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1 ed to contribute to the development of human diabetic autonomic neuropathy.
2 y a role in the pathogenesis of experimental diabetic autonomic neuropathy.
3 G which is relatively spared in experimental diabetic autonomic neuropathy.
4 ing GSK3beta as a new therapeutic target for diabetic autonomic neuropathy.
5 remain potential pathogenetic mechanisms for diabetic autonomic neuropathy.
6 idiopathic gastrointestinal dysmotility, or diabetic autonomic neuropathy (9 percent), and in none o
7 hat may have implications for both long-term diabetic autonomic neuropathies and insulin-induced hypo
15 ganglion neuritic dystrophy, the hallmark of diabetic autonomic neuropathy in mouse prevertebral symp
19 The striking improvement in the severity of diabetic autonomic neuropathy shown with IGF-I treatment
20 ly part of a larger disease process, such as diabetic autonomic neuropathy, traumatic brain injury, a
21 thesis in an in vivo streptozotocin model of diabetic autonomic neuropathy using an unbiased counting
23 tion in the areas of diabetic amyotrophy and diabetic autonomic neuropathy will also be reviewed.
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