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1 s underlying scale selection are impaired in developmental dyslexia.
2 it in noise-exclusion, a hallmark symptom in developmental dyslexia.
3 ter a behavioral intervention in adults with developmental dyslexia.
4 ficits in word reading and picture naming in developmental dyslexia.
5 ed support for a neurobiological etiology of developmental dyslexia.
6 impaired perceptual learning as a factor in developmental dyslexia.
7 he isolation of one of the genes involved in developmental dyslexia.
8 mirrored by its functional disconnection in developmental dyslexia.
9 reading in normal subjects and subjects with developmental dyslexia.
10 deficits have been reported in the brain in developmental dyslexia.
11 these systems where dysfunction may lead to developmental dyslexia.
12 , right-handed children with a normal IQ and developmental dyslexia (16 male, three female; age range
16 w, we discuss recent data on the genetics of developmental dyslexia and consider broader issues invol
18 cell density in the temporo-parietal lobe in developmental dyslexia and that the altered cerebral str
19 enic variant primary progressive aphasia and developmental dyslexia both manifest with phonological d
20 oding of speech in children with and without developmental dyslexia by measuring auditory brainstem r
21 word reading and picture naming deficits in developmental dyslexia can be reduced to a common neurol
23 ted in the pathogenesis of such disorders as developmental dyslexia, congenital amusia and tinnitus.
25 most extensively studied candidate genes for developmental dyslexia (DD) owing to its important role
26 is in a Finnish pedigree of eight cases with developmental dyslexia (DD) revealed several regions sha
30 e-organisation within the reading system; in developmental dyslexia, functional imaging is being used
31 underlying cause of reading difficulties in developmental dyslexia; however, existing evidence also
32 in order to assess three leading theories of developmental dyslexia: (i) the phonological theory, (ii
33 relatively less gray matter volume (GMV) in developmental dyslexia in bilateral temporoparietal and
40 ior temporal cortex, much in the same way as developmental dyslexia is associated with hypoactivation
43 A general problem in studying children with developmental dyslexia is how to separate inefficiency i
46 al study investigates cortical signatures of developmental dyslexia, particularly from the perspectiv
48 ed from these regions in men with persistent developmental dyslexia, suggesting that the anatomical d
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