ライフサイエンスコーパス: diastolic volume

1 nulus, E/E' septal annulus, left ventricular diastolic volume).

2 iated with greater RV mass and larger RV end-diastolic volume.

3 ump failure revealed a rapid decrease in end diastolic volume.

4 impaired ventricular filling with decreased diastolic volume.

5 nificantly reduced because of diminished end-diastolic volume.

6 adjustment for baseline left ventricular end-diastolic volume.

7 ncing the saline-mediated increase in LV end-diastolic volume.

8 e of 15% or more in the left ventricular end-diastolic volume.

9 ed with the postoperative decrease in LV end-diastolic volume.

10 Allogeneic MSCs reduced LV end-diastolic volumes.

11 s associated with lower left ventricular end-diastolic volume (0.01 mm/mL; P<0.01), a lower left vent

12 tio of elevated left ventricular mass to end-diastolic volume (0.02 g/mL per unit).

13 as percent reduction in left ventricular end-diastolic volume 1 year after CRT-D implantation) and to

14 with stable LV chamber volumes (DeltaLV end-diastolic volume, 1+/-1 mL/m(2); P=0.48).

15 women exhibited RV cavity dilatation (RV end-diastolic volume, +1.0 mL per BMI point increase; P<0.00

16 5% vs. 56%; p < 0.001), increased median end-diastolic volume (100 ml/body surface area [BSA](1.3) vs

17 ated right ventricles (right ventricular end-diastolic volume=101+/-26 mL/m(2)) with good systolic fu

18 and 65+/-4%; P<0.05), greater indexed LV end-diastolic volume (102+/-34 versus 84+/-14 and 85+/-16 mL

19 t associated with changes in LV volumes (end-diastolic volume 106 +/- 15 versus 110 +/- 22 mL; end-sy

20 ared with patients with GLS</=-15.0% (LV end-diastolic volume 123+/-44 versus 106+/-36 mL and 121+/-4

21 and increased echocardiographic volumes (end-diastolic volume, 126+/-39 versus 112+/-33 mL/BSA(1.3),

22 e was mild with significant decreases in end-diastolic volume (139 to 107 mL; P=0.03) and left atrial

23 reduction in right ventricular volumes (end diastolic volume 142+/-43 to 91+/-18, end systolic volum

24 A significant reduction of LV end-diastolic volume (149.7 +/- 41.4-140.1 +/- 43.9 mL; P=0.

25 s a larger increase in right ventricular end-diastolic volume (15 mL; 95% confidence interval, 3-28 m

26 rized by dilation of the left ventricle (end-diastolic volume, 156+/-26 versus 172+/-28 mL, P<0.001),

27 se of 15 mL at >500 ng/mL, p=0.0026) and end-diastolic volumes (16 mL, p=0.0096) that might have been

28 ncing the saline-mediated increase in LV end-diastolic volume (+17+/-1 versus +10+/-2 mL; P=0.016).

29 significant decrease in left ventricular end-diastolic volume (-18 mL; P=0.009) and end-systolic volu

30 .80; P<0.00001), and tended to reduce LV end-diastolic volume (-2.26 mL; 95% confidence interval, -4.

31 sed E-wave velocity and left ventricular end-diastolic volume, 2) exhibit a higher plasma volume, and

32 CMR at 1 year demonstrated a decrease in end diastolic volume (208.7+/-20.4 versus 167.4+/-7.32 mL; P

33 odeling (>20% change in left ventricular end-diastolic volume; 21.91 [2.75-174.29]; P=0.004).

34 on (30+/-10% versus 29+/-7%, P=0.79), LV end-diastolic volume (220+/-70 versus 228+/-57 mL, P=0.68),

35 +/- 72 to 145 +/- 23 ml, p < 0.001), LV end-diastolic volume (242 +/- 85 ml to 212 +/- 63 ml, p < 0.

36 lar [LV] ejection fraction 23 +/- 9%, LV end-diastolic volume 275 +/- 127 ml) underwent evaluation.

37 22 years were inversely associated with end-diastolic volume (-3.0 mL per unit mean hemoglobin A(1c)

38 (mean LV ejection fraction 22 +/- 7%, LV end-diastolic volume 323 +/- 140 ml, 40% ischemic) had exper

39 elated to reductions in left ventricular end-diastolic volume (-4.1 ml; 95% confidence interval [CI],

40 ; p = 0.002), and a trend toward reduced end-diastolic volume (-4.6 ml [95% CI -10.4 to 1.1]; p = 0.1

41 (94% versus 57%) and less LV dilatation (end-diastolic volume, 46+/-8 versus 85+/-32 microL), mechani

42 5) greater reduction in left ventricular end-diastolic volume (-49+/-16% versus -35+/-20%), left vent

43 val, -11.57 to -6.25; P<0.00001), and LV end-diastolic volume (-5.23 mL; 95% confidence interval, -7.

44 r age was associated with lower volumes (end-diastolic volume, -5 mLdecade; end-systolic volume, -3 m

45 Changes in end-diastolic volume (-6+/-14 versus -4+/-16 mL; P=0.67) at

46 ficant attenuation in the increase in LV end-diastolic volume (64.8 mL [60.7-71.3] to 83.5 mL [74.7-9

47 6+/-4.3 cm(2); P=0.02), and left atrium (end-diastolic volume, 65+/-19 versus 72+/-19; P=0.02).

48 s in the highest quintile had smaller LV end-diastolic volumes (68+/-13 versus 58+/-12 mL/m(2); P<0.0

49 in; p = 0.03), but no difference in peak end-diastolic volume (77 +/- 18 ml vs. 77 +/- 17 ml; p = 0.5

50 associated with a small right ventricle (end diastolic volume, 79.8+/-13.2 versus 88.5+/-11.8 mL/m(2)

51 more than mild FMR despite increased LV end-diastolic volume (82 +/- 22, 86 +/- 23, and 51 +/- 12 cm

52 rophy was driven by LV dilation (DeltaLV end-diastolic volume, 9+/-3 mL/m(2); P=0.004) with stable LV

53 : -4.3 [11.3] versus 7.4 [11.8], P=0.02; end-diastolic volume: -9.1 [14.9] versus 7.4 [15.8], P=0.02)

54 V ejection fraction and left ventricular end-diastolic volume, although correlation coefficients were

55 LV end-diastolic volume, an index of LV remodeling, increased b

56 On echocardiography, left ventricular end diastolic volume and deceleration time improved with 1.5

57 the right ventricle were used to measure end-diastolic volume and ejection fraction.

58 results showed a decrease of indexed RV end-diastolic volume and end-systolic volume (98 ml/m(2) to

59 RV volumes (end-diastolic volume and end-systolic volume), stroke volume

60 women were classified as having abnormal end-diastolic volume and internal diameter by ASE 2015 cutof

61 d was highly reflective of changes in LV end-diastolic volume and LA pressure.

62 , including structural (left ventricular end-diastolic volume and left ventricular ejection fraction)

63 placebo did not change left ventricular end-diastolic volume and left ventricular mass nor did it im

64 ated the progressive deterioration in LV end-diastolic volume and LV end-systolic volume.

65 s the change in indexed left ventricular end diastolic volume and LVEF.

66 Indexed left ventricular end-diastolic volume and mass correlated with O2max (r=0.45,

67 ); P<0.001) and indexed left ventricular end-diastolic volume and right ventricular end-diastolic vol

68 During PEI, LV end-diastolic volume and stroke volume were increased in bot

69 and G6PDX mice but significantly greater end diastolic volume and wall thinning in G6PDX mice.

70 n in CHF rats increased cardiac systolic and diastolic volumes and further increased the elevated LVE

71 ate and midterm reductions in indexed RV end-diastolic volumes and RV end-systolic volumes (RVESVi) (

72 es in left and right ventricular masses, end-diastolic volume, and diastolic dysfunction, and an incr

73 We quantified associations with RV mass, end-diastolic volume, and ejection fraction after control fo

74 +/- 13 years of age), noncontrast 3D EF, end-diastolic volume, and end-systolic volume had significan

75 ere directly correlated with LV mass, LV end-diastolic volume, and left atrial dimension.

76 asures of left ventricular (LV) mass, LV end-diastolic volume, and left atrial dimension.

77 6-minute walk distance, left ventricular end-diastolic volume, and left ventricular ejection fraction

78 ographic data, risk factors for scar, LV end-diastolic volume, and LV mass.

79 vascular risk factors, calcium score, LV end-diastolic volume, and mass in addition to resting heart

80 ociated with preserved stroke volume, LV end-diastolic volume, and mass/volume ratio as measured by c

81 ffective regurgitant orifice, indexed LV end-diastolic volume, and right ventricular systolic pressur

82 Before training, Vo(2)max, LV mass, LV end-diastolic volume, and stroke volume were significantly s

83 an values for the left ventricular mass, end diastolic volume, and stroke volume, but not with ejecti

84 left ventricular ejection fraction, systolic/diastolic volumes, and wall motion indexes.

85 Improvement in end-diastolic volume at 1 year was predictive of subsequent

86 t relationship between GLS groups and LV end-diastolic volume at 3 and 6 months (adjusted for clinica

87 ependently of load/extrinsic forces, the end-diastolic volume at a common end-diastolic pressure on t

88 ed LV compliance, measured as reduced LV end-diastolic volume at an idealized LV end-diastolic pressu

89 ric remodeling characterized by lower LV end-diastolic volume (beta=-0.21), higher concentricity (bet

90 beta=-0.02/%; P=0.015), left ventricular end-diastolic volume (beta=0.01/mL; P<0.0001), and left vent

91 aneous fat was associated with higher LV end-diastolic volume (beta=0.48), reduced concentricity (bet

92 r end-diastolic volume (left ventricular end-diastolic volume/body surface area, 104+/-13 and 69+/-18

93 /-18 mL/m(2); P<0.001; right ventricular end-diastolic volume/body surface area, 110+/-22 and 66+/-16

94 point variables for primary outcomes: LV end-diastolic volume/body surface area, LV ejection fraction

95 on fraction (P<0.01), reduced stroke and end-diastolic volumes (both P<0.001), decreased peak E' velo

96 [7.3-28.5], then decreased by 14 months (end-diastolic volume/BSA(1.3), end-systolic volume/BSA(1.3),

97 higher ratio of left ventricular mass to end-diastolic volume (by 8%).

98 to -0.23; P=0.028) and left ventricular end-diastolic volume (coefficient, 7.85; 95% confidence inte

99 The 3DE end-diastolic volume correlated well (r=0.96) but was smalle

100 ndices ([RA+aRV]/[fRV+LA+LV]) and fRV/LV end-diastolic volume corresponded only to some parameters.

101 In both men and women, LV end-diastolic volume decreased (-9.8 and -13.3 mL per decade

102 Left ventricular end-diastolic volume decreased (from 125.1 +/- 40.1 ml to 10

103 iac index increased and left ventricular end-diastolic volume decreased significantly only with 1.5 m

104 odeling was defined as an increase in LV end-diastolic volume (DeltaEDV>0) between discharge and foll

105 significant increase in left ventricular end-diastolic volume, demonstrating increased blood flow and

106 e mean left ventricular end systolic and end diastolic volumes did not differ between the groups.

107 mL of CHAM (n=5) or saline (n=13) and LV end-diastolic volume (EDV) and MMP/TIMP profiles in the MI r

108 mall difference in left ventricular (LV) end-diastolic volume (EDV) and thus, LV stroke volume and EF

109 13.0 mL; P < 0.0001), difference between end-diastolic volume (EDV) at rest and stress (DeltaEDV[stre

110 n (LVEF), end systolic volume (ESV), and end diastolic volume (EDV) using 4 different commercial soft

111 ft ventricular ejection fraction (LVEF), end-diastolic volume (EDV), and end-systolic volume (ESV) ar

112 d PET images, LV ejection fraction (EF), end-diastolic volume (EDV), and end-systolic volume (ESV) we

113 tensive versus conventional treatment in end diastolic volume (EDV), end systolic volume, stroke volu

114 al function was assessed by measuring LV end-diastolic volume (EDV), end-systolic volume (ESV), and e

115 erall bias and limits of agreement of LV end-diastolic volume (EDV), end-systolic volume (ESV), and E

116 yzed to provide the reference limits for end-diastolic volume (EDV), end-systolic volume (ESV), eject

117 eters, including ejection fraction (EF), end-diastolic volume (EDV), end-systolic volume (ESV), strok

118 End-diastolic volume (EDV), end-systolic volume (ESV), strok

119 ventricular ejection fraction (LVEF) and end-diastolic volume (EDV).

120 c (ePAD) pressures and an increase in LV end diastolic volume (EDV).

121 Filling was assessed by changes in LV end-diastolic volume (EDV; conductance catheter technique).

122 LV end-diastolic volumes (EDV) were measured by echocardiograph

123 fication based on LV dilatation (high LV end-diastolic volume [EDV] index) and concentricity (mass/en

124 ar events and a dilated LV (increased LV end-diastolic volume [EDV] indexed to body surface area) at

125 diographic measures of EF: cardiac size (end-diastolic volume [EDV]); contractile function (the end-s

126 End-systolic and end-diastolic volumes, ejection fraction, and left ventricle

127 The mean LV end-diastolic volume, end-systolic volume, and ejection frac

128 efficacy end points included changes in end-diastolic volume, end-systolic volume, and ejection frac

129 However, changes in end-diastolic volume, end-systolic volume, and LVEF did not

130 Biventricular end-diastolic volume, end-systolic volume, stroke volume, an

131 demonstrated increasing left ventricular end-diastolic volumes, end-systolic volumes, stroke volumes,

132 justment for city attenuated the RV mass/end-diastolic volume findings.

133 med to compare stroke, end-systolic, and end-diastolic volumes for the left ventricle (LV) and the ri

134 ter than 20% relative increase in the LV end-diastolic volume from 1 to 12 wk (percentage injected do

135 8 mm Hg (p = 0.016) and a decrease in LV end-diastolic volume from 172 +/- 37 ml to 158 +/- 38 ml (p

136 ow-up documented a stepwise reduction in end-diastolic volume (from 147 mL [IQR, 95-191 mL] to 127 mL

137 >/=10 U, combined with a decrease in LV end-diastolic volume >/=10% at follow-up.

138 tion for heart failure, left ventricular end-diastolic volume >/=125 mL/m(2), and left atrial volume

139 fined as an increase in left ventricular end-diastolic volume >/=20% at 6 months.

140 -losing enteropathy, ventricular indexed end-diastolic volume >125 mL/body surface area raised to the

141 CMR-derived left ventricular end-diastolic volume >246 mL had good, although lower, discr

142 achycardia, low blood pressure, enlarged end-diastolic volume, high ejection fraction, and high cardi

143 encing MACE showed higher left ventricle end-diastolic volume, higher left ventricle end-systolic vol

144 strain (HR, 1.63; P=0.005), exercise LV end-diastolic volume (HR, 1.38; P=0.048), and resting RV str

145 ase and dilatation, but left ventricular end-diastolic volume improved because of reduced blood retur

146 line to 12 months, left ventricular (LV) end-diastolic volume improved from 161 +/- 56 ml to 143 +/-

147 Left ventricular end-diastolic volume improved from 172 ml to 140 ml and end-

148 e MRI demonstrated a >40% increase in LV end-diastolic volume in both groups, consistent with a failu

149 ion initially induced an expansion of LV end-diastolic volume in IPAH (+7%; P < 0.05), whereas end-di

150 P<0.001), BMI was not correlated with RV end-diastolic volume in men (R=0.04; P=0.51).

151 167), and after replacing LV mass by LV end-diastolic volume in the regression models.

152 recognized for stroke, end-systolic, or end-diastolic volumes in either the LV or the RV.

153 ntal value of LVGLS for prediction of LV end-diastolic volume increase (0.14 [95% confidence interval

154 (2), P<0.0001), whereas left ventricular end diastolic volume increased (66+/-12 to 73+/-13 mL/m(2),

155 Cine-magnetic resonance-derived LV end-diastolic volume increased 2-fold (P=0.005), and LV ejec

156 After intervention, LV mass and end-diastolic volume increased and exercise capacity improve

157 Right ventricular end diastolic volume increased by 49.4% after transannular p

158 trol animals, LV end-systolic volume and end-diastolic volume increased from 6 to 30 weeks (median an

159 LV end diastolic volume increased in all groups but was attenua

160 of trastuzumab, indexed left ventricular end diastolic volume increased in patients treated with peri

161 End-diastolic volume increased in placebo, but not in CDC-tr

162 Subsequently, RV end-systolic and end-diastolic volume increased more in both groups, resultin

163 the Frank-Starling relationship, greater end-diastolic volume increases ventricular output.

164 ery at 5 years, 90% for left ventricular end-diastolic volume index <100 mL/m(2) versus 48% for >/=10

165 scular lung water (<10 mL/kg) and global end-diastolic volume index (<850 mL/m) in the transpulmonary

166 greater improvement in left ventricular end-diastolic volume index (-26.2 versus -7.4 mL/m(2)), left

167 +/- 5% to 54 +/- 9%, p < 0.0001) and LV end-diastolic volume index (108 +/- 28 ml/m(2) to 78 +/- 24

168 The decrease in end-diastolic volume index (14+/-3) was different from that

169 shortening (B=-0.1; P=0.0001), lower LV end-diastolic volume index (B=0.6; P=0.0001), and lower LV e

170 01), obesity (beta=1.3 mL/m(2), P<0.01), end-diastolic volume index (beta=0.4 mL/m(2), P<0.0001), Chi

171 vs 3.8 L/min/m(2); P = .001), and global end-diastolic volume index (GEDVI) (726 vs 775 mL/m(2); P =

172 come was mean change from baseline in LV end-diastolic volume index (LVEDVI) at 6 months.

173 ll, stable reduction in left ventricular end-diastolic volume index (P<0.001), with a concomitant sma

174 stable increase in the right ventricular end-diastolic volume index (P<0.001).

175 Stroke volume index (P=0.015) and end-diastolic volume index (P=0.001) decreased during exerci

176 e index (P<0.0001), and left ventricular end-diastolic volume index (P=0.0015).

177 Patients with a right ventricular end-diastolic volume index above 150 ml/m(2) and peak exerci

178 Patients with a right ventricular end-diastolic volume index above 150 ml/m(2) and peak exerci

179 n change from baseline right ventricular end-diastolic volume index and a 429 ml (P < 0.001) reductio

180 re severe renal dysfunction, yet smaller end-diastolic volume index and cardiac output and increased

181 Left ventricle end-diastolic volume index and end-systolic volume index wer

182 reater ECE is associated with reduced LV end-diastolic volume index and LV end-diastolic mass index i

183 ed in a greater reduction of LV systolic and diastolic volume index and LV mass index as compared wit

184 The left ventricular end-diastolic volume index decreased (90.1 +/- 28.2 ml/m(2)

185 Resting left ventricular end-diastolic volume index increased (P=0.001) within the si

186 Right ventricular end-diastolic volume index measured by means of cardiovascul

187 CMR-derived end-diastolic volume index showed a weaker association with

188 a greater extent of LGE and a higher LV end-diastolic volume index than other groups, but levels of

189 hange from baseline in right ventricular end-diastolic volume index versus placebo.

190 All patients were hypovolemic (global end-diastolic volume index<680 mL/m) on enrollment.

191 ral venous pressure, 0.56 for the global end-diastolic volume index, and 0.64 for the left ventricula

192 c of the central venous pressure, global end-diastolic volume index, and left ventricular end-diastol

193 infarct volume, LV ejection fraction, LV end-diastolic volume index, and LV end-systolic volume index

194 ympathetic denervation, left ventricular end-diastolic volume index, creatinine, and no angiotensin i

195 re no significant differences in mean LV end-diastolic volume index, end-systolic volume index and LV

196 tors (age, body mass index, diabetes, LV end-diastolic volume index, LGE, EF) (hazard ratio = 2.051 p

197 87.3+/-18.7 mL/m(2) at 3-year follow-up (end-diastolic volume index, P=0.0056; end-systolic volume in

198 volume variation monitoring, and global end-diastolic volume index.

199 tic peptide, and larger left ventricular end-diastolic volume index.

200 olume index, and larger left ventricular end-diastolic volume index.

201 (LV mass index: r = 0.35, p < 0.0001; LV end-diastolic volume index: r = 0.43, p < 0.0001) and LVEF (

202 imal NC/C ratio and preceding changes in end-diastolic volume indexed (EDVi) to body surface area and

203 In the 50-mg group, end-systolic and end-diastolic volume indexes decreased relative to baseline

204 nt improvements in both LV end-systolic and -diastolic volume indexes.

205 ) ejection fraction, LV end-systolic and end-diastolic volumes, infarct size, and major adverse cardi

206 CMR-derived ventricular indexed end-diastolic volume is an independent predictor of transpla

207 s defined as (LA end-systolic volume--LA end-diastolic volume) / LA end-systolic volume.

208 d-diastolic volume and right ventricular end-diastolic volume (left ventricular end-diastolic volume/

209 nderwent TTE and CMR, and left ventricle end-diastolic volume, left ventricle end-systolic volume, an

210 provements in LV end-systolic volume, LV end-diastolic volume, left ventricular ejection fraction, le

211 rcentage of myocardium, left ventricular end-diastolic volume, left ventricular end-systolic volume,

212 e by CMR is superior to left ventricular end-diastolic volume, left ventricular end-systolic volume,

213 s in ejection fraction, left ventricular end-diastolic volume, left ventricular end-systolic volume,

214 ctors of death were: larger left ventricular diastolic volume, left ventricular systolic volume and E

215 t in TAVR patients, and low left ventricular diastolic volume, low stroke volume, and greater severit

216 s; and smaller left ventricular systolic and diastolic volumes, low stroke volume, smaller EOA, and p

217 e predicted by a preoperative indexed RV end-diastolic volume </=158 mL/m(2) and RVESVi </=82 mL/m(2)

218 A relatively aggressive PVR policy (end diastolic volume <150 mL/m(2)) leads to normalization of

219 reduction in LV wall stress, reduced LV end-diastolic volume, LV end-systolic volume and increase in

220 percent change in left ventricular (LV) end-diastolic volume, LV end-systolic volume, LV ejection fr

221 LV function, who showed reduction in LV end-diastolic volume, LV wall stress, no change in LV end-sy

222 thickening fraction, LV end-systolic and end-diastolic volumes, LV ejection fraction) and hemodynamic

223 ion fraction (LVEF) and left ventricular end diastolic volume (LVEDV) on cardiac magnetic resonance i

224 ed as percent change in left ventricular end-diastolic volume (LVEDV), was analyzed in 3 prespecified

225 ume [EDV] index) and concentricity (mass/end-diastolic volume [M/EDV](2/3)) in hypertensive patients.

226 normalization of LV stroke volume and LV end-diastolic volume/mass ratio, there was a persistent sign

227 CMR showed higher left ventricle end-diastolic volume (mean difference: 43+/-22.5 mL), higher

228 decreased the temporal variability of LV end-diastolic volume measurements by the 2D biplane method.

229 right/left-volume index was defined from end-diastolic volume measurements in CMR: total right/left-v

230 reased from pre-Norwood to pre-stage II (end-diastolic volume [milliliters]/body surface area [BSA](1

231 nance (1.5 T) to measure RV mass (g), RV end-diastolic volume (mL), RV mass/volume ratio, and LV dias

232 Left ventricular systolic and diastolic volumes, New York Heart Association functional

233 .002), exercise indexed left ventricular end-diastolic volume (odds ratio, 1.04; P=0.026), exercise e

234 c distensibility index (ratio of ePAD to end-diastolic volume) of 0.11+/-0.06 mm Hg/mL.

235 t ventricular (LV) ejection fraction, LV end-diastolic volume, or New York Heart Association (NYHA) f

236 ; 95% confidence interval, -0.2 to 2.1), end-diastolic volume, or systolic volume were observed compa

237 ejection fraction (p = 0.002), increased end-diastolic volume (p < 0.001), increased mass(i) (p < 0.0

238 t not CT-significantly overestimated the end-diastolic volume (p < 0.001), whereas 2D Echo and 3D Ech

239 redictor of increase in left ventricular end-diastolic volume (p < 0.001).

240 y associations with baseline and DeltaLV end diastolic volume (P<0.0001 for each) and not wall thickn

241 ere observed for the ratio of LV mass to end-diastolic volume (P=0.02) and with hyperinflation measur

242 on left ventricular (LV) mass (P=0.016), end-diastolic volume (P=0.029), and end-systolic volumes (P=

243 02), and increased ventricular dilation (end-diastolic volume, P=0.01).

244 (Delta ejection fraction: P<0.04, Delta end-diastolic volume: P<0.02, Delta end-systolic volume: P<0

245 and IMH correlated with the change in LV end-diastolic volume (Pearson's rho of 0.64, 0.59, and 0.66,

246 Indexed RV end-diastolic volume plotted against neoaortic stroke volume

247 nd-systolic volumes (RVESVi) (indexed RV end-diastolic volume pPVR versus immediately after PVR versu

248 ic pressure (LVEDP) and left ventricular end-diastolic volume (preload) in CHF rats, which was not ob

249 ial lidocaine paradoxically decreased LV end-diastolic volume (preload) in CHF rats, which was not ob

250 c geometry, defined by higher LV mass to end-diastolic volume quartiles, were associated with higher

251 s a significant predictor of increase in end-diastolic volume (r = 0.51, P < 0.05).

252 ction correlated closely with indexed RV end-diastolic volume (R = 0.79, p < 0.001) and modestly with

253 95), end-systolic volume (r = 0.93), and end-diastolic volume (r = 0.90), and slightly lower correlat

254 ne strongly correlated with reduction of end diastolic volume (r(2)=0.69, P=0.04) and end systolic vo

255 chanical dyssynchrony correlated with RV end-diastolic volume (r=0.39; P=0.03).

256 arlier and accounted for the increase in end-diastolic volume (r=0.65), and VO2max (r=0.74, both p<0.

257 overall explained variance for volumes (end-diastolic volume, R(2)=0.43; end-systolic volume, R(2)=0

258 ional decline in the RV/left ventricular end-diastolic volume ratio (P=0.05) and trended toward impro

259 % CI: 0.04, 0.36)] and a greater RV mass/end-diastolic volume ratio conditional on LV parameters.

260 ociated with greater RV mass and RV mass/end-diastolic volume ratio conditional on the LV; however, a

261 ociated with greater RV mass and RV mass/end-diastolic volume ratio relative to the LV.

262 ures of PM2.5, whereas those for RV mass/end-diastolic volume ratio were attenuated.

263 e to assess LV remodeling (LV mass-to-LV end diastolic volume ratio), function, tissue characterizati

264 e directly associated with LV mass to LV end-diastolic volume ratio, a marker of cardiac remodelling

265 of LVH and abnormal/normal LV mass to LV end-diastolic volume ratio.

266 who developed asymmetry, the wall thickness/diastolic volume ration remained normal (0.09+/-0.02 mmm

267 , preload-recruitable stroke work, dP/dt-end-diastolic volume relation) were significantly depressed,

268 n LV mass index and the ratio of LV mass:end-diastolic volume (relative wall mass).

269 ercentiles of LV end systolic volume, LV end diastolic volume, relative wall and septal thickness, LV

270 dels showed that the addition of indexed end-diastolic volume resulted in a significantly improved en

271 57% increase in LV mass (no change in LV end diastolic volume, resulting in an increase in the LV mas

272 easurements of left ventricular end-systolic/diastolic volumes revealed that the least amount of vent

273 Right ventricular end-diastolic volume (RVEDV) was reduced in COPD compared wi

274 f 15 to 30 mL in 3DTTE right ventricular end-diastolic volume; sample sizes were 2x to 2.5x those req

275 re inversely related to left ventricular end-diastolic volume, stroke volume, and cardiac output amon

276 Systolic function (EF, end-systolic and end-diastolic volumes, stroke volumes) was not different in

277 relaxation velocity and left ventricular end diastolic volume to produce higher left ventricle maximu

278 tion was 34+/-17%, and right ventricular end-diastolic volume was 114+/-39 cc/m(2).

279 LV end-diastolic volume was 2-fold (P=0.005) higher in MR patie

280 nd treatment group; each 10% decrease in end-diastolic volume was associated with a 40% reduction in

281 LA end-diastolic volume was increased in patients with mitral r

282 V mass and left atrial dimension, but LV end-diastolic volume was not consistently associated with ad

283 In CAP-treated dogs, end-diastolic volume was unchanged; ejection fraction increa

284 rct size, LV end-systolic volume, and LV end-diastolic volume were analyzed with random-effects meta-

285 roke volume (SV), ejection fraction, and end-diastolic volume were assessed by echocardiography.

286 rct size, LV end-systolic volume, and LV end-diastolic volume were estimated with random-effects meta

287 ulmonary capillary wedge pressure and LV end-diastolic volume were measured at baseline, during decre

288 By CMR, right ventricular end-diastolic volumes were 118 +/- 31 cc/m(2), 108 +/- 22 cc

289 End-systolic and end-diastolic volumes were calculated using blood pool image

290 LV end-systolic and end-diastolic volumes were defined with a phase analysis and

291 Both the LV and RV end-systolic and LV end-diastolic volumes were increased compared with reference

292 lmonary capillary wedge pressures and LV end-diastolic volumes were measured at baseline, during decr

293 low-up, left atrial and left ventricular end-diastolic volumes were significantly more reduced in pat

294 iated with greater RV mass and larger RV end-diastolic volume with or without further adjustment for

295 ions of RV mass and, to a lesser extent, end diastolic volume with PM10-2.5 mass among susceptible po

296 decreased left ventricular (LV) systolic and diastolic volumes with little effect on LV end-diastolic

297 ocardiogram, the median left ventricular end-diastolic volume z score was +1.7 (range, -1.3 to +8.2),

298 ormal values, left and right ventricular end-diastolic volume z scores were mildly enlarged (0.48+/-1

299 LV end-diastolic volume z-score increased (P=0.02), LV mass was

300 The mean right ventricular (RV) end-diastolic volume z-score was 1.8 +/- 1.9; RV dilation (z