ライフサイエンスコーパス: dextrose

1 pon restoration of ATP levels by addition of dextrose.

2 dration is often treated more slowly with 5% dextrose.

3 days' postfeeding with ethanol, but not with dextrose.

4 ) achieved by intra-arterial infusion of 50% dextrose.

5 d in groups fed unsaturated fatty acids with dextrose.

6 iets containing either ethanol or isocaloric dextrose.

7 hat were pair-fed with isocaloric amounts of dextrose.

8 d to 90% hepatectomy (Hpx) and given 5% oral dextrose.

9 that ethanol was isocalorically replaced by dextrose.

10 RNA targets in the fermentable carbon source dextrose.

11 lt, d-(+)-galactose, sorbitol, glycerol, and dextrose.

12 rbonate-buffered solutions that contained 4% dextrose.

13 red in Adsol or EAS 25 (mmol/L: adenine 2/2, dextrose 122/110, mannitol 42/55, glycerol 0/150, NaCl 1

14 g(-1) x min(-1)) and left intrarenal [6,6-2H]dextrose (14 micromol x kg(-1) x min(-1)) to achieve and

15 g(-1) x min(-1)) and left intrarenal [6,6-2H]dextrose (14 micromol x kg(-1) x min(-1)) to achieve and

16 in cells treated with high concentrations of dextrose, 2) statin treatment of endothelial cells norma

17 om-limited treadmill exercise testing during dextrose 5% infusion and after double-blind intravenous

18 ours of hyperglycemia (300 mg/dL) created by dextrose (50%) intra-arterial infusion, and with coadmin

19 In plasma prepared with acid citrate dextrose a binding pattern identical to that of serum wa

20 d samples collected in EDTA and acid-citrate-dextrose (ACD) were compared by cytomegalovirus (CMV) pp

21 IV-1-infected patients in EDTA, acid citrate dextrose (ACD), and heparin tubes, aliquoted, and stored

22 etected in EDTA-, heparin-, and acid citrate dextrose (ACD)-anticoagulated plasma samples were compar

23 in(-1)) with a variable infusion of [6-(2)H2]dextrose adjusted to maintain plasma glucose at either a

24 Hyperglycemia was induced by intraperitoneal dextrose administration prior to ischemia.

25 The only predictor of hyperglycemia was a dextrose administration rate >4 mg/kg/min.

26 PN regimens should not exceed a dextrose administration rate of 4 mg/kg/min to avoid hyp

27 h culture grown at 30 degrees C on Sabouraud dextrose agar (Emmons modification) by following the rec

28 occus neoformans using 2% dextrose Sabouraud dextrose agar (SabDex) with fluconazole was compared to

29 ain heart infusion agar (BHI), and Sabouraud dextrose agar (SDA) and into a BACTEC 13A bottle.

30 38 isolates grown on prepoured BBL Sabouraud dextrose agar (SDA) and prepoured Remel SDA.

31 specimens were streaked onto CAC, Sabouraud dextrose agar (SDA), inhibitory mold agar (IMA), and Myc

32 ia, inhibitory mold agar (IMA) and Sabouraud dextrose agar (SDA), were compared with respect to recov

33 lysis, yeasts were inoculated onto Sabouraud dextrose agar and incubated at 28 degrees C for 24 h.

34 ast isolates were subcultured onto Sabouraud dextrose agar and were incubated at 28 degrees C for 24

35 d on double-pour agar plates by using potato dextrose agar in both.

36 Each scraping was inoculated onto Sabouraud Dextrose Agar in C-shaped streaks and incubated at 25 de

37 the same isolate of yeast grown on Sabouraud dextrose agar simultaneously.

38 posing 90 mm settle plates containing potato dextrose agar to the air for 10 min.

39 ve fungal culture media, including Sabouraud dextrose agar with gentamicin (SDA), inhibitory mold aga

40 agar, sheep blood chocolate agar, Sabouraud dextrose agar, brain heart infusion, thioglycolate broth

41 on a variety of substrates including potato dextrose agar, olive oil, glyceride trioleate, oleic aci

42 ains were grown at 37 degrees C on Sabouraud dextrose agar, only C. dubliniensis strains coaggregated

43 ly, at 37 degrees C on yeast extract-peptone-dextrose agar, suggesting that T6P accumulation in R265t

44 Isolates were grown on Sabouraud-Dextrose agar, swabbed, and prepared in suspension, and

45 sing three different cereal agars and potato dextrose agar.

46 fied equally as well as those from Sabouraud dextrose agar.

47 strains passaged on CHROMagar and on potato dextrose agar.

48 tration of perfluorocarbon-exposed sonicated dextrose albumin (PESDA) and transcutaneous delivery of

49 ously that perfluorocarbon-exposed sonicated dextrose albumin (PESDA) microbubbles bind to injured va

50 trast agent perflurocarbon exposed sonicated dextrose albumin (PESDA) to increase uptake of VEGF in t

51 ntravenous perfluorocarbon-exposed sonicated dextrose albumin contrast injection.

52 ections of perfluorocarbon-exposed sonicated dextrose albumin microbubble contrast medium in humans.

53 ntravenous perfluorocarbon-exposed sonicated dextrose albumin, and ultrasound can be significantly im

54 andard PN containing conventional amounts of dextrose, amino acids, micronutrients, and lipid (120 kJ

55 DTIC 220 mg/m(2) IV for 1 hour in 500 mL of dextrose and (1/2) NaCl on days 1 to 3 of a 3-week cycle

56 ously (IV) for 30 to 45 minutes in 500 mL of dextrose and (1/2) normal saline (NS) on days 1 to 3 of

57 rs (Mueller-Hinton agar supplemented with 2% dextrose and 0.5 microg/ml methylene blue [MGM], Shadomy

58 media (supplemented Mueller-Hinton agar [2% dextrose and 0.5 microg/ml methylene blue] and plain Mue

59 2) IV for 2 to 3 hours in 750 to 1,000 mL of dextrose and 5% water on day 1 of every odd 3-week cycle

60 ired blood samples collected in acid-citrate-dextrose and EDTA were compared for human immunodeficien

61 Rats were fed intragastrically ethanol or dextrose and either medium-chain triglycerides, corn oil

62 own on a mixed carbon source containing both dextrose and methanol, ubiquitin is found in small stora

63 Ume6p acetylation occurs in medium lacking dextrose and results in a partial destruction of the rep

64 ith fasting insulin (57 pmol/L), achieved by dextrose and somatostatin (octreotide) infusion (150 mg/

65 after which the stomach was loaded with acid dextrose and the recording continued for 2 hours with in

66 pid, 170 micromol h(-1), chylous lymph) or a dextrose and/or electrolyte solution (control lymph).

67 iet contained 30% of calories as ethanol (or dextrose) and 25% of calories as corn oil.

68 blood (anticoagulated with citrate-phosphate-dextrose) and crystalloid and observed for the next 6 or

69 ined on solidified (1.5% agar) RPMI 1640 (2% dextrose), and zone diameters were obtained on supplemen

70 18-, and 3-fold in deletion strains grown on dextrose, and 2-, 7-, and 5-fold in deletion strains gro

71 pH 4.5 to 8.0, low O2 tension, 0.1 to 0.3 M dextrose, and absence of iron for 2 h.

72 ne HDP-cCDV was synthesized, suspended in 5% dextrose, and injected into the rabbit's vitreous at 10,

73 gned infusions of either insulin at 2.5 U/h, dextrose, and potassium (GIK) or normal saline and potas

74 nhibitory mold, cornmeal, Czapek-Dox, potato dextrose, and V8 juice agars; all isolates were sequence

75 dia, standard RPMI 1640 (RPMI), RPMI with 2% dextrose, antibiotic medium 3 (M3), and M3 with 2% dextr

76 d by a variety of sugars, including sucrose, dextrose, arabinose, fructose, and maltose.

77 d, regardless of use of EDTA or acid citrate dextrose as anticoagulant, and despite the presence of t

78 on of deuterated glucose and infusion of 20% dextrose as required to maintain euglycemia.

79 und, and the fellow eye was injected with 5% dextrose as the control.

80 CR, and 3) HS + CR + PR with a hyperosmolar dextrose-based solution (Delflex 2.5%).

81 rin and amino acids; EDEN trial: a different dextrose-based solution, icodextrin and amino acids) and

82 ention group (IMPENDIA trial: combination of dextrose-based solution, icodextrin and amino acids; EDE

83 ch competitor were inoculated into Sabouraud dextrose broth and incubated at 37 degrees C with vigoro

84 ntrolled by a GAL1 promoter grew normally on dextrose but not on galactose medium.

85 ated to receive either 546C88 or placebo (5% dextrose) by intravenous infusion for up to 72 hrs.

86 H rather than acidifying it, as occurs after dextrose catabolism.

87 iluted drugs in 7H9 broth-oleic acid albumin dextrose catalase.

88 ller-Hinton broth with 5% oleic acid-albumin-dextrose-catalase and 7H9 broth with casein) and by macr

89 serum) is used instead of oleic acid-albumin-dextrose-catalase to support good growth of M. tuberculo

90 s, the sensor has been characterized for the dextrose concentration range of 0 mM-10mM including the

91 oglycemia that was managed with concentrated dextrose containing fluids.

92 If alcoholic ketoacidosis is suspected, dextrose-containing fluids are recommended over normal s

93 t the 3' end of the GAL10 coding sequence in dextrose-containing growth medium that induces antisense

94 ubsequently, yeast cells were transferred to dextrose-containing growth medium to stop HO expression,

95 itrate-dextrose regional anticoagulation and dextrose-containing replacement fluids in the CVVH circu

96 (BCDTP) (collected in EDTA- or acid citrate dextrose-containing tubes in 1996 and 1997, had a 2-h ma

97 atin (sufficient to induce neurotoxicity) or dextrose control injections.

98 n the presence of ethanol (ethanol group) or dextrose (control group).

99 withdrawal, were refed ethanol or isocaloric dextrose (control) diets intragastrically for 7 days.

100 volume bolus and intravenous infusions of 5% dextrose (control, n = 8).

101 atectomy model in mice given standard or 10% dextrose (D10)-supplemented drinking water.

102 .p.) 60 min after DZ deposits, but not after dextrose deposits, further reduced the MRF-evoked EMG re

103 After 5 d of drinking 5% dextrose, desmopressin does not increase the osmolality

104 s; both Sod1(+/+) and Sod1(-/-) mice fed the dextrose diet had normal histology.

105 For comparing icodextrin and 4.25% dextrose during the long dwell of automated peritoneal d

106 Masking was achieved by infusion of 5% dextrose (during acetylcysteine delivery) or saline (for

107 polymerization (DP; liquid chromatography), dextrose equivalency (reducing sugar assays), and preval

108 corn syrup solids glucose polymers used had dextrose equivalent (DE) values of 17 or 38, respectivel

109 ferences between the quantities of calories, dextrose, fat, and protein provided to the two groups.

110 were not significantly different between the dextrose-fed controls and saturated fat/ethanol-fed rats

111 omparison of symptoms during the lactose and dextrose feeding periods showed no significant differenc

112 er 15 mins and then 50 mg/kg in 250 mL of 5% dextrose for 45 mins at an infusion rate of 62.5 mL/hr.

113 LCTs alone or 100% of nonprotein energy from dextrose for 6 or 12 d.

114 sponse was assessed by the administration of dextrose for 80 min following a 4-h clamp with either sa

115 ransporters, icodextrin is superior to 4.25% dextrose for long-dwell fluid and solute removal.

116 arable at baseline (all patients using 4.25% dextrose for the long dwell) with regard to mean (+/-SEM

117 or anticoagulants (EDTA versus acid citrate dextrose) for either test.

118 medium (up to 23% of the cell dry weight in dextrose-free tryptic soy broth [TSB]).

119 ) and the placebo group (n = 45) received 5% dextrose, from a minimum of 3 days up to a maximum of 5

120 Control deposits of dextrose had no significant effect.

121 yperglycemia was achieved by infusion of 50% dextrose (i.v.) prior to KCl-induced cardiac arrest glob

122 A solution of 5% dextrose in 0.9 % saline was continuously infused via fe

123 's solution (n = 6), 0.9% saline (n = 6), 5% dextrose in lactated Ringer's solution (D5RL) (n = 6), o

124 uperiority of icodextrin compared with 4.25% dextrose in optimizing peritoneal ultrafiltration (UF),

125 marate, separated by a 10-min infusion of 5% dextrose in sterile water, or three successive 10-min IV

126 unted, acute insulin response to intravenous dextrose in the patients with diffuse hyperinsulinism.

127 ated Ringer's solution (D5RL) (n = 6), or 5% dextrose in water (D5W) (n = 6).

128 avenous infusion of NAC (150 mg/kg/day in 5% dextrose in water [D5W]) or placebo (D5W) for up to 7 da

129 e (0.8 mg/kg ip) and were either starved (5% dextrose in water ad libitum) or fed (water and rat chow

130 ardial tamponade was induced by injecting 5% dextrose in water into the pericardial space until blood

131 a necessary mechanism for USV production, 5% dextrose in water or blood was infused intravenously int

132 -acetylcysteine at 150 mg/kg in 250 mL of 5% dextrose in water over 15 mins and then 50 mg/kg in 250

133 d divided into 3 groups: group I received 5% dextrose in water throughout the study period; group II

134 usion of normal saline (control; n=9) or 70% dextrose in water to increase blood glucose to 350 to 40

135 G-CSF or 5% dextrose in water was administered subcutaneously to cir

136 without a 2-microg/kg bolus) or placebo (5% dextrose in water) for 48 h in addition to their usual c

137 gnesium sulfate (2 g, 8 mmol) or placebo (5% dextrose in water) infused over 30 mins every 6 hrs for

138 Under control conditions (intracoronary 5% dextrose in water), atrial-pacing tachycardia decreased

139 dy wt) and one after placebo administration (dextrose) in a double-blind randomized design.

140 s may be attributed to the inhibition of the dextrose-induced increase in superoxide anions, whereas

141 astatin (10 nmol/l) normalized the 500 mg/dl dextrose-induced permeability changes.

142 ative recovery of the liver was postponed in dextrose-infused mice (versus vehicle control) by an int

143 opolysaccharide (1 mg/kg) in the presence of dextrose infusion (100 microL/hr).

144 in levels increased significantly during the dextrose infusion (P<.001).

145 A regimen of parenteral dextrose infusion that delays PH-induced hypoglycemia fo

146 onset of hyperglycemia (10 mmol/l; variable dextrose infusion) under fixed hormonal conditions (soma

147 ma and p27 expression, was also disrupted by dextrose infusion.

148 00 mg/dL]), achieved by insulin and variable dextrose infusion; (2) hyperglycemia (12.5 mmol/L [225 m

149 ated by severe hypoglycemia and the need for dextrose infusion; C-peptide levels were initially low a

150 d to receive either midazolam or vehicle (5% dextrose) infusion for 6 hrs.

151 ly to groups that were given NAC or placebo (dextrose) infusion for 72 hours.

152 Acute hyperglycemia induced by dextrose ingestion does not restore their retinal functi

153 ination of media, which included Sabouraud's dextrose, inhibitory mold, cornmeal, Czapek-Dox, potato

154 Hypertonic dextrose injections (prolotherapy) is an emerging treatm

155 ntrol animals were fed isocaloric amounts of dextrose instead of ethanol with the same diets.

156 Control rats received isocaloric amounts of dextrose instead of ethanol.

157 cted with 3x10(5) IU of IL-2 or 0.1 ml of 5% dextrose intraperitoneally every 8 h for 6 d, then kille

158 Controls were pair-fed and received dextrose isocaloric to ethanol.

159 Dextrose isocalorically replaced ethanol in control rats

160 Dextrose isocalorically replaced ethanol in controls.

161 ther placebo (5% dextrose) (n=160), GIK (40% dextrose, K+ 100 mmol.L(-1), insulin 70 u.L(-1)) (0.75 m

162 e-ischemic placebo (5% dextrose) or GIK (40% dextrose, K+ 100 mmol.L(-1), insulin 70 u.L(-1); 0.75 mL

163 age (20 ml/kg of body weight, 0.9% NaCl, 15% dextrose), KO mice had impaired natriuresis (37 +/- 10 v

164 he fasting level (6.1+/-2.73 mmol/l) with 5% dextrose labeled with 6,6[2H2]glucose throughout the AH

165 There was no acid production from dextrose, lactose, maltose, or sucrose.

166 ecognized being hypoglycaemic with the blood dextrose level falling down to 2.2mM or less.

167 %], antibiotic medium 3 [M3], and M3 with 2% dextrose [M3-2%]) and two criteria of MIC determination

168 tion induces partial destruction of Ume6p in dextrose medium and accelerates meiotic degradation by t

169 maintenance of mtDNA in cells grown on rich dextrose medium, but is dispensible in glycerol grown ce

170 nance of mtDNA in rho+ cells growing on rich dextrose medium, we find that it is not required for the

171 g vegetative growth on yeast extract-peptone-dextrose medium.

172 rmal blood cells mixed together in a sucrose/dextrose medium.

173 se, antibiotic medium 3 (M3), and M3 with 2% dextrose, MFCs were determined for each isolate-medium-d

174 cruited and randomized to either placebo (5% dextrose) (n=160), GIK (40% dextrose, K+ 100 mmol.L(-1),

175 ibited decreased growth on medium containing dextrose, oleic acid, and cerulenin, an inhibitor of fat

176 TP depletion (5 mM cyanide in the absence of dextrose), on the distribution and function of beta 1 in

177 and Sod1 knockout (Sod1(-/-)) mice were fed dextrose or ethanol (10% of total calories) liquid diets

178 FAT1 deletion strains grown on either dextrose or oleic acid medium accumulated very long-chai

179 l) and (2) continuous infusion of either 50% dextrose or saline (control).

180 llocated to receive pre-ischemic placebo (5% dextrose) or GIK (40% dextrose, K+ 100 mmol.L(-1), insul

181 N ranibizumab monotherapy (sham infusion [5% dextrose] PDT and ranibizumab 0.5 mg).

182 iod (9 +/- 38 ppm.h) compared with after the dextrose period (385 +/- 52 ppm.h, P < 0.001).

183 end of the lactose period compared with the dextrose period.

184 ming small colonies on yeast extract/peptone/dextrose plates.

185 mM-10mM including the cases of normal blood dextrose range.

186 ystemic caloric contribution of acid-citrate-dextrose regional anticoagulation and dextrose-containin

187 ydrate calories and nutritional support, and dextrose replacement for hypoglycemia prevention and tre

188 and 6 were fed fish oil-ethanol and fish oil-dextrose, respectively, for 8 weeks.

189 dia (standard RPMI-1640 [RPMI], RPMI with 2% dextrose [RPMI-2%], antibiotic medium 3 [M3], and M3 wit

190 tibility of Cryptococcus neoformans using 2% dextrose Sabouraud dextrose agar (SabDex) with fluconazo

191 cterized using surface plasmon resonance for dextrose sensing.

192 ype of anticoagulant used, with acid-citrate-dextrose solution B (ACD-B) providing the best results.

193 12.6% ACD/NS solution (anticoagulant citrate dextrose, solution A, USP mixed with 0.9% NaCl, v/v) was

194 signed in a 1:1 manner to the control group (dextrose solutions only) or to the low-glucose intervent

195 and conventional oral diets with intravenous dextrose (standard care), are compared with parenteral n

196 adults were randomly assigned to lactose or dextrose supplementation for 10 d (days 1-10), crossing

197 hese data show that the inhibitory effect of dextrose supplementation on liver regeneration is associ

198 The inhibitory effect of dextrose supplementation on liver regeneration was first

199 Following culture in medium containing dextrose, the added Q(6) was detected in the plasma memb

200 Intravenous fluid and dextrose therapy for illnesses during the first 2 years

201 ed state (i.v. Glamin to double amino acids, dextrose to sustain glucose approximately 7-7.5 mmol l(-

202 nerative response to PH was compared between dextrose-treated and control mice.

203 thally injured MPT cells but not to control, dextrose-treated cells, indicating that the beta 1 integ

204 t 50 microg/kg) or vehicle (100 microL of 5% dextrose) treatment was initiated at 1 hr after cecal li

205 A dialysis solution with 2.5% dextrose was administered continuously via one of the ca

206 roxide anion production induced by 500 mg/dl dextrose was inhibited by therapeutic concentrations of

207 Once each minute, 2 mL of cooled 5% dextrose was injected through the pulmonary catheter.

208 -480 mins, phenylephrine, normal saline, and dextrose were administered to maintain cerebral perfusio

209 of crystalline mixtures of DZ (20-80 ng) in dextrose were delivered to the medullary reticular forma

210 MGM], Shadomy [SHA], and RPMI 1640 [RPMI, 2% dextrose]) were obtained at 24 to 72 h.

211 to five rats per group) were fed ethanol or dextrose with either corn oil or saturated fat for 1-, 2

212 were switched to isocaloric diets containing dextrose with fish oil (group 2), palm oil (group 3), or

213 ditional group of rats fed either ethanol or dextrose with fish oil or corn oil were supplemented wit

214 media, including Bacto yeast extract-peptone-dextrose (yeast culture medium) broth, Luria-Bertani (ba

215 clinical strain was plated on yeast-peptone-dextrose (YPD) agar with 1 mM CuSO(4), three colony colo