ライフサイエンスコーパス: dentinal

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1 namel, the dentin-enamel junction, and outer dentinal acellular tubules, and then concentrates in odo

2 , OMZ175, Cnm mediates stringent adhesion to dentinal and root tissues as well as collagen-coated sur

3 Due to the collagenous composition of dentinal and root tissues, we hypothesized that Cnm may

4 ouse trigeminal ganglia, we demonstrate that dentinal application of LPS increases the expression of

5 cular interest was the disease trajectory of dentinal caries in the permanent teeth in groups defined

6 groups defined by the presence or absence of dentinal caries in the primary teeth.

7 The mechanical removal of dentinal caries traditionally involves the use of tactil

8 A significantly lower number of enamel and dentinal carious lesions was observed for the mutant-inf

9 With these stimuli dentinal fluid flow could not be recorded in vivo for te

10 After the dentinal fluid flow measurements, specimens were also ev

11 The thresholds of these units in terms of dentinal fluid flow were in the range 0.3-2.1 nl s(-1) m

12 resent in saliva, mineralized dentin, and/or dentinal fluid may affect the dentin caries process at t

13 rupt influx, because of neural regulation of dentinal fluid movement, but it did not.

14 ion of dental sensory neurons in response to dentinal fluid movements.

15 h the initial enamel or root lesion, through dentinal involvement, to eventual cavitation.

16 net increments of occlusal surfaces and deep dentinal lesions SBCPRs (H = 46.4-56.2).

17 er, exposing the soft underlying collagenous dentinal matrix and allowing further infiltration by the

18 s that were caries-lesion-free (PF) and from dentinal (PD) and enamel (PE) caries lesions.

19 Dentinal repair in the postnatal organism occurs through

20 fects of aging, injury, neural function, and dentinal repair on its influx into vital rat teeth.

21 s and distinctive in the demineralization of dentinal root surfaces for periodontal regeneration and

22 e to dentin following the preparation of the dentinal surface with either an Er:YAG laser (lambda = 2

23 ewer carious lesions in the buccal enamel or dentinal surfaces than the sham-immunized animals (P < 0

24 as also associated with abnormalities in the dentinal tubule system and delayed formation of the thir

25 gth tested perpendicular to the direction of dentinal tubules (undemineralized crown = 140.4 +/- 48.6

26 y and fail to form proper dentin with normal dentinal tubules and activate terminal differentiation,

27 mineralization as well as the defects in the dentinal tubules and third molar development.

28 ed by estimating the number and diameters of dentinal tubules exposed.

29 anomalies in the number and organization of dentinal tubules in MT-DLX3 TG mice.

30 In particular, the dentinal tubules in the DGI-II patient were very irregul

31 test the hypothesis that fluid flow through dentinal tubules is part of the mechanism involved in th

32 ectively removed the surface smear layer and dentinal tubules remained partially to totally occluded

33 re based on the physical obliteration of the dentinal tubules to reduce hydraulic conductance.

34 consequence of fluid movement within exposed dentinal tubules.

35 resent in the predentin matrix and along the dentinal tubules.

36 ansparent carious dentin containing occluded dentinal tubules.

37 ormation, and a relatively high branching of dentinal tubules.

38 antigens that diffuse into the pulp through dentinal tubules.

39 w tracts that exist in human dentine forming dentinal tubules.

40 few mature odontoblasts, and sharply reduced dentinal tubules; and (5) a dramatic change in Osx and n

41 tiation into odontoblast-like cells near the dentinal walls.

42 losure of immature permanent teeth with thin dentinal walls.

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