ライフサイエンスコーパス: diesel exhaust particle

1 anin in conjunction with adjuvant intranasal diesel exhaust particles.

2 species and detoxify xenobiotics present in diesel exhaust particles.

3 nasal allergic responses in the presence of diesel exhaust particles.

4 /L [-0.2-19.6], p=0.01) after challenge with diesel exhaust particles and allergens.

5 The model pollutant, diesel exhaust particles, can participate with allergens

6 studies have reported an association between diesel exhaust particle (DEP) exposure, allergic sensiti

7 in the pathogenesis of lung injury caused by diesel exhaust particles (DEP) and bacterial lipopolysac

8 Diesel exhaust particles (DEP) and their organic constit

9 The redox activity of diesel exhaust particles (DEP) collected from a light-du

10 Diesel exhaust particles (DEP) contain organic chemicals

11 Ambient PM and diesel exhaust particles (DEP) contain redox cycling org

12 of the mice were co-exposed to mycotoxins or diesel exhaust particles (DEP) during pregnancy.

13 Inhaled diesel exhaust particles (DEP) exert proinflammatory eff

14 Increased exposure to air pollutants such as diesel exhaust particles (DEP) has been proposed as one

15 Diesel exhaust particles (DEP) have strong, selective Th

16 cies are involved in the adjuvant effects of diesel exhaust particles (DEP) in a murine OVA sensitiza

17 demonstrate that methanol extracts made from diesel exhaust particles (DEP) induce apoptosis and reac

18 usly shown that in vivo nasal challenge with diesel exhaust particles (DEP) induces both quantitative

19 dies have correlated inflammatory effects of diesel exhaust particles (DEP) with its organic constitu

20 We hypothesized that exposure to diesel exhaust particles (DEP), a major component of urb

21 effects of particulate pollutants, including diesel exhaust particles (DEP), are related to their con

22 nce that particulate air pollutants, such as diesel exhaust particles (DEP), potentiate chronic infla

23 Ambient particulate matter, including diesel exhaust particles (DEP), promotes the development

24 ncluding ambient particulate matter (PM) and diesel exhaust particles (DEP).

25 encephalic neuron-glia cultures treated with diesel exhaust particles (DEP; 0.22 microM) (5-50 microg

26 ntly exposed via oropharyngeal aspiration to diesel exhaust particles (DEP; 50 mug x 6 doses) or vehi

27 Diesel exhaust particles (DEPs) and SHS can interact wit

28 Diesel exhaust particles (DEPs) are a major component of

29 Diesel exhaust particles (DEPs) are a major component of

30 Oxidant pollutants such as diesel exhaust particles (DEPs) can initiate and exacerb

31 The inhalation of diesel exhaust particles (DEPs) is associated with incre

32 re given repeated intranasal applications of diesel exhaust particles (DEPs) or PBS.

33 Here, we report that diesel exhaust particles (DEPs), a major constituent of

34 pollution particulate matter, predominantly diesel exhaust particles (DEPs), increases the risk of a

35 xposure to environmental pollutants, such as diesel exhaust particles (DEPs).

36 of traffic-related particulate matter (e.g., diesel exhaust particles [DEPs]) is associated with acut

37 The diesel exhaust particles enhancement was largest in pati

38 5-Aza-2'-deoxycytidine and diesel exhaust particle exposure in human bronchial epit

39 ell-established allergic adjuvant effects of diesel exhaust particle exposure.

40 mon lipophilic pollutants benzo[a]pyrene and diesel exhaust particles impact on the activation of lip

41 y with allergen alone and with allergen plus diesel exhaust particles in a randomised order at separa

42 Insights into the role of diesel exhaust particles in patients with severe asthma

43 f temperature on the number concentration of diesel exhaust particles in the nucleation mode in a pre

44 STM1 and GSTP1 modify the adjuvant effect of diesel exhaust particles on allergic inflammation.

45 re key regulators of the adjuvant effects of diesel exhaust particles on allergic responses.

46 t has been described that exposure to ozone, diesel exhaust particles, or tobacco smoke exacerbates a

47 4.7] vs 7.4 nmol/L [1.2-12.3], p=0.02) after diesel exhaust particles plus allergen challenge.

48 Examples of UFPs are diesel exhaust particles, products of cooking, heating,

49 Similarly, diesel exhaust particles showed a marginal inhibitory ef