ライフサイエンスコーパス: diet

1 cits caused by the consumption of a high-fat diet.

2 for 3 days followed by 14 days of customary diet.

3 PUFA diet, or 5) a high fat monounsaturated diet.

4 stent organic pollutants (POPs) in the human diet.

5 e risk of FGR, one of which is poor maternal diet.

6 nt source of Cu, Fe, Mg, and Zn to the human diet.

7 ity and insulin resistance under a lipogenic diet.

8 8 cups per day, as part of a weight-reducing diet.

9 crophage populations by the mutation and the diet.

10 adverse metabolic consequences of a high fat diet.

11 re undetectable or at low abundance in their diet.

12 e impact of CR in terms of optimal onset and diet.

13 and glucose tolerance on a regular chow (RC) diet.

14 ual to or higher than their exposure through diet.

15 two soybean oils, coconut oil, and a low-fat diet.

16 not influenced by a post-weaning obesogenic diet.

17 of these mice fed a control chow or high-fat diet.

18 se tolerance in mice fed with a calorie-rich diet.

19 d low-fat, plant-polysaccharide rich (LFPPD) diets.

20 bioassays employ carbohydrate-biased rearing diets.

21 dants in concentrations reflecting different diets.

22 e seen strong tendencies toward high-protein diets.

23 et (VLCD; 500 kcal/d) or a 12-wk low-calorie diet (1250 kcal/d) (WL period) with a subsequent 4-wk WS

24 enriched in palmitate) or a purified regular diet (16.9% kcal from fat) for 3, 6, 9 and 12 weeks.

25 he following: 1) a normal low fat (13% kcal) diet, 2) a low fat diet containing n-3 PUFAs, 3) a high

26 e, in wild-type C57BL/6J mice fed a high fat diet, 2-weeks supplementation with Lab4 probiotic consor

27 (WT/KO) dams were fed a control breeder chow diet (25% fat) or a semi-purified HFD (45% fat) 4 weeks

28 cheese diet were lower than after the butter diet (-3.3%, P < 0.05) but were higher than after the ca

29 Ptrend = 0.002] and 1.41 for early adulthood diet (95% CI, 1.11-1.78; Ptrend = 0.006) compared with w

30 opausal breast cancer of 1.35 for adolescent diet [95% confidence interval (95% CI), 1.06-1.73; Ptren

31 A high-fat diet accelerated stroke incidence.

32 Following 3 weeks diet adapting, a multi-dose intraperitoneal injections o

33 y bloodletting associated with lifestyle and diet advice (LFDA) to those of LFDA only.

34 x n = 29), diet (Diet n = 28), exercise plus diet (AED n = 29), or no-intervention (NI n = 29) groups

35 both maternal diet groups were fed the same diet after hatch.

36 ition from a milk-based diet to a chow-based diet after weaning.

37 d-type mice fed an unsupplemented ad libitum diet, age-associated hypomethylation was enriched at sup

38 Retinas of mice fed a high fat/sucrose diet also exhibited elevated levels of activated JNK as

39 On a high-fat diet, although no differences in body weight and composi

40 s and 935+/-329.9mg estimated by a duplicate diet analyses.

41 ated the results with the use of a duplicate diet analyses.

42 Diet analysis indicated that the consumption of invasive

43 a single amount of vitamin D (511 IU/d from diet and a cholecalciferol supplement) for 10 wk.

44 obially derived metabolites affected by both diet and antibiotic treatment, which conformed to previo

45 borative Cohort Study (1990-1994), the Malmo Diet and Cancer Study (1991-1996), and the Northern Swed

46 es from 1421 participants of the MDCS (Malmo Diet and Cancer Study).

47 While typically managed by gluten-free diet and dapsone, treatment of DH refractory to standard

48 Creatine levels are maintained by diet and endogenous synthesis from arginine and glycine.

49 We studied the effects of a diet and exercise weight-loss intervention on skeletal m

50 anthropometrics, cardiac and blood measures, diet and exercise, physical and mental health, medicatio

51 ral adipose tissue of TRPC1 KO mice fed a HF diet and exercised.

52 l changes place commensurate demands on good diet and health; and the adolescent phase of growth and

53 developed urinary metabolite models for each diet and identified the associated metabolic profiles, a

54 ocytes in multiple tissues during a high-fat diet and in skin during hair follicle growth.

55 number of risk factors, including lifestyle, diet and inflammation.

56 ortant source of micronutrients in the human diet and is extensively consumed around the world.

57 understand the mechanisms linking unhealthy diet and mental disorders.

58 luded: smoking, heavy alcohol use, unhealthy diet and physical inactivity.

59 ns in human skeletal muscle after a high-fat diet and resistance exercise.

60 t clear how the complex interactions between diet and the intestinal microbiota affect development of

61 rminal center response to a cholesterol-rich diet and uncover a PDL1-dependent mechanism through whic

62 food staples incorporated into contemporary diets and how the nutrient landscapes of these staples v

63 t gnotobiotic mice fed a polysaccharide-rich diet, and (ii) in situ hybridization and spectral imagin

64 n intervention comprising physical activity, diet, and cognitive training improved several cognitive

65 ctors such as caloric restriction, ketogenic diet, and hyperglycemia influence the inflammatory respo

66 T2DM arises largely from obesity, poor diet, and lack of exercise, but it also involves genetic

67 social activity, physical activity, healthy diet, and light-to-moderate alcohol consumption were pos

68 us insects, most crustaceans have very broad diets, and the increased richness of taxa that include p

69 To assess whether a high-salt diet, as measured by urinary sodium concentration, is as

70 c findings found initiation of a gluten-free diet associated with small improvement in gastrointestin

71 food frequency questionnaires that assessed diet at ages 12-13 years and 10 years previously.

72 in intake within countries, with plant-based diets being the most vulnerable.

73 of heterogeneity in herbivore body size and diet breadth (i.e. the diversity of host plants used) fo

74 result of prey partitioning by body size and diet breadth, the combined effects of birds and ants on

75 thy Diet Indicator (HDI), and the Baltic Sea Diet (BSD)] with serum metabolites.

76 the VTA decreased wheel running in standard diet but not in WD F1 female offspring.

77 Although the diet can affect the metabolic response to exercise, the

78 olic diseases, while calorie-restricted (CR) diets can improve health and extend lifespan.

79 illions of people worldwide whose inadequate diet causes iron deficiency anemia.

80 e we show that in rats on a high-cholesterol diet, cholesterol levels in hippocampal neurons are incr

81 , and grains (40%; n = 19,541) or to a usual diet comparison (60%; n = 29,294).

82 ydrate) (CHO) as well as a eucaloric control diet (CON).

83 n of polar bears and observed changes in the diet, condition and/or reproduction of four other verteb

84 normal low fat (13% kcal) diet, 2) a low fat diet containing n-3 PUFAs, 3) a high fat (41% kcal) diet

85 ignificantly reduced as compared to rats fed diet containing SFO.

86 implanted with zeranol and three were fed a diet containing zearalenone.

87 Ppara(-/-) double-knockout (dKO) mice liquid diets containing corn oil resulted in a percentage fat-d

88 Diets containing three different lipid levels (high [HFD

89 ion of this functional food in a daily human diet could contribute to improve consumers' health.

90 or sodium and zinc, in which analyses of 24h diet dairies overestimated intake by 35% and 52%, respec

91 Maternal DHT exposure, regardless of diet, decreased fetal liver Pparg mRNA expression and in

92 is often produced through the deposition of diet-derived carotenoid pigments, yet the mechanisms of

93 aily intakes of 956+/-327.9mg estimated from diet diary analyses and 935+/-329.9mg estimated by a dup

94 Daily intake estimation comparisons through diet diary analyses and duplicate diet for other element

95 andomly assigned into exercise (AEx n = 29), diet (Diet n = 28), exercise plus diet (AED n = 29), or

96 stinal symptoms compared with no gluten-free diet (difference less than 1 point on a scale of 1 to 7)

97 rowth rate lower than expected when fed on a diet different to which they were raised, possibly due t

98 n proposed as an adaptation to a starch-rich diet driven by the widespread adoption of agriculture in

99 The AP and PP diets each reduced liver fat by 36%-48% within 6 weeks (

100 ring weight stability while consuming 3 test diets, each for a 4-wk period according to a crossover l

101 steoblastic cell senescence, and that ST-SPI diet early in life has modest but persistent programming

102 ccurs rapidly upon consumption of a high-fat diet, even prior to significant weight gain.

103 To assess the in vivo efficacy, western-type diet fed apoE(-/-) mice were treated daily with 15a or v

104 Standard and cafeteria (CAF) diet fed rats, a robust model of metabolic syndrome (MeS

105 Western diet-fed LDL receptor-deficient (Ldlr-/-) mice with myel

106 e and weight gain in lean mice upon high-fat diet feeding, and this injection paradigm reduced high-f

107 ponse to LXR activation and high-cholesterol diet feeding.

108 sulin sensitivity despite prolonged high-fat diet feeding.

109 consumed in low amounts as part of a healthy diet, few children achieve such levels, making this an i

110 weeks or a methionine- and choline-deficient diet for 1, 4, 8, or 12 weeks to induce a continuum of s

111 ), C57BL/6J mice were fed a low- or high-fat diet for 16 weeks with or without 2% (w/w) fenugreek sup

112 articipants reported following a gluten-free diet for at least 1 year before the study began.

113 gut microbiota, which relies heavily on host diet for metabolic substrates and the gastrointestinal t

114 ns through diet diary analyses and duplicate diet for other elements showed good agreement, except fo

115 Rats were fed six protein diets for 14 days, including casein (control), and prote

116 bese twin pairs consumed recommended low fat diets for 6 weeks before they received a 6-week high fat

117 individuals on a self-instituted gluten-free diet, for whom celiac disease had been excluded.

118 long the mammalian tree and to infer ancient diets from the predicted microbiomes of mammalian ancest

119 line for future work to optimize recommended diets further.

120 When the data from the 2 diet groups were combined, the mean methylation of 1444

121 Chicks from both maternal diet groups were fed the same diet after hatch.

122 jects (HS), and IBS patients on a 4-week LFM diet had improved IBS symptoms and reduced fecal LPS lev

123 Fish fed the CS diet had significantly more long chain polyunsaturated f

124 The optimised diets had up to 30% lower blue water footprints and gene

125 , low-carbohydrate diets, known as ketogenic diets, have been used as a non-pharmacological treatment

126 d with either a high-cholesterol atherogenic diet (HCD) or matching normal diet (ND), respectively.

127 FD) and moderately high fat plus cholesterol diet (HFC)-on wildtype (WT) and liver-specific Foxo1/3/4

128 effects of two different diets-very high fat diet (HFD) and moderately high fat plus cholesterol diet

129 in D-enriched mushrooms extracts on high-fat diet (HFD) animal model of non-alcoholic steatohepatitis

130 rmation in transgenic CCDC3 mice on high-fat diet (HFD) by reducing the expression of hepatic PPARgam

131 Under conditions of high fat diet (HFD) consumption, glucose dyshomeostasis develops

132 -12(-/-) (p35(-/-)) mice were fed a high-fat diet (HFD) for 12 weeks.

133 h significant weight reduction in a high-fat diet (HFD) induced diabetic mouse model and a geneticall

134 Consumption of a high-fat diet (HFD) results in suppression of ATP citrate-lyase l

135 erosclerosis was induced by feeding high fat diet (HFD) to mice for 10 weeks, followed by five oral d

136 ion accumulation in mice exposed to high-fat diet (HFD), injected with streptozotocin, or both in com

137 rom epidemiology studies have indicated that diets high in animal fat and low in fruits and vegetable

138 We investigated the effects of diets high in animal protein (AP) vs plant protein (PP),

139 opposite is true for consumers with broader diets (i.e., generalists).

140 ngs underline the role of fat content in the diet in altering gut microbiota community by shifting ph

141 s the lifespan of animals fed a high glucose diet in an AMPK-dependent manner.

142 ates an adaptive response to a high-fructose diet in mice and that loss of this transcription factor

143 Hypertension (AASK) and 761 Modification of Diet in Renal Disease (MDRD) Trial participants previous

144 ate was calculated using the Modification of Diet in Renal Disease formula.

145 rst instar when fed a nutritionally complete diet in the absence of a gut microbiota.

146 it the implementation of empiric elimination diets in patients with eosinophilic esophagitis (EoE).

147 Nine healthy men ingested two hypercaloric diets (in 75% excess of habitual caloric intake) for 3 d

148 an primates, a 2-year high-fat, high-sucrose diet increased hepatic mIndy expression.

149 Strikingly, a ketogenic diet increased lysine acetylation in Cpt2M(-/-) hearts 2

150 Furthermore, both the SFA and PUFA diets increased the mean degree of DNA methylation in ad

151 We hypothesized that a high-FODMAP (HFM) diet increases visceral nociception by inducing dysbiosi

152 terranean Diet Score (aMED), the WHO Healthy Diet Indicator (HDI), and the Baltic Sea Diet (BSD)] wit

153 means of managing cholesterol metabolism and diet induced dyslipidaemia, as well as insulin sensitivi

154 We used a mouse model of maternal-diet induced obesity to define predictive correlations b

155 ce of cholesterol metabolism by the host for diet-induced changes of the gut microbiota and energy me

156 ZIP14 during pharmacologically and high-fat diet-induced ER stress using Zip14(-/-) (KO) mice, which

157 SLC13A5 deletion protects mice from high-fat diet-induced hepatic steatosis and that mutation of the

158 and increased as expected in the setting of diet-induced insulin resistance.

159 , we show that Zbtb20 ablation protects from diet-induced liver steatosis and improves hepatic insuli

160 rbates or alleviates, respectively, high-fat diet-induced mitochondrial dysfunction, hepatosteatosis,

161 halamic expression of Ctbp2 was increased in diet-induced obese (DIO) mice as compared with age-match

162 adigm reduced high-fat intake and obesity in diet-induced obese (DIO) mice.

163 Here we report that in the NTS of high-fat diet-induced obese (DIO) rats, the apoA-IV mRNA level is

164 Diet-induced obese animals received either Lactobacillus

165 ng body weight gain relative to control in a diet-induced obese dog model, suggesting the importance

166 n acetylation-defective SIRT3-K57R mutant in diet-induced obese mice decreased acetylation of mitocho

167 am signals TNFSF11A or NDUFAB1 in the MBH of diet-induced obese mice reverses mitochondrial elongatio

168 Here we show that in hypercaloric diet-induced obese mice, persistently activated microgli

169 ood glucose and improve glucose tolerance in diet-induced obese mice.

170 For this purpose, we used diet-induced obese rats and rats administered thapsigarg

171 issues were resistant to developing high-fat diet-induced obesity and had significantly reduced white

172 epresents a promising approach to ameliorate diet-induced obesity and leptin resistance.

173 mproves insulin sensitivity substantially in diet-induced obesity by both peripheral and central mech

174 MH-specific inhibition of TBK-1 in mice with diet-induced obesity impaired glucose metabolism and AKT

175 Maternal diet-induced obesity increased miR-126 expression howeve

176 Relative to the overweight and diet-induced obesity regimens, CR decreased body weight,

177 Our findings suggest that the combination of diet-induced obesity with other risk factors may increas

178 ation of hepatic Dpp4 in young mice prone to diet-induced obesity.

179 electrophysiological properties observed in diet-induced obesity.

180 and carbohydrates consumption) combined with diet-induced overweight/obesity on the risk of periodont

181 total cholesterol levels and suppression of diet-induced weight gain.

182 behavioral tests including chow and high-fat diet intake, meal patterns, conditioned place preference

183 Furthermore, the gene-diet interaction was confirmed in the cross-sectional Me

184 In addition, significant gene-diet interactions were shown for the rs1440581 PPM1K gen

185 tic minipump), or PCP+glycine (16% by weight diet) interventions.

186 The Western diet is characterized by high protein, sugar, fat, and l

187 ased nutritional intake from an energy-dense diet is known to disrupt metabolic homeostasis and contr

188 High-fat, low-carbohydrate ketogenic diets (KDs) have shown beneficial effects in mouse model

189 High-fat, low-carbohydrate diets, known as ketogenic diets, have been used as a non

190 When dairy was introduced into the diet, lactose-fermenting Roseburia species increased fro

191 ichness of taxa that include plants in their diet likely results from access to a novel resource base

192 Mice maintained on a vitamin A-free diet lose HSCs and show a disrupted re-entry into dorman

193 s given counselling to follow a sham diet or diet low in FODMAPs for 4 weeks, along with a placebo or

194 dies examining the effects of consumption of diets low in advanced glycation end products (AGEs) on c

195 nation of reduced sodium intake and the DASH diet lowered SBP throughout the range of pre- and stage

196 Female mice fed a high-fat diet maintain CX3CL1-CX3CR1 levels while male mice show

197 ketogenic low carbohydrate, high fat (LCHF) diet markedly increases rates of whole-body fat oxidatio

198 DIET may also have a role in anaerobic methane oxidation

199 emphasize that the importance of a balanced diet may extend beyond the mere physical benefits of ade

200 The results indicated cows consuming RS diets may have had depressed milk protein synthesis beca

201 fatty acid supplementation, and gluten-free diet, may have additional benefits, as do potential noni

202 3) more on the low-fat and high-carbohydrate diet [mean group difference: 2.47 kg (95% CI: 0.20, 4.75

203 nverse association between the Mediterranean diet (MedDiet) and cardiovascular disease (CVD).We evalu

204 y assigned into exercise (AEx n = 29), diet (Diet n = 28), exercise plus diet (AED n = 29), or no-int

205 s; n=100), with respect to a low-fat control diet (n=96).

206 ol atherogenic diet (HCD) or matching normal diet (ND), respectively.

207 We report differences in morphology and diet of the termite-eating gecko Gymnodactylus amarali b

208 r in vivo studies revealed the combinatorial diets of GTPs and BSp significantly inhibited breast tum

209 ent seem to have a stronger influence on the diets of young adults.

210 such as adipose and liver, but the impact of diet on acetyl-CoA and histone acetylation in these tiss

211 nuates the inflammatory impact of a high fat diet on glucose tolerance and insulin resistance.

212 NA, the effects of a post-weaning obesogenic diet on IRS-1 are mediated by miR-126 independent mechan

213 The impact of diet on the metabolism-epigenome axis is poorly understo

214 We investigated the impact of six protein diets on oxidation and anti-oxidation status in the musc

215 g of DL-alpha-tocopheryl acetate/kg of basal diet) on physicochemical and fatty acid stability of fre

216 thanotrophic bacteria comprises 21% of their diet, on average.

217 to groups given counselling to follow a sham diet or diet low in FODMAPs for 4 weeks, along with a pl

218 ormal daily routines without restrictions on diet or physical activity.

219 Specific diets or agonists that target these chemosensory signali

220 et rich in n-3 PUFAs, 4) a high fat n-6 PUFA diet, or 5) a high fat monounsaturated diet.

221 liver fat by 36%-48% within 6 weeks (for AP diet P = .0002; for PP diet P = .001).

222 ithin 6 weeks (for AP diet P = .0002; for PP diet P = .001).

223 ate (+2.6%), MUFA (+5.3%), and PUFA (+12.3%) diets (P < 0.05 for all).

224 Alternatively, DIET partners can plug into conductive carbon materials,

225 nseling interventions to promote a healthful diet, physical activity, or both improve health outcomes

226 n State (1996-2008), reported information on diet, physical activity, smoking, and stress during earl

227 sham diet/probiotic, 24 receiving low FODMAP diet /placebo, and 27 receiving low FODMAP diet/probioti

228 on, resulting in 4 groups (27 receiving sham diet/placebo, 26 receiving sham diet/probiotic, 24 recei

229 e designed to assess type of control strain, diet, presence of the misty allele, and parity as potent

230 P diet /placebo, and 27 receiving low FODMAP diet/probiotic).

231 ceiving sham diet/placebo, 26 receiving sham diet/probiotic, 24 receiving low FODMAP diet /placebo, a

232 xcess cholesterol to a high-fat/high-sucrose diet produced greater steatosis in LCR and high capacity

233 We propose that the Western lifestyle and diet promote innate danger signals and immune responses

234 e present study, we show that a reduced BCAA diet promotes rapid fat mass loss without calorie restri

235 Diets promoting obesity and insulin resistance can lead

236 Consumption of a high-fat diet protects mice from ventilator-induced lung injury i

237 Subgroup analyses showed that diets providing the largest total energy intake and ener

238 EI) later in the day is associated with poor diet quality and obesity.

239 There is substantial room for improvement in diet quality in all sociodemographic subgroups.

240 & AIMS: Healthy eating patterns assessed by diet quality indexes (DQIs) have been related to lower r

241 is study was to examine the correlation of 4 diet quality indexes [the Healthy Eating Index (HEI) 201

242 Better diet quality was associated with greater moderate-to-vig

243 pathways were most strongly associated with diet quality.

244 nd energy intake assessed with 3-day weighed diet records at 7, 12, and 24 months.

245 cted diversity induced by a typical American diet reflects a durable loss of taxa that is replenished

246 how nutritional metabolomics might identify diet-related exposures that modulate cancer risk.

247 of health behaviours, socio-demographic and diet-related factors, adiposity and other diseases.

248 otential of probiotics to attenuate high-fat diet-related metabolic disorder.

249 with 16%, 13% and 10% crude protein (CP) in diets, respectively, were investigated using Illumina Mi

250 lished in Cell Host & Microbe showing that a diet rich in fat and simple sugars alters the gut microb

251 ntaining n-3 PUFAs, 3) a high fat (41% kcal) diet rich in n-3 PUFAs, 4) a high fat n-6 PUFA diet, or

252 A diet rich in phytochemicals confers benefits for health

253 Clinical studies suggest that diets rich in omega-3 polyunsaturated fatty acids (PUFAs

254 fferences in hepatic proteins when comparing diets rich in the two soybean oils, coconut oil, and a l

255 ndex (HEI) 2010, the Alternate Mediterranean Diet Score (aMED), the WHO Healthy Diet Indicator (HDI),

256 ] = 1.47) and children with higher unhealthy diet scores (RR = 1.08) complied more, but overweight/ob

257 of a 25% calorie restricted (CR) or standard diet (SD).

258 e factors including body mass index, healthy diet, sedentary lifestyle, alcohol consumption, smoking,

259 tion of longevity by other factors including diet, sex, insulin signalling and population density.

260 to a level close to that reported for human' diet since weaning lead to hypertension, which appears t

261 Our results show that early diet specialization may be reinforced by the elevated co

262 Furthermore, we found evidence of diet specialization with all fish maintaining a standard

263 Larvae were reared on artificial diets spiked with contaminants of emerging concern (CECs

264 141 food samples from the first French total diet study on infants and toddlers.

265 291 food samples from the first French total diet study on infants and toddlers.

266 Hens were fed diets supplemented (2.8% wt:wt) with corn oil (CO; n-6)

267 m T4 and very low serum T3 levels when fed a diet supplying the minimum I(-) requirement for rodents.

268 7) more on the high-fat and low-carbohydrate diet than on the low-fat and high-carbohydrate diet, whe

269 nt of energy from free sugars with a control diet that provides the same amount of energy from comple

270 a-analysis of intervention trials to compare diets that provide a given amount of energy from free su

271 Beetles were maintained on one of five diets that varied in their ratio of protein to fat for 4

272 rceived absolute requirement for a ketogenic diet, the assumed lack of structural brain defects, and

273 tion during the transition from a milk-based diet to a chow-based diet after weaning.

274 help elucidate mechanistic pathways linking diet to chronic disease risk.

275 g therapy, a resurgence of interest in using diet to manage and treat DM has emerged in recent years.

276 xercise, the contribution of a self-selected diet to the interindividual variability in the MFO requi

277 were placed on control and low protein (LP) diets to assess changes in energy expenditure, food inta

278 before they received a 6-week high fat (HF) diet under isocaloric conditions.

279 se individuals who were randomly assigned to diets varying in macronutrient content.

280 ents to examine the effects of two different diets-very high fat diet (HFD) and moderately high fat p

281 ants followed either a 5-wk very-low-calorie diet (VLCD; 500 kcal/d) or a 12-wk low-calorie diet (125

282 Western diet was associated with an increased incidence of CRC (

283 is when a combined high-fat and high-glucose diet was given, seemingly due to suppression of autophag

284 C57BL/6 mice were fed a Western diet (WD) (35% kcal from fat enriched in palmitate) or a

285 C57BL/6 male mice were fed a Western diet (WD) +/-75 mg PDX twice daily by oral gavage for 14

286 Western-style diets (WD) high in fat and scarce in fiber and vitamin D

287 e liver disease following a 16-week 'western diet' (WD) high in fat (45% kcal), cholesterol (1% w/w)

288 -cholesterol concentrations after the cheese diet were lower than after the butter diet (-3.3%, P < 0

289 receptor-null (Ldlr(-/-)) mice on a high-fat diet were orally administered with vehicle control or UF

290 Diets were liberalized at 8 months in both groups.

291 et than on the low-fat and high-carbohydrate diet, whereas normoglycemic individuals lost a mean of 0

292 A palmitate-rich diet, which increases serum 27OHC, or APP ablation, abro

293 The functional consequence of this diet will now be tested in a clinical trial.

294 its of 2 key components of the Mediterranean diet (wine and VOO).

295 Promoting DIET with conductive materials shows promise for stabili

296 e unhealthy high-calorie, high-sugar Western diet with reduced levels of BCAAs lost weight and fat ma

297 species abundance in response to changes in diets with minimal additional imposed constraints on the

298 ith Lcn2-depleted animals and fed isocaloric diets with varying amounts of fat content.

299 n through an elemental analysis of duplicate diets, with a mean+/-sd daily intakes of 956+/-327.9mg e

300 with littermate control mice fed a ketogenic diet, yet it did not improve cardiac hypertrophy.