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1 in further insight into the pathogenesis of amyloidosis and develop therapeutic strategies to inhibit fibril formation we
2 ave focused on dissecting the underlying causes, as well as developing therapeutic strategies to replenish dopamine defic
3 minants of LDL susceptibility to oxidation is essential for developing therapeutic strategies to inhibit this process.
4 mechanisms that regulate its transcription is important for developing therapeutic strategies to treat pathologies of the
6 ed IL-6 expression in chondrocytes may provide insights for developing therapeutic strategies to combat osteoarthritis.
7 ilial BRCA1 carriers and establish a unique mouse model for developing therapeutic strategies to target both luminal and
8 model of glutamate injury-induced epileptogenesis may help develop therapeutic strategies to prevent epileptogenesis aft
9 This study will pave a way for targeting TLR-2 in developing therapeutic strategies to treat chronic diseases i
10 cteristics should be strongly considered as advantageous in developing therapeutic strategies to assist in the recovery o
11 Together, these observations may aid in developing therapeutic strategies to improve the outcome of s
15 elopment and growth has stimulated comprehensive efforts to develop therapeutic strategies to block mutant RAS function f
16 knowledge of the pCRP activation mechanism is essential to develop therapeutic strategies to minimize tissue injury.
17 antitumor immune response, highlights the critical need to develop therapeutic strategies to target the Rac pathway in a
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