ライフサイエンスコーパス: dental plaque

1 onema socranskii, and Treponema vincentii in dental plaque.

2 as each new bacterial cell binds to existing dental plaque.

3 onsortium in the microbiome of supragingival dental plaque.

4 an oral commensal and an early coloniser of dental plaque.

5 ing are required for the periodic removal of dental plaque.

6 its a general inflammatory response to local dental plaque.

7 t of the complex oral biofilm referred to as dental plaque.

8 . gingivalis and S. cristatus in subgingival dental plaque.

9 cterial surfaces and to colonize subgingival dental plaque.

10 abundant species in the oral biofilm called dental plaque.

11 d-species colonies during formation of early dental plaque.

12 an inflammatory reaction to the bacteria in dental plaque.

13 ntitis in response to challenge by microbial dental plaque.

14 es disease by surviving acidic conditions in dental plaque.

15 the mixed-species environment of subgingival dental plaque.

16 to the initiation of the oral biofilm called dental plaque.

17 mportant role in the ecology of S. mutans in dental plaque.

18 reptococcus cristatus, an early colonizer of dental plaque.

19 alence of mixed-species communities in early dental plaque.

20 ispecies bacterial biofilm commonly known as dental plaque.

21 and plays a pivotal role in the formation of dental plaque.

22 the inflammatory response of the gingiva to dental plaque.

23 e fluids reflect the cariogenic potential of dental plaque.

24 he complex multiple species biofilm known as dental plaque.

25 ly significant in the establishment of early dental plaque.

26 gordonii-A. naeslundii communities in early dental plaque.

27 a substantial proportion of the bacteria in dental plaque (30 to 40%) bear PC antigen; this antigen

28 The formation of dental plaque, a highly complex biofilm that causes ging

29 a natural microbial community, namely, human dental plaque, a microbial biofilm.

30 in this association, our hypothesis was that dental plaque accumulation in healthy subjects would eli

31 We hypothesized that dental plaque accumulation over 21 days in the experimen

32 differences in the inflammatory response to dental plaque accumulation.

33 nly from FBS, but also from human saliva and dental plaque after the incubation of 0.45-microm membra

34 ltispecies biofilm on tooth surfaces forming dental plaque and a potential agent of endocarditis.

35 llied food industries on enzymes to break up dental plaque and a vaccine against tooth decay with que

36 cer mortality was positively associated with dental plaque and gingival inflammation.

37 tococcus sanguinis are pioneer colonizers of dental plaque and important agents of bacterial infectiv

38 t the glutamine endoprotease is derived from dental plaque and likely microbial in origin.

39 o examine the prevalence of P. gingivalis in dental plaque and of serum immunoglobulin G (IgG) antibo

40 Dental plaque and prostatic secretion samples were used

41 Although bacteremias from dental plaque and/or elevated circulating inflammatory c

42 However, PCR of whole dental plaques and subsequent analysis of up to 130 indi

43 Bacterial levels, gingivitis, dental plaque, and caries experience were assessed.

44 ucation, income, smoking, diabetes mellitus, dental plaque, and presence of any of 8 subgingival micr

45 lis creates a dysbiosis between the host and dental plaque, and this may represent one mechanism by w

46 levels of Treponema denticola in subgingival dental plaque are associated with severe periodontal dis

47 y half of the bacteria in the saliva and the dental plaque are not cultivable.

48 ity and complexity of the microbial biota in dental plaque are significantly less in S-ECC children t

49 Porphyromonas gingivalis is present in dental plaque as early as 4 h after tooth cleaning, but

50 It is generally recognized that bacteria in dental plaque at sites of periodontal diseases are not c

51 have shown that a significant proportion of dental plaque bacteria contain PC as determined by react

52 Once thought to occur exclusively between dental plaque bacteria, there are increasing reports of

53 ard pertains to only a few of the acidogenic dental plaque bacteria.

54 lectin-like adhesins on other members of the dental plaque biofilm community.

55 on of these bacteria by other members of the dental plaque biofilm community.

56 Dental plaque biofilm formation proceeds through a devel

57 atypica, two early colonizing members of the dental plaque biofilm, have been postulated to participa

58 atypica, two early-colonizing members of the dental plaque biofilm, participate in a relationship tha

59 Acidogenic bacteria within dental plaque biofilms are the causative agents of carie

60 and Streptococcus intermedius in subgingival dental plaque biofilms may contribute to forms of period

61 device for the in vivo generation of intact dental plaque biofilms on natural tooth surfaces in huma

62 Streptococcus mutans in dental plaque biofilms play a role in caries development

63 is the anaerobic environment of subgingival dental plaque, but initial colonization of the oral cavi

64 ary for initial or sustained colonization in dental plaque by this major dental pathogen.

65 from five specimens of Neanderthal calcified dental plaque (calculus) and the characterization of reg

66 lysis of urea by ureases of oral bacteria in dental plaque can cause a considerable increase in plaqu

67 port the hypothesis that the accumulation of dental plaque can result in a measurable systemic inflam

68 to determine if proteases produced by early dental plaque colonizers such as Streptococcus gordonii

69 We propose that dental plaque communities originate as a result of intim

70 ve advantages over nonureolytic organisms in dental plaque, constituting an important determinant of

71 ommonly occurring bacterial species found in dental plaque contain PC antigen and that immunization w

72 Calcified dental plaque (dental calculus) preserves for millennia

73 examination was performed in order to assess dental plaque, dental calculus and gingival inflammation

74 ay determine polymicrobial succession during dental plaque development, but the ecological constraint

75 d presence of P. gingivalis at all stages of dental plaque development.

76 streptococci and actinomyces, are central to dental plaque development.

77 al bacteria and individual species counts in dental plaque did not differ significantly between basel

78 The microbiomes of supragingival dental plaque differ substantially among tooth surfaces

79 Newly formed biofilms and more mature dental plaque each have a level of spatial organization

80 Overall, these data demonstrate that dental plaque eDNA is potentially an important target fo

81 The predominant species group in developing dental plaque films during density-dependent growth was

82 saturation with respect to enamel mineral in dental plaque fluid following sucrose exposure.

83 m disulfide bonds plays an important role in dental plaque formation and fitness for the bacteria.

84 pportunistic pathogen, is thought to promote dental plaque formation by serving as a bridge bacterium

85 al oral hygiene measures was used to compare dental plaque formation following use of chlorhexidine (

86 The initial stages of dental plaque formation involve the adherence of early c

87 h surfaces colonization and contributions to dental plaque formation, as well as their potential role

88 During initial dental plaque formation, the ability of a species to gro

89 nii, are pioneer oral bacteria that initiate dental plaque formation.

90 is interaction may play an important role in dental plaque formation.

91 aining chewing gum appears useful to control dental plaque formation.

92 The microbial composition of dental plaque from 42 severe ECC children was compared w

93 in both prostatic secretion and subgingival dental plaque from the same individual.

94 e microbiomes of site-specific supragingival dental plaques from children with different caries statu

95 To define the role of dental plaque fructans and the enzymes involved in their

96 All-cause mortality risk were raised with dental plaque, gingival inflammation, >10 missing teeth

97 ty were also positively associated with high dental plaque (HR = 3.30, [95% CI: 1.76-6.17]), high gin

98 constituent of supragingival and subgingival dental plaque in children and adults.

99 gy or the physicochemical characteristics of dental plaque in such a way as to reduce its cariogenic

100 l flora and pH-lowering capacity of the same dental plaques in relation to caries.

101 nd the same DNA mixed with DNA isolated from dental plaque, indicating that P. gingivalis levels can

102 om the biofouling of ocean-going vessels, to dental plaque, infections of the urinary tract, and cont

103 These biofilms were grown from dental plaque inoculum (oral microcosms) and were obtain

104 Dental plaque is a complex biofilm that accretes in a se

105 Dental plaque is a complex multispecies biofilm, and is

106 It also suggests that the overall effect of dental plaque is a function of the balance between patho

107 nths, independent of age, smoking status, or dental plaque levels.

108 gate the effects on dentin bond strength and dental plaque microcosm biofilms for the first time.

109 Streptococcus gordonii DL1, a nonureolytic, dental plaque microorganism.

110 We propose that PC-bearing dental plaque microorganisms may induce an antibody resp

111 Future studies assessing a larger panel of dental plaque microorganisms, with shorter intervals bet

112 lammatory disease initiated and sustained by dental plaque microorganisms.

113 ajor direct cause of chronic inflammation is dental plaque, much of the new research is directed at m

114 Eikenella corrodens isolates recovered from dental plaque, mucosal surfaces, and saliva of 24 subjec

115 ry pathogens have been shown to colonize the dental plaque of hospitalized intensive care and nursing

116 ative phase of treatment, and in subgingival dental plaque of periodontitis patients, indicating that

117 and Fusobacterium nucleatum, in subgingival dental plaque of pregnant women in the OPT Study and the

118 ssociation was observed for number of teeth, dental plaque, or detectable oral mucosal lesions and PD

119 gnificantly higher percentages of sites with dental plaque (P <0.0001), gingival bleeding (P <0.05),

120 sis did not reveal susceptibility of certain dental plaque pathogens to light, and it was not possibl

121 and this ability has an effect on a range of dental plaque-related phenotypes.

122 Pi was detected in all dental plaque samples but not in the prostatic secretion

123 of cells of individual bacterial species in dental plaque samples is needed for understanding the ba

124 An extensive analysis of dental plaque samples over the years has led to the iden

125 Pooled dental plaque samples were collected from 20 children ag

126 tatus (smoker or non-smoker), and full-mouth dental plaque score.

127 e data suggest that some early colonizers of dental plaque, such as S. cristatus, may be beneficial t

128 ans is the principal acidogenic component of dental plaque that demineralizes tooth enamel, leading t

129 disease, is caused by the bacterial biofilm (dental plaque) that accumulates on teeth adjacent to the

130 ispecies oral biofilm known as supragingival dental plaque; they grow by fermentation of sugars to or

131 ranspose these interactions from undisturbed dental plaque to an experimentally tractable in vitro bi

132 cies colonies of bacteria, e.g., biofilms or dental plaque, to behave as pseudomulticellular organism

133 Supragingival dental plaque was collected from tooth surfaces that wer

134 Percentages of tooth surfaces carrying dental plaque were 41% and 36% for right and left sides,

135 e result of infection by anaerobic bacteria; dental plaque would seem to be a logical source of these