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1 a) and viability (insulin-stimulated (18)F-2-deoxyglucose).
2 n the presence of the glycolysis inhibitor 2-deoxyglucose.
3 coupled with tracer radioactively labeled 2-deoxyglucose.
4 s if glucose is replaced with nonmetabolized deoxyglucose.
5 ravenous injection of the glucopenic agent 2-deoxyglucose.
6 reased in Arabidopsis cells in response to 2-deoxyglucose.
7 e a functional isoform with low affinity for deoxyglucose.
8 e of metabolic inhibition with cyanide and 2-deoxyglucose.
9 ly different sensitivities to lysozyme and 2-deoxyglucose.
10 We also compared uptake of FLT and deoxyglucose.
11 RNAs inhibits insulin-stimulated uptake of 2-deoxyglucose.
12 e as a function of growth than did uptake of deoxyglucose.
13 S42, UW479 and RES186) using metformin and 2-deoxyglucose.
14 5Y neuroblastoma cells with sodium azide and deoxyglucose.
15 Glucose uptake was measured using (3)H-deoxyglucose.
16 se that converts UDP-glucose to UDP-4-keto-6-deoxyglucose.
17 glucose analogs alpha-methyl glucoside or 2-deoxyglucose.
18 th of these UOK257 cells by treatment with 2-deoxyglucose.
20 imaging with fluorine-18-labeled 2-fluoro-2-deoxyglucose ((18)FDG) ligand with kinetic analysis demo
21 13)N-ammonia and of the glucose tracer (18)F-deoxyglucose ((18)FDG) was graded on a five-point scale
22 tomography with (11)C-acetate, (18)F-fluoro-deoxyglucose ((18)FDG), and (18)F-fluoro-thiaheptadecano
23 m(-2)), using [(15)O]H(2)O and [(18)F]fluoro-deoxyglucose ([(18)F]FDG) to quantify tissue perfusion a
24 e accumulation of two cytotoxic compounds, 2-deoxyglucose (2-DG) and copper(II)diacetyl-bis(N(4)-meth
27 gher uptake of radio-labeled [14C]2-fluoro-2-deoxyglucose (2-DG) in the preoptic area (25%) and signi
28 ats show fourth ventricular application of 2-deoxyglucose (2-DG) inhibits NST neurons and activates d
36 , we show that the hexose kinase inhibitor 2-deoxyglucose (2-dG) preferentially kills cancer cells wi
40 The study objective was to determine if 2-deoxyglucose (2-DG), a glucose analogue that blocks its
41 al MAN perfusion of the glucoprivic agent, 2-deoxyglucose (2-DG), under normal and hypoglycemic condi
46 thoxyphenylhydrazone (FCCP, 50 nmol/L) and 2-deoxyglucose (2-DG, 10 mmol/L), there was a decrease in
47 ficacy of F1,6BP was compared with that of 2-deoxyglucose (2-DG; an inhibitor of glucose uptake and g
48 s is reciprocally regulated by glucose and 2-deoxyglucose (2-DG; inhibitor of cellular glucose metabo
50 059; and (c) effects of AICAR on aPKCs and 2-deoxyglucose (2-DOG) uptake were inhibited by genistein,
53 iple brain structures during neglect using 2-deoxyglucose (2DG) as a metabolic marker of neural activ
59 ons of primary visual cortex and measuring 2-deoxyglucose (2DG) uptake to assess neural activity in s
61 teral neuronal and hemodynamic changes and 2-deoxyglucose (2DG) uptake, as measured by autoradiograph
62 the impact of glycolysis inhibition, using 2-deoxyglucose (2DG), in combination with cytotoxic agents
63 metabolism with the glycolysis inhibitor, 2-deoxyglucose (2DG), is a viable therapeutic strategy, bu
67 , we used pharmacological agents (insulin, 2-deoxyglucose, 3-nitropropionic acid, and kainic acid) to
68 ond gene encodes a bifunctional UDP-4-keto-6-deoxyglucose-3,5-epimerase/-4-reductase that converts UD
69 elated positively with in vitro assays of 3H-deoxyglucose (3H-DG) uptake in cells harvested via bronc
70 -deoxyglucose (8 and 9) > compounds with 2,6-deoxyglucose (5 and 6) > compounds with 2,3,6-deoxygluco
71 cose analogues 3-O-methylglucose (3OMG) or 6-deoxyglucose (6DOG) has been cited as evidence for metab
76 degrees of overlap in their monomolecular 2-deoxyglucose activation patterns to test the theory in a
78 ose analogs such as 3-O-methyl-glucose and 2-deoxyglucose also caused an induction, suggesting that s
80 sing two-photon imaging of a near-infrared 2-deoxyglucose analogue (2DG-IR), that glucose is taken up
81 h glucose deprivation combined with 0.5 mm 2-deoxyglucose and 5 mm azide ("chemical ischemia") to mod
83 lidinedione-derived ERMA, CG-12, vis-a-vis 2-deoxyglucose and glucose deprivation, we obtain evidence
84 rences in the insulin-stimulated uptake of 2-deoxyglucose and in the activity of carnitine palmitoyl
85 re decreased by the glucose antimetabolite 2-deoxyglucose and increased by high blood glucose concent
88 is observed under metabolic inhibition with deoxyglucose and oligomycin, indicating an energy-indepe
89 lls are 10 and 4.9 times more sensitive to 2-deoxyglucose and oxamate, respectively, than wt cells.
91 ound that ABT-263 increased sensitivity to 2-deoxyglucose and promoted rapid and extensive cell death
93 n the presence of the glycolysis inhibitor 2-deoxyglucose and radiation treatment followed by PBMC ch
94 e synergy between the glycolytic inhibitor 2-deoxyglucose and rapamycin in decreasing cell viability.
96 rocess, because cells depleted of ATP with 2-deoxyglucose and sodium azide were unable to properly re
99 lar layer was measured as uptake of [(14)C]2-deoxyglucose and was mapped into anatomically standardiz
100 patterns were measured as uptake of [(14)C]2-deoxyglucose and were mapped into standardized data matr
101 ty (euglycemic-hyperinsulinemic clamp with 2-deoxyglucose) and fat utilization during 1 h of exercise
102 creases in permeability, aliphatic alcohols, deoxyglucose, and chilling trigger the reversible dissoc
108 sing c-Fos early gene expression and (14)C 2-deoxyglucose autoradiography during mother-to-infant fea
110 tumors had similar uptake of [(18)F]fluoro-2-deoxyglucose before and after 2 weeks of 2-DG treatment
112 lucosensors detect mannose, d-glucose, and 2-deoxyglucose but not galactose, l-glucose, alpha-methyl-
113 Hydralazine activated more neurons than 2-deoxyglucose but similar numbers of catecholaminergic ne
114 by the non-metabolizable glucose analogue 2-deoxyglucose, but not by stimulating intracellular ATP p
115 uated by fructose, galactose, mannose, and 2-deoxyglucose, but not by the non-metabolizable glucose a
116 bles that of cortical metabolism seen with 2-deoxyglucose, but the increase in vascular density prece
117 erregulatory responses to hypoglycemia and 2-deoxyglucose, but the mechanisms that mediate these resp
118 of conversion of dTDP-6FGlc to dTDP-4-keto-6-deoxyglucose by each Asp135 variant was identical to tha
119 onversion of dTDP-glucose into dTDP-4-keto-6-deoxyglucose by Escherichia coli dTDP-glucose 4,6-dehydr
121 7-nitrobenz-2-oxa-1, 3-diazol-4-yl) amino)-2 deoxyglucose compared with those from HIV(-) controls.
122 ing intact Mpi(-/-) fibroblasts with 2-[(3)H]deoxyglucose confirmed mannose-dependent hexokinase inhi
123 low uptake activity for the glucose analog 2-deoxyglucose, consistent with a role in the transport of
125 al, we have measured the uptake of tritiated deoxyglucose (DG) in neutrophils isolated from human per
126 ulin-stimulated glucose uptake ((18)F-fluoro-deoxyglucose) during euglycemic (5.6 mmol/l), physiologi
127 he presence of an inhibitor of glycolysis, 2-deoxyglucose, enhanced the generation of memory cells an
131 heterogeneity in the uptake of [(18)F]fluoro-deoxyglucose (FDG) in single cells, which was found cons
132 emission tomography (PET) imaging with (18)F deoxyglucose (FDG) is a molecular imaging modality that
133 ism was assessed with (18)F-labeled fluoro-2-deoxyglucose (FDG) positron emission tomography in 236 y
135 ositron emission tomography with fluorine-18-deoxyglucose (FDG-PET) detects active lymphoid tissues d
136 by positron emission tomography using (18)F-deoxyglucose (FDG-PET) has not been established after sa
137 n vitro work and confirms the selectivity of deoxyglucose for viable cells over necrotic regions and
138 sures derived from the comparison of [14C]-2-deoxyglucose glomerular activity pattern data yielded a
139 ia and knob can incorporate and accumulate 2-deoxyglucose (glucose analog), but not when blocking GLU
140 sitivity to radiation with or without 25mM 2-deoxyglucose (glycolytic inhibitor) was evaluated in clo
141 diopharmaceutical used in PET imaging - [18F]deoxyglucose - has a limited role in diagnosing primary
143 rceptual similarity and comparability with 2-deoxyglucose imaging data from the olfactory bulb are de
146 of excised plaques confirmed accumulation of deoxyglucose in macrophage-rich areas of the plaque.
148 tic agents (ERMAs) such as resveratrol and 2-deoxyglucose in suppressing carcinogenesis in animal mod
149 catalyzes C-3 deoxygenation of CDP-4-keto-6-deoxyglucose in the biosynthesis of 3,6-dideoxyhexoses,
155 re layer was assessed by mapping uptake of 2-deoxyglucose into anatomically standardized data matrice
156 ation of AMPK in response to ionomycin and 2-deoxyglucose is not impaired in LKB1(-/-) murine embryo
157 animals studied using the metabolic marker 2-deoxyglucose, layer 4 was 25% denser than the other laye
158 ng p53, we showed that CR mimetics such as 2-deoxyglucose led to a decrease in Mcl-1 expression and s
159 nd pdk1, lung fluorine-18-labeled 2-fluoro-2-deoxyglucose ligand uptake was significantly increased i
160 nthesis (oligomycin, 2,4-dinitrophenol, or 2-deoxyglucose) made them more susceptible to cell death b
161 sults fail to confirm predictions based on 2-deoxyglucose maps of bulbar activity that enantiomers of
163 e distinguishable functionally, we used [14C]deoxyglucose metabolic mapping in the rat and tested whe
164 avioral abnormalities, we applied the [(14)C]deoxyglucose method for the determination of cerebral me
166 uctures in this system, we used the [(14)C]2-deoxyglucose method to determine glomerular responses to
169 and glycolysis (as measured by (18)fluoro-2-deoxyglucose microPET) of glioblastoma xenografts engine
170 ffects of metabolic blockade (cyanide plus 2-deoxyglucose) on Ca2+ release from the sarcoplasmic reti
174 s, Thr49 was phosphorylated in response to 2-deoxyglucose or phenformin, stimuli that activate the AM
177 were exposed to the glycolytic inhibitor, 2-deoxyglucose, or fatty acid synthase inhibitors to pertu
178 cell lines, 5 small-molecule perturbagens (2-deoxyglucose, oxamate, oligomycin, rapamycin, and wortma
180 glucose metabolism (imaged with [(18)F]fluro-deoxyglucose PET), and structural atrophy (imaged by MRI
181 static lesions in both tumor types with [18F]deoxyglucose PET, as compared with previous studies.
182 urrent prostate cancer than (18)F-2-fluoro-D-deoxyglucose-PET and monoclonal antibody imaging with th
183 lity, validity and reproducibility of fluoro-deoxyglucose-PET/CT for imaging of atherosclerotic plaqu
187 We retrospectively evaluated (18)fluoro-2-deoxyglucose positron emission tomography (FDG-PET) scan
189 that contained fluorine 18 ((18)F) fluoro-2-deoxyglucose positron emission tomography (PET) and mess
190 iew addresses technical improvements in [18F]deoxyglucose positron emission tomography (PET) and new
191 al blood to clinical outcomes and (18)fluoro-deoxyglucose positron emission tomography combined with
192 icient rationale given the utility of fluoro-deoxyglucose positron emission tomography in diagnostic
193 cs, magnetic resonance imaging and 18-fluoro-deoxyglucose positron emission tomography results, and n
194 lizing on the diagnostic utility of 18fluoro-deoxyglucose positron emission tomography that relies on
198 n brain glucose metabolism (measured by [18F]deoxyglucose-positron emission tomography) and on its re
200 In addition, treatment of NOD mice with 2-deoxyglucose resulted in improved beta cell granularity.
201 tudies using intrinsic optical imaging and 2-deoxyglucose) resulted in increased detection thresholds
203 ose however, combination of metformin with 2-deoxyglucose significantly reduced cell proliferation co
204 ndent manner, whereas oxidative stress and 2-deoxyglucose stimulated phosphorylation at this site via
206 is, functional magnetic resonance imaging, 2-deoxyglucose studies, and induction of gene expression h
207 results are consistent with a previous (14)C-deoxyglucose study of the isoflurane-anesthetized rat.
208 of pyruvate or alpha-ketocaproate, but not 2-deoxyglucose, suggesting that mitochondrial metabolism w
209 We demonstrate that the parthenolide, 2-deoxyglucose, temsirolimus (termed PDT) regimen is a pot
210 determined by NMR spectroscopy, including 2-deoxyglucose, the glucose analogue used for tumor detect
212 hen prediabetic NOD mice were treated with 2-deoxyglucose to block aerobic glycolysis, there was a re
213 of ATP by the addition of sodium azide and 2-deoxyglucose to block ATP production by oxidative phosph
214 positron emission tomography (PET) and [18F] deoxyglucose to compare the brain metabolic responses (m
215 er, we used the quantitative method of (14)C-deoxyglucose to reveal changes in activity, in the corte
217 e, we show that low doses of verapamil and 2-deoxyglucose, to accentuate the cost of resistance and t
218 ies examining the LC values for radiolabeled deoxyglucose tracers used to estimate the glucose metabo
219 dipocytes also attenuated insulin-stimulated deoxyglucose transport and Myc-GLUT4-EGFP translocation
221 onse relationship for insulin stimulation of deoxyglucose transport in primary adipocytes derived fro
224 cells failed to attenuate insulin-stimulated deoxyglucose transport or Myc-tagged GLUT4-GFP transloca
227 on preconditioning (1 h of antimycin A and 2-deoxyglucose treatment followed by 1 h of recovery), ade
228 g p38 vectors reduced apoptosis induced by 2-deoxyglucose treatment, whereas overexpression of wild-t
229 [(14)C]glucose into glycogen (60%) and [(3)H]deoxyglucose uptake (40%) but did not inhibit phosphoryl
230 ose incorporation into glycogen (60%), [(3)H]deoxyglucose uptake (60%), and protein kinase B phosphor
231 We therefore examined the effects of HGF on deoxyglucose uptake (DOGU), glucose utilization, and fat
232 assessments of cold-induced changes in BAT 2-deoxyglucose uptake (increased 2.7-fold), BAT lipogenesi
234 ice, caCaMKKalpha increased in vivo [(3)H]-2-deoxyglucose uptake 2.5-fold and AMPKalpha1 and -alpha2
235 ocytes, and their membrane concentrations, 2-deoxyglucose uptake activities, and sensitivities to pCM
236 s N terminus suppressed insulin-stimulated 2-deoxyglucose uptake and Glut4 translocation to roughly t
238 bitor LY-294002 display a decrease in both 2-deoxyglucose uptake and hexokinase activity as compared
240 significant correlation between posterior 2-deoxyglucose uptake and molecular properties associated
242 aCaMKKalpha increased basal in vivo [(3)H]-2-deoxyglucose uptake approximately twofold, insulin incre
243 t with DMOG or DHB reverses the decline in 2-deoxyglucose uptake caused by NGF withdrawal and suppres
244 ron emission tomography of 2-[(18)F]fluoro-2-deoxyglucose uptake combined with computed tomography.
245 Cip4-null mice exhibited increased [(14)C]2-deoxyglucose uptake compared with cells from wild-type m
246 by individual odorant chemicals, we mapped 2-deoxyglucose uptake during exposures to vapors arising f
247 les with the greatest UBX-Cter expression, 2-deoxyglucose uptake during fasting was similar to that i
248 nfusion rate and 90% greater muscle [(3)H]-2-deoxyglucose uptake during hyperinsulinemic-euglycemic c
249 evious studies, we mapped glomerular layer 2-deoxyglucose uptake evoked by hundreds of both systemati
251 d with c-Fos immunohistochemistry and [14C]2-deoxyglucose uptake implicate a prominent involvement of
252 nduced PPAR gamma-dependent adipogenesis and deoxyglucose uptake in 3T3-L1 preadipocytes at a potency
253 on of [(14)C]glucose into glycogen and [(3)H]deoxyglucose uptake in L-CPT I-transduced, palmitate-tre
254 rogressive but similar levels of increased 2-deoxyglucose uptake in macrophages that reached up to si
255 first investigated glomerular patterns of 2-deoxyglucose uptake in response to aromatic compounds th
256 ent with the 2.5- to threefold increase in 2-deoxyglucose uptake in skeletal muscle, heart, and white
257 ytokine, increases macrophage glycolysis and deoxyglucose uptake in vitro and acutely enhances (18)F-
258 ke in transfected muscles, we measured [3H]2-deoxyglucose uptake in vivo following intravenous glucos
259 tified activity patterns by mapping [(14)C]2-deoxyglucose uptake into anatomically standardized data
261 glucose infusion rate and markedly reduced 2-deoxyglucose uptake into skeletal muscle (85-90%) and wh
262 tive to WL5, submaximal insulin-stimulated 2-deoxyglucose uptake into the epitrochlearis muscle was l
263 rug triester 70 did induce enhancements in 2-deoxyglucose uptake into two different cell lines with c
264 e together with impaired exercise-mediated 2-deoxyglucose uptake into white but not red muscles.
265 robust and surprisingly focal patterns of 2-deoxyglucose uptake involving clusters of neighboring gl
266 Ischemia stimulated a 2.5-fold increase in 2-deoxyglucose uptake over base line in WT, whereas the in
267 ozygote matings exhibited reduction of the 2-deoxyglucose uptake rate: one by 50% (presumed heterozyg
268 ly active cdc42 (CA-cdc42; V12) stimulated 2-deoxyglucose uptake to 56% of the maximal insulin respon
270 activity of the alpha2 isoform of AMPK and 2-deoxyglucose uptake were assessed in incubated rat exten
272 q), and CA-cdc42 on GLUT4 translocation or 2-deoxyglucose uptake were inhibited by microinjection of
273 entiated adipocytes and insulin-stimulated 2-deoxyglucose uptake were slightly lower than in adipocyt
274 osely with decreases in glucose transport (2-deoxyglucose uptake), measured during a subsequent 20-mi
275 testing, measurement of in vivo myocardial 2-deoxyglucose uptake, and echocardiography were performed
277 ipocytes, we analyzed Akt phosphorylation, 2-deoxyglucose uptake, and Glut4 translocation by immunofl
287 tory activation and uptake of radiolabeled 2-deoxyglucose was assessed before and after GM-CSF exposu
288 Rather, IRF3 activation by tunicamycin and 2-deoxyglucose was inhibited by 4-(2-aminoethyl)-benzenesu
290 depletion, equivalent ATP loss induced by 2-deoxyglucose was without toxicity, arguing that bioenerg
291 cancer cells to ERMAs, including CG-12 and 2-deoxyglucose, we demonstrated that this beta-TrCP accumu
292 7-nitrobenz-2-oxa-1, 3-diazol-4-yl) amino)-2 deoxyglucose were analyzed by flow cytometry on monocyte
294 ked by the energy poisons sodium azide and 2-deoxyglucose, whereas staining of the nucleus (nucleolus
295 rated by using an inhibitor of glycolysis, 2-deoxyglucose, which almost totally abolished low-dose ar
296 lucose-inhibited neurons were activated by 2-deoxyglucose, which also activates counterregulatory res
297 form glucose, the nonmetabolizable sugars 2-deoxyglucose, which is still converted to G-6-P as well
298 adiotracer for oncologic PET is (18)F-fluoro-deoxyglucose, which measures glucose accumulation as a s
299 radiation response after administration of 2-deoxyglucose, which significantly (p<0.05) potentiated e
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