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1 ntly by lanthanum (IC50 = 2 microM) and were desensitized by 1 microM 2S,4R-4-methylglutamate (SYM 20
2 In contrast to the WT CXCR2, which is 93% desensitized by 20 nM ligand, the 331T, 342T, and 4A CXC
3 vo, EBI2-deficient B cells or normal B cells desensitized by 7alpha,25-OHC pre-treatment showed reduc
4 of immature thymocytes is not significantly desensitized by a negative concentration gradient of CKb
5 eptor is exposed to bright light, the rod is desensitized by a process known as bleaching adaptation.
6 ntrast, GABA(B)R-mediated GIRK currents were desensitized by a similar amount after only 2 h of agoni
7 majority of G protein-coupled receptors are desensitized by a uniform two-step mechanism: phosphoryl
11 A-induced Ca(2+) flux in monocytes was cross-desensitized by an agonistic ligand MMK-1 specific for f
14 a(2+)channel coupling in dorsal root ganglia desensitized by ARM390 and the rate of resensitization w
15 ed by pertussis toxin and suramin and can be desensitized by ATP and ADP, suggesting a P2Y type nucle
18 adapted, and show that, for rods permanently desensitized by bleaching, the desensitization is not du
20 protein signaling is rapidly terminated (or "desensitized") by calcium influx through voltage-gated c
24 gger deletion, however, when the BCRs become desensitized by chronic stimulation with self-antigens o
26 lization by S3 and DeltaCCR1 were also cross-desensitized by CXCR1 and CXCR2 despite lack of receptor
27 5aR nor FR was cross-phosphorylated or cross-desensitized by CXCR2 activation, although CXCR1 did med
29 tor neurons in male antennae were completely desensitized by direct application of metyrapone into th
30 was lower than that for CCR9B, and CCR9A was desensitized by doses of CCL25 that failed to silence CC
32 A, like DeltaST, could not be significantly desensitized by exposure to agonist, mutant B exhibited
37 bers of this receptor family are regulated ("desensitized") by G protein-coupled receptor kinase (GRK
39 AdipoR1 is phosphorylatively modified and desensitized by GRK2 in failing cardiomyocytes, contribu
44 R2-specific calcium flux and chemotaxis were desensitized by injury, returning toward normal after 1
47 CRAMP-induced calcium flux in monocytes was desensitized by MMK-1, an agonistic ligand specific for
49 he alpha5 subunit are potently activated and desensitized by nanomolar concentrations of nicotine.
53 om rat tail artery and vas deferens are also desensitized by OXY, but not by NE or PE, indicating tha
54 PAFR was homologously phosphorylated and desensitized by PAF, and cross-phosphorylated and cross-
56 entration in response to Alternaria that was desensitized by peptide and protease ligands for PAR-2 a
57 ranulation was pertussis toxin sensitive and desensitized by preincubating cells with G protein-coupl
60 rowth-related oncogene alpha (Groalpha), was desensitized by prior exposure to the chemoattractants N
69 H1R-mediated adenylyl cyclase responses were desensitized by repetitive PTH challenges in a concentra
70 mobilization in lymphocytes, which could be desensitized by SDF-1, suggesting possible cross-regulat
72 ed alpha7-mediated nicotinic EPSCs also were desensitized by superfusion with 1 microM nicotine, had
73 , yet nearly all affected individuals can be desensitized by the administration of graded doses of as
74 ly restored the response of the B2 receptor, desensitized by the agonist (1 mumol/L [Hyp3-Tyr(Me)8]BK
75 Ca2+ entry as the response to capsaicin was desensitized by the same amount whether prior exposure t
76 erization in eosinophils and neutrophils was desensitized by the same combinations of chemoattractant
79 ugh Flag-hkorS358N was not phosphorylated or desensitized by (-)U50,488H stimulation, Flag-rkorN358S
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