ライフサイエンスコーパス: diabetic foot

1 ntuated in the presence of neuropathy in the diabetic foot.

2 nopathy, nephropathy, peripheral neuropathy, diabetic foot, and ischemic heart disease were 21.9%, 17

3 he large vessels and microcirculation of the diabetic foot are important in the development of foot u

4 s useful in planning surgery of the infected diabetic foot, as it enables reliable distinction betwee

5 Specific recommendations and pathways for diabetic foot care have been devised.

6 Microvascular disease, diabetic foot, cerebrovascular disease, cardiovascular d

7 (Explorer) in 43 hospitals with specialised diabetic foot clinics in France, Spain, Italy, Germany,

8 When assessing the economic effects of diabetic foot disease, it is important to remember that

9 olism of the lower extremity were factors in diabetic foot disease.

10 words "probe to bone," "osteomyelitis," and "diabetic foot" from 1946 to May 2015.

11 Diabetic foot infections (DFIs) typically begin in a wou

12 ctam for the treatment of moderate-to-severe diabetic foot infections (DFIs), we obtained 454 pretrea

13 Diabetic foot infections are a common and serious proble

14 Diabetic foot infections are a leading cause of lower ex

15 Diabetic foot infections cause substantial morbidity and

16 , that is absent from invasive isolates from diabetic foot infections, including osteomyelitis.

17 ould be considered for parenteral therapy of diabetic foot infections, when deemed appropriate.

18 ude that the PTB test can accurately rule in diabetic foot OM in the high-risk patients and rule out

19 iew the accuracy of the PTB test to diagnose diabetic foot OM.

20 The incidence of nonhealing wounds (diabetic foot, pressure, venous, and arterial ulcers) is

21 Diabetic foot problems are common throughout the world,

22 hase (GM3S) expression is increased in human diabetic foot skin, ob/ob and diet-induced obese diabeti

23 Diabetic foot ulcer (DFU) infections are challenging.

24 of diabetic neuropathy patients developed a diabetic foot ulcer by the one-year follow-up.

25 h diabetes and a non-infected neuroischaemic diabetic foot ulcer greater than 1 cm(2) and of grade IC

26 oot self-care behavior on the development of diabetic foot ulcer has received little empirical invest

27 the demographic variables and the number of diabetic foot ulcer hospitalizations, however, the effec

28 r predictors (neuropathy severity, number of diabetic foot ulcer hospitalizations, insulin treatment,

29 Diabetic foot ulcer microbiota were found to exist in on

30 er controlling for demographic variables and diabetic foot ulcer predictors (neuropathy severity, num

31 at significantly predicted the occurrence of diabetic foot ulcer, even after controlling for demograp

32 behavior as it relates to the occurrence of diabetic foot ulcer.

33 be insufficient to prevent the occurrence of diabetic foot ulcer.

34 pathophysiological deficit in the nonhealing diabetic foot ulcer.

35 ant in wound healing deficiency, typified by diabetic foot ulcer.

36 Diabetic foot ulceration is a major complication of diab

37 Diabetic foot ulceration is a severe complication of dia

38 n and axon-reflex vasodilation contribute to diabetic foot ulceration, early and prolonged NGF treatm

39 s viewed as beneficial for the prevention of diabetic foot ulceration, the effect of foot self-care b

40 Most wounds were diabetic foot ulcers (19.0%), venous leg ulcers (26.1%),

41 Nonhealing diabetic foot ulcers (DFUs) are a common and costly comp

42 Diabetic foot ulcers (DFUs) are a debilitating complicat

43 Diabetic foot ulcers (DFUs) are a severe complication of

44 Diabetic foot ulcers (DFUs) threaten limbs and prompt ho

45 Diabetic foot ulcers (DFUs), a leading cause of amputati

46 Diabetic foot ulcers (DFUs), a life-threatening complica

47 ined the effect of NETosis on the healing of diabetic foot ulcers (DFUs).

48 onizing potential of S. aureus isolated from diabetic foot ulcers (DFUs).

49 ation in the development and failure to heal diabetic foot ulcers (DFUs).

50 5) may decrease amputations in patients with diabetic foot ulcers and possibly accelerate closure of

51 Diabetic foot ulcers are serious and challenging wounds

52 Diabetic foot ulcers have been neglected in health-care

53 pplying behavior predicted the occurrence of diabetic foot ulcers in diabetic patients with neuropath

54 t >50% of S. aureus isolates from uninfected diabetic foot ulcers in French patients harbor a prophag

55 f the feet, and while its pathogenic role in diabetic foot ulcers is difficult to establish, it may b

56 tly improved wound closure of neuroischaemic diabetic foot ulcers without affecting safety after 20 w

57 mporal dynamics of the microbiota colonizing diabetic foot ulcers, a common and costly complication o

58 studies in the prevention and management of diabetic foot ulcers, including studies that focus on of

59 ail to heal in a timely manner, for example, diabetic foot ulcers, pose a health, economic, and socia

60 zer of skin and as a possible participant in diabetic foot ulcers, we used a selective medium to cult

61 rains are maintained in a commensal state in diabetic foot ulcers.

62 ound closure in patients with neuroischaemic diabetic foot ulcers.

63 cal need to develop therapies for nonhealing diabetic foot ulcers.

64 sing as a local treatment for neuroischaemic diabetic foot ulcers.

65 e a safe and effective treatment for complex diabetic foot wounds, and could lead to a higher proport

66 Diabetic foot wounds, particularly those secondary to am