ライフサイエンスコーパス: dermal

1 n is preserved), as well as craniofacial and dermal abnormalities and the absence of a corpus callosu

2 Dermal absorption of BPA from thermal paper receipts occ

3 Compared to dietary BPA exposure, dermal absorption of BPA leads to prolonged exposure and

4 Dermal absorption of PAHs measured using pig skin was di

5 lected due to the small quantity and limited dermal absorption rate.

6 For both the oral bioavailability and dermal absorption studies, the aggregate data do not pro

7 s (PFRs) via inhalation, dust ingestion, and dermal absorption using different sampling and assessmen

8 to PFRs via inhalation, dust ingestion, and dermal absorption was conducted with individual personal

9 0days in vitro while significantly enhancing dermal accumulation of the ATRA in explant pig skin.

10 E16.5 and P2 resulted in a reduction in the dermal adipocyte layer with a corresponding increase in

11 Depletion of Shh from HF-TACs abrogates both dermal adipogenesis and hair follicle growth.

12 ACs) play an essential role in orchestrating dermal adipogenesis through secreting Sonic Hedgehog (SH

13 hat overexpression of localized Wnt converts dermal adipose cells into a distinct fibroblast subtype,

14 The dermal adipose layer expands concomitantly with hair fol

15 and maintenance of adipocyte progenitors in dermal adipose tissues.

16 One participant repeated the dermal administration with extended monitoring of urine

17 he NLRP3 inflammasome in the pathogenesis of dermal and airway inflammation, and they highlight the u

18 To compare the pharmacokinetics of dermal and dietary BPA exposure, six male participants h

19 Dermal and inhalation exposure levels were assessed by c

20 R levels in treated textiles and measures of dermal and inhalation exposure.

21 luding prothoracic, metathoracic, abdominal, dermal, and anal glands, are revealing unforeseen trophi

22 cipants provided information on respiratory, dermal, and eye irritation health.

23 to the dispersants and adverse respiratory, dermal, and eye irritation symptoms.

24 aturally heal with scar, as characterized by dermal appendage restoration and organized collagen arch

25 thyreophoran dinosaurs' possessing extensive dermal armor, some of the most extreme examples of anti-

26 hyperplasia and is functionally required for dermal ASC proliferation.

27 pogenic differentiation of ADSCs, leading to dermal augmentation.

28 g vessel contraction frequency and decreased dermal backflow.

29 w, effusions in more than 1 compartment, and dermal backflow.

30 METHODS AND Primary endothelial cells from dermal blood and lymphatic vessels (blood vascular endot

31 CGRP-containing nerves innervate dermal blood vessels and lymph nodes.

32 dochondral skeletal elements and exoskeletal dermal bones.

33 vity across the entire skin reveals a latent dermal capacity to undergo spatially patterned self-orga

34 al Col5a2 knockdown results in pathognomonic dermal cauliflower-contoured collagen fibril aggregates,

35 as skin hyperextensibility at low strain and dermal cauliflower-contoured collagen fibril aggregates,

36 epidermal gammadelta T cells, and increased dermal CD4(+) T cells.

37 Concurrently, this dermal cell aggregation triggers the mechanosensitive ac

38 affecting the differentiation of pluripotent dermal cells.

39 uced the severity of skin disease as well as dermal cellularity.

40 erapeutic modality and following irradiation dermal changes, including fibrosis and atrophy, may lead

41 actor-beta (TGF-beta) signalling mediate the dermal changes.

42 ive hair loss, and excessive accumulation of dermal cholesterol, triglycerides, and ceramides.

43 onfirmed by apple-green birefringence within dermal collagen, sweat glands, and arrector pili that en

44 ngs demonstrated objective thickening of the dermal collagen.

45 l for the Hh pathway, was impaired in mutant dermal condensate cells, suggesting that Gorab may be re

46 uppressed hedgehog (Hh) signaling pathway in dermal condensates in vivo.

47 e contribution of inhalation, ingestion, and dermal contact pathways to these risks.

48 a are Langerhans cells (LC) and interstitial dermal DC (iDDC).

49 ersely, KLF4 is maintained or induced during dermal DC and monocyte-derived dendritic cell/inflammato

50 LC and dermal DC subsets often show functional redundancy, but

51 Unlike skin-resident dermal DCs (dDCs)/interstitial-type DCs and inflammatory

52 at not only delayed lymphatic trafficking of dermal DCs but also blunted their capacity to prime CD8(

53 y ELISA; the percentage of migratory CD1a(+) dermal DCs was significantly decreased in the DSS patien

54 CD11b(+)CD4(+) splenic DCs, but not CD11b(+) dermal DCs, were reduced, indicating cDC2s in the lung a

55 otential of coated microneedles for improved dermal delivery of 5-aminolevulinic acid (5-ALA), which

56 Dermal dendritic cells and epidermal Langerhans cells ar

57 ection of Langerhans cells, macrophages, and dermal dendritic cells by 75-90% and reduced the overall

58 ss from skin and enables the accumulation of dermal dendritic cells in skin-draining LN.

59 fection of Langerhans cells and interstitial dermal dendritic cells results in an impaired ability to

60 A to the skin can target distinct subsets of dermal dendritic cells to confer a superior immune respo

61 ection of Langerhans cells, macrophages, and dermal dendritic cells, and these cells emigrated from s

62 ls, including Langerhans cells, macrophages, dermal dendritic cells, mast cells, fibroblasts, and lym

63 yte-derived dendritic cells and interstitial dermal dendritic cells, yet the virus fully replicates o

64 ic cells, Langerhans cells, and interstitial dermal dendritic cells.

65 mal injection by LECs was similar to that of dermal dendritic cells.

66 with teeth, the principal skeletal tissue of dermal denticles is dentine.

67 in fact, give rise to odontoblasts of trunk dermal denticles.

68 ACKR4-mediated scavenging of dermal-derived CCL19, rather than CCL21, is critical dur

69 ords tuneability of properties necessary for dermal drug delivery including octanol-water partitionin

70 rmis and provide an explanation for how such dermal dysbiosis results in increased inflammatory cytok

71 presence of 3 or more atypical cells at the dermal-epidermal junction (DEJ) by RCM correlated with h

72 that the basement membrane components at the dermal-epidermal junction are subject to ongoing remodel

73 infection and to ganglia and persist at the dermal-epidermal junction for up to 12 weeks after lesio

74 cell culture media and also localized to the dermal-epidermal junction in organotypic skin culture.

75 es, eosinophils induced separation along the dermal-epidermal junction of ex vivo skin.

76 asts also deposited type VII collagen at the dermal-epidermal junction of human RDEB skin xenografts

77 ion induced type VII collagen and AFs at the dermal-epidermal junction of treatment sites.

78 en VII, our study challenges the view of the dermal-epidermal junction zone as a static structure wit

79 The dermal-epidermal junction zone is believed to be a rathe

80 ential for anchoring fibril formation at the dermal-epidermal junction.

81 stained delivery of type VII collagen at the dermal-epidermal junction.

82 and regeneration of anchoring fibrils at the dermal-epidermal junction.

83 formation and to define conditions inducing dermal-epidermal separation (DES).

84 Dermal-epidermal separation by IL-5-activated eosinophil

85 Dermal-epidermal separation was assessed by light micros

86 Dermal-epidermal separation was significantly reduced by

87 Topical gentamicin corrected dermal-epidermal separation, improved wound closure, and

88 oportions of free BPA-d16 in urine following dermal exposure (0.71%-8.3% of total BPA-d16) were gener

89 The participant repeating the dermal exposure had detectable BPA-d16 in urine for 9 da

90 ts occurs but BPA pharmacokinetics following dermal exposure is not understood.

91 The median dermal exposure on hand wipes was 0.32 ng.kg bw(-1).day(

92 These results demonstrate that dermal exposure to FRs occurs from handling camping tent

93 After dermal exposure, cumulative excretion increased linearly

94 thworm tissue was possible after only 4 h of dermal exposure, when the uptake of (68)Zn-E had increas

95 The results also have implications for dermal exposure.

96 is of anti-p200 confirmed by immunoblot with dermal extract.

97 treatment with SR1001 reduces epidermal and dermal features of MC903-induced atopic dermatitis-like

98 rix production, cross-linking, thickening of dermal fibrils, and tissue stiffening.

99 ice with inducible deletion of MMP-14 in the dermal fibroblast (MMP-14(Sf-/-)).

100 In addition, P311 induced dermal fibroblast activation and proliferation.

101 cytotoxic effects on SHSY5Y, MRC5, and human dermal fibroblast cells compared with the dissolved PhIP

102 ed in vitro studies on the response of human dermal fibroblast cells toward pristine titania nanotube

103 is variability, we established primary human dermal fibroblast cultures from several ODDD patients an

104 tained from a patient biopsy by reprograming dermal fibroblasts (DF), hiPSc present the same properti

105 human embryonic stem cells (hESCs) and human dermal fibroblasts (hDFs) derived hiPSCs.

106 chymal stem cells (hMSCs) and human neonatal dermal fibroblasts (hNDFs) within a customized extracell

107 Furthermore, normal human dermal fibroblasts (primary cells) are also seeded on th

108 sing cell death also in normal cells such as dermal fibroblasts and endometrial mesenchymal stem cell

109 rm of IL-36Ra was confirmed in human primary dermal fibroblasts and keratinocytes and in skin equival

110 al promoters of TGFbeta signaling in primary dermal fibroblasts and of bleomycin-induced fibrosis in

111 13 in mediating the induction of collagen in dermal fibroblasts and that blockade with IL-13 antibodi

112 liferative phase of cutaneous wound healing, dermal fibroblasts are recruited into the clotted wound

113 dary effect of SAg-stimulated PBMCs on human dermal fibroblasts as judged by C/EBP delta expression.

114 ure and ex vivo approaches to identify human dermal fibroblasts as natural host cells that support pr

115 anced uptake of apoptotic PMN (51%) by human dermal fibroblasts at concentrations as low as 0.1 nM.

116 d after the dedifferentiation of human adult dermal fibroblasts by overexpression of pluripotency tra

117 Since dermal fibroblasts can have profound impacts on melanoma

118 t, P4HA1 protein level and C-P4H activity in dermal fibroblasts compared to age-matched control sampl

119 , we examined whether age-related changes in dermal fibroblasts could drive melanoma metastasis and r

120 Blimp1 ablation in E12.5 mouse dermal fibroblasts delayed HF morphogenesis and growth a

121 oupled to the surrounding matrix for primary dermal fibroblasts embedded in a 3D fibrin matrix.

122 , etc.), in primary cultures of normal human dermal fibroblasts exposed to visible and near infra-red

123 sent a long-term cell culture model of human dermal fibroblasts expressing fluorescence-labelled huma

124 We demonstrate that normal dermal fibroblasts expressing high PEDF levels attenuate

125 hese results highlight a central function of dermal fibroblasts for skin protection, opening new poss

126 re, we demonstrate unexpectedly that primary dermal fibroblasts from pre-symptomatic mutation carrier

127 Primary dermal fibroblasts from R258C patients exhibited increas

128 Dermal fibroblasts from two CMT1A pedigrees with confirm

129 Dermal fibroblasts from two unrelated patients harboring

130 00A12 by nearly 70%, which in turn activated dermal fibroblasts in vitro.

131 for achieving transdifferentiation of human dermal fibroblasts into induced cardiomyocyte-like cells

132 te and sebocyte differentiation, its role in dermal fibroblasts is unclear.

133 nerated induced pluripotent stem cells using dermal fibroblasts obtained from patients with TSC.

134 contrast, IL-1alpha-dependent stimulation of dermal fibroblasts optimally stimulates epidermal stem c

135 In vitro, primary dermal fibroblasts readily express podoplanin in respons

136 mulation of D1 dopamine receptors present in dermal fibroblasts restores vascular endothelial growth

137 In vitro studies using murine and human dermal fibroblasts showed that P311 stimulated TGF-beta1

138 tic endothelial cells (LECs) cocultured with dermal fibroblasts spontaneously organize into a stable

139 in all cancer cell lines and normal primary dermal fibroblasts studied.

140 transforming growth factor-beta signaling in dermal fibroblasts through the down-regulation of thromb

141 ctin expression and secretion and stimulated dermal fibroblasts to express EDA-fibronectin.

142 e describe the transdifferentiation of human dermal fibroblasts towards the cardiac cell lineage via

143 mal stability of collagen in patient-derived dermal fibroblasts versus age-matched control samples.

144 mmortalized lines of control and R258C human dermal fibroblasts were established and SM alpha-actin e

145 Isolated dermal fibroblasts were incubated with recombinant IL-13

146 ion of cell-derived matrices (CDMs) by human dermal fibroblasts with stable knockdown of COL6A1 revea

147 r, Fn14, is upregulated in keratinocytes and dermal fibroblasts, and TWEAK induces these cytokines an

148 We obtained similar findings in dermal fibroblasts, demonstrating that the IFN-gamma/TNF

149 nd in vitro matrix fiber assembly by primary dermal fibroblasts, EMILIN-1 and -2 are deposited on and

150 We used dermal fibroblasts, from patients with retinal pathology

151 inhibitory factor (MIF), more strongly than dermal fibroblasts, thereby creating a MIF gradient in s

152 lasts or selectively targeting Dlk1(+) lower dermal fibroblasts, we found that beta-catenin stabiliza

153 drives two waves of gene expression in human dermal fibroblasts.

154 EDA-dependent fibro-inflammatory response in dermal fibroblasts.

155 l motility in serum-stimulated primary mouse dermal fibroblasts.

156 and gelatine, was performed by seeding human dermal fibroblasts.

157 nduces collagen production by normal and SSc dermal fibroblasts.

158 growth factor-beta, at least in the case of dermal fibroblasts.

159 d VZV peptides, as well as kill VZV-infected dermal fibroblasts.

160 ct of targeted beta-catenin stabilization in dermal fibroblasts.

161 c function in new matrix deposition by human dermal fibroblasts.

162 in different concentrations on primary human dermal fibroblasts.

163 AP in mediating the profibrotic responses in dermal fibroblasts.

164 that dermal invasion is directly impeded by dermal fibroblasts.

165 ocyte layer with a corresponding increase in dermal fibrosis and an altered hair cycle.

166 Glycyrrhizin significantly ameliorated dermal fibrosis in bleomycin-treated mice, which was par

167 t glycyrrhizin ameliorates bleomycin-induced dermal fibrosis through the inhibition of fibroblast act

168 Contrast treatment led to dermal fibrosis, and this was exacerbated in recipients

169 ther DMF is effective as a treatment for SSc dermal fibrosis.

170 pports the use of DMF as a treatment for SSc dermal fibrosis.

171 e lacking adiponectin mounted an exaggerated dermal fibrotic response, while transgenic mice with con

172 nt spine and tendon, aspiration biopsies and dermal fillers (DF).

173 atory in the short term but may also promote dermal, heart, liver, and lung fibrosis with repetitive

174 ing in adult skin and largely contributes to dermal homeostasis underlying its pathogenic role in fib

175 Dermal hyperneury and multiple sclerotic fibromas should

176 ion, with 2 of the relatives presenting with dermal hyperneury, cutaneous lesions classically describ

177 urfacing (AFR) within treated areas of focal dermal hypoplasia (FDH).

178 logy of skin, including the establishment of dermal identity.

179 ersus-host disease skin and markedly reduced dermal IFN-alpha levels.

180 d by enhanced neutrophil accumulation at the dermal infection site.

181 The dermal infiltrate from the 33 study patients (20 female;

182 The dermal infiltrate of cutaneous lesions of histiocytoid S

183 In some cases, cytogenetic studies of the dermal infiltrate were also performed.

184 eous inflammation characterized by erythema, dermal infiltrates of CD45(+) leukocytes, and a local pr

185 n in epidermal hyperplasia (Ki67) and in the dermal infiltration of inflammatory cytokines (IL36alpha

186 elevated ear thickening, mono/MPhi-dominated dermal inflammation, and increased iNOS and IL-6 express

187 or IFNAR1, we observed a marked reduction in dermal inflammation, vasculopathy, and fibrosis compared

188 ciated with eruptive KAs with characteristic dermal inflammation, which improved with corticosteroid

189 ll populations and C. albicans revealed that dermal invasion is directly impeded by dermal fibroblast

190 es significantly increased the epidermal and dermal layer of the healed wound, as compared to the oth

191 1 permitted extension of VACV infection into dermal layers of the skin.

192 with maculopapular or nodular post-kala-azar dermal leishmaniasis (PKDL).

193 rom healthy, healed VL (HVL), post kala-azar dermal leishmaniasis(PKDL) and VL subjects.

194 ka presented to an urgent care clinic with a dermal lesion consistent with poxvirus infection.

195 These data implicate dermal liberation of specific chemokines in the recruitm

196 Here we studied PD-L1 expression in human dermal lymphatic endothelial cells (HDLECs), which play

197 levance of this interaction in primary human dermal lymphatic endothelial cells (HDLECs).

198 astructural features of native in vivo human dermal lymphatic microvasculature and is stable over man

199 and immunity using a mouse model that lacks dermal lymphatic vessels (K14-VEGFR3-Ig mice).

200 CCR7, which directs egress from the skin via dermal lymphatic vessels and extravasation into the LN p

201 We conclude that embryonic-derived, MR(hi) dermal macrophages are permissive for parasite growth ev

202 etion of MR or selective depletion of MR(hi) dermal macrophages by anti-CSF-1 receptor antibody rever

203 The origin and functional specialization of dermal macrophages in cutaneous infections have been lit

204 s) in vitro and by mannose receptor (MR)(hi) dermal macrophages in vivo compared with a healing strai

205 cell-driven inflammatory states, the MR(hi) dermal macrophages showed M2 characteristics.

206 The dermal macrophages were radio resistant and not replaced

207 istry showed CXCL9 co-localized with CD68(+) dermal macrophages.

208 Dermal mast cell migration from the skin to the draining

209 ve IgG autoantibodies to FcepsilonRIalpha on dermal mast cells and basophils, which on activation rel

210 that type VI collagen is a key regulator of dermal matrix assembly, composition, and fibroblast beha

211 cytes, which triggers the differentiation of dermal MCs.

212 Here, we discover that extensive dermal melanocytosis and phakomatosis pigmentovascularis

213 hakomatosis pigmentovascularis and extensive dermal melanocytosis are therefore diagnoses in the grou

214 Mongolian blue spots (dermal melanocytosis) are usually localized and transien

215 Gorab-deficient dermal mesenchymal cells also displayed a significantly

216 mis and prominent clustering of the adjacent dermal mesenchymal cells.

217 at myosin II contractility drives the smooth dermal mesenchyme into a pattern of surface bumps that t

218 A non-cross-linked porcine acellular dermal mesh was sutured to the pelvic floor remnants in

219 Human dermal microvascular cells exposed to conditioned medium

220 Human dermal microvascular ECs (HDMECs) treated with TLR3 [Pol

221 Exposure of primary murine dermal microvascular ECs (pDMECs) to CGRP followed by co

222 trical resistance compared with normal human dermal microvascular ECs.

223 Dermal microvascular endothelial cells (ECs) isolated fr

224 a-associated herpesvirus (KSHV) enters human dermal microvascular endothelial cells (HMVEC-d), its na

225 nrelated functions of ICAM-1 in cerebral and dermal microvascular endothelial cells (MVECs).

226 is the major entry pathway of KSHV in human dermal microvascular endothelial cells, the natural targ

227 ne Colo320 (high Nrp-2 expression) and human dermal microvascular lymphatic endothelial cells (LECs).

228 Functional assessment of gated individual dermal microvessels is therefore of outstanding interest

229 rve as the key progenitors of intestinal and dermal MPhis.

230 rrow-derived MPhis and microglia, but not in dermal MPhis.

231 Lip scaffolds were cultivated with dermal, muscle progenitor and endothelial cells, either

232 dent expression of TNF-alpha in cerebral and dermal MVECs, and CXCL8, CCL3, CCL4, VCAM-1, and cycloox

233 ERK was additionally required for TEM across dermal MVECs.

234 Dermal nest and combined melanoma were associated with t

235 classification: dendritic cell, round cell, dermal nest, combined, and nonclassifiable types.

236 dendritic cell (40%), 21 round cell (37%), 2 dermal nests (4%), 2 combined (4%), and 9 nonclassifiabl

237 atures, such as junctional thickening, dense dermal nests, and nucleated cells within papillary dermi

238 mulated radiation on the potential source of dermal NO, the effective doses and wavelengths, the resp

239 branching tubules in three-dimensional human dermal organoid ex vivo culture.

240 he close reciprocal relationship between the dermal papilla and adjacent HF epithelial cells.

241 Dermal papilla cells (DPCs) located in the hair bulb are

242 show that Blimp1 is dynamically regulated in dermal papilla cells during hair follicle (HF) morphogen

243 mary DPCs in hair-related studies often lack dermal papilla characteristics.

244 e functional role of Blimp1 in promoting the dermal papilla inductive signaling cascade that initiate

245 limp1 is both a target and a mediator of key dermal papilla inductive signaling pathways including tr

246 d et al show that JAK/STAT5 signaling in the dermal papilla is required for anagen onset in the murin

247 nd the epithelial stem cells that respond to dermal papilla signaling.

248 oth BAB and BAN retained high proportions of dermal papilla signature gene and versican protein expre

249 At the core is the dermal papilla, the organizing center, and the epithelia

250 leads to endogenous EDN3 upregulation in the dermal papilla, the secondary hair germ cells, and the e

251 unravel new molecular landscapes within the dermal papilla.

252 talized DPCs have high resemblance to intact dermal papilla.

253 ected anchors (37 of 44 images [84.1%]) were dermal papillae.

254 By contrast, polarized mitochondria in dermal papillar fibroblasts produced minimal ROS.

255 Mincle-deficient mice had milder dermal pathology and a tenth of the parasite burden comp

256 study, we describe a novel subset of murine dermal perivascular macrophages that extend protrusions

257 In vivo dermal pharmacokinetics showed that delivery of just 350

258 gerstatte (Teruel, Spain) [9, 10], preserves dermal pigment cells (chromatophores)-xanthophores, irid

259 xceptionally preserved fossils [16, 17], and dermal pigment cells generate coloration in numerous rep

260 with SS reveal that the basement membrane of dermal postcapillary venules undergoes changes in struct

261 The key initiators of heterogeneity are dermal progenitors, which spontaneously aggregate throug

262 monitoring in the interstitial fluid in the dermal region, in contrast to larger state-of-the-art sy

263 growth factor (EGF) is a critical element in dermal repair, but EGF-containing wound dressings have n

264 genes involved in epidermal homeostasis and dermal repair.

265 these pathways prevents beta-catenin-induced dermal reprogramming and EF formation.

266 ed MMP9 expression in vivo in full thickness dermal scalp wounds created in experimental K14.Cre (+)

267 100A12 is a potential therapeutic target for dermal scarring.

268 okines from the epidermis is associated with dermal scarring.

269 Dermal sclerosis and adnexal atrophy are additional feat

270 lcium deposits in the muzzle skin containing dermal sheath of vibrissae and in aorta.

271 Patients were selected based on a dermal side binding on 1-mol/L salt (sodium chloride)-sp

272 g., sharks and skates) possess a postcranial dermal skeleton consisting of tooth-like "denticles" emb

273 nd co-localize with And1, a component of the dermal skeleton.

274 dentine throughout the ancestral vertebrate dermal skeleton.

275 e regeneration of both the epidermis and the dermal stroma.

276 cesses that govern the establishment of each dermal subset remain unknown.

277 ntrast, NIR stimulated cells when exposed to dermal tissue oxygen levels (approx. 2%).

278 Acute dermal toxicity was done, and drug diffusion in skin lay

279 This polymer microneedle has no dermal toxicity.

280 of interferon regulatory factor 4-dependent dermal type 2 conventional DC subsets and not by epiderm

281 intake from dust ingestion, inhalation, and dermal uptake from air, and then identified hazard trait

282 nd chemical properties, we hypothesized that dermal uptake from clothing could contribute to the body

283 udies where high dosing levels were used and dermal uptake is dominant.

284 ictions of steady-state models, suggest that dermal uptake of BP-3 from clothing could meaningfully c

285 re, flow status, or morphology of the deeper dermal vessels.

286 s and systemic manifestations in response to dermal vibration, with coincident degranulation of mast

287 ficiency during winter because of negligible dermal vitamin D3 production.

288 lerosis (SSc) is accompanied by attrition of dermal white adipose tissue (dWAT) and reduced levels of

289 sis typical of scleroderma is atrophy of the dermal white adipose tissue (DWAT).

290 Col5a2 knockdown also led to loss of dermal white adipose tissue (WAT) and markedly decreased

291 Paired urine and dermal wipe samples (i.e., pre- and post-event) as well

292 ary PAH metabolite levels, levels of PAHs in dermal wipes and personal air samples, and urinary mutag

293 forming ability in wounds reflects elevated dermal Wnt/beta-catenin activation in the wound bed, inc

294 esis and the postnatal hair cycle, preceding dermal Wnt/beta-catenin activation.

295 polymer would significantly improve impaired dermal wound healing in diabetes.

296 main I and VEGF189 loaded scaffolds promoted dermal wound healing in normal and diabetic rats.

297 nalyzed in vivo for their ability to promote dermal wound healing.

298 ted in a diabetic murine splinted excisional dermal wound model using gross observation, histology, i

299 formation and healing was also observed in a dermal wounding model with deletion of Mapk14.

300 The repair of dermal wounds, particularly in the diabetic population,