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1 he hypogastric (presumably inhibitory to the detrusor muscle).
2 ient contraction and prolonged relaxation of detrusor muscles.
3 -labeling, was up-regulated in fetal bladder detrusor muscle and lamina propria cells and that this w
5 an explanation for purinergic relaxation in detrusor muscles and show that there are no discrete inh
6 eported a new class of interstitial cells in detrusor muscles and showed that these cells could be id
7 e pelvic nerve (presumably excitatory to the detrusor muscle); and a pathway involving the pelvic ner
8 ereas PAR-1 and PAR-2 are predominant in the detrusor muscle, and PAR-4 is expressed in peripheral ne
9 ammation, increased mast cell numbers in the detrusor muscle have been reported in a subset of IC pat
12 ccount of this evidence, we propose that the detrusor muscle is arranged into modules, which are circ
13 ooth muscle cells (SMCs) in vitro and in the detrusor muscle of a mechanically overloaded bladder in
15 s assumption by studying E-C coupling in the detrusor muscle of wild type and Homer1(-/-) mice and by
16 mooth muscle cells (SMCs) were isolated from detrusor muscles of PDGFRalpha(+)/eGFP and smMHC/Cre/eGF
17 al recording from smooth muscle cells of the detrusor muscle revealed spontaneous depolarizations, di
19 ls subjacent to the epithelium and a thinner detrusor muscle that was not attributable to disruption
20 as associated with higher sensitivity of the detrusor muscle to muscarinic stimulation and membrane d
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