ライフサイエンスコーパス: desynchronization

1 during baseline conditions (i.e. hippocampal desynchronization).

2 significance of reducing forced ventricular desynchronization.

3 noting whether stimulation produced cortical desynchronization.

4 MS/DB neurons, thereby producing hippocampal desynchronization.

5 l resources by facilitating movement-related desynchronization.

6 y, caused a delay of alpha parieto-occipital desynchronization.

7 inergic modulation, was sufficient to induce desynchronization.

8 f global cortical network synchronization or desynchronization.

9 tates are not equivalent to brief periods of desynchronization.

10 cell resolution through a chemically induced desynchronization.

11 pup by removing littermates induced further desynchronization.

12 ctions can mediate either synchronization or desynchronization.

13 ation and inhibition of GoC firing and spike desynchronization.

14 amplitude of melatonin release under forced desynchronization.

15 identify several mechanisms responsible for desynchronization: (1) shared inhibitory inputs in local

16 spection of individual profiles of localized desynchronization (10-18Hz) revealed left hemispheric do

17 tricular conduction, and prevent ventricular desynchronization (530 patients).

18 The degree and duration of desynchronization among SCN neurons depended on both the

19 ogenetic inhibition of SOM responses blocked desynchronization and decorrelation, demonstrating that

20 n LC neuronal discharge, and a transient EEG desynchronization and decrease in mitral cell discharge.

21 ated weaker post-stimulus beta event-related desynchronization and earlier and shorter event-related

22 Here, we show that two competing hypotheses, desynchronization and entrainment in a population of mod

23 ventricular pacing, which causes ventricular desynchronization and has been linked to an increased ri

24 n of cortical network disorganization (i.e., desynchronization and hypersynchronization) that affects

25 causal relationship between prefrontal beta desynchronization and memory formation.

26 al dual-chamber pacing, prevents ventricular desynchronization and moderately reduces the risk of per

27 This decrease was caused by a desynchronization and overall reduction in frequency of

28 ignificantly improve the stimulation-induced desynchronization and reduce the amount of the administe

29 owing they are involved in ACh-dependent EEG desynchronization, and others suggesting that this effec

30 eme deals explicitly with the problem of TEC desynchronization as transcript synthesis proceeds, and

31 es at similar rates gives way to substantial desynchronization at larger firing rate differences.

32 I and Type II neurons, we observe clustered desynchronization at many pulsing frequencies.

33 rrhythmia at the single-cell level and phase desynchronization at the network level--can account for

34 This triggers network desynchronization because heterogeneous coupling to surr

35 ind that transcranially inducing oscillatory desynchronization between the frontopolar and -parietal

36 (dm) SCN, and a jetlag paradigm that induces desynchronization between these SCN subregions, we show

37 ing revealed that posterior cingulate cortex desynchronization can be explained by increased excitabi

38 pected parieto-occipital low-alpha (8-10 Hz) desynchronization contralateral to the cued location.

39 ignment that results from circadian internal desynchronization could preserve the ability of light to

40 ork synchronization or trigger rapid network desynchronization depending on the synaptic input.

41 nal peptide-expressing neurons did not block desynchronization, despite these neurons being activated

42 ent-related analysis showed time-locked beta desynchronization during WAKE movements.

43 cal responses: powerful cortical and VTA EEG desynchronization, EMG activation, a large brain tempera

44 D showed attenuated alpha band event-related desynchronization (ERD) during encoding.

45 Event-related desynchronization (ERD) in alpha (8-12Hz) and low beta b

46 lp EEG studies have shown that event-related desynchronization (ERD) in the alpha (8-13 Hz) and beta

47 h much of it, in parallel with event-related desynchronization (ERD) in the alpha band.

48 MI led to event-related desynchronization (ERD) of oscillatory beta activity in

49 f brain oscillations is termed event-related desynchronization (ERD).

50 DN corresponded to a period of event-related desynchronization extending across a wide low-frequency

51 thy human subjects is a beta (13-30 Hz) band desynchronization followed by a postmovement event-relat

52 analysis showed that the amplitude of alpha-desynchronization followed the time course of temporal e

53 e, synchronization along short distances and desynchronization for long distances, and the decrease o

54 The extent of the decreased event-related desynchronization for median nerve-innervated digits was

55 Furthermore, beta-band desynchronizations for antisaccades started earlier, wer

56 In contrast, the beta-band event-related desynchronization from the motor cortex was preserved.

57 orienting was correlated with alpha/low-beta desynchronization (i.e., power suppression).

58 ng paradigm, we show that picrotoxin-induced desynchronization impairs the discrimination of molecula

59 Ventricular desynchronization imposed by ventricular pacing even whe

60 different scalp sites, ii) the event-related desynchronization in alpha and synchronization in theta,

61 In each patient, there was alpha/beta band desynchronization in M1 for stop trials.

62 However, the degree of desynchronization in M1 was less for successfully than u

63 The effect of pacing-induced ventricular desynchronization in patients with normal baseline QRSd

64 nia have impaired movement-related beta band desynchronization in primary motor and sensory cortices.

65 activity, and cause surround inhibition and desynchronization in response to excitatory input.

66 Desynchronization in steady light lowers the sensitivity

67 that seizure-like events are associated with desynchronization in such networks is consistent with re

68 and sentence comprehension tasks revealed a desynchronization in the 10-18Hz range, localized to the

69 EEG sensorimotor power spectra (ie, stronger desynchronization in the alpha and beta bands) occurred

70 We demonstrated that beta-band event-related desynchronization in the auditory cortex differentiates

71 otor affordance signatures: an event-related desynchronization in the mu frequency and an increased P

72 ST-ARM group had impaired beta and low gamma desynchronization in the primary motor cortex.

73 Beta-band desynchronization in the STN may reflect the additional

74 Previous studies have shown that beta-band desynchronization in the subthalamic nucleus (STN) is re

75 -13 Hz) and beta band (13-35 Hz) local field desynchronizations in sensorimotor and parietal cortex,

76 Alpha desynchronization increased rhythmically, peaking just b

77 with vIPS, reduced the high-alpha (10-12 Hz) desynchronization induced by shifting attention into bot

78 or the first time that robust alpha and beta desynchronization is a shared feature of sensorimotor co

79 , it remains elusive whether prefrontal beta desynchronization is causally relevant for memory format

80 presenting the sensorimotor mu rhythm, whose desynchronization is indicative for the degree of engage

81 Detailed network models predict that desynchronization is robust, local, and dependent on syn

82 The adverse effects of ventricular desynchronization may explain the association of RVA pac

83 ght ventricular stimulation, and ventricular desynchronization may result.

84 ramework that integrates synchronization and desynchronization mechanisms to explain how the two syst

85 n Parkinson's disease and the greatest alpha desynchronization occurring in essential tremor.

86 ssential tremor, with the greatest high-beta desynchronization occurring in Parkinson's disease and t

87 The underlying cause is an Abeta-induced desynchronization of action potential generation in pyra

88 visual WM task revealed that the prestimulus desynchronization of alpha oscillations predicts the acc

89 ention is associated with spatially specific desynchronization of alpha-band activity over visual cor

90 he circadian system also occur when there is desynchronization of clock phase with that of the outsid

91 We found that movement-related desynchronization of cortical activity in the upper alph

92 Desynchronization of electrical stimuli have shown benef

93 ontaneous release and suppression as well as desynchronization of evoked release, recapitulating the

94 Syt2 to achieve a significant reduction and desynchronization of fast release.

95 an raphe serotonergic neurons results in the desynchronization of hippocampal electroencephalographic

96 d in 17.8 +/- 1.4 days of training rewarding desynchronization of ipsilesional oscillatory sensorimot

97 processing and brain states, causing robust desynchronization of local field potentials and strong d

98 Specifically, desynchronization of local neuronal assemblies in the le

99 both an attenuation of spike frequency and a desynchronization of neighbors.

100 development and progression of AD including desynchronization of neuronal action potentials, consequ

101 ective effects of psychedelics result from a desynchronization of ongoing oscillatory rhythms in the

102 ovements are associated with a dampening and desynchronization of oscillatory activity in STN neurons

103 ative differences in the synchronization and desynchronization of responding neuronal populations.

104 This led to a desynchronization of rhythmic immune parameters, which m

105 elated behavior following UCMS and suggest a desynchronization of rhythms in the brain with an indepe

106 ms, such as VIP-VPAC2 signaling, can lead to desynchronization of SCN neuronal clocks and loss of beh

107 noic acid (RA) in chicken embryos leads to a desynchronization of somite formation between the two em

108 ring of ON DS cell neighbors, resulting in a desynchronization of spike activity.

109 Therefore, developmental desynchronization of spontaneous neuronal activity is a

110 The selective desynchronization of stimulus-evoked oscillating neural

111 Conversely, clinical relapse may reflect a desynchronization of the clock, indicative of a reactiva

112 A direct role for the MR in the desynchronization of the electroencephalographic activit

113 fe is probably due, in part, to cold-induced desynchronization of the ripening program involving ethy

114 he pattern of melatonin release under forced desynchronization of these SCN subregions.

115 ings of the SCN from these mice showed rapid desynchronization of unit oscillators.

116 ors provide increasing evidence showing that desynchronization of ventricular electrical activation a

117 phase walk ascribable to weakened coupling (desynchronization) of two oscillators, AP-SND phase walk

118 This reduced desynchronization on successful stop trials could relate

119 response to VIP and that the transient phase desynchronization, or "phase tumbling", could arise from

120 opographic differentiation revealed stronger desynchronization over the (ipsilateral) right-hemispher

121 e, MI, EF) and pacing promoters (ventricular desynchronization-paced QRSd and Cum%VP, and AV desynchr

122 ynchronization-paced QRSd and Cum%VP, and AV desynchronization-pacing mode).

123 g of location persists into brief periods of desynchronization prevalent in slow-wave sleep.

124 es and modeling studies indicated that this "desynchronization" process was dependent on presynaptic

125 hat transient periods of synchronization and desynchronization provide a mechanism for dynamically in

126 We posit that active desynchronization reduces summation of synaptic potentia

127 cally, we argue that the synchronization and desynchronization reflect a division of labor between a

128 nerve, can also cause cortical and thalamic desynchronization, resulting in a reduction of seizure a

129 Awareness of the problem of desynchronization should also lead to more regular monit

130 Preservation of event-related desynchronization suggests that the cells of origin diff

131 20, and 20-30 Hz) by using the event-related desynchronization technique.

132 is stimulation produced electroencephalogram desynchronization that was blocked by systemic and corti

133 d onset of phase synchronization and delayed desynchronization to the click train.

134 Spontaneous desynchronization under constant conditions was limited,

135 ulus combinations, the extent of oscillatory desynchronization varies with stimulus disparity.

136 No event-related desynchronization was detected; rather, there was a tran

137 tion of correlation accompanying brain state desynchronization was largely explained by a decrease in

138 Phase errors induced by sweep-trigger desynchronization were effectively reduced by spectral p

139 Saccade-related beta-band desynchronizations were observed just before and during

140 band power changes and broadband (4-150 Hz) desynchronization, which predicted significant reduction