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1 mes (3-5 and 6-10 h) following 0.5 mg kg(-1) dextroamphetamine.
2 ation for improved cognitive efficiency with dextroamphetamine.
3 ntially to treatment with methylphenidate or dextroamphetamine.
6 LAC)-controlled study of the effects of oral dextroamphetamine (AMPH, 0.25 mg/kg) treatment on brain
9 lness, and DA transporter inhibitors such as dextroamphetamine and methylphenidate are effective for
10 e sulfate, dextro-, levoamphetamine sulfate, dextroamphetamine aspartate, levoamphetamine aspartate,
11 al behavioral sensitization mechanisms after dextroamphetamine challenge is seen in subjects with sch
12 like movements would increase after repeated dextroamphetamine challenge to the dopaminergic system.
16 portion of the left inferior frontal gyrus, dextroamphetamine increased rCBF during WCST but decreas
17 ne aspartate, levoamphetamine aspartate, and dextroamphetamine saccharate) in the treatment of adult
18 mixed amphetamine salts compound (Adderall, dextroamphetamine sulfate, dextro-, levoamphetamine sulf
21 ever, the effect of dextroamphetamine (i.e., dextroamphetamine vs placebo) on task-dependent rCBF act
22 osite to that seen in a previous study using dextroamphetamine, we postulated a dopaminergic mechanis
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