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1 type detergents (i.e., Chapso, glycocholate, deoxycholate).
2 ental strain when treated with the detergent deoxycholate.
3  protein complex because it was abolished by deoxycholate.
4 mily member YegM and pump out novobiocin and deoxycholate.
5 pecific antibodies after solubilization with deoxycholate.
6 ge when cells without ygiS are stressed with deoxycholate.
7 d tolerance during stress from the bile salt deoxycholate.
8 lymyxin B but did not display sensitivity to deoxycholate.
9 th in the presence of large sugars or sodium deoxycholate.
10 hysiologic levels of the secondary bile acid deoxycholate.
11 wth of C. jejuni cultured in the presence of deoxycholate.
12 g with 0.4% dioleoylphosphatidylcholine/0.4% deoxycholate.
13 gent but lacking dioleoylphosphatidylcholine/deoxycholate.
14  and plate cultures supplemented with sodium deoxycholate.
15 8011 were screened for sensitivity to sodium deoxycholate.
16 rsion of graphene oxide/few-layered graphene/deoxycholate.
17                           Treatment with AmB deoxycholate (1 mg/kg/day) or liposomal AmB (5 mg/kg/day
18 ton X-100 (0.3%) or the mild ionic detergent deoxycholate (20 mM); however, higher concentrations of
19 ncreased plasma levels of glycodeoxycholate, deoxycholate 3-sulfate, and bilirubin.
20 4 patients (71%) treated with amphotericin B deoxycholate, 4/12 (33%) treated with a triazole alone,
21 nthetically link 2 of the following ligands: deoxycholate, 5-leuenkephalin, triiodothyronine, thyroni
22 yes treated with L-AmB (75%), amphotericin B deoxycholate (78%), and ABLC (91%) than with the control
23 B host strain to novobiocin (16-fold) and to deoxycholate (8-fold).
24  greater in eyes treated with amphotericin B deoxycholate (81%), L-AmB (91%), and ABLC (97%) than wit
25 man diarrheal illness worldwide, responds to deoxycholate, a component of bile, by altering global ge
26 ound that growth of C. jejuni in medium with deoxycholate, a component of bile, caused DNA damage con
27  effects, zebrafish were treated with sodium deoxycholate, a known inducer of NF-kappaB or NF-kappaB
28 nsitivity to and biofilm induction by sodium deoxycholate, a major bile component that acts as an ext
29                                              Deoxycholate, a metabolite of cholate produced by the no
30 cholelithogenesis by augmenting formation of deoxycholate, a pro-lithogenic secondary bile salt, and
31 us 10, 20, 30, and 50 micro g amphotericin B deoxycholate, ABLC, and L-AmB.
32  isolates grown on blood agar, xylose lysine deoxycholate agar (XLD), Hektoen enteric agar (HE), salm
33 d persistent infection, despite therapy with deoxycholate AmB or liposomal AmB.
34  effect of the combination of amphotericin B deoxycholate (AmB) and 5-fluorocytosine (5FC) by use of
35 erosolized administrations of amphotericin B deoxycholate (AmBd) and amphotericin B lipid complex (AB
36  late 1950s, intrathecal (IT) amphotericin B deoxycholate (AmBd) has been successfully used to treat
37 to receive either intravenous amphotericin B deoxycholate (amphotericin) (219 patients), at a dose of
38 arly, in OE19 human esophageal cancer cells, deoxycholate and acid induced expression of GC-C.
39 encies in minimal medium, in the presence of deoxycholate and bile, and in competition assays with wi
40 ng less active than unconjugated bile salts (deoxycholate and cholate).
41 sistance specifically to the bile components deoxycholate and conjugated forms of chenodeoxycholate,
42  mutant was more sensitive to the detergents deoxycholate and dodecyl sulfate and the antimicrobial p
43 mbination antifungal therapy (amphotericin B deoxycholate and flucytosine) is the recommended treatme
44  increased 7-8-fold with bile duct ligation; deoxycholate and hyodeoxycholate disappeared.
45                                          AmB deoxycholate and liposomal AmB had the greatest antifung
46  chenodeoxycholate, the secondary bile acids deoxycholate and lithocholate, and 3alpha,7alpha,12alpha
47 d mutants exhibited increased sensitivity to deoxycholate and showed increased resistance to polymyxi
48 he bile salts sodium taurocholate and sodium deoxycholate and significantly increased sensitivity to
49  of toxic compounds, including the bile salt deoxycholate and the hydrophobic dye crystal violet.
50 nt was assayed for its sensitivity to sodium deoxycholate and to the antimicrobial cationic peptide,
51  mutant demonstrated enhanced sensitivity to deoxycholate and was impaired in DNA double strand break
52  IpaD specifically binds fluorescein-labeled deoxycholate and, based on energy transfer measurements
53 d-state and gas-phase structures of cyclotri(deoxycholate) and cyclotetra(24-norcholate).
54  buffer which contains 1% Nonidet P-40, 0.5% deoxycholate, and 0.1% SDS.
55 d in the presence of the bile salts cholate, deoxycholate, and chenodeoxycholate, and EMSA showed tha
56 etergents such as sodium dodecyl sulfate and deoxycholate, and it showed increased susceptibility to
57 e extracted with a buffer containing Triton, deoxycholate, and SDS but not with a buffer containing T
58 tergents such as Triton X-100, Nonidet P-40, deoxycholate, and SDS tended to destabilize the CFTR dim
59 the anal verge were incubated in 1 mM sodium deoxycholate, and the percentage of goblet cells undergo
60                         They are elicited by deoxycholate applied to either side of the membrane, wit
61   Micromolar concentrations of the bile salt deoxycholate are shown to rescue the activity of an inac
62 n of the addAB mutant restored resistance to deoxycholate, as well as function of the DNA double stra
63 ivated protein kinases did not attenuate the deoxycholate-associated increase in Mcl-1 protein, the R
64 ed upon addition of chlorpromazine or sodium deoxycholate at concentrations below the critical micell
65                                       Sodium deoxycholate at pH 9.0 solubilized about 35% of the rece
66 the amino acids proposed to be important for deoxycholate binding by IpaD appear to have significant
67  the wild-type enzyme reveals, as predicted, deoxycholate bound with its carboxyl group at the entran
68 ed from these marginal edges with a Tween-40/deoxycholate buffer that solubilizes the actin cytoskele
69 ommend initial treatment with amphotericin B deoxycholate, but this drug has substantial side effects
70 ere more sensitive than the parent strain to deoxycholate by varying degrees.
71  (serotonin, 2,4-dichlorophenoxyacetic acid, deoxycholate) can each partially substitute for the red
72 ents, e.g., Tween 20, sodium cholate, sodium deoxycholate, CHAPS, or CHAPSO, are completely ineffecti
73 cluding sodium dodecyl sulfate (SDS), sodium deoxycholate, Chaps, Triton X-100, Triton X-114, NP-40,
74 ected by the interaction with the bile salts deoxycholate, chenodeoxycholate, and taurodeoxcholate.
75 lta ompU2 strain was deficient for growth in deoxycholate compared to wild-type, Delta ompT, and Delt
76 centration (MBC) of bile, the bile component deoxycholate (DC), and the anionic detergent sodium dode
77 r four bile acids, chenodeoxycholate (CDCA), deoxycholate (DCA), cholate (CA), and ursodeoxycholate (
78          Toward anionic Trition X-100/sodium deoxycholate/diC8PC (4:2:1) mixed micelles, L20W and L20
79 of the detergents sodium dodecyl sulfate and deoxycholate disrupted this interaction.
80 ric stoichiometry for the oligomer formed in deoxycholate (DOC) micelles, the high-resolution unproce
81 appendages in response to bile salts such as deoxycholate (DOC), and that the formation of these appe
82 tolC mutant to grow in the presence of 0.05% deoxycholate (DOC).
83 escence and by insolubility in the detergent deoxycholate (DOC).
84 pylobacter may be triggered by the bile acid deoxycholate (DOC).
85                         The bile salt sodium deoxycholate (DOC, 1-4 mM) caused a dose-dependent incre
86    In nerves exposed to the bile salt sodium deoxycholate (DOC, 6 min, 4 mM), challenge with elevated
87 ively; Phadebact, 100 and 98%, respectively; deoxycholate drop test, 99 and 98%, respectively; deoxyc
88 ic simulations revealed that the presence of deoxycholate enhances NaCl rejection in these graphene-b
89 ly inhibited by the detergents, Sarkosyl and deoxycholate, even at 0.025%, but it was not inhibited b
90 deoxycholate uptake and reduces tolerance to deoxycholate exposure.
91  off-patent antifungal drugs: amphotericin B deoxycholate, flucytosine, and fluconazole.
92 occal induction regimens: (1) amphotericin B deoxycholate for 4 weeks; (2) amphotericin and flucytosi
93             All three enzymes require sodium deoxycholate for optimal activities; other detergents ex
94 ompared voriconazole to amphotericin B (AmB) deoxycholate for the primary therapy of IA.
95 h AIDS, the preferred treatment has been the deoxycholate formulation of amphotericin B.
96 th cultures containing 0.05% (wt/vol) sodium deoxycholate, growth of the mutant was significantly inh
97  conferring full resistance to the bile salt deoxycholate, improving the efficiency of cell division
98 ling predicted that IpaD binds the bile salt deoxycholate in a cleft formed by the N-terminal domain
99 the higher efficacy of voriconazole over AmB deoxycholate in mycologically documented IA.
100 t and independent attachment and invasion by deoxycholate in Shigella flexneri, deoxycholate negative
101 ce immunized with larval antigens soluble in deoxycholate in which protective immunity was shown to b
102 es the addition of a bile salt (e.g., sodium deoxycholate) in trace amounts to solutions of the phosp
103                                              Deoxycholate increased cellular Mcl-1 protein in a conce
104                                 Furthermore, deoxycholate increased NF-kappaB activity, associated wi
105 ow that submicellar concentrations of sodium deoxycholate induce time-resolved blocking events of Omp
106                                     Although deoxycholate induced apoptosis and activated all three c
107                                  Whereas the deoxycholate-induced increase in Mcl-1 reduced Fas-media
108 t negative construct for NF-kappaB prevented deoxycholate-induced p50 nuclear translocation and activ
109  decrease fibronectin incorporation into the deoxycholate insoluble matrix, and prevent fibronectin's
110 hich, over time, becomes incorporated in the deoxycholate-insoluble ECM in a similar fashion to FN.
111 corporation of FN(syn-) into fibrils and the deoxycholate-insoluble matrix could be stimulated by Mn2
112 and/or 70-kDa fragments to the cell surface, deoxycholate-insoluble matrix, and adsorbed 160-kDa cell
113  and chenodeoxycholate, and EMSA showed that deoxycholate is able to abolish the formation of BreR-P(
114                              We propose that deoxycholate is able to interact with BreR and induce a
115     The voltage dependence remains even when deoxycholate is applied symmetrically, indicating that i
116 ons preferentially binds multiple conjugated deoxycholate ligands in a novel 3:1 binding mode essenti
117                                          The deoxycholate-mediated increase of cellular Mcl-1 protein
118 vasion by deoxycholate in Shigella flexneri, deoxycholate negatively regulates IcsA and MAM in S. son
119 d into approximately 70-kDa components after deoxycholate/Nonidet P-40 treatment.
120  to be less toxic than either amphotericin B deoxycholate or ABLC.
121 o a control arm or to receive amphotericin B deoxycholate or caspofungin treatment while undergoing s
122  We solubilized HisACAT-1 with the detergent deoxycholate or CHAPS (3-[(3-cholamidopropyl)-dimethylam
123 eveloped in eyes treated with amphotericin B deoxycholate or L-AmB (P < 0.05).
124 aB at the TTSA needle tip in the presence of deoxycholate or other bile salts.
125  amphotericin B compared with amphotericin B deoxycholate (OR 0.09, 95% CI 0.02-0.50, P=0.006) and su
126  of bile salts, e.g., sodium cholate, sodium deoxycholate, or CHAPS.
127 e containing 5 mmol/L each of the BS, sodium deoxycholate, PC, or Indo, alone and in combination, was
128                                              Deoxycholate prolongation of Mcl-1 turnover was blocked
129                             We now show that deoxycholate promotes the stable recruitment of IpaB to
130  C. jejuni in physiological levels of sodium deoxycholate released all three CDT proteins, as well as
131 e dihydroxy bile acids chenodeoxycholate and deoxycholate resulted in an approximately 10-fold increa
132 h S. typhimurium and S. typhi in bile and in deoxycholate resulted in the induction or repression of
133 ni on plates supplemented with the bile salt deoxycholate retarded the inhibitory effect of chloramph
134   Treatment of the organelles with Na2CO3 or deoxycholate reveal that calpain I, 78-kDa calpain II, a
135                         The bile salt sodium deoxycholate (SDC) enabled efficient extraction and disa
136 uids were obtained by doping lecithin/sodium deoxycholate (SDC) reverse micelles with a photochromic
137 ti bacA mutants are symbiotically defective, deoxycholate sensitive, and bleomycin resistant.
138 ion, cell surface hydrophobicity, and sodium deoxycholate sensitivity.
139          Cell surface-bound FBG is initially deoxycholate-soluble, which, over time, becomes incorpor
140 exposure of a number of enteric pathogens to deoxycholate stimulates a conserved survival response to
141 tively resistant to in vitro dissociation by deoxycholate, suggesting a change in cellular IkappaB co
142                          With the detergent, deoxycholate, the endocytosis rate was restored to inter
143 icator (PRODAN), and a hydrophobic additive (deoxycholate) to detect terpenes.
144                                       Sodium deoxycholate treatment mimicked the effect of heat-kille
145 tivity nor its mRNA stability was altered by deoxycholate treatment.
146 cholate drop test, 99 and 98%, respectively; deoxycholate tube test, 100 and 99%, respectively; optoc
147                    Surprisingly, cholate and deoxycholate, two of the most abundant and very closely
148   The latter was activated by treatment with deoxycholate under the same conditions as mammalian NF-k
149  encodes a periplasmic protein that promotes deoxycholate uptake and reduces tolerance to deoxycholat
150 bance in the test tube containing 10% sodium deoxycholate versus a blank control tube, after incubati
151 ot reduced by dithionite until the detergent deoxycholate was added to disrupt membranes.
152 ction of COX-2 mRNA by chenodeoxycholate and deoxycholate was due to increased transcription.
153 cifically, continuous growth of C. jejuni in deoxycholate was found to: 1) induce the production of r
154        Cellular sensitivity to the detergent deoxycholate was increased for each deletion mutant, imp
155     In turn, induction of Cdx2 expression by deoxycholate was mediated by binding sites in the proxim
156 y heating at 56 degrees C in the presence of deoxycholate were able to function as templates in an in
157 ecting cell integrity; in contrast to sodium deoxycholate which enhanced insulin permeability but was
158                                              Deoxycholate, which interferes with the protein-protein
159 ed that the transporter binds to cholate and deoxycholate with micromolar affinity, and transport ass

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