ライフサイエンスコーパス: deoxy-ATP

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1 o thin filament activation by substituting 2 deoxy-ATP (dATP; a strong cross-bridge augmenter) for AT

2 Replacing ATP with 2 deoxy-ATP (dATP) increased F-actin speed for both groups

3 utanedione monoxime) or augmentation (with 2 deoxy-ATP) had no greater effect in cardiac muscle with

4 ucture of hOAS1 in complex with dsRNA and 2'-deoxy ATP at 2.7 A resolution, which reveals the mechani

5 The time-course of VCF responses to ATP, 2'-deoxy ATP, 3'-deoxy ATP, Ap5A and alphabetameATP were ag

6 Substitution of 2'-deoxy ATP (dATP) for ATP as substrate for actomyosin res

7 ion (NO3 (-) ) and N(6) -(2-phenylethyl)-2'-deoxy-ATP (d-PATP), which almost completely rectifies th

8 e custom synthesized N(6)-(2-phenylethyl)-2'-deoxy-ATP (P-dATP), an analog combining the chemical mod

9 ngly, an ATP analogue, N6-(2-phenylethyl)-2'-deoxy-ATP (P-dATP), can increase the open probability (P

10 Here we show that 2'-deoxy-ATP (dATP), but not 3'-deoxy-ATP, increases the ac

11 ATP, 2-deoxy ATP (dATP), CTP, and UTP support isometric force a

12 s of cardiac trabeculae with either ATP or 2-deoxy-ATP (dATP) as the substrate for contraction.

13 y demonstrated that cardiac myosin can use 2-deoxy-ATP (dATP) as an energy substrate, that it enhance

14 ntrol by substitution of ATP (5.0 mM) with 2-deoxy-ATP (dATP) (5.0 mM) or by lowering [ATP] to 0.5 mM

15 were altered by either replacing ATP with 2-deoxy-ATP (dATP) or by reducing [ATP].

16 oss-bridge cycling rate was increased with 2-deoxy-ATP.

17 se of VCF responses to ATP, 2'-deoxy ATP, 3'-deoxy ATP, Ap5A and alphabetameATP were agonist dependen

18 differences with respect to inhibition by 3'-deoxy-ATP.

19 -(7-diethylaminocoumarin-3-carbonylamino)-3'-deoxy-ATP (deac-aminoATP), which undergoes a 20-fold inc

20 luenza RNA polymerase, the K(i) value for 3'-deoxy-ATP (0.4-0.6 microM) is approximately 100-fold low

21 Whereas the K(i) value for 3'-deoxy-ATP (105-117 microM) is similar to the K(m) value

22 MANT-3'-dATP [2'-O-(N-methylanthraniloyl)-3'-deoxy-ATP] (Ki, 16.7 nM).

23 we show that 2'-deoxy-ATP (dATP), but not 3'-deoxy-ATP, increases the activity of G551D-CFTR by appro

24 In the active site, 3'-deoxy-ATP and a single metal ion are well positioned for

25 of the 2'-deoxyribose leads to ligands (mant-deoxy-ATP [dATP], mant-deoxy-ADP) with inverse agonist a

26 as active with ATP and partially active with deoxy-ATP, but lacked measurable activity with other nuc

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