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1 ide a structural framework for understanding desmosome assembly.
2 Dsg3, thereby driving Dsg3 biosynthesis and desmosome assembly.
3 ey may also participate in the regulation of desmosome assembly.
4 termine the role of plakophilin-1 in de novo desmosome assembly.
5 cruitment of desmoplakin to the membrane and desmosome assembly.
6 ese effects, indicating a key role for PG in desmosome assembly.
7 tion, suggesting that EGFR blockade promotes desmosome assembly.
8 erin is apparently not sufficient to disrupt desmosome assembly.
9 hown to play an important role in initiating desmosome assembly.
10 e, promoting its activation and facilitating desmosome assembly.
11 xpected to compete with endogenous DP during desmosome assembly.
12 ulate DP-IF interactions required for normal desmosome assembly.
13 contribute to actin-dependent regulation of desmosome assembly.
14 o protein plakophilin 3 (Pkp3) mediates both desmosome assembly and E-cadherin maturation through Rap
15 obin to desmoglein 3 is an important step in desmosome assembly and leads to the incorporation of des
19 Nguyen et al. demonstrate a role for Perp in desmosome assembly and trafficking and pemphigus IgG-med
20 derived cell lines was sufficient to disrupt desmosome assembly and weaken intercellular adhesive str
21 at SERCA2-deficiency is sufficient to impede desmosome assembly and weaken intercellular adhesive str
22 on of truncated Dsg2 protein interferes with desmosome assembly and/or maintenance to disrupt cell-ce
24 ent to rescue the defective DP localization, desmosome assembly, and intercellular adhesive strength
25 koglobin, suggesting that p0071 may regulate desmosome assembly by controlling plakoglobin availabili
26 rnalization of newly synthesized Dsg3 during desmosome assembly, correlating with their pathogenic ac
27 Dsg1.myc, but not Dsc1a, Dsc1b, disrupted desmosome assembly in a cell-type-specific manner, and d
28 esults suggest that EGFR inhibition promotes desmosome assembly in oral squamous cell carcinoma cells
29 To define the function of plakophilin-1 in desmosome assembly, interactions among the desmosomal ca
30 Instead, impaired adhesion through aberrant desmosome assembly may explain the diminished tumor deve
31 nction assembly but dramatically compromised desmosome assembly, resulting in keratin filament retrac
32 e data suggest that in addition to mediating desmosome assembly, the nuclear pool of Pkp can influenc
33 , a known regulator of DP-IF association and desmosome assembly, to the plasma membrane by up to 70%.
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