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1 e water samples (0.8 ng/L to 1.14 mug/L, for deoxynivalenol).
2 h and development: aflatoxin, fumonisin, and deoxynivalenol.
3 enzymes toward B-type trichothecenes such as deoxynivalenol.
4 method limits of detection were 8mug/kg for deoxynivalenol, 10mug/kg for enniatin A1 and 5mug/kg for
5 ricin (BEA), deoxynivalenol (DON), 15-acetyl-deoxynivalenol (15-ADON), 3-acetyl-deoxynivalenol (3-ADO
6 ine trichothecenes (deoxynivalenol, 3-acetyl-deoxynivalenol, 15-acetyl-deoxynivalenol, nivalenol, neo
7 of 4 out of 12 studied trichothecenes: DON (deoxynivalenol), 15AcDON (15-acetyl-deoxynivalenol), T2-
9 15-acetyl-deoxynivalenol (15-ADON), 3-acetyl-deoxynivalenol (3-ADON), nivalenol (NIV), sterigmatocyst
11 of eighteen mycotoxins, nine trichothecenes (deoxynivalenol, 3-acetyl-deoxynivalenol, 15-acetyl-deoxy
12 otoxins in beer (deoxynivalenol-3-glucoside, deoxynivalenol, 3-acetyldeoxynivalenol, 15-acetyl-deoxyn
13 The stability of deoxynivalenol (DON) and deoxynivalenol-3-glucoside (DON-3-glucoside) during the
15 s spectrometry method capable of determining deoxynivalenol-3-glucoside (DON-3G), which is the main k
16 deoxynivalenol, 3- and 15-deoxynivalenol and deoxynivalenol-3-glucoside in 84 durum wheat samples, fr
20 also deoxynivalenol and its conjugated form (deoxynivalenol-3-glucoside) were determined in almost al
22 on the following mycotoxins: deoxynivalenol, deoxynivalenol-3-glucoside, and the minor metabolite cul
23 oxins including modified mycotoxins in beer (deoxynivalenol-3-glucoside, deoxynivalenol, 3-acetyldeox
24 ted to determine the fate of deoxynivalenol, deoxynivalenol-3-glucoside, HT-2 toxin and T-2 toxin, du
25 y to conjugate DON into a glucosylated form, deoxynivalenol-3-O-glucose (D3G), by secondary metabolis
26 , we found that F. graminearum produced more deoxynivalenol, a mycotoxin, in the primed treatment.
27 en analysed, and certain mycotoxins, such as deoxynivalenol, aflatoxin B1, aflatoxin B2, aflatoxin G1
29 ng of the five dry pasta samples, 60% of the deoxynivalenol and 83-100% of the enniatins were retaine
30 The occurrence of deoxynivalenol, 3- and 15-deoxynivalenol and deoxynivalenol-3-glucoside in 84 duru
33 nstrating contamination of most samples with deoxynivalenol and high frequency of zearalenone in samp
35 tigated the fungal diversity and presence of deoxynivalenol and zearalenone in 150 samples of freshly
36 samples containing relatively high levels of deoxynivalenol and/or enniatins were selected for the co
37 wheat plants (0.1-133 mg/kg(dry weight), for deoxynivalenol), and drainage water samples (0.8 ng/L to
39 More than 60% of the samples analysed showed deoxynivalenol contamination, followed by HT-2 toxin and
40 type of trichothecene produced (nivalenol or deoxynivalenol) cosegregated with the TRI5 gene (which e
42 the mycotoxins quantified in wheat (3-acetyl-deoxynivalenol, deoxynivalenol, fusarenone-X, nivalenol,
44 mainly focused on the following mycotoxins: deoxynivalenol, deoxynivalenol-3-glucoside, and the mino
45 ation was conducted to determine the fate of deoxynivalenol, deoxynivalenol-3-glucoside, HT-2 toxin a
46 antitative column-based rapid immunotest for deoxynivalenol detection with IC50 of 473 and 20 ng/ml,
47 15-acetyldeoxynivalenol (15-ADON), de-epoxy-deoxynivalenol (DOM-1) and ochratoxin A (OTA) during the
49 to determine the incidence of the mycotoxins deoxynivalenol (DON) and fumonisin B1 (FB1) in industria
50 ification of mycoflora and the occurrence of deoxynivalenol (DON) and fumonisins (FBs) in malting bar
52 parameters), the bioactive compound content, deoxynivalenol (DON) contamination and the physical prop
59 A significant incidence of HT-2 toxin and deoxynivalenol (DON) were found in 9.1% and 59.7% of tot
60 ochratoxin A (OTA), aflatoxin B1 (AFB1) and deoxynivalenol (DON) which are strictly regulated food c
61 (DAS), three fumonisins, beauvericin (BEA), deoxynivalenol (DON), 15-acetyl-deoxynivalenol (15-ADON)
63 ochratoxin A (OTA), aflatoxin B1 (AFB1) and deoxynivalenol (DON), are subject to strict regulations
67 of the early wheat response to the mycotoxin deoxynivalenol (DON), which is a virulence factor produc
68 type B trichothecenes on cereals, including deoxynivalenol (DON), which is harmful for humans and an
69 ped and validated for three Fusarium toxins, deoxynivalenol (DON), zearalenone (ZEA) and T-2 toxin.
72 on of aflatoxins, ochratoxin A, zearalenone, deoxynivalenol, fumonisins, T-2 and HT-2 toxins, fusaren
73 s (ochratoxin A, fumonisin B1, fumonisin B2, deoxynivalenol, fusarenon-X, T-2 and HT-2 toxin, citrini
74 uantified in wheat (3-acetyl-deoxynivalenol, deoxynivalenol, fusarenone-X, nivalenol, HT-2 toxin, T-2
75 was 33%, 6.5%, 2%, 27%, 7%, 10% and 43% for deoxynivalenol, HT-2, T-2, nivalenol, zearalenone, beauv
76 nivalenol, 3-acetyldeoxynivalenol, 15-acetyl-deoxynivalenol, HT2-toxin, T2-toxin, enniatin B, B1, A1,
77 abels for immunoassay detection of mycotoxin deoxynivalenol in food and feed, CdSe/CdS/ZnS core-shell
78 essfully used for determination of mycotoxin deoxynivalenol in wheat and maize samples by fluorescenc
81 sult of this remarkable symbiosis is reduced deoxynivalenol mycotoxin, potentially benefiting million
82 ivalenol, 3-acetyl-deoxynivalenol, 15-acetyl-deoxynivalenol, nivalenol, neosolaniol, diacetoxyscirpen
84 verning trichothecene toxin amount and type (deoxynivalenol or nivalenol) map on linkage groups IV an
85 role in growth, asexual/sexual sporulation, deoxynivalenol production and virulence in F. graminearu
87 All the commercial cultivars transformed deoxynivalenol to its glucosylated form at conversion ra
90 t the same extent as the prominent mycotoxin deoxynivalenol, while NX-2 is far less toxic, similar to
92 toxins A and B, HT-2 and T-2 toxins, deepoxy-deoxynivalenol, zearalenone, sterigmatocystin and fumoni
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