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1 erstitial cells of Cajal (ICCs) is common in diabetic gastroparesis.
2 is and compare findings in idiopathic versus diabetic gastroparesis.
3 strointestinal motility disorders, including diabetic gastroparesis.
4 ht be a therapeutic option for patients with diabetic gastroparesis.
5 ing studies on the morbidity associated with diabetic gastroparesis.
6 changes in the gastric wall in patients with diabetic gastroparesis.
7 in patients with diabetes) in patients with diabetic gastroparesis.
8 a loss of Kit expression and development of diabetic gastroparesis.
9 has shown to be beneficial in idiopathic and diabetic gastroparesis.
10 cells could contribute to the development of diabetic gastroparesis.
11 expression in ICC may directly contribute to diabetic gastroparesis.
12 cterized clinical profiles in idiopathic and diabetic gastroparesis and are defining roles of gastric
14 no1 gene in gastric muscles of patients with diabetic gastroparesis and nondiabetic control tissues.
17 proposed that Kit expression is lost during diabetic gastroparesis due to increased levels of oxidat
21 e) intended to reduce the aspiration risk of diabetic gastroparesis is likely over-utilized and may o
24 ession and splicing of Ano1 in patients with diabetic gastroparesis that alter the electrophysiologic
25 an age, 55 y; 88% with type 2 diabetes) with diabetic gastroparesis with moderate to severe symptoms
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