantly reduced in these gr
287 samples of patients or populations; and (2) dental caries was assessed clinically, and criteria were
288 l sugar consumption groups, the increment of dental caries was slightly higher between ages 6 and 12
289 oth loss in the LJP and GJP groups, and that dental caries was the principal cause for tooth extracti
291 ptococcus mutans, a causative agent of human dental caries, was detected for the aqueous leaf extract
292 ns, one of the principal causative agents of dental caries, was dramatically impaired by growth of th
293 abolism in the initiation and progression of dental caries, we constructed otherwise-isogenic mutants
294 development of broader-spectrum vaccines for dental caries, we explored the immune potential of const
296 th the development of periodontal disease or dental caries were overabundant in the group that did fl
297 ide varnish (FV) are effective in preventing dental caries when compared with a no-treatment control.
298 nd in biofilms of children exhibiting severe dental caries, with different degrees of lesion severity
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