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1 ients undergoing retinal surgery to repair a detached retina.
2 of patients who undergo surgery to correct a detached retina.
3 own to improve photoreceptor survival in the detached retina.
4 increased CD11b+ macrophage/microglia in the detached retina.
5 es of retinal Po(2) in both the attached and detached retina.
6 toreceptors for metabolism in the normal and detached retina.
7 1 and by counting monocytes/microglia in the detached retinas.
8 me-dependent increase in their expression in detached retinas.
9 ion of caspase-3, -7, and -9 and PARP in the detached retinas.
10 of the antiapoptotic Bcl-2 was increased in detached retinas after edaravone treatment, whereas the
11 An acute efflux of neuronal glutamate in detached retina could contribute to excitotoxicity and t
12 d to 47% +/- 18% of the control value in the detached retina during normoxia; hyperoxia increased Q(a
19 of zLEHD.fmk into the subretinal space of a detached retina resulted in decreased caspase-9 activity
20 opy were used to localize IRBP in intact and detached retina-retinal pigment epithelium (RPE) eyecups
22 lation of Muller glia pERK expression in the detached retina, suggesting that Muller survival pathway
24 e average inner retinal Po(2) (P(IR)) in the detached retina to a level higher than that during normo
26 is study tested whether axonal retraction in detached retina was due to the activation of the small G
29 rapid depletion from neuronal cell bodies in detached retina, whereas Muller cells, which normally se
30 efined as subfoveal fluid accumulation under detached retina with or without overlying foveal thicken
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