ライフサイエンスコーパス: desflurane

1 n and cardiovascular and airway effects with desflurane.

2 [161-176]) > sevoflurane (164% [150-177]) > desflurane (119% [109-129]).

3 hy were enhanced by surgery as compared with desflurane alone.

4 matched 3xTgAD and WT mice exposed to air or desflurane alone.

5 However, treatment with a combination of 12% desflurane and hypoxia (18% O(2)) (desflurane/hypoxia) f

6 Here, we set out to assess the effects of desflurane and hypoxia on caspase activation, amyloid pr

7 the favorable binding enthalpy observed for desflurane and isoflurane, with an opposing increase of

8 reduction strategies including avoidance of desflurane and occupancy-based ventilation have the pote

9 Rapid recovery after desflurane and single-breath inductions with sevoflurane

10 In contrast, several volatile (isoflurane, desflurane) and i.v. (propofol) general anesthetics exci

11 Recovery after desflurane anesthesia is more rapid than with the other

12 r disease (3xTgAD) and C57BL/6 WT mice under desflurane anesthesia.

13 mine, inhalational anaesthetics (isoflurane, desflurane), antiepileptic drugs (topiramate, lacosamide

14 ional agents, usage costs of sevoflurane and desflurane are 10 and 25 times, respectively, that of is

15 of emergence delirium after sevoflurane and desflurane are discussed.

16 tile anesthetics isoflurane, sevoflurane and desflurane at clinically relevant concentrations did not

17 ession systems, sevoflurane, isoflurane, and desflurane at subsurgical concentrations potentiated del

18 In summary, desflurane can induce Abeta production and caspase activ

19 Sevoflurane and desflurane continue to challenge our abilities to anesth

20 that underwent surgery compared with air or desflurane controls persisted to at least 14 weeks after

21 INTERPRETATION: These findings suggest that desflurane could be a safer anesthetic for AD patients a

22 Finally, the anesthetic desflurane does not induce activation of mitochondrial p

23 librium association constants (K(a) values): desflurane > isoflurane approximately enflurane > haloth

24 he inhalation anesthetic isoflurane, but not desflurane, has been shown to induce caspase activation

25 Sevoflurane and desflurane have important advantages over isoflurane and

26 Propofol and desflurane have reliable anticonvulsant effects, whereas

27 Desflurane/hypoxia also increased levels of beta-site AP

28 on of 12% desflurane and hypoxia (18% O(2)) (desflurane/hypoxia) for 6 h induced caspase-3 activation

29 In addition, desflurane/hypoxia-induced Abeta generation could be red

30 8 attenuated caspase-3 activation induced by desflurane/hypoxia.

31 Here we show that isoflurane, but not desflurane, induces opening of mitochondrial permeabilit

32 Desflurane is one of the most commonly used inhalation a

33 on of closely related anesthetic haloethers (desflurane, isoflurane, enflurane, and sevoflurane), a h

34 Neither 12% desflurane nor hypoxia (18% O(2)) alone affected caspase

35 However, the effects of desflurane on AD neuropathogenesis have not been previou

36 out to assess the effects of isoflurane and desflurane on mitochondrial function, cytotoxicity, lear

37 tions of isoflurane, halothane, sevoflurane, desflurane or the nonimmobilizer 1,2-dichlorohexafluoroc

38 ents (isoflurane (IF), sevoflurane (SF), and desflurane) or MF were added to rat proximal tubular seg

39 ly nondefluorinated inhalational anesthetic (desflurane) or with a nonfluorinated anesthetic (pentoba

40 Preferential use of desflurane resulted in a ten-fold difference in anaesthe

41 Molecular simulations of desflurane reveal a binding pathway to GLIC via a membra

42 s of isoflurane, sevoflurane, enflurane, and desflurane sensitize TRPV1 to capsaicin and protons and

43 f TASK-3 to several anesthetics (isoflurane, desflurane, sevoflurane, halothane, alpha-chloralose, 2,

44 t comparable tubular necrosis, resulted with desflurane use.

45 ced by isoflurane, halothane, sevoflurane or desflurane was 221, 173, 184 and 929 microM, respectivel

46 ion anesthetics isoflurane, sevoflurane, and desflurane when used in routine clinical practice.

47 n studies using sevoflurane, isoflurane, and desflurane, which poorly quench tryptophan fluorescence,

48 expensive than administering sevoflurane or desflurane with premedication using antiemetics.