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1 of systemic morphine is due to activation of descending inhibition.
2 iac nociceptive information were under tonic descending inhibition.
3 for the full development of noxiously evoked descending inhibition.
4 n oral to anal direction because of impaired descending inhibition; (6) reduced CMMCs and inhibitory
5                          The balance between descending inhibition and facilitation is thought to be
6 onous inhibitory neuromuscular inputs during descending inhibition and synchronous excitatory neuromu
7 l activation, reactive astrogliosis, loss of descending inhibition, and spasticity are responsible fo
8 ytes, and activation of LC neurons to induce descending inhibition, and that this paradoxical action
9 /- mice, with no evidence for recruitment of descending inhibition at the lumbar cord level after for
10                 The UG reflex is under tonic descending inhibition by neurons in the region of the nu
11 by intradermal injection of capsaicin on the descending inhibition driven from the periaqueductal gra
12 dystonia-like symptoms result from lapses in descending inhibition, exposing excess activity in intri
13  with the UG reflex and demonstrate that the descending inhibition from the nPGi is by means of prega
14 ent pain, and may not mediate opioid-induced descending inhibition in acute pain.
15                                              Descending inhibition induced by intra-RVM electrical st
16 ed a role for endogenous cannabinoids in the descending inhibition of dorsal horn cells via a suprasp
17 t suggests that the mechanism of PAG-induced descending inhibition of dorsal horn neuron activity inv
18  effects may be associated with an increased descending inhibition of dorsal horn neurons.
19 teral dorsolateral funiculus (DLF) removed a descending inhibition of multireceptive spinal neurones
20 to HCN channel function, which can influence descending inhibition of pain pathways in neuropathic co
21 erstanding regulatory mechanisms involved in descending inhibition of pain scientifically and clinica
22 ocus ceruleus that have substantial roles in descending inhibition of pain.
23 tem inhibitory system is related to both the descending inhibition of spinal motoneurons and suppress
24  produces lasting analgesia by enhancing the descending inhibition of spinal nociceptive neurons.
25 ired' polysynaptic circuit in the ENS evokes descending inhibition of the intestinal circular muscle
26 (ascending excitation) to, and relaxed anal (descending inhibition) to, a peristaltic wave.

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