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1 ons were observed between TWA and general or diastolic hypertension.
2 al and high-normal BP than from "burned-out" diastolic hypertension.
3 lic hypertension but not in individuals with diastolic hypertension.
4 e of future graft function in the setting of diastolic hypertension.
5 t childhood OSAS is associated with systemic diastolic hypertension.
6 range 20-31), five had systolic and two had diastolic hypertension.
7 ce lacking one or both D1A alleles developed diastolic hypertension.
8 and less than those associated with systolic diastolic hypertension.
9 isolated diastolic hypertension and systolic diastolic hypertension.
10 .53 (2.39 to 2.68) for combined systolic and diastolic hypertension, 1.78 (1.69 to 1.87) and 1.68 (1.
11 o 1.76) and 1.45 (1.27 to 1.65) for isolated diastolic hypertension, 2.01 (1.64 to 2.48) and 1.61 (1.
13 t, a stepped-care drug treatment program for diastolic hypertension (after an initial attempt at bloo
14 ntly associated with higher odds of systolic/diastolic hypertension (AHI 15 to 29.9, OR=2.38 [95% CI
15 lood pressure and risks of both systolic and diastolic hypertension among women aged 40 to 59 years.
16 han in those with high-normal BP or isolated diastolic hypertension and less than those associated wi
19 and less than those associated with isolated diastolic hypertension and systolic diastolic hypertensi
22 ence or absence of diabetes and systolic and diastolic hypertension; body-mass index; and other tradi
23 on was not related to renin transcription or diastolic hypertension but was correlated with histologi
25 plus hypopneas per hour of sleep), systolic/diastolic hypertension (> or =140 and > or =90 mm Hg), a
27 nction is associated with protection against diastolic hypertension in humans, underscoring the impor
28 ated an association between SDB and systolic/diastolic hypertension in the elderly by categorizing in
29 sociation was found between SDB and systolic/diastolic hypertension in those aged > or =60 years or b
31 stmenopausal women, in whom adjusted ORs for diastolic hypertension increased with increasing quartil
32 a support the hypothesis that posttransplant diastolic hypertension is a result of TGF-beta-induced,
34 ip between blood lead level and systolic and diastolic hypertension is most pronounced in postmenopau
35 in SBP, cannot be explained by "burned out" diastolic hypertension or by "selective survivorship" bu
36 mm Hg and DBP >/=90 mm Hg); and 5) systolic diastolic hypertension (SBP >/=140 mm Hg and DBP >/=90 m
37 btypes were defined as combined systolic and diastolic hypertension (SBP >or=140 and DBP >or=90 mm Hg
38 on (SBP >or=140 and DBP <90 mm Hg), isolated diastolic hypertension (SBP <140 and DBP >or=90 mm Hg),
39 to 139/85 to 89 mm Hg); 3) ISH; 4) isolated diastolic hypertension (SBP <140 mm Hg and DBP >/=90 mm
40 ce, longer duration, and higher systolic and diastolic hypertension than the non-Hispanic and Hispani
42 or tacrolimus has been postulated to lead to diastolic hypertension through the induction of transfor
43 the general population (eg, for systolic or diastolic hypertension versus the hazard ratio of mortal
44 1.42 [95% CI, 1.13-1.78]), whereas isolated diastolic hypertension was associated with increased ris
46 stolic blood pressure; treatment of isolated diastolic hypertension was modeled as a reduction in dia
48 tolic hypertension and combined systolic and diastolic hypertension were modeled as a reduction in sy
49 Most middle-aged hypertensives have systolic/diastolic hypertension, whereas isolated systolic hypert
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