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1 postoperative macular edema in patients with diabetic macular edema.
2 d vascular leakage observed in patients with diabetic macular edema.
3 either proliferative diabetic retinopathy or diabetic macular edema.
4 e nonsurgical management options in treating diabetic macular edema.
5 er-reviewed literature regarding therapy for diabetic macular edema.
6 al therapeutic approach for the treatment of diabetic macular edema.
7 mproving visual acuity in some patients with diabetic macular edema.
8 ical trials for the treatment of uveitis and diabetic macular edema.
9 otentially abate or restore vision loss from diabetic macular edema.
10 rs a protective effect on the development of diabetic macular edema.
11 of optical coherence tomography detection of diabetic macular edema.
12 diabetic retinopathy (DR) in the absence of diabetic macular edema.
13 vascular age-related macular degeneration or diabetic macular edema.
14 ates with increased vascular permeability in diabetic macular edema.
15 vascular age-related macular degeneration or diabetic macular edema.
16 such as age-related macular degeneration or diabetic macular edema.
17 lar degeneration, retinal vein occlusion and diabetic macular edema.
18 ) and other factors is an important cause of diabetic macular edema.
19 lying capillary nonperfusion in eyes without diabetic macular edema.
20 dothelial growth factor for the treatment of diabetic macular edema.
21 se in diabetic retinopathy in the absence of diabetic macular edema.
22 injections during 6 months in patients with diabetic macular edema.
23 (3-fold) and serum samples of patients with diabetic macular edema (1.6-fold) measured by Western bl
26 omenon of temporary paradoxical worsening of diabetic macular edema after insulin treatment has been
27 eakage from the retinal vasculature leads to diabetic macular edema, an important cause of vision los
28 of randomized trial data on 660 adults with diabetic macular edema and decreased VA (Snellen equival
31 s (of 486 participants) with center-involved diabetic macular edema and no preexisting open-angle gla
33 re nonproliferative diabetic retinopathy and diabetic macular edema and of a normal nondiabetic eye w
34 e understanding, diagnosis, and treatment of diabetic macular edema and other ocular diseases with ou
35 f endothelial dysfunction, which can lead to diabetic macular edema and proliferative diabetic retino
36 ed retinal vascular leakage in patients with diabetic macular edema and reduced the rate of sustained
38 nosed as having diabetic retinopathy without diabetic macular edema and underwent fluorescein angiogr
39 treal Aflibercept Injection in Patients With Diabetic Macular Edema) and VIVID (Intravitreal Afliberc
40 treal Aflibercept Injection in Patients With Diabetic Macular Edema) and VIVID (Intravitreal Afliberc
41 nts with retinal disease, including uveitis, diabetic macular edema, and age-related macular degenera
42 raded 3 to 7 times for diabetic retinopathy, diabetic macular edema, and image gradability by a panel
46 improved vision in eyes with center-involved diabetic macular edema, but the relative effect depended
49 n included age-related macular degeneration, diabetic macular edema, central and branch retinal vein
50 ized the treatment of clinically significant diabetic macular edema (CSDME); yet these agents are exp
51 who had at least 1 eye with central-involved diabetic macular edema, defined as Stratus central subfi
53 gns of retinopathy (n = 9) and patients with diabetic macular edema (DME) (n = 31) were compared with
54 7 mg (DEX 0.7) was approved for treatment of diabetic macular edema (DME) after demonstration of its
56 10 eyes of healthy subjects, 10 eyes with diabetic macular edema (DME) and 10 eyes with neovascula
58 cation and characterization of patients with diabetic macular edema (DME) are important for individua
59 mpairing (VA 20/32 or worse) center-involved diabetic macular edema (DME) at baseline were required t
60 average, a shorter duration of diabetes and diabetic macular edema (DME) at baseline, were less like
61 brovascular accidents (CVA) in patients with diabetic macular edema (DME) compared with diabetic pati
62 travitreal aflibercept injections (IAIs) for diabetic macular edema (DME) during the phase III VISTA
63 helial growth factor (anti-VEGF) therapy for diabetic macular edema (DME) favorably affects diabetic
65 ), and ranibizumab (0.3 mg) for treatment of diabetic macular edema (DME) involving the center of the
67 ntravitreal dexamethasone implant therapy in diabetic macular edema (DME) is associated with long-ter
71 flibercept, bevacizumab, and ranibizumab for diabetic macular edema (DME) might influence interpretat
74 d aqueous pro-permeability factors (PPFs) in diabetic macular edema (DME) patients before and after i
75 tereoscopic fundus photographs, we evaluated diabetic macular edema (DME) progression and DR progress
76 itial monthly dosing period) (2q8) and other diabetic macular edema (DME) therapies at doses licensed
77 ser, and sham in the first-line treatment of diabetic macular edema (DME) to inform technology assess
78 pathy severity score (DRSS) in patients with diabetic macular edema (DME) treated with intravitreal r
81 (PDR) without macular edema, and 27 eyes had diabetic macular edema (DME) with either NPDR or PDR.
83 t-effectiveness analysis of the treatment of diabetic macular edema (DME) with ranibizumab plus promp
84 and functional characteristics in eyes with diabetic macular edema (DME) with subfoveal neuroretinal
85 dothelial growth factor therapy in eyes with diabetic macular edema (DME) with vision loss after macu
86 dothelial growth factor therapy in eyes with diabetic macular edema (DME) with vision loss after macu
87 ure visual acuity (VA) in eyes with baseline diabetic macular edema (DME) would substantively improve
88 nonproliferative DR (NPDR), 51 with NPDR and diabetic macular edema (DME), and 18 with proliferative
90 ular age-related macular degeneration (AMD), diabetic macular edema (DME), and retinal vein occlusion
91 ular age-related macular degeneration (AMD), diabetic macular edema (DME), central and branch retinal
93 useful therapeutic target for patients with diabetic macular edema (DME), perhaps in combination wit
94 lar age-related macular degeneration (nAMD), diabetic macular edema (DME), retinal vein occlusion, ch
95 with DR of varying severity, with or without diabetic macular edema (DME), using en face Doppler OCT.
115 t age-related macular degeneration (AMD) and diabetic macular edema (DME).All patients were operated
116 related macular degeneration (AMD, n = 400), diabetic macular edema (DME, n = 400), or retinal vein o
117 ith type 2 diabetes and severe stages of DR (diabetic macular edema [DME] and proliferative diabetic
118 rative DR [NPDR], proliferative DR [PDR], or diabetic macular edema [DME]) or "any DR" (further subcl
119 vascular age-related macular degeneration or diabetic macular edema does not respond to an initial an
120 vascular age-related macular degeneration or diabetic macular edema does not respond to an initial an
121 included 5 pregnant women who presented with diabetic macular edema during pregnancy in the period fr
122 ) grade during the Fluocinolone Acetonide in Diabetic Macular Edema (FAME) A and B Phase III clinical
123 ctiveness of ranibizumab in the treatment of diabetic macular edema has been proven with large clinic
126 cept for age-related macular degeneration or diabetic macular edema in a 9-member retinal specialty p
128 omated detection of diabetic retinopathy and diabetic macular edema in retinal fundus photographs.
129 ess levels to have reasonable certainty that diabetic macular edema involving the CSF is present usin
132 ed 660 adults (mean age, 61+/-10 years) with diabetic macular edema involving the macular center to r
135 is of proliferative diabetic retinopathy and diabetic macular edema, leading causes of vision loss in
136 reomacular separation occurring in eyes with diabetic macular edema may facilitate spontaneous resolu
138 ore, evaluating the effects of therapies for diabetic macular edema on development or worsening of PD
139 ade, and presence of morphologic features of diabetic macular edema on Spectralis optical coherence t
140 or severe diabetic retinopathy as defined by diabetic macular edema or proliferative diabetic retinop
142 te and worse diabetic retinopathy, referable diabetic macular edema, or both, were generated based on
148 tment of various neovascular diseases (e.g., diabetic macular edema, retinal vein occlusion, choroida
149 dothelial growth factor agent ranibizumab in diabetic macular edema, retinal vein occlusion, pseudoph
150 Ranibizumab therapy in the treatment of diabetic macular edema seems to improve retinal function
158 (MMP-9), which is increased in patients with diabetic macular edema, was capable of cleaving netrin-1
160 in optical coherence tomography features of diabetic macular edema were seen with ranibizumab therap
161 Long-term visual outcomes for treatment of diabetic macular edema with ranibizumab are excellent, b
162 ee eyes of 33 patients with center-involving diabetic macular edema, with best corrected visual acuit
163 CT is a useful tool to detect and to measure diabetic macular edema without the need for pupil dilata
164 rated clinical efficacy for the treatment of diabetic macular edema without visible signs of retinal
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