ライフサイエンスコーパス: deprivation

1 isk factors (age, sex, ethnicity, and social deprivation).

2 Fe/S cluster biogenesis proteins during iron deprivation.

3 ioral adjustment to a novel task after sleep deprivation.

4 and proliferation defect caused by glutamine deprivation.

5 eviation of the behavioural constellation of deprivation.

6 s (P&N's) hypothesised adaptive responses to deprivation.

7 al in flies counteracts the effects of sleep deprivation.

8 eeking and the consumption of salt following deprivation.

9 y metabolic programs required under nutrient deprivation.

10 dent and exhibit little death during cystine deprivation.

11 SD caused by K+ perfusion and oxygen-glucose deprivation.

12 h of the evidence and the timing of economic deprivation.

13 olecular properties that respond to androgen-deprivation.

14 reas this was not observed after acute sleep deprivation.

15 ns, undergo long periods of enteral nutrient deprivation.

16 lar to what is observed during acute glucose deprivation.

17 sponse to low socioeconomic status (SES), or deprivation.

18 , including a session after full-night sleep deprivation.

19 unting is a functional, adaptive response to deprivation.

20 group of UK adoptees who did not experience deprivation.

21 -seq analysis of N. gaditana during nitrogen deprivation.

22 riod mutant mice was also robust during food deprivation.

23 m auditory sensory brain areas due to visual deprivation.

24 Chlamydomonas subject to sulfur or phosphate deprivation.

25 high myopia and astigmatism induced by form deprivation.

26 ify novel noncoding RNAs regulated by oxygen deprivation.

27 but a beneficial effect in countering water deprivation.

28 ltaneity after either monocular or binocular deprivation.

29 logical, behavioral, and health responses to deprivation.

30 reached a matured level before the monocular deprivation.

31 gocytosis, are upregulated after acute sleep deprivation.

32 at they call the behavioral constellation of deprivation.

33 growth limitations associated with nutrient deprivation.

34 systemic cardiovascular adaptation to oxygen deprivation.

35 creases during prolonged periods of nutrient deprivation.

36 its genetic underpinnings following sensory deprivation.

37 eprivation, rather than the average level of deprivation?

38 Importantly, under nutrient deprivation, 16:4 and omega-3 C18 polyunsaturated FAs ac

39 ompensatory contact lenses (mean duration of deprivation = 4.4 months; range = 0.3-28.8 months).

40 ring differ after monocular versus binocular deprivation [8-11].

41 umvent the normally lethal effects of oxygen deprivation, a mechanism that could be harnessed to mini

42 evidence from animal models, we propose that deprivation accelerates the neurodevelopmental process o

43 Glucose deprivation activates AMP-activated protein kinase (AMPK

44 ime: contrary to intuition, monocular visual deprivation actually improves the deprived eye's competi

45 Randomization 2:1 to arginine deprivation (ADI-PEG20, 36.8 mg/m2, weekly intramuscular

46 It remains unclear whether early visual deprivation affects visuotactile perception similarly to

47 Food deprivation also challenges the gut microbiota, which re

48 he auditory cortex (ACx), and early auditory deprivation alters intrinsic and synaptic properties in

49 tome data were collected under normal, sleep-deprivation and abnormal sleep-timing conditions to asse

50 Stresses such as hypoxia, nutrient deprivation and acidification disturb protein folding in

51 in response to lesion-induced somatosensory deprivation and activity loss, and can be controlled by

52 Both sleep deprivation and caffeine treatment potentiated light-ind

53 Both sleep deprivation and caffeine treatment potentiated light-ind

54 mmalian body provide resilience against food deprivation and dietary stress.

55 h of England characterised by high levels of deprivation and diverse populations.

56 nked with relative state - personal relative deprivation and envy - that may play an important role i

57 he depletion of energy induced by acute food deprivation and excessive storage of energy by high-fat

58 n 15 healthy male adults after 52 h of sleep deprivation and following 14 h of recovery sleep.

59 required for TAG accumulation under nitrogen deprivation and for glycerol synthesis under high salini

60 Here, we demonstrate that glutamine deprivation and hypoxia result in inhibition of mTOR-med

61 luences how cancer cells respond to nutrient deprivation and hypoxic stress, two hallmarks of the tum

62 vironmental energetic stressors such as food deprivation and physical exertion.

63 mitochondrial translation during amino-acid deprivation and predicts novel ORFs in 5'UTRs, long nonc

64 gies help explain the health correlations of deprivation and provide additional pathways for feedback

65 regulated already after a few hours of sleep deprivation and shows a further significant increase aft

66 ral manifestations of sleep, wake, and sleep deprivation and specific measurable changes in the netwo

67 microscopy, we examined the effect of visual deprivation and subsequent recovery on dendritic spine r

68 policy responses to the relationship between deprivation and temporal discounting.

69 hibits CCNE1 expression during growth factor deprivation and that miR-874 down-regulation in osteosar

70 The ZI cells were excited by food deprivation and the gut hunger signal ghrelin.

71 ediated the relationship between sex, social deprivation, and Charlson Comorbidity Index with inciden

72 t in regions with the greatest socioeconomic deprivation, and deficiencies exist in training programm

73 ty), socioeconomic (education, neighbourhood deprivation, and household income), and psychological (d

74 ty, socioeconomic status, neighborhood-level deprivation, and population density.

75 stablished negative health outcomes of sleep deprivation, and the suggestion that availability of ele

76 0 phosphorylation are required for glutamine deprivation- and hypoxia-induced autophagy and brain tum

77 poxia and survive 18 minutes of total oxygen deprivation (anoxia) without apparent injury.

78 mited, early-life exposures to institutional deprivation are associated with disorders in childhood,

79 r hearing impairment, dehydration, and sleep deprivation are effective for delirium prevention and al

80 strategy, in which those who experience such deprivation are more risk-averse.

81 3 months of age, after which the effects of deprivation are thought to be permanent and without capa

82 as drought, temperature, salinity, nutrient deprivation, bacteria, virus and others.

83 's theory of the behavioral constellation of deprivation (BCD) would benefit from teasing apart the c

84 features of the behavioral constellation of deprivation (BCD).

85 onocular deprivation (MD), but not binocular deprivation (BD), increased dendritic spine elimination

86 Nutrient deprivation, biochemical analysis, and metabolite quanti

87 During water deprivation, body water preservation is ensured by the s

88 promotes cancer cell survival upon glutamine deprivation both in vitro and in vivo.

89 Moreover, sleep deprivation brings about vehicle accidents and medical e

90 a reversal of processes directly following N deprivation, but a distinct cellular state.

91 lular survival and homeostasis upon nutrient deprivation, but is repressed after feeding.

92 Sirt1 can be activated by energy deprivation, but the mechanism is poorly understood.

93 lanogaster males, sleep pressure after sleep deprivation can be counteracted by raising their sexual

94 ries provide insight into how extreme energy deprivation can impact Sirt1 activity and underscore the

95 quit smoking and improve living conditions (deprivation) can be also employed, however, may lead to

96 d activity-dependent plasticity by monocular deprivation caused rapid changes in single unit activity

97 oked firing rates decreased, suggesting that deprivation causes a wider range of perceptual problems

98 ce that, when growth is arrested by nutrient deprivation, cells induce rapid clearance of Pol I-Rrn3

99 Serum starvation resembles the growth factor deprivation characteristic of the poorly vascularized tu

100 it group-average PVT data during acute sleep deprivation, chronic sleep restriction, and recovery.

101 restriction and after 1 night of acute sleep deprivation compared to a regular sleep condition in a w

102 A mutant with slow recovery from N deprivation, compromised hydrolysis of triacylglycerols7

103 oalgae is lacking, especially under nitrogen deprivation conditions known to trigger lipid accumulati

104 homeostatic sleep-pressure response to sleep deprivation correlated negatively with the decrease in A

105 S1 immunohistochemistry, and longer arginine deprivation correlated with improved PFS.

106 oeconomic status (Scottish Index of Multiple Deprivation decile 1: RRR 2.27, 2.22-2.31) significantly

107 K(+) deprivation decreased root biomass and external K(+) upt

108 Unilateral whisker deprivation decreased the strength and spatial range of

109 We further demonstrate that our deprivation did not impair the maturation of disparity s

110 nvy - that may play an important role in the deprivation-discounting link.

111 matching is completely blocked by monocular deprivation during the critical period.

112 ANCE STATEMENT We demonstrate that monocular deprivation during the developmental critical period imp

113 Sensory deprivation during these periods permanently compromises

114 order to survive low temperatures and water deprivation during winter.

115 s unnecessary for producing this paradoxical deprivation effect: interocular suppression of an ordina

116 ormonally intact prostate but, upon androgen deprivation, exclusively labels a type of luminal stem c

117 state human EEG data during a 40-hour sleep deprivation experiment by evaluating the decay in autoco

118 Visual-deprivation experiments have suggested that the onset of

119 also be sensitive to subjective feelings of deprivation for critical nonfood resources (e.g., social

120 Nutrient deprivation further severely down-regulated the conversi

121 Here, we show that under glucose deprivation (GD) conditions, decreased expression of pre

122 his ansorgei, were aroused at night by sleep deprivation (gentle handling) or caffeine treatment that

123 t gonorrhoea after adjustment for ethnicity, deprivation, geographical area, and sex was 31% (95% CI

124 During nutrient deprivation, glucagon-mediated increase in calcium (Ca(2

125 ort that in various cancer cells upon oxygen deprivation, HIF-1 activation down-modulates LD cataboli

126 events during and after acute oxygen glucose deprivation highlights a possible important difference,

127 In contrast, chronic ACh deprivation hindered whisker-evoked CBF responses and th

128 adjusting for age, marital status, material deprivation history, smoking, drinking and socioeconomic

129 During sleep deprivation, homeostatic and circadian processes interac

130 As such, oxygen deprivation (hypoxia) limits cholesterol synthesis throu

131 irium, including cognitive impairment, sleep deprivation, immobility and visual and hearing impairmen

132 structural plasticity in response to whisker deprivation, impaired texture novel object recognition a

133 atic changes in spine size following sensory deprivation in a subset of inhibitory (layer 2/3 GAD65-p

134 tory cortex 15 days after permanent auditory deprivation in adult rats, which is partly reversed 90 d

135 A new study reveals the effects of visual deprivation in early life on the development of multisen

136 Sleep deprivation in early night, but not late night, potentia

137 lone may worsen pain, and experimental sleep deprivation in humans supports this claim.

138 arplugging-induced, early transient auditory deprivation in male and female Mongolian gerbils caused

139 in vivo, resulting in overeating after food deprivation in mice.

140 pithelial differentiation induced by insulin deprivation in normal human bronchial epithelial cells c

141 ted within areas of high social and economic deprivation in South Wales.

142 rodent, the Grass rat, indicates that sleep deprivation in the early rest period induces phase delay

143 States that links child outcomes to economic deprivation in the first several years of life.

144 g to characterize neurons activated by water deprivation in the hypothalamic median preoptic nucleus

145 Heme deprivation in the tsetse fly anterior midgut might repr

146 the NLRP3 inflammasome response to nutrient deprivation in wild-type and SIRT3 knock-out mice.

147 Early visual deprivation, in both animals and humans, leads to abnorm

148 ed increase in alpha power by means of sleep deprivation increased the average duration of individual

149 scientific commentary on this article.Sleep deprivation increases amyloid-beta, suggesting that chro

150 S and innexin2 are increased following sleep deprivation, indicating that GS and innexin2 genes are d

151 Early night, but not late night, sleep deprivation induced a significant phase shift.

152 Bcl-w conferred sensitivity to growth factor deprivation-induced B cell apoptosis.

153 Arginine deprivation-induced cell cycle arrest was mediated in pa

154 amma-secretase protects neurons from glucose deprivation-induced death by regulating miR-212 and PEA1

155 Moreover, GLD4 depletion impairs glucose deprivation-induced GLUT1 up-regulation.

156 ways that render them susceptible to cystine deprivation-induced necrosis.

157 the cortex, BC1 RNA is required for sensory deprivation-induced structural plasticity of dendritic s

158 Oxygen deprivation induces a range of cellular adaptive respons

159 Preclinical studies show that arginine deprivation is synthetically lethal in argininosuccinate

160 ocular dominance to regulation by monocular deprivation is the canonical model of plasticity confine

161 Nutrient deprivation is therefore likely to exacerbate environmen

162 out salt when under a novel state of sodium deprivation, is a classic example of how homeostatic sys

163 The resulting sensory deprivation jeopardizes auditory cortex (AC) maturation.

164 inic: electroconvulsive shock therapy, sleep deprivation, ketamine, scopolamine, GLYX-13 and pindolol

165 The induction of autophagy by nutrient deprivation leads to a rapid increase in the formation o

166 the spatial pattern of apoptosis and sensory deprivation leads to exacerbated amounts of apoptotic ne

167 thors make a compelling case that early-life deprivation leads to present orientation, we believe tha

168 uisition of resistance to long-term-estrogen-deprivation (LTED) and subsequent resistance to fulvestr

169 Correspondingly, androgen deprivation markedly attenuates the frequency and severi

170 ome frequency bands over the course of sleep deprivation may falsely indicate LRTC changes that do no

171 se results suggest that new inputs following deprivation may not maintain the precise spatial relatio

172 postmenopausal women suggests that estrogen deprivation may play a facilitating role, probably media

173 Monocular deprivation (MD) during the visual critical period cause

174 Monocular deprivation (MD) imposed early in postnatal life elicits

175 Brief monocular deprivation (MD) shifts ocular dominance and reduces the

176 We found that monocular deprivation (MD), but not binocular deprivation (BD), in

177 activity during the first 48 h of monocular deprivation (MD), we show that PNN removal resets the ne

178 cing adverse conditions such as nitrogen (N) deprivation, microalgae enter cellular quiescence, a rev

179 and highlights novel pathways through which deprivation might confer risk for internalizing and exte

180 After 2 days of arginine deprivation, mTORC1 activity declined paralleling a sele

181 Cells respond to deprivation of certain nutrients such as glucose or nitr

182 ies, tumor cells experience acute or chronic deprivation of nutrients and oxygen and induce tumor vas

183 We found that a 1-h deprivation of the main energetic nutrients is an approp

184 We conclude that beside a role in sugar deprivation of the pathogen by competing for sugar avail

185 ebral ischemia results in oxygen and glucose deprivation (OGD) and consequent delayed cell death of v

186 Brain ischemia causes oxygen and glucose deprivation (OGD) in neurons, triggering a cascade of ev

187 es of sleep duration, and experimental sleep deprivation on genomic, cellular, and systemic markers o

188 ere, we evaluated the impact of early visual deprivation on the perception of simultaneity for audiov

189 in cognitive performance during acute sleep deprivation (one prolonged wake episode), chronic sleep

190 Androgen deprivation or AR inhibition significantly increased CXC

191 As a result, glutamine deprivation or glutaminase inhibitor treatment triggers

192 In cell culture, glutamine deprivation or inhibition of glutaminase prevents EC pro

193 Here, we show in mice that food deprivation or optogenetic activation of AgRP neurons in

194 More gradual glucose starvation, amino acid deprivation or rapamycin did not trigger micro-lipophagy

195 onse to tunicamycin-induced ER stress, serum deprivation or reduced levels of mitofusin 2 (MFN2).

196 ; 95%CI 5.85-7.07), and higher socioeconomic deprivation (OR 2.90; 95%CI 2.72-3.09 for highest vs. lo

197 ation was tested by comparing change in area deprivation over time.

198 itions within tumor masses, such as nutrient deprivation, oxygen limitation, high metabolic demand, a

199 H3K4me3 to examine effects of early maternal deprivation (peer-rearing, PR) in archived rhesus macaqu

200 studies have demonstrated that early visual deprivation prevents the automatic remapping of touch in

201 Water deprivation produces a drive to seek and consume water.

202 th both measures that LRTCs decline as sleep deprivation progresses.

203 , ethnicity, age at death, index of multiple deprivation quintile, year of death, liver disease causi

204 Prostate cancer risk grouping, androgen deprivation, race, age-adjusted CCI, L5HU, and PsoasL4-5

205 , the article reviews evidence on compounded deprivation, racial cleavages, civic engagement, institu

206 nctions counteracting neutrophil-induced ion deprivation, radical stress, and nutritional restraints.

207 be affected by the variation in the level of deprivation, rather than the average level of deprivatio

208 and rodents it was found that, during sleep deprivation, regional 'sleep-like' slow and theta (slow/

209 Here, we show that glucose deprivation regulates the ART protein Csr2/Art8 at multi

210 odal plasticity in the case of early sensory deprivation relates to the original functional specializ

211 Sensory deprivation reorganizes neurocircuits in the human brain

212 We conclude that the recovery from N deprivation represents not simply a reversal of processe

213 ed on census tracts (family income, poverty, deprivation, residential stability, and percent white, b

214 Sleep deprivation resulted in a higher A1AR availability in th

215 and IKZF1 enforce a state of chronic energy deprivation, resulting in constitutive activation of the

216 Sleep deprivation results in a sleep rebound.

217 We demonstrate here that glucose deprivation results in AMP-activated protein kinase (AMP

218 r 6-8 h of sleep, spontaneous wake, or sleep deprivation (SD) and after chronic ( approximately 5 d)

219 Acute sleep deprivation (SD) can trigger or exacerbate psychosis- an

220 Yet the effect of sleep deprivation (SD) on decision making and performance is o

221 strategies, low-dose ketamine (KT) and sleep deprivation (SD) therapies, dramatically reduce depressi

222 hereas Hcrt(ko/ko) mice respond to 6-h sleep deprivation (SD) with a slow-wave sleep (SWS) EEG delta

223 Although different studies associated sleep deprivation (SD) with systemic inflammatory changes, the

224 ring undisturbed 24 h, and after a 6-h sleep deprivation (SD).

225 Sex, age, ethnicity, marital status, social deprivation, severity of psychopathology, duration of in

226 ontralateral eye input and contralateral eye deprivation shifts mouse V1 neurons toward more balanced

227 Sensory deprivation shows competitive interactions between the i

228 g process is completely blocked by monocular deprivation spanning the entire critical period.

229 idopsis thaliana) experiences a local energy deprivation state and confirm previous findings that the

230 dition, we previously showed that asparagine deprivation such as that mediated by l-asparaginase II o

231 expression is observed upon prolonged serum deprivation, supporting the concept that ST6Gal-I confer

232 p53 proteins are more resistant to glutamine deprivation than cells with wild-type p53.

233 is unresolved whether the permanent auditory deprivation that deaf people experience leads to the enh

234 d specific transcriptional responses to K(+) deprivation that seem to temper these negative effects.

235 in a higher mammal model of complete sensory deprivation, the congenitally deaf cat.

236 Under nitrogen deprivation, the green alga C. reinhardtii showed substa

237 and its activity can be blocked by androgen-deprivation therapies (ADTs).

238 er cells respond heterogeneously to androgen deprivation therapies and reveals characteristics of sub

239 Concordance was greatest for androgen deprivation therapy (ADT) (86.0%, n = 308) alone or comb

240 of evidence supports a link between androgen deprivation therapy (ADT) and cognitive dysfunction, inc

241 Twenty-four patients received androgen-deprivation therapy (ADT) and were excluded for outcomes

242 ated with postoperative response to androgen deprivation therapy (ADT) in a subset analysis in our re

243 key driver of prostate cancer, and androgen-deprivation therapy (ADT) is a standard treatment for pa

244 drogen receptor (AR) signaling, and androgen deprivation therapy (ADT) is the accepted treatment for

245 randomized clinical trial comparing androgen-deprivation therapy (ADT) plus docetaxel with ADT alone

246 djuvant therapy in combination with androgen deprivation therapy (ADT) to prevent androgen-independen

247 were 1.19 (95% CI, 1.05-1.34) after androgen deprivation therapy (ADT) vs no ADT and 1.21 (95% CI, 1.

248 studies have associated the use of androgen deprivation therapy (ADT) with an increased risk of deme

249 the 118 patients, 45 were receiving androgen-deprivation therapy (ADT) within at least 6 mo before th

250 who have a poor response to initial androgen-deprivation therapy (ADT), as reflected by a prostate-sp

251 mbination in men starting long-term androgen-deprivation therapy (ADT), using a multigroup, multistag

252 The primary end point was androgen deprivation therapy (ADT)-free survival.

253 r who were initiating or continuing androgen deprivation therapy (ADT).

254 ulated in prostate cancer following androgen-deprivation therapy (ADT).

255 efore and 4 wk after treatment with androgen deprivation therapy (ADT).

256 rs to extend survival compared with androgen deprivation therapy alone.

257 cterized by abbreviated response to androgen-deprivation therapy and in approximately 30% of castrati

258 n 0.5 ng/mL following radiation and androgen deprivation therapy appears to identify men prior to PSA

259 ) differs between those who receive androgen-deprivation therapy by surgical castration and those who

260 vanced prostate cancer treated with androgen deprivation therapy experience relapse with relentless p

261 reasing stroke risk include medical androgen deprivation therapy for ischemic and any stroke and erec

262 Androgen-deprivation therapy has been identified to induce oxidat

263 PARP inhibitors in combination with androgen-deprivation therapy upfront in advanced or high-risk pro

264 g the impact on survival of salvage androgen deprivation therapy with or without agents shown to prol

265 ion therapy followed by 6 months of androgen deprivation therapy, and followed for a median 16.62 yea

266 rostatectomy, radical radiotherapy, androgen deprivation therapy, and watchful waiting).

267 prostate cancer receiving EBRT and androgen-deprivation therapy, brachytherapy boost (LDR or HDR) sh

268 Despite resistance to initial androgen deprivation therapy, most men respond to second-line hor

269 can improve survival compared with androgen deprivation therapy.

270 selection as dominant clones after androgen deprivation therapy.

271 herapy or radiation and 6 months of androgen deprivation therapy.

272 e poised for clonal selection after androgen-deprivation therapy.

273 ties within 1.5 years of initiating androgen-deprivation therapy.

274 owever, we have previously shown that visual deprivation through dark exposure (DE) reactivates criti

275 tivate p53-target gene CDKN1A upon glutamine deprivation, thus triggering cell cycle arrest and promo

276 associations provide some tolerance to K(+) deprivation to host plants, revealed that AM symbiosis m

277 Moreover, Gln3 inactivation links nutrient deprivation to increased mutagenesis.

278 d that is required for amino acid or glucose deprivation to inhibit mTORC1 in cultured human cells.

279 significance of macrophage-mediated arginine deprivation to T cells.

280 nd protects cells from death caused by serum deprivation, toxicity of xenobiotics or high concentrati

281 Using a slow-onset water deprivation treatment in Arabidopsis (Arabidopsis thalia

282 This screen revealed that cystine deprivation triggered rapid programmed necrosis, but not

283 and mechanistic basis to explain how cystine deprivation triggers necrosis by activating pre-existing

284 with repeated exposures to acute total sleep deprivation (TSD) within a short-time interval (weeks).

285 cial class, household income, and area-based deprivation) using Cox proportional hazards models.

286 Recovery from sleep deprivation was associated with a decrease in A1AR avail

287 sion of cognitive impairment, extended early deprivation was associated with long-term deleterious ef

288 Social deprivation was negatively associated with some outcomes

289 ch we call "the behavioural constellation of deprivation." We propose that the relatively limited con

290 conomic variables, such as smoking, diet and deprivation which are potentially modifiable.

291 female mice before and after a 7 d monocular deprivation, which allowed us to examine both the depres

292 sters during Ramadan shows that self-imposed deprivation, which carries no implications regarding the

293 tress markers, and cell death during glucose deprivation, which could be rescued by inhibition of rRN

294 me the detrimental effects of early maternal deprivation, which could have translational implications

295 hanges in AMP and G6P levels during nutrient deprivation, which provides insights into how a PYK OR g

296 nsity EEG after normal sleep and after sleep deprivation while participants observed a Necker cube im

297 itive cancer cells could not survive glucose deprivation, while the chemoresistant cells displayed ad

298 In this trial, arginine deprivation with ADI-PEG20 improved PFS in patients with

299 In contrast, visual deprivation with binocular lid suturing resulted in incr

300 ia in the visual cortex respond to monocular deprivation with increased lysosome content, but signali