ライフサイエンスコーパス: diesel exhaust

1 for secondary organic aerosol formation from diesel exhaust.

2 myocytes as a result of in utero exposure to diesel exhaust.

3 susceptibility through prenatal exposure to diesel exhaust.

4 y childhood exposures, including exposure to diesel exhaust.

5 ic material (ACM), that were challenged with diesel exhaust.

6 ) formed from the photochemical oxidation of diesel exhaust.

7 d at rest in a randomized, balanced order to diesel exhaust (200 mug/m(3) particulate matter with an

8 se in the ischemic burden during exposure to diesel exhaust (-22+/-4 vs. -8+/-6 millivolt seconds, P<

9 tudy, 30 healthy men were exposed to diluted diesel exhaust (300 microg/m3 particulate concentration)

10 exposed, in two separate sessions, to dilute diesel exhaust (300 mug per cubic meter) or filtered air

11 nvestigated electrode-assisted deposition of diesel exhaust aerosol (DEA) on human lung epithelial ce

12 In utero exposure to diesel exhaust air pollution has been associated with in

13 Exposure to allergen alone, diesel exhaust alone, or allergen and diesel exhaust tog

14 and suspected human carcinogen identified in diesel exhaust and air pollution.

15 y to this environmental contaminant found in diesel exhaust and ambient air pollution.

16 late carbon is obtained by increasing mobile diesel exhaust and area-source particulate carbon emissi

17 the increase was similar during exposure to diesel exhaust and exposure to filtered air (P=0.67).

18 cardiac myocytes after in utero exposure to diesel exhaust and found that the promoter for Mir133a-2

19 a, we estimated SOA formation potential from diesel exhaust and predict the contribution from UCM vap

20 en the same lung was exposed to allergen and diesel exhaust but separated by approximately 4 weeks, s

21 +/- 2% of gasoline exhaust and 26 +/- 1% of diesel exhaust by mass.

22 utagenicity and the genotoxicity of complete diesel exhaust compared to an organic exhaust particle e

23 Under laboratory-simulated diesel exhaust conditions, this mixed-phase oxide materi

24 sly demonstrated that short-term exposure to diesel exhaust (DE) for 1 h induced a marked leukocytic

25 nt mice were exposed to filtered air (FA) or diesel exhaust (DE) on embryonic days (E) 9-17.

26 We hypothesized that a single exposure to diesel exhaust (DE) would increase the risk of adverse c

27 Diesel exhaust (DE), in addition to generating other pol

28 ne whether exposure to allergen, exposure to diesel exhaust (DE), or coexposures modulate miRNA, gene

29 Exposure to diesel exhaust did not aggravate preexisting vasomotor d

30 Air pollution, primarily consisting of diesel exhaust emissions, has increased at a similar rat

31 hose observed in laboratory studies of fresh diesel exhaust emissions.

32 Diesel exhaust enhances allergic inflammation, and pollu

33 stantially reduce SOA formation potential of diesel exhaust, except at low speed operations.

34 ht to investigate the effect of allergen and diesel exhaust exposure on bronchial epithelial DNA meth

35 With emerging evidence that diesel exhaust exposure poses distinct risks to human he

36 experimental evidence on cigarette smoke and diesel exhaust exposure.

37 le organic compounds (VOCs) were measured in diesel exhaust from three heavy-duty trucks equipped wit

38 Diesel exhaust gaseous sulphuric acid (GSA) concentratio

39 e and compare ozone production potentials of diesel exhaust, gasoline exhaust, and nontailpipe gasoli

40 Diesel exhaust has been considered to be a probable lung

41 n an urban environment, inhalation of dilute diesel exhaust impairs 2 important and complementary asp

42 ganic exhaust particle extract from the same diesel exhaust in a bacterial and a eukaryotic system, t

43 We confirmed that short-term exposure to diesel exhaust in healthy subjects is associated with ac

44 er-controlled exposure study to allergen and diesel exhaust in humans, and measured single-site (CpG)

45 We conducted a controlled exposure to dilute diesel exhaust in patients with stable coronary heart di

46 +/-3 years) were exposed to filtered air and diesel exhaust in the presence or absence of a particle

47 adult hearts from mice that were exposed to diesel exhaust in utero and that have subsequently under

48 No association was found between diesel exhaust inhalation and flow-mediated dilation.

49 study aim was to investigate the effects of diesel exhaust inhalation on vascular and endothelial fu

50 Compared with filtered air, diesel exhaust inhalation was associated with reduced va

51 Diesel exhaust inhalation, which is the model traffic-re

52 from gasoline and diesel vehicles, and find diesel exhaust is seven times more efficient at forming

53 ominant nitropolycyclic hydrocarbon found in diesel exhaust, is a mutagen and tumorigen.

54 Short-term exposure to diesel exhaust leads to increased eosinophil activation

55 s of 1-nitropyrene (1-NP), a highly specific diesel exhaust marker, at the neighborhood scale.

56 Exposure to diesel exhaust may play a role in the development and pr

57 Sulfur driven diesel exhaust nucleation particle formation processes w

58 ow or inflammatory markers after exposure to diesel exhaust or air.

59 studies have reported an association between diesel exhaust particle (DEP) exposure, allergic sensiti

60 5-Aza-2'-deoxycytidine and diesel exhaust particle exposure in human bronchial epit

61 ell-established allergic adjuvant effects of diesel exhaust particle exposure.

62 in the pathogenesis of lung injury caused by diesel exhaust particles (DEP) and bacterial lipopolysac

63 Diesel exhaust particles (DEP) and their organic constit

64 The redox activity of diesel exhaust particles (DEP) collected from a light-du

65 Diesel exhaust particles (DEP) contain organic chemicals

66 Ambient PM and diesel exhaust particles (DEP) contain redox cycling org

67 of the mice were co-exposed to mycotoxins or diesel exhaust particles (DEP) during pregnancy.

68 Inhaled diesel exhaust particles (DEP) exert proinflammatory eff

69 Increased exposure to air pollutants such as diesel exhaust particles (DEP) has been proposed as one

70 Diesel exhaust particles (DEP) have strong, selective Th

71 cies are involved in the adjuvant effects of diesel exhaust particles (DEP) in a murine OVA sensitiza

72 demonstrate that methanol extracts made from diesel exhaust particles (DEP) induce apoptosis and reac

73 usly shown that in vivo nasal challenge with diesel exhaust particles (DEP) induces both quantitative

74 dies have correlated inflammatory effects of diesel exhaust particles (DEP) with its organic constitu

75 We hypothesized that exposure to diesel exhaust particles (DEP), a major component of urb

76 effects of particulate pollutants, including diesel exhaust particles (DEP), are related to their con

77 nce that particulate air pollutants, such as diesel exhaust particles (DEP), potentiate chronic infla

78 Ambient particulate matter, including diesel exhaust particles (DEP), promotes the development

79 ncluding ambient particulate matter (PM) and diesel exhaust particles (DEP).

80 encephalic neuron-glia cultures treated with diesel exhaust particles (DEP; 0.22 microM) (5-50 microg

81 ntly exposed via oropharyngeal aspiration to diesel exhaust particles (DEP; 50 mug x 6 doses) or vehi

82 Diesel exhaust particles (DEPs) and SHS can interact wit

83 Diesel exhaust particles (DEPs) are a major component of

84 Diesel exhaust particles (DEPs) are a major component of

85 Oxidant pollutants such as diesel exhaust particles (DEPs) can initiate and exacerb

86 The inhalation of diesel exhaust particles (DEPs) is associated with incre

87 re given repeated intranasal applications of diesel exhaust particles (DEPs) or PBS.

88 Here, we report that diesel exhaust particles (DEPs), a major constituent of

89 pollution particulate matter, predominantly diesel exhaust particles (DEPs), increases the risk of a

90 xposure to environmental pollutants, such as diesel exhaust particles (DEPs).

91 of traffic-related particulate matter (e.g., diesel exhaust particles [DEPs]) is associated with acut

92 /L [-0.2-19.6], p=0.01) after challenge with diesel exhaust particles and allergens.

93 The diesel exhaust particles enhancement was largest in pati

94 mon lipophilic pollutants benzo[a]pyrene and diesel exhaust particles impact on the activation of lip

95 y with allergen alone and with allergen plus diesel exhaust particles in a randomised order at separa

96 Insights into the role of diesel exhaust particles in patients with severe asthma

97 f temperature on the number concentration of diesel exhaust particles in the nucleation mode in a pre

98 STM1 and GSTP1 modify the adjuvant effect of diesel exhaust particles on allergic inflammation.

99 re key regulators of the adjuvant effects of diesel exhaust particles on allergic responses.

100 4.7] vs 7.4 nmol/L [1.2-12.3], p=0.02) after diesel exhaust particles plus allergen challenge.

101 Similarly, diesel exhaust particles showed a marginal inhibitory ef

102 The model pollutant, diesel exhaust particles, can participate with allergens

103 t has been described that exposure to ozone, diesel exhaust particles, or tobacco smoke exacerbates a

104 Examples of UFPs are diesel exhaust particles, products of cooking, heating,

105 anin in conjunction with adjuvant intranasal diesel exhaust particles.

106 species and detoxify xenobiotics present in diesel exhaust particles.

107 nasal allergic responses in the presence of diesel exhaust particles.

108 The particle trap markedly reduced diesel exhaust particulate number (from 150 000 to 300 0

109 d the inflammatory response to inhalation of diesel exhaust particulates (DEP) in normal volunteers.

110 We investigated how diesel exhaust particulates (DEPs) aggravate asthma-like

111 In utero exposure to diesel exhaust particulates is associated with an altere

112 uated, with rank-ordered responses of CFA1 > diesel exhaust PM > crystalline silica; TRP melastatin-8

113 an health, the need for fine-scale models of diesel exhaust pollutants is growing.

114 ent aftertreatment system for the removal of diesel exhaust pollutants.

115 hybrid modeling was successful in predicting diesel exhaust pollution at a very fine scale and identi

116 oraging for flowers; we investigated whether diesel exhaust pollution could interrupt these floral od

117 (NPAHs) to identify fine-scale gradients in diesel exhaust pollution in two Seattle, WA neighborhood

118 identified from oilseed rape, was exposed to diesel exhaust pollution.

119 Brief exposure to dilute diesel exhaust promotes myocardial ischemia and inhibits

120 Diesel exhaust-related vasoconstriction was primarily ob

121 Pretreatment with antioxidants augmented diesel exhaust-related vasoconstriction with a mean chan

122 Compared with filtered air, exposure to diesel exhaust resulted in a significant reduction in BA

123 age in activities that result in exposure to diesel exhaust, solvents, welding fumes, and other respi

124 alone, diesel exhaust alone, or allergen and diesel exhaust together (coexposure) led to significant

125 s show promise for a considerably lower-cost diesel exhaust treatment system.

126 The effective SOA yield of diesel exhaust was similar to that of unburned diesel fu

127 P<0.001) infusions 2 hours after exposure to diesel exhaust, which persisted at 6 hours.

128 ents were created in a chamber that combined diesel exhaust with an urban-like mixture.

129 rrhythmia during or after exposure to dilute diesel exhaust, wood smoke, ozone, concentrated ambient

130 Ignoring secondary chemistry from diesel exhaust would lead to underestimates of both orga