ライフサイエンスコーパス: dermal fibroblast

1 AP in mediating the profibrotic responses in dermal fibroblasts.

2 ellular retention of collagen XII in patient dermal fibroblasts.

3 ile biocompatibility was verified with human dermal fibroblasts.

4 eticulum (ER) retention of COL4A2 in primary dermal fibroblasts.

5 ession in gingival fibroblasts compared with dermal fibroblasts.

6 )] mitochondrial DNA (mtDNA) damage in human dermal fibroblasts.

7 y reduced the PDGF-BB-dependent migration in dermal fibroblasts.

8 of the canonical TGFbeta pathway, in primary dermal fibroblasts.

9 ns and other cell types, including patients' dermal fibroblasts.

10 teraction with alphaVbeta3 integrin in human dermal fibroblasts.

11 that dermal invasion is directly impeded by dermal fibroblasts.

12 authentic versican core protein produced by dermal fibroblasts.

13 Decorin was predominantly produced by dermal fibroblasts.

14 human retinal pigmented epithelium cells and dermal fibroblasts.

15 ntial KF apoptosis when compared with normal dermal fibroblasts.

16 ing on keratinocytes and importantly also on dermal fibroblasts.

17 ed the expression of HAS3 and versican V2 in dermal fibroblasts.

18 EDA-dependent fibro-inflammatory response in dermal fibroblasts.

19 OA FLS) but not in nondiseased primary human dermal fibroblasts.

20 stinct gene expression signature from non-DP dermal fibroblasts.

21 t activities when transfected in orbital and dermal fibroblasts.

22 votal role in regulation of the ECM genes in dermal fibroblasts.

23 ients were elevated 2-5-fold above wild-type dermal fibroblasts.

24 pecific sites on DNA is greater than that in dermal fibroblasts.

25 a negative regulator of the ERalpha gene in dermal fibroblasts.

26 O1 and p63 in skin-derived keratinocytes and dermal fibroblasts.

27 how wound electric fields guide migration of dermal fibroblasts.

28 ated the effects of dhS1P and S1P in control dermal fibroblasts.

29 d electric field activated PI3 kinase/Akt in dermal fibroblasts.

30 alyze the IRA-induced transcriptome in human dermal fibroblasts.

31 ctin module, FnIII-1c, on gene expression in dermal fibroblasts.

32 drives two waves of gene expression in human dermal fibroblasts.

33 ytes could induce a profibrotic phenotype in dermal fibroblasts.

34 inocytes is activated by IGF-1 secreted from dermal fibroblasts.

35 e phosphorylation and acetylation of Fli1 in dermal fibroblasts.

36 uced a profibrotic phenotype in normal human dermal fibroblasts.

37 ake in both established cell lines and human dermal fibroblasts.

38 in human primary epidermal keratinocytes and dermal fibroblasts.

39 -response protein heme oxygenase 1 (HO-1) in dermal fibroblasts.

40 led to an attenuation of Smad3 signaling in dermal fibroblasts.

41 l motility in serum-stimulated primary mouse dermal fibroblasts.

42 and gelatine, was performed by seeding human dermal fibroblasts.

43 nduces collagen production by normal and SSc dermal fibroblasts.

44 growth factor-beta, at least in the case of dermal fibroblasts.

45 d VZV peptides, as well as kill VZV-infected dermal fibroblasts.

46 ct of targeted beta-catenin stabilization in dermal fibroblasts.

47 c function in new matrix deposition by human dermal fibroblasts.

48 K, were down-regulated in LTBP4 mutant human dermal fibroblasts.

49 ernate but inefficient receptor for HSV-1 on dermal fibroblasts.

50 in different concentrations on primary human dermal fibroblasts.

51 tion and wound-healing properties of patient dermal fibroblasts.

52 mparable entry pathways in keratinocytes and dermal fibroblasts.

53 es of human skin epidermal keratinocytes and dermal fibroblasts.

54 ll sources by comparing costal chondrocytes, dermal fibroblasts, a mixture of the two, and TMJ disc c

55 Here, we show that in primary normal human dermal fibroblasts, A2AR stimulation with CGS21680 elici

56 In addition, P311 induced dermal fibroblast activation and proliferation.

57 ata suggest that in the Abcc6(-/-) genotype, dermal fibroblasts actively contribute to changes that p

58 investigated the hypothesis that autologous dermal fibroblast (ADF) injection into the AV nodal area

59 Human endothelial cells and dermal fibroblasts adhered to rScl proteins, indicating

60 nd PDGF-BB (KD=200 nM), enhanced adult human dermal fibroblast (AHDF) survival under serum starvation

61 factor BB (PDGF-BB) and promote adult human dermal fibroblast (AHDF) survival under stress.

62 Interestingly, in dermal fibroblasts, although complete inhibition of IRAK

63 elialization, and angiogenesis compared with dermal fibroblast and PBS treated wounds.

64 nditions and exhibit minimal cytotoxicity on dermal fibroblast and PC-12 cells.

65 and proliferation of diabetic mouse primary dermal fibroblasts and 3T3 fibroblasts, which express ve

66 ists of a dermal equivalent containing human dermal fibroblasts and a fully stratified, biologically

67 lyplexes also distributed more broadly among dermal fibroblasts and allowed greater interaction with

68 demonstrate that NRG1 is highly expressed by dermal fibroblasts and cancer-associated fibroblasts (CA

69 sing cell death also in normal cells such as dermal fibroblasts and endometrial mesenchymal stem cell

70 mis, but Ing3A also activated ERK1/2 in skin dermal fibroblasts and endothelial cells.

71 -38 cells and other elastogenic cells, human dermal fibroblasts and fetal bovine chondrocytes.

72 , it does not form elastic fibers with human dermal fibroblasts and forms fewer atypical fibers with

73 Moreover, the patient's dermal fibroblasts and induced pluripotent stem cell (iP

74 pressed in human epidermal keratinocytes and dermal fibroblasts and is regulated via RAR/RXR-mediated

75 rm of IL-36Ra was confirmed in human primary dermal fibroblasts and keratinocytes and in skin equival

76 cal electric fields may regulate motility of dermal fibroblasts and keratinocytes differently, albeit

77 B1 protein levels decline in senescent human dermal fibroblasts and keratinocytes, mediated by reduce

78 pholipids on global gene expression in human dermal fibroblasts and keratinocytes.

79 s exchanged between epidermal keratinocytes, dermal fibroblasts and leukocytic cells.

80 al promoters of TGFbeta signaling in primary dermal fibroblasts and of bleomycin-induced fibrosis in

81 Human neonatal dermal fibroblasts and primary human adult keratinocyte

82 ceptors promotes collagen synthesis by human dermal fibroblasts and that blockade or deletion of this

83 13 in mediating the induction of collagen in dermal fibroblasts and that blockade with IL-13 antibodi

84 talk between PDGFRbeta and TbetaRI occurs in dermal fibroblasts and that CD44 negatively modulates si

85 B and TGFbeta interact physically in primary dermal fibroblasts and that stimulation with PDGF-BB ind

86 omas, normal melanocytes, keratinocytes, and dermal fibroblasts and utilized The Cancer Genome Atlas

87 es in healing skin, including keratinocytes, dermal fibroblasts, and infiltrating macrophages, but th

88 rometry in human keratinocytes, melanocytes, dermal fibroblasts, and melanoma cells.

89 sis in systemic sclerosis (SSc; scleroderma) dermal fibroblasts, and such cells in scleroderma skin l

90 role of Akt in collagen deposition by normal dermal fibroblasts, and to determine the sensitivity of

91 r, Fn14, is upregulated in keratinocytes and dermal fibroblasts, and TWEAK induces these cytokines an

92 o, HEp-2, LLC-MK2, primary human and macaque dermal fibroblasts, and U373 human glioblastoma cells.

93 of fibrillin-1 into microfibrils produced by dermal fibroblasts; and (iii) the requirement of the pro

94 Dermal fibroblasts are a simple, relevant, and much unde

95 do not usually develop in adult skin, adult dermal fibroblasts are competent to contribute to DP dur

96 ist for the four neuroectoderm lineages, and dermal fibroblasts are not progenitors for fin ray osteo

97 liferative phase of cutaneous wound healing, dermal fibroblasts are recruited into the clotted wound

98 r is mediated by the lower lineage and upper dermal fibroblasts are recruited only during re-epitheli

99 Dermal fibroblasts are required for structural integrity

100 and highlight the RalA signaling cascade in dermal fibroblasts as a potential anticancer target.

101 dary effect of SAg-stimulated PBMCs on human dermal fibroblasts as judged by C/EBP delta expression.

102 ure and ex vivo approaches to identify human dermal fibroblasts as natural host cells that support pr

103 ogether, our findings identify primary human dermal fibroblasts as responder cells to IFNlambda.

104 38alpha MAPK is confirmed in Werner syndrome dermal fibroblasts at 1.0 microM concentration by immuno

105 anced uptake of apoptotic PMN (51%) by human dermal fibroblasts at concentrations as low as 0.1 nM.

106 lA executes this tumor-promoting function of dermal fibroblasts, at least in part, by mediating hepat

107 We found a novel human dermal fibroblast attachment and spreading site on tropo

108 , impaired hydrogel formation, and decreased dermal fibroblast attachment compared to wild-type tropo

109 tions that suppression of RalA expression in dermal fibroblasts blocked tumorigenic keratinocytes fro

110 ntry mechanism suggests that HSV-1 can enter dermal fibroblasts both by direct fusion with the plasma

111 e in the regulation of TGFbeta1 signaling in dermal fibroblasts both in vivo and in vitro.

112 F-beta(1), with it inducing proliferation in dermal fibroblasts but an anti-proliferative response in

113 Finally, induction of MMP-1 expression in dermal fibroblasts by CCN1 N-terminal domains resulted i

114 d after the dedifferentiation of human adult dermal fibroblasts by overexpression of pluripotency tra

115 In primary dermal fibroblasts, C/EBPalpha activates a different set

116 Since dermal fibroblasts can have profound impacts on melanoma

117 .0009) and TGF-beta2 (P=0.0001) secretion in dermal fibroblast cell lines from subjects with FMD comp

118 ir effect on UVA-induced senescence in human dermal fibroblast cells (FB) and the associated mechanis

119 cytotoxic effects on SHSY5Y, MRC5, and human dermal fibroblast cells compared with the dissolved PhIP

120 ed in vitro studies on the response of human dermal fibroblast cells toward pristine titania nanotube

121 This suggests that the properties of dermal fibroblasts change during postnatal development.

122 ECM protein production by normal dermal fibroblasts cocultured with SSc CD8+ T cell super

123 t, P4HA1 protein level and C-P4H activity in dermal fibroblasts compared to age-matched control sampl

124 sensitivity to mtDNA damage was observed in dermal fibroblasts compared with keratinocytes at wavele

125 d biochemically superior to the TMJ disc and dermal fibroblast constructs, and their compressive prop

126 , we examined whether age-related changes in dermal fibroblasts could drive melanoma metastasis and r

127 is variability, we established primary human dermal fibroblast cultures from several ODDD patients an

128 transforming growth factor-beta1-stimulated dermal fibroblasts decreased the formation of contractil

129 Blimp1 ablation in E12.5 mouse dermal fibroblasts delayed HF morphogenesis and growth a

130 ell survival assays in normal neonatal human dermal fibroblasts demonstrated that RS-1 promotes a dos

131 We obtained similar findings in dermal fibroblasts, demonstrating that the IFN-gamma/TNF

132 ed in vitro by exposing platelets to TSP2 KO dermal fibroblast (DF)-derived ECM.

133 tained from a patient biopsy by reprograming dermal fibroblasts (DF), hiPSc present the same properti

134 h factor, acting as a mitogen and motogen of dermal fibroblasts (DFs), for skin wound healing.

135 fficiently reprogrammed postnatal cardiac or dermal fibroblasts directly into differentiated cardiomy

136 ols cellular properties of keratinocytes and dermal fibroblasts during early stages of skin developme

137 Here, we describe methods to use dermal fibroblasts easily obtained from an individual hu

138 oupled to the surrounding matrix for primary dermal fibroblasts embedded in a 3D fibrin matrix.

139 nd in vitro matrix fiber assembly by primary dermal fibroblasts, EMILIN-1 and -2 are deposited on and

140 Furthermore, in activated dermal fibroblasts, ERbeta-selective compounds also inhi

141 , etc.), in primary cultures of normal human dermal fibroblasts exposed to visible and near infra-red

142 We report here that dermal fibroblasts express increased levels of collagen-

143 sent a long-term cell culture model of human dermal fibroblasts expressing fluorescence-labelled huma

144 We demonstrate that normal dermal fibroblasts expressing high PEDF levels attenuate

145 ombinant human C7 (rhC7) purified from human dermal fibroblasts (FB-rhC7), we showed previously that

146 Experiment 1) and versus nerve-derived SC or dermal fibroblast (Fibro) transplantation (Experiment 2)

147 CD26 expression was increased in dermal fibroblasts following skin wounding but was downr

148 ts5(-/-);Vcan(hdf/+) mice and isolated their dermal fibroblasts for comparison with dermal fibroblast

149 hese results highlight a central function of dermal fibroblasts for skin protection, opening new poss

150 ion and are epigenetically suppressed in SSc dermal fibroblasts, Friend leukaemia integration 1 (Fli1

151 the generation of human ESCs via SCNT using dermal fibroblasts from 35- and 75-year-old males.

152 Dermal fibroblasts from Adamts5(-/-) mice, which lack a

153 their dermal fibroblasts for comparison with dermal fibroblasts from Adamts5(-/-) mice.

154 Cultured dermal fibroblasts from affected individuals showed acce

155 Cultured dermal fibroblasts from affected individuals showed enha

156 ed human keratinocytes and in whole skin and dermal fibroblasts from Cdkn2a-deficient mice.

157 differentiation into specifically of palmar dermal fibroblasts from Dupuytren's patients in to myofi

158 proteomics to characterize the phenotype of dermal fibroblasts from healthy subjects of various ages

159 Dermal fibroblasts from p110gamma (a PI3 kinase catalyti

160 Primary dermal fibroblasts from patients with diffuse SSc (dSSc)

161 teomic analysis of the secretome of cultured dermal fibroblasts from patients with systemic sclerosis

162 Dermal fibroblasts from patients with various inherited

163 eta) signaling biomarkers in plasma and from dermal fibroblasts from patients with VEDS.

164 re, we demonstrate unexpectedly that primary dermal fibroblasts from pre-symptomatic mutation carrier

165 Primary dermal fibroblasts from R258C patients exhibited increas

166 Cultured dermal fibroblasts from the most severely affected infan

167 Dermal fibroblasts from the Syrian hamster (SHD cells) a

168 Dermal fibroblasts from two CMT1A pedigrees with confirm

169 Dermal fibroblasts from two unrelated patients harboring

170 In contrast, dermal fibroblasts from Vcan haploinsufficient (Vcan(hdf

171 We used dermal fibroblasts, from patients with retinal pathology

172 , beta2AR antagonism increased angiogenesis, dermal fibroblast function, and re-epithelialization, bu

173 Estrogen is an important regulator of dermal fibroblast functions, including extracellular mat

174 enzyme, indoleamine 2,3-dioxygenase (IDO) in dermal fibroblasts generates a tryptophan-deficient envi

175 Compared to dermal fibroblasts, gingival fibroblasts are less respon

176 Here we show that, compared with dermal fibroblasts, gingival fibroblasts show reduced ex

177 In vitro, OPN(-/-) dermal fibroblasts had decreased migratory capacity but

178 s necessary for reprogramming of human adult dermal fibroblasts (hADFs) into undifferentiated induced

179 tion, angiogenesis, and differentiated human dermal fibroblast (HDF ) function contribute to scarring

180 In primary human dermal fibroblasts (HDF) from both photoprotected and ph

181 an skin equivalents (HSE) by comparing human dermal fibroblasts (HDF) incorporated into the three-dim

182 Human dermal fibroblasts (HDF) were also subjected to PMB stre

183 in steroid-resistant BT-549 cells and human dermal fibroblasts (HDF-a) using AP and WB.

184 no-associated virus type 2 (wtAAV2) in human dermal fibroblasts (HDFs) and HeLa cells revealed that j

185 human embryonic stem cells (hESCs) and human dermal fibroblasts (hDFs) derived hiPSCs.

186 ts role in aging, we demonstrated that human dermal fibroblasts (HDFs) from older human subjects were

187 ys both migration and proliferation of human dermal fibroblasts (HDFs) were inhibited by the 5-LO pha

188 Treatment of human dermal fibroblasts (HDFs) with combinations of cell-perm

189 enase type 1 (11beta-HSD1) in cultured human dermal fibroblasts (HDFs).

190 iPSCs), as well as in control parental human dermal fibroblasts (HDFs).

191 (non-contact) cocultures with primary human dermal fibroblasts (hDFs).

192 s, and spreads rapidly in confluent cultured dermal fibroblasts (HFFs).

193 tumor-promoting epigenetic modifications in dermal fibroblasts, highlighting further the importance

194 chymal stem cells (hMSCs) and human neonatal dermal fibroblasts (hNDFs) within a customized extracell

195 Human dermal fibroblasts, however, are not used for commercial

196 In dermal fibroblasts, IFNlambda induced the expression of

197 was expressed in epidermal keratinocytes and dermal fibroblasts in human skin and also in outer hair

198 Our findings suggest a crucial role for dermal fibroblasts in regulating the differentiation and

199 alone did not induce CD44v7, but stretching dermal fibroblasts in the presence of OPN increased huma

200 Using healthy dermal fibroblasts in vitro, we analyzed the signaling p

201 00A12 by nearly 70%, which in turn activated dermal fibroblasts in vitro.

202 ated protein, is elevated in aged human skin dermal fibroblasts in vivo and stimulates MMP-1 expressi

203 )-responsive gene expression in SSc skin and dermal fibroblasts, in particular the effect of differen

204 cated that the addition of FnIII-1c to human dermal fibroblasts induced the expression of several inf

205 (MESP) homolog may be used to convert human dermal fibroblasts into cardiac progenitors.

206 d that YAP1 regulates the differentiation of dermal fibroblasts into highly contractile myofibroblast

207 for achieving transdifferentiation of human dermal fibroblasts into induced cardiomyocyte-like cells

208 ession of human ETS2 to convert normal human dermal fibroblasts into replicative cells expressing the

209 Unlike strain-matched dermal fibroblasts, intraperitoneally administered ICC-S

210 A balanced turnover of dermal fibroblasts is crucial for structural integrity a

211 hat expression of TbetaRII in differentiated dermal fibroblasts is essential for normal wound healing

212 progression to reveal that RalA function in dermal fibroblasts is required for tumor progression of

213 that a physiological function of ADAMTS5 in dermal fibroblasts is to maintain optimal versican conte

214 te and sebocyte differentiation, its role in dermal fibroblasts is unclear.

215 MMP-1, principally derived from dermal fibroblasts, is the major protease capable of ini

216 ave now extended our study of HSV-1 entry to dermal fibroblasts isolated from nectin-1- or HVEM-defic

217 C5 in different human cell lines and primary dermal fibroblasts leads to reduced MHC class I expressi

218 ue-engineered human skin, Tiam1 silencing in dermal fibroblasts led to increased invasiveness of epid

219 noted that versican overexpression in human dermal fibroblasts led to increased SMA expression, enha

220 OL7A1, the defective gene in RDEB, in normal dermal fibroblasts led to increased type XII collagen, t

221 out the disease process and that analyses of dermal fibroblasts might lead to the discovery of promis

222 Second, COL7A1 supported dermal fibroblast migration and regulates their cytokine

223 ice with inducible deletion of MMP-14 in the dermal fibroblast (MMP-14(Sf-/-)).

224 mal human epidermal keratinocytes (NHEK) and dermal fibroblasts (NHDF) and to confine all tumorigenic

225 tructures when co-cultured with normal human dermal fibroblasts (NHDFs).

226 Human dermal fibroblasts obtained by skin biopsy can be reprog

227 nerated induced pluripotent stem cells using dermal fibroblasts obtained from patients with TSC.

228 Human dermal fibroblasts of both primary and cell-line culture

229 uced pluripotent stem cells (iPSCs) from the dermal fibroblasts of TLR3- and UNC-93B-deficient patien

230 contrast, IL-1alpha-dependent stimulation of dermal fibroblasts optimally stimulates epidermal stem c

231 ivo, we injected CSp-EMV-primed or -unprimed dermal fibroblasts (or CSp-EMVs) in a chronic rat model

232 ic mice were treated either with fibrocytes, dermal fibroblasts, or phosphate buffered saline (PBS) t

233 ctivity of ADAMTS5 profoundly influenced the dermal fibroblast phenotype and may regulate a phenotypi

234 Uniform Wnt signaling activity is present in dermal fibroblast precursors preceding hair follicle ini

235 in a transition wherein the abundant CD34(+) dermal fibroblasts present in healthy human skin disappe

236 Furthermore, normal human dermal fibroblasts (primary cells) are also seeded on th

237 Our previous studies show that whereas dermal fibroblasts proliferate in response to TGF-beta1,

238 ing into nonkeratinocytes, particularly into dermal fibroblasts, promotes cellular infiltration and,

239 In vitro, primary dermal fibroblasts readily express podoplanin in respons

240 GF knockdown and TGF-beta blockade in normal dermal fibroblasts reduced procollagen expression, where

241 ha and ERbeta; however, regulation of ERs in dermal fibroblasts remains poorly understood.

242 Dermal fibroblasts represent a heterogeneous population

243 mulation of D1 dopamine receptors present in dermal fibroblasts restores vascular endothelial growth

244 RNA analysis of transgenic dermal fibroblasts revealed elevated expression of key m

245 Gene-expression profiles of primary dermal fibroblasts revealed overexpression of matrix met

246 Cultured dermal fibroblast samples from patients with major depre

247 VEDS dermal fibroblasts secreted more TGF-beta2, whereas down

248 Affected dermal fibroblasts showed enhanced basal and epidermal g

249 In vitro studies using murine and human dermal fibroblasts showed that P311 stimulated TGF-beta1

250 tic endothelial cells (LECs) cocultured with dermal fibroblasts spontaneously organize into a stable

251 IL-1alpha treatment of dermal fibroblasts stimulated CD26 activity, and therefo

252 we show that forced expression of MRTF-A in dermal fibroblasts stimulates contraction of a collagen

253 in all cancer cell lines and normal primary dermal fibroblasts studied.

254 Microbe, Liu et al. (2016) report that human dermal fibroblasts support productive MCPyV infection.

255 extracellular 2-D matrix derived from human dermal fibroblasts supports GAS adherence and biofilm fo

256 f each receptor during HSV-1 entry in murine dermal fibroblasts that were deficient in expression of

257 of MEK1 in the epidermis leads to changes in dermal fibroblasts that, like the skin inflammatory infi

258 a/Smad/CTGF axis is significantly reduced in dermal fibroblasts, the major collagen-producing cells,

259 inhibitory factor (MIF), more strongly than dermal fibroblasts, thereby creating a MIF gradient in s

260 ol-sensitive PTP1B is affected by ROS in SSc dermal fibroblasts, thereby enhancing the phosphorylatio

261 e role of TGF-beta signaling specifically in dermal fibroblasts through the development of a novel, i

262 transforming growth factor-beta signaling in dermal fibroblasts through the down-regulation of thromb

263 al differentiation, is sufficient to convert dermal fibroblasts to a keratinocyte phenotype.

264 acellular matrix may regulate the ability of dermal fibroblasts to engage in such cross talk.

265 ctin expression and secretion and stimulated dermal fibroblasts to express EDA-fibronectin.

266 It took more than 1 hour for dermal fibroblasts to manifest detectable directional mi

267 est-derived melanocytes and mesoderm-derived dermal fibroblasts, to identify SE differentially methyl

268 e describe the transdifferentiation of human dermal fibroblasts towards the cardiac cell lineage via

269 f the myofibroblast protein alpha-SMA in SSc dermal fibroblasts treated with a caspase 1 inhibitor.

270 re, intraray glia, lateral line, osteoblast, dermal fibroblast, vascular endothelium, and resident bl

271 mal stability of collagen in patient-derived dermal fibroblasts versus age-matched control samples.

272 Inhibition of HA synthesis in dermal fibroblasts was shown to abrogate the TGF-beta(1)

273 Using knockout dermal fibroblasts, we confirmed its role for Brucella b

274 By using human dermal fibroblasts, we demonstrate that NO-np increased

275 lasts or selectively targeting Dlk1(+) lower dermal fibroblasts, we found that beta-catenin stabiliza

276 sing primary mouse embryonic fibroblasts and dermal fibroblasts, we show that TGF-beta-mediated, Smad

277 Using patient-matched oral and dermal fibroblasts, we show that TGF-beta1-dependent pro

278 These nuclear abnormalities in patient dermal fibroblast were restored by expression of WT NSMC

279 g cancer (NSCLC) cell lines and normal human dermal fibroblasts were co-cultured.

280 Human dermal fibroblasts were contaminated with gammaFe(2)O(3)

281 mmortalized lines of control and R258C human dermal fibroblasts were established and SM alpha-actin e

282 d monocytes on the phenotype of normal human dermal fibroblasts were examined by real-time PCR and We

283 Isolated dermal fibroblasts were incubated with recombinant IL-13

284 Dermal fibroblasts were isolated from systemic sclerosis

285 beta (TGF-beta) cytokines in patient-derived dermal fibroblasts were measured by ELISA.

286 es from KO mice were more migratory, whereas dermal fibroblasts were more proliferative compared with

287 Dermal fibroblasts were obtained from 2 patients with AR

288 Dermal fibroblasts were obtained from a CPVT patient due

289 ee cell strains each of normal, SSc, and NSF dermal fibroblasts were pooled separately, and each pool

290 Dermal fibroblasts were primed with CSp-EMVs for 24 h fo

291 The functional properties of fibrocytes and dermal fibroblasts were tested by using reverse-transcri

292 Our in vitro experiments showed that dermal fibroblasts, which are an important source of gro

293 nd induced a profibrotic phenotype in normal dermal fibroblasts, which was inhibited by an anti-IL-13

294 We aimed to study affected dermal fibroblasts with a view to inform therapeutic stu

295 We measured PIP3 concentrations in dermal fibroblasts with endogenous PIK3CA mutations and

296 r growth, and co-injection of Cav1-deficient dermal fibroblasts with melanoma cells is sufficient to

297 being made to generate neurons directly from dermal fibroblasts with neuron-specific transcription fa

298 ion of cell-derived matrices (CDMs) by human dermal fibroblasts with stable knockdown of COL6A1 revea

299 We show that treatment of human dermal fibroblasts with transforming growth factor-beta

300 r inducing apoptosis of primary normal human dermal fibroblasts without affecting the overall cell vi