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1 differentiate to neutrophils by exposure to dibutyryl-cyclic AMP.
2 ther insulin (5 microM) nor a combination of dibutyryl cyclic AMP (5 mM) and the phosphodiesterase in
5 rphine's modulatory function, the effects of dibutyryl cyclic AMP and forskolin were investigated.
6 he ability to extend neurites in response to dibutyryl cyclic AMP and to express various antigens cha
9 yclic AMP elevation with either forskolin or dibutyryl cyclic AMP, and this increase was inhibitable
11 ssing c-Myc with the growth inhibitory agent dibutyryl cyclic AMP (Bt2cAMP) arrested these cells G1,
12 in the livers of 19-day fetal rats, whereas dibutyryl cyclic AMP (Bt2cAMP) increases the expression
13 ) M PGE2, 10(-6) M misoprostol, and 10(-4) M dibutyryl cyclic AMP, but not by the EP3R agonist sulpro
14 ll lines to a NE phenotype can be induced by dibutyryl cyclic AMP (cAMP), suggesting that physiologic
16 Phorbol 12-myristate 13-acetate (PMA) and dibutyryl cyclic AMP (dbcAMP) increased expression of th
17 with the cell permeable cyclic AMP analogue dibutyryl cyclic AMP (dbcAMP) resulted in a strong upreg
18 ith either phorbolmyristate acetate (PMA) or dibutyryl cyclic AMP (dbcAMP) to activate either protein
20 2 (Mrp2) and the effect of pretreatment with dibutyryl-cyclic AMP (DBcAMP; 20 micromol/kg) on these m
22 protein kinase (PKA), such as PGE2 itself or dibutyryl cyclic AMP, did not modify PGE2 production by
24 rol cultures after 3 weeks of treatment with dibutyryl-cyclic AMP, medroxyprogesterone acetate, and E
27 22536 was reversed by prostaglandin E(2) and dibutyryl cyclic AMP, respectively, demonstrating that i
28 nhibitors and was unaffected by brefeldin A, dibutyryl cyclic AMP, taurocholate, or PI 3-kinase inhib
30 the increase of intracellular cyclic AMP by dibutyryl cyclic AMP were able to inhibit cytotoxicity d
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