ライフサイエンスコーパス: depolarization-evoked

1 gonist, CGP52432, which was shown to amplify depolarization-evoked [(3)H]d-aspartate release in the v

2 e cholinergic presynaptic terminal membrane; depolarization evoked [3H]glycine release that was calci

3 during embryonic day (E) 11-17 have enhanced depolarization-evoked acetylcholine (ACh) release from h

4 On P18, IGF2/depolarization-evoked ACh release from hippocampal slice

5 brain ACh levels as well as ACh content and depolarization-evoked ACh release in hippocampal slices

6 ChC and ChS rats and an inability to sustain depolarization-evoked ACh release relative to the ChS an

7 ted by HACU, were lowest in ChS rats whereas depolarization-evoked ACh release was the highest.

8 nel blocker lanthanum (0.5 mM) inhibited the depolarization-evoked acid shifts.

9 s of the basal forebrain (BFB) to study both depolarization-evoked adenosine release and the steady s

10 Strong stepwise depolarization evoked an exocytic burst that lasted 10 m

11 The membrane depolarization evoked by Ba2+ or CRH increased the cell

12 under basal conditions and during localized depolarization evoked by infusion of a high-K(+) solutio

13 neurovascular coupling holds that glial cell depolarization evoked by neuronal activity leads to the

14 (1) Depolarizations evoked by orthogonal stimuli are determi

15 (2) Depolarizations evoked by preferred stimuli saturate at

16 perm similarly correlates with an absence of depolarization evoked Ca(2+) entry.

17 croscopy revealed that, at the axon, somatic depolarization evoked Ca(2+) influx through voltage-sens

18 Our examination of depolarization-evoked Ca(2+) entry indicates that mature

19 s (RRP and IRP, respectively) in response to depolarization-evoked Ca(2+) influx.

20 inhibitory effects of AEA and URB597 on the depolarization-evoked Ca(2+) transient were increased in

21 an increase in the amplitude and duration of depolarization-evoked Ca(2+) transients in putative noci

22 ects on insulin secretion, including reduced depolarization-evoked Ca(2+)-influx and beta-cell exocyt

23 enotype and is associated with an absence of depolarization-evoked Ca(2+)entry.

24 ptical imaging of exocytosis and submembrane depolarization-evoked [Ca(2+)](i).

25 we show that mouse CatSper1 is essential for depolarization-evoked Ca2+ entry and for hyperactivated

26 n rat portal vein smooth muscle cells during depolarization-evoked Ca2+ entry.

27 (36 degrees C) resulted in a more sustained depolarization-evoked Ca2+ increase compared with more t

28 The depolarization-evoked Ca2+ increase was present in Ca2+-

29 racellular dialysis with heparin blocked the depolarization-evoked Ca2+ increase, indicating a role f

30 evoke exocytosis, nor does it interfere with depolarization-evoked Ca2+ influx and exocytosis.

31 The delta Cm,J was proportional to depolarization-evoked Ca2+ influx with initial exocytoti

32 idly and reversibly sequestering Ca2+ during depolarization-evoked Ca2+ loads.

33 nstream of surface membrane receptors in the depolarization-evoked Ca2+ response.

34 cin increased the duration of spontaneous or depolarization-evoked Ca2+ sparks 6- to 7-fold.

35 The amplitude of the depolarization-evoked Ca2+ transient is larger in dorsal

36 absence of this region selectively abolished depolarization-evoked Ca2+ transients without affecting

37 pluronic F-127 appeared to be affecting the depolarization-evoked [Ca2+]cyt transient in DRG neurons

38 ate that pluronic F-127 significantly alters depolarization-evoked [Ca2+]cyt transients, which may re

39 also substantially reduced the amplitude of depolarization-evoked [Ca2+]i increases, providing evide

40 Melatonin exerted an inhibitory influence on depolarization-evoked calcium increases, and the melaton

41 The observation that depolarization-evoked calcium release can occur after ry

42 l axons, the opposite appears to occur since depolarization-evoked calcium rises in retinotectal axon

43 tration (1.5%) that had little effect on the depolarization-evoked calcium spike.

44 ubstantiated by a linear correlation between depolarization-evoked capacitance increases and EPSC cha

45 It was concluded that the depolarization-evoked current was activated by Ca2+.

46 A depolarization-evoked endocytosis was observed that shar

47 The G100V/C103V mutation nearly abolishes depolarization-evoked exocytosis (measured by membrane c

48 Depolarization-evoked exocytosis and Ca(2+) currents in

49 The reduction of depolarization-evoked exocytosis in du/du IHCs reflected

50 In addition, depolarization-evoked exocytosis is markedly facilitated

51 ctivity is hardly evident, alpha-LT augments depolarization-evoked exocytosis probably by second mess

52 to the jump in capacitance that accompanies depolarization-evoked exocytosis.

53 Blocking P/Q-type Ca(2+)-currents abolished depolarization-evoked exocytosis.

54 te normal intracellular Ca(2+) signaling and depolarization-evoked exocytosis.

55 decrease spontaneous firing while increasing depolarization-evoked firing in a DA receptor dependent

56 ls in pigment epithelial (PE) cells, whereas depolarization evoked [Formula: see text] elevations in

57 hat two components evident previously in the depolarization-evoked gating currents from voltage-gated

58 Both treatments reversed the reduction in depolarization-evoked glutamate release and in the expre

59 rats was inversely related to the extent of depolarization-evoked glutamate release in the ventral h

60 +/-3% from DKO nerve terminals and potassium depolarization-evoked glutamate release was also decreas

61 The relationship between depolarization-evoked ICa and rise in [Ca2+]i was examin

62 as multiple action potentials and prolonged depolarizations evoked in layer II stellate cells of epi

63 ndane revealed an additive inhibition of the depolarization-evoked increase in [Ca(2+)](i), whereas t

64 t Ca(2+)-induced Ca2+ release contributes to depolarization-evoked increases in [Ca2+]i in rat resist

65 dependence and the voltage dependence of the depolarization-evoked increases in ICa and [Ca2+]i were

66 pplication of glycine effectively suppressed depolarization-evoked increases in intracellular Ca2+ at

67 Depolarization-evoked increases in PKA activity were blo

68 time- and concentration-dependently inhibit depolarization-evoked influx of Ca(2+).

69 NQ1, and TCF7L2 were associated with reduced depolarization-evoked insulin exocytosis.

70 pendymal layer (SEL) undergo spontaneous and depolarization-evoked intracellular calcium transients m

71 Strong depolarization evoked irregular current fluctuations, wh

72 evetoxins that produce neural insult through depolarization-evoked Na+ load, glutamate release, relie

73 ctions in membrane Ca2+ channel currents and depolarization-evoked neurotransmitter release have been

74 Vigabatrin enhanced this depolarization-evoked nonvesicular GABA release and also

75 The depolarization-evoked potentiation of the muscarinic sig

76 produce DISC attenuation through blockade of depolarization-evoked Purkinje cell Ca transients.

77 T) potently enhances both "spontaneous" and "depolarization-evoked" quantal secretion from neurons.

78 xpressed L-, N-, and P-type Ca channels, and depolarization evoked rapid catecholamine secretion reco

79 Depolarization evoked rapid positive bundle deflections

80 voltagegated Ca(2+) channels, increasing the depolarization-evoked rate of rise of intracellular Ca(2

81 Surprisingly, sucrose or potassium depolarization-evoked release of 14C-GABA was increased

82 Interestingly, depolarization-evoked release of [(14)C]-GABA was signif

83 Depolarization-evoked release of [(3)H]-NE from amygdala

84 effects of opioid agonists and CP 55,940 on depolarization-evoked release of calcitonin gene-related

85 On the other hand, the depolarization-evoked release of K+ suppressed by ATP co

86 However, enhancement of depolarization-evoked release, seen in many cells at <50

87 isappearance of the pool of DA available for depolarization-evoked release.

88 These observations suggest that the depolarization-evoked rise in [Ca2+]i was tightly couple

89 increase in omega-conotoxin GVIA-sensitive, depolarization-evoked rises in [Ca(2+)](i).

90 Single 250 msec depolarizations evoked saturating capacitance responses

91 ound receptor contributing to enhancement of depolarization-evoked secretion as well as spontaneous r

92 Depolarization-evoked sparks elicited with small pulses

93 Ryanoids also abolished depolarization-evoked sparks elicited with small pulses,

94 bursts induced an increase in the number of depolarization-evoked spikes in some neurones, but in ot

95 ion or current injection, we show that brief depolarization evoked spiking and suppressed firing duri

96 In a small subset of neurons, depolarization evoked stronger calcium elevations, appro

97 ed little change in TEP and did not modulate depolarizations evoked through the B pathway.

98 In all patches, step depolarizations evoked transient current, and step repol

99 ic acid also dose-dependently potentiated K+-depolarization evoked vasopressin release.

100 Consistent with fusion, depolarization-evoked vesicle disappearance paralleled e

101 Depolarization evoked voltage-dependent Ca(2+) currents