ライフサイエンスコーパス: deoxycorticosterone acetate

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1 +) intake regardless of the presence of high deoxycorticosterone acetate.

2 cement therapy with either corticosterone or deoxycorticosterone acetate.

3 studied 2 weeks later) mice without and with deoxycorticosterone acetate administration, all in the s

4 Exogenous deoxycorticosterone acetate and deletion of the P2Y(2) r

5 vels in thoracic aortas when challenged with deoxycorticosterone acetate and high-salt diet (DOCA-sal

6 er the induction of severe hypertension with deoxycorticosterone acetate and salt, proteinuria, impai

7 ust sodium appetite (e.g., sodium depletion, deoxycorticosterone acetate) decrease lateral hypothalam

8 ase-3 (NOS3) expression in rats treated with deoxycorticosterone acetate (DOCA) and high salt.

9 (SD), spontaneously hypertensive (SHR), and deoxycorticosterone acetate (DOCA) hypertensive single c

10 sverse aortic constriction (TAC) surgery and deoxycorticosterone acetate (DOCA) pellet implantation.

11 Arteries from deoxycorticosterone acetate (DOCA)-salt and N(omega)-nit

12 ertensive mice with kidney injury induced by deoxycorticosterone acetate (DOCA)-salt compared to the

13 Deoxycorticosterone acetate (DOCA)-salt hypertension is

14 ugmented by increased endothelin-1 (ET-1) in deoxycorticosterone acetate (DOCA)-salt hypertension, a

15 scular superoxide level is also increased in deoxycorticosterone acetate (DOCA)-salt hypertension, wh

16 ound that CXCL16 is induced in the kidney in deoxycorticosterone acetate (DOCA)-salt hypertension.

17 th CTLA4-Ig reduced both angiotensin II- and deoxycorticosterone acetate (DOCA)-salt-induced hyperten

18 hat are administered the aldosterone-mimetic deoxycorticosterone acetate (DOCA).

19 Administration of the mineralocorticoid deoxycorticosterone-acetate (DOCA) in combination with h

20 ropriate for salt status, mineralocorticoid (deoxycorticosterone acetate) excess causes hypertrophy,

21 Compared with sham mice, sham plus deoxycorticosterone acetate mice had mild hypertrophy wi

22 on mice, transverse aortic constriction plus deoxycorticosterone acetate mice had similar left ventri

23 prevented decreases in the ECu50 induced by deoxycorticosterone acetate-no salt treatment.

24 taneous implantation of a controlled-release deoxycorticosterone acetate pellet, and given 1% saline

25 ion per se were studied in uninephrectomized deoxycorticosterone acetate salt-treated rats, where the

26 In aortas of mice with deoxycorticosterone acetate-salt (DOCA-salt) hypertensio

27 se in RMR in response to a high-fat diet and deoxycorticosterone acetate-salt (DOCA-salt) treatments,

28 pes in the brain, ADAM17 upregulation during deoxycorticosterone acetate-salt hypertension occurs sel

29 To test this hypothesis, we used the deoxycorticosterone acetate-salt model of neurogenic hyp

30 ebrospinal fluid of nontransgenic mice after deoxycorticosterone acetate-salt treatment and were acco

31 Deoxycorticosterone acetate-salt treatment in nontransge

32 have shown that stimuli like angiotensin II, deoxycorticosterone acetate-salt, and excessive catechol

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