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1 in this investigation clearly show that 4,6-diamidino-2 phenylindole (DAPI) is superior to both of t
2 d to study the excited triplet state of 4',6-diamidino-2-phenyl indole (DAPI) and its complexes with
4 d for intracapsid steric restriction of 4',6-diamidino-2-phenylindole (DAPI) binding to packaged DNA.
6 show that the ICD of minor-groove-bound 4',6-diamidino-2-phenylindole (DAPI) originates from an intri
7 nd labeling (TUNEL) in conjunction with 4'6'-diamidino-2-phenylindole (DAPI) staining and by fluoresc
11 groove binding compounds, netropsin and 4,6-diamidino-2-phenylindole (DAPI), and a DNA hairpin havin
12 ell extracts, and staining of cells with 4,6-diamidino-2-phenylindole (DAPI), and the polyP-binding d
14 ng toxin (Cdt), connective tissue (CT), 4',6-diamidino-2-phenylindole (DAPI), human gingival epitheli
15 microscope after labeling in vitro with 4',6-diamidino-2-phenylindole (DAPI), intracellular injection
17 ei frequently do not stain equally with 4',6-diamidino-2-phenylindole (DAPI), suggesting that byr4 is
18 finity exhibited by 5PTB, netropsin and 4',6-diamidino-2-phenylindole (DAPI), two AT-specific minor g
19 ent of changes in the fluorescence of a 4',6-diamidino-2-phenylindole (DAPI)-dsDNA complex upon RecA
27 hods: staining intact chloroplasts with 4',6-diamidino-2-phenylindole (DAPI); staining at the single-
28 romosome probes, single-copy genes, and 4'-6-diamidino-2-phenylindole (DAPI-) and G-banded chromosome
29 l level using double immunostaining plus 4,6-diamidino-2-phenylindole (for nuclei) counterstaining.
30 on of polyP in the dense granules using 4',6-diamidino-2-phenylindole and by its release together wit
31 ng with anti-alpha-tubulin antibody and 4',6-diamidino-2-phenylindole and flow cytometric analysis of
32 tochemical staining of cellular DNA with 4,6-diamidino-2-phenylindole demonstrated TNF-alpha-induced
33 ive fluorescent reagents SYPRO Ruby and 4',6-diamidino-2-phenylindole dihydrochloride (DAPI), to dete
35 kit and of dead (apoptotic) cells using 4',6-diamidino-2-phenylindole dihydrochloride nuclear stainin
36 cent dye molecules per CPMV using DAPI (4',6-diamidino-2-phenylindole dihydrochloride), propidium iod
37 thermore, microtubule immunostaining and 4,6-diamidino-2-phenylindole DNA staining demonstrated that
38 These cells were later stained with 4', 6-diamidino-2-phenylindole for simultaneous DNA analysis a
39 ate nuclear localization correlated with 4,6-diamidino-2-phenylindole light regions and is excluded f
41 specific probes, in addition to inverted 4,6-diamidino-2-phenylindole or conventional G-banding analy
42 xed permeabilized red blood cells with 4',6'-diamidino-2-phenylindole or YOYO-1, a sensitive nucleic
43 rast microscopy, and in selected cases 4',6'-diamidino-2-phenylindole staining and DNA fragmentation.
46 was developed based on the patterns of 4',6-diamidino-2-phenylindole staining of the Nipponbare pach
47 bution of the actin cytoskeleton, DNA (4', 6-diamidino-2-phenylindole staining), and calcofluor-stain
48 tin fragmentation by acridine orange and 4'6-diamidino-2-phenylindole staining, and (3) agarose gel e
49 ic, as assessed by Annexin V staining, 4',6'-diamidino-2-phenylindole staining, and DNA fragment ELIS
50 ediated dUTP nick-end labeling (TUNEL), 4',6-diamidino-2-phenylindole staining, and immunohistochemis
51 hanges in nuclear morphology detected by 4,6-diamidino-2-phenylindole staining, DNA fragmentation ass
52 c apoptotic changes, as demonstrated by 4',6-diamidino-2-phenylindole staining, lack of either DNA la
53 localized to the contractile vacuole by 4',6-diamidino-2-phenylindole staining, suggesting, with the
56 e for cytokeratins 8, 18, and/or 19 and 4',6-diamidino-2-phenylindole were considered to be CTCs.
59 ragmentation of cellular DNA, and DAPI (4',6-diamidino-2-phenylindole) staining revealed condensation
61 uridine (BrdU) pulse-labeling and DAPI (4',6-diamidino-2-phenylindole) staining, which precisely esta
62 escent foci that colocalize with DAPI (4',6'-diamidino-2-phenylindole)-positive material and follow D
66 ondensation, as observed with the use of 4,6-diamidino-2-phenylindole-fluorescent staining, and by de
68 mly; locally high levels accumulated in 4',6-diamidino-2-phenylindole-negative zones containing euchr
70 going apoptosis verifies that the FD of 4',6-diamidino-2-phenylindole-stained nuclear structures does
75 oncept, we evaluated the selectivity of 4',6-diamidino-2-phenylindone (an AT-specific binder), ethidi
78 ts in mice combining retrograde transport of diamidino yellow after spinal cord injections and immuno
79 ither Cholera Toxin-fluorescein (CT-FITC) or Diamidino Yellow into TPNs revealed that RPM and TPN mot
81 fluorescent retrograde tracers Fast blue and Diamidino yellow were placed into different locations wi
82 retrogradely transported dyes Fast Blue and Diamidino Yellow were placed within areas V4 and MT, or
84 ish this, fluorescent tracers (fast blue and diamidino yellow) were injected into NTS and RVLM, after
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