1 We determined 1,200 mg to be the recommended single-agent d
2 sert, milkshake, pancake and omelettete, was determined after their manufacturing and during in vitro
3 Thus, our objectives were to determine and compare levels of glutathione, as well as
4 Protein synthesis rates are determined, at the translational level, by properties of
5 Computational analysis of gene expression to determine both the sequence of lineage choices made by m
6 followed by surgery and further chemotherapy determined by histology.
7 The metabolic phenotype of a cancer cell is determined by its genetic makeup and microenvironment, w
8 elative to conventional paleoseismic records determined by trenching.
9 To determine direct targets, we performed a chromatin immun
10 ype) and fluoroquinolone susceptibility were determined from whole genome sequences.
11 Using prion permissive cells, we determined how impairment of endocytosis affects product
12 We determined how the chemical composition of primary ambie
13 f cell-conditioned medium prior to a non-NTA-determined (i.e., nanoparticle tracking analysis) exosom
14 We aimed to determine if associations between maternal body mass ind
15 Thus, we sought to determine if intrinsic MAVS signaling is required for pa
16 To determine if testosterone treatment compared with placeb
17 d how a protein's 3-dimensional conformation determines its capacity to interact with potential ligan
18 tablished the optical trapping technique for determining membrane mechanics of cultured primary affer
19 of female sex hormones as a major factor in determining migraine risk and characteristics, which acc
20 nitions of response, relapse, and methods of determining minimal residual disease.
21 vealed that germline-encoded complementarity-determining region 1beta residues present exclusively in
22 ired t test and chi(2) analysis were used to determine significant differences.
23 The prepared immunosensor was validated by determining TGF-beta1 in real saliva samples.
24 Here we determined that increased expression of XRN2 induced EMT
25 Our laboratory previously determined that mast cells are activated via a non-Fceps
26 It was determined that neither naphthalene-based modifiers with
27 Logistic regressions were used to determine the association of migration on CVD risk.
28 The purpose of the current study is to determine the efficacy of a PI3K/mTOR dual inhibitor, LY
29 To determine the frequency of emergency department (ED) vis
30 Purpose To determine the frequency of, and yield after, provider ov
31 mmatory SF modulate Th cell responses and to determine the immunosuppressive efficacy of RASF.
32 To determine the lineage contribution of the Nkx2.5+ cardio
33 erent candidates to be directly compared and determine the minimal antibody titers required to halt t
34 of Ebola viruses, it would be challenging to determine the propensity of acquiring mutations in respo
35 We sought to determine the proportions of IgA coating together with t
36 The goal of this study is to determine the underlying mechanisms of sequential energy
37 ver, to date, there are no studies that have determined the cellular or enzymatic function of UBE3B.
38 ntified autophagy as a pivotal cell response determining the efficiency of AAVs intracellular process
39 Secondary objectives include determining the incidence rate, relative risk (RR), and
40 e role of sulfur vacancies and the mechanism determining the reaction kinetics are still unknown.
41 ise from different acetylation patterns, but determining the sequences of paCOS and producing paCOS w
42 e, we tested the influence of temperature by determining the X-ray structure of Aqy1 at room temperat
43 also localizes to cilia similar to TULP3 and determines trafficking of a subset of these GPCRs to neu
44 anistic understanding of the trade-offs that determine trait diversity.
45 omer-dimer equilibrium affinity constant was determined using native MS, revealing a nearly eightfold
46 neurokinin-1 receptor (NK-1R), we sought to determine whether itch induced by SP can also be mediate
47 The objectives of this study were to determine whether the blood eosinophil molecular pattern
48 on and prior and concurrent malignancies) to determine whether to modify definitions to extend trials
49 n between LC n-3 PUFA intake and CVD risk.We determined whether a PCSK9 variant (rs11206510), which h
50 us rate constant (k0) for CO2 reduction were determined with different quaternary ammonium electrolyt
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