ライフサイエンスコーパス: diet-induced

1 The cholesterol-enriched diet induced ABL in group Hc; groups HcP and P had more

2 The resistance to diet-induced adiposity was the result of an increased me

3 we reveal that harmine antagonizes high fat diet-induced adiposity.

4 s for p32 are resistant to age- and high-fat diet-induced ailments, including obesity, hyperglycemia,

5 independent models of renal failure, adenine diet induced and 5/6 nephrectomy.

6 ly in the liver, protects mice from high-fat diet-induced and aging-induced obesity and hepatic fat a

7 ttenuated both methionine choline-deficient, diet-induced and choline-deficient, high-fat diet-induce

8 ed in hyperlipidemia and exacerbated Western diet-induced atherogenesis.

9 elet function, vasculature inflammation, and diet-induced atherosclerosis and myocardial infarction.

10 ceride and cholesterol levels and attenuates diet-induced atherosclerosis development.

11 , a skin disease that is not associated with diet-induced atherosclerosis in humans.

12 with Arhgef1-deficient BM prevented high-fat diet-induced atherosclerosis, while reconstitution of Ld

13 tion, commensal microbial TMA production, on diet-induced atherosclerosis.

14 LR) and Arhgef1 were protected from high-fat diet-induced atherosclerosis.

15 ST overexpression did not influence high fat diet-induced body weight and fat mass gain throughout th

16 lasma glutamate concentration and alleviated diet-induced body-weight gain and adiposity in mice.

17 However, the exact pathways linking diet-induced changes (e.g., hyperlipidemia) and the ensu

18 ce of cholesterol metabolism by the host for diet-induced changes of the gut microbiota and energy me

19 In contrast, the vast majority of diet-induced chromatin accessibility changes in A/J mice

20 We have now examined the persistence of diet-induced chromatin accessibility changes upon diet r

21 dependent, indicating a genetic component in diet-induced chromatin alterations.

22 ich requires domains that influence high-fat-diet-induced chronic inflammation and alter cell functio

23 bjective of this study was to assess whether diet-induced coronary plaque ruptures trigger atherothro

24 VWR exercise in obese mice rescued high-fat diet-induced decreased muscle GLUT4 protein and improved

25 ain, improves sleep, and attenuates age- and diet-induced deterioration in cardiac performance.

26 increased body fat, which are signatures of diet-induced diabetes (type 2 diabetes) and obesity in h

27 To determine if high-fat diet-induced diabetes in mice can model retinal disease,

28 ry proteins produced by macrophages improves diet-induced diabetes, but how nutrient-dense diets indu

29 stevioside prevents development of high-fat-diet-induced diabetic hyperglycaemia in wild-type mice,

30 ly improved in normal mice and high fat (HF) diet-induced diabetic mice with hyperinsulinemia in ECIR

31 This is the first evidence that diet-induced disruption to vagal afferent signaling may

32 alizing antibody are resistant to obesogenic diet-induced disturbances in metabolism.

33 means of managing cholesterol metabolism and diet induced dyslipidaemia, as well as insulin sensitivi

34 ol levels were investigated in hamsters with diet-induced dyslipidemia.

35 ty liver disease; 3) DKO mice demonstrate HF diet-induced elevations of plasma leptin, resistin, fed-

36 Although diet-induced epigenetic reprogramming via paternal linea

37 ZIP14 during pharmacologically and high-fat diet-induced ER stress using Zip14(-/-) (KO) mice, which

38 diet-induced and choline-deficient, high-fat diet-induced ER stress, inflammation, steatohepatitis, a

39 In high-fat diet induced fatty-liver mice, AM580 reduced ApoC-III le

40 tal AsIII exposure exacerbated Western-style diet-induced fatty liver disease.

41 salutary role in the protection against the diet-induced fatty liver disease.

42 on of MDA epitopes plays a major role during diet-induced hepatic inflammation and can be ameliorated

43 CM, integrins, are key to the development of diet-induced hepatic insulin resistance.

44 nsrT1150A mice) were protected from high fat diet-induced hepatic insulin resistance.

45 of integrin signaling in the pathogenesis of diet-induced hepatic insulin resistance.

46 eficient mice are protected against high-fat diet-induced hepatic steatosis and insulin resistance.

47 cific disruption of Hif2a, in which high-fat-diet-induced hepatic steatosis and obesity were substant

48 SLC13A5 deletion protects mice from high-fat diet-induced hepatic steatosis and that mutation of the

49 0 knockout mice were protected from high-fat diet-induced hepatic steatosis as well as from insulin r

50 ractive effect of PM2.5 exposure on high-fat diet-induced hepatic steatosis was mediated through PM2.

51 ochondrial function and ameliorates high-fat-diet-induced hepatic steatosis, glucose tolerance, and i

52 lipogenic response to feeding, exacerbating diet-induced hepatic steatosis.

53 ipose tissue lipolysis and prevents high-fat diet-induced hepatic steatosis.

54 Hepatic DsbA-L protects mice from diet-induced hepatosteatosis and insulin resistance.

55 adipose tissue and have shown that high-fat diet-induced (HFD-induced) insulin resistance is mitigat

56 However, diet-induced high Hcy resulted in a significant increase

57 d pulmonary homeostasis, we are wondering if diet induced hypercholesterolemia would influence the su

58 y of a miR-30c mimic to the liver diminished diet-induced hypercholesterolemia in C57BL/6J mice.

59 mouse liver but protects mice from high-fat diet-induced hyperglycemia.

60 sphate (Pi) were reduced by 50% in mice with diet-induced hypophosphatemia as well as in sodium-depen

61 SOCS3 from brain neurons delays the onset of diet-induced infertility.

62 how that macrophage FAS is indispensable for diet-induced inflammation.

63 fic Dnmt3a knock-out mice are protected from diet-induced insulin resistance and glucose intolerance

64 Deleting Fasn in macrophages prevents diet-induced insulin resistance, recruitment of macropha

65 ression in muscle is a critical component of diet-induced insulin resistance, which possibly acts by

66 itochondrial function, and susceptibility to diet-induced insulin resistance.

67 affecting mechanisms implicated in high-fat diet-induced insulin resistance.

68 mpair glucose metabolism in a mouse model of diet-induced insulin resistance.

69 ing adipose tissue DNL as an early target in diet-induced insulin resistance.

70 inhibited obesity and prevented the onset of diet-induced insulin resistance.

71 and increased as expected in the setting of diet-induced insulin resistance.

72 lly enforced guanylin replacement eliminated diet-induced intestinal tumorigenesis in mice.

73 ysis-regulatory enzyme that protects against diet-induced intestine inflammation.

74 Deficient leptin signaling attributable to diet-induced leptin resistance is associated with infert

75 ctivation of CES1 aggravated alcohol- or MCD diet-induced liver inflammation and liver injury, likely

76 eased the susceptibility to high cholesterol diet-induced liver injury and abolished the protective e

77 ) and methionine and choline-deficient (MCD) diet-induced liver injury.

78 l role in protection against alcohol- or MCD diet-induced liver injury.

79 , we show that Zbtb20 ablation protects from diet-induced liver steatosis and improves hepatic insuli

80 hese results simplify the pathophysiology of diet-induced liver tumorigenesis by focusing attention o

81 high-fat diet and tested the hypothesis that diet-induced metabolic disease promotes retinopathy.

82 lines were found to be highly susceptible to diet-induced metabolic disease, as evidenced by impairme

83 ) mice are known to be highly susceptible to diet-induced metabolic disease, this notion stems primar

84 may mediate intergenerational inheritance of diet-induced metabolic disorders.

85 K3 inactivation for adiponectin formation in diet-induced metabolic syndrome.

86 production of adiponectin and protects from diet-induced metabolic syndrome.

87 We sought to test whether diet-induced mitochondrial dysfunction and oxidative str

88 rbates or alleviates, respectively, high-fat diet-induced mitochondrial dysfunction, hepatosteatosis,

89 th liver-specific luciferase expression in a diet-induced model of nonalcoholic fatty liver disease r

90 When administered to various genetic and diet-induced mouse models of obesity, FGF21 can attenuat

91 Diet-induced muscle insulin resistance is associated wit

92 ILK, a key component of the IPP complex, in diet-induced muscle insulin resistance.

93 PLD1 expression was decreased in high-fat diet-induced NAFLD.

94 In the diet-induced NASH model, an increased CNR was also found

95 that CXCL10(-/-) mice are protected against diet-induced NASH, in an obesity-independent manner.

96 ith a data set of blood serum samples from 7 diet induced obese (DIO) and 7 nonobese (NO) C57BL/6J mi

97 is independent of obesity as weight matched diet induced obese mice do not display systolic dysfunct

98 glucose, lipid and cholesterol metabolism in diet induced obese rodents.

99 e NAD tissue levels in liver and muscle in a diet-induced obese (DIO) C57BL/6 mouse model.

100 halamic expression of Ctbp2 was increased in diet-induced obese (DIO) mice as compared with age-match

101 tion, apoA-I injections in insulin-resistant diet-induced obese (DIO) mice lead to increased glucose-

102 Studies in normal and diet-induced obese (DIO) mice with the preferred compoun

103 adigm reduced high-fat intake and obesity in diet-induced obese (DIO) mice.

104 abetic db/db mice and reduced body weight in diet-induced obese (DIO) mice.

105 Here we report that in the NTS of high-fat diet-induced obese (DIO) rats, the apoA-IV mRNA level is

106 ride and diacylglycerol content in livers of diet-induced obese and genetically obese and insulin-res

107 Diet-induced obese animals received either Lactobacillus

108 model based on a single miRNA deficiency in diet-induced obese atherogenic mice.

109 ivation in adipose tissue under the high fat diet-induced obese condition.

110 ng body weight gain relative to control in a diet-induced obese dog model, suggesting the importance

111 Kbtbd2 was down-regulated in diet-induced obese insulin-resistant mice in a leptin-de

112 Changes in VMH AKT activation of diet-induced obese Lin28aKI(VMH) or Lin28aKD(VMH) mice w

113 ed elevated levels of FGF21 in both high-fat diet-induced obese mice and in genetically obese-diabeti

114 n acetylation-defective SIRT3-K57R mutant in diet-induced obese mice decreased acetylation of mitocho

115 am signals TNFSF11A or NDUFAB1 in the MBH of diet-induced obese mice reverses mitochondrial elongatio

116 GM-CSF neutralization in diet-induced obese mice significantly reduced immunosupp

117 posity, and improve blood glucose control in diet-induced obese mice with pre-existing metabolic synd

118 d liver steatosis and glucose intolerance in diet-induced obese mice, but these beneficial effects we

119 Here we show that in hypercaloric diet-induced obese mice, persistently activated microgli

120 g insulin resistance and fat accumulation in diet-induced obese mice.

121 ood glucose and improve glucose tolerance in diet-induced obese mice.

122 sity and improves glucoregulatory control in diet-induced obese mice.

123 tic glucose and lipid metabolism in high-fat diet-induced obese mice.

124 sed metabolic rate and energy expenditure in diet-induced obese mice.

125 expression ameliorates hepatic steatosis in diet-induced obese mice.

126 of UGN reduces weight gain and adiposity in diet-induced obese mice.

127 olism, and also reduced hepatic steatosis in diet-induced obese mice.

128 lin resistance, was significantly reduced in diet-induced obese mice.

129 osure relieved hepatic steatosis in high-fat diet-induced obese mice.

130 bone marrow-derived macrophages and high-fat diet-induced obese mice.

131 In a diet-induced obese mouse model, these angiogenesis-targe

132 ebrafish and in live tissues from a high-fat diet-induced obese mouse model.

133 eported that lean juvenile offspring born to diet-induced obese rats (OffOb) exhibit sympathetic-medi

134 For this purpose, we used diet-induced obese rats and rats administered thapsigarg

135 Diet-induced obese rats were randomized to isocaloric di

136 In the diet-induced obese state, the effects of GCG neuronal st

137 Oral administration of Pep19 into diet-induced obese Wistar rats significantly reduces adi

138 We used a mouse model of maternal-diet induced obesity to define predictive correlations b

139 Global deletion of Ip6k1 protects mice from diet-induced obesity (DIO) and insulin resistance, but t

140 ought to establish whether the propensity to diet-induced obesity (DIO) is associated with addictive-

141 In db/db or diet-induced obesity (DIO) mice, hepatic expression of A

142 Impaired adipogenic differentiation during diet-induced obesity (DIO) promotes adipocyte hypertroph

143 t received low perinatal n-6/n-3 ratios were diet-induced obesity (DIO) resistant and had a lower pos

144 und that withaferin-A treatment of mice with diet-induced obesity (DIO) resulted in a 20-25% reductio

145 gh behavioural rhythmicity was maintained in diet-induced obesity (DIO), gene expression profiling re

146 a cannabinoid receptor 1 (CB1) antagonist on Diet-Induced Obesity (DIO), specifically whether such a

147 e long-term impact of transient peripubertal diet-induced obesity (ppDIO, induced between 4 and 10 we

148 define whether activated calpains influence diet-induced obesity and adipose tissue macrophage accum

149 O) was sufficient to protect adult mice from diet-induced obesity and associated metabolic alteration

150 issues were resistant to developing high-fat diet-induced obesity and had significantly reduced white

151 ally reducing dietary BCAAs rapidly reverses diet-induced obesity and improves glucoregulatory contro

152 Importantly, loading relieves diet-induced obesity and improves glucose tolerance.

153 tivation of Hh signaling suppresses high-fat-diet-induced obesity and improves whole-body glucose tol

154 In addition, diet-induced obesity and insulin resistance are exacerba

155 nd adipose tissue mass and were resistant to diet-induced obesity and insulin resistance due to a com

156 NSG mice were completely protected from diet-induced obesity and insulin resistance with signifi

157 homeostasis in a preclinical murine model of diet-induced obesity and insulin resistance, making the

158 ial fatty acid oxidation, thereby preventing diet-induced obesity and insulin resistance.

159 ound, lymphocytes, and cytokine signaling in diet-induced obesity and insulin resistance.

160 ion of Tst in adipocytes protected mice from diet-induced obesity and insulin-resistant diabetes.

161 epresents a promising approach to ameliorate diet-induced obesity and leptin resistance.

162 y regulated in white and brown fat depots of diet-induced obesity and leptin-deficient ob/ob mouse mo

163 geted activation of Hh signaling ameliorates diet-induced obesity and may be explored for pharmaceuti

164 potential therapeutic targets for combating diet-induced obesity and metabolic disease.

165 dy suggests that blocking of CB1 ameliorates Diet-Induced Obesity and metabolic disorder by modulatin

166 egulated microbiota protect BALB/c mice from diet-induced obesity and metabolic dysfunction.

167 Finally, moderate alcohol prevented high-fat diet-induced obesity and metabolic dysfunction.

168 lacking hepatic ZFP36L1 were protected from diet-induced obesity and steatosis.

169 Shp inactivation may be beneficial to combat diet-induced obesity and uncover that hepatic SHP is nec

170 ic insulin resistance and hepatosteatosis in diet-induced obesity are associated with various metabol

171 confers long-term metabolic protection from diet-induced obesity at the cost of moderate skin oxidat

172 Since diet-induced obesity attenuates the responsiveness of ga

173 lin resistance and insulin resistance due to diet-induced obesity both depend on muscle TRIB3.

174 mproves insulin sensitivity substantially in diet-induced obesity by both peripheral and central mech

175 eered mice on different diets, we found that diet-induced obesity caused a loss of guanylin expressio

176 Diet-induced obesity causes chronic macrophage-driven in

177 Diet-induced obesity compromises the excitability and re

178 Diet-induced obesity drove early production of interleuk

179 d muscle glucose metabolism and resistant to diet-induced obesity due to increased energy expenditure

180 dipose tissue development, but its effect on diet-induced obesity during postnatal life is not known.

181 PQQ, provided prenatally in a mouse model of diet-induced obesity during pregnancy, could protect obe

182 and suggest a novel mechanism through which diet-induced obesity during puberty imposes its long-las

183 exercise on type II diabetes risk under a HF diet-induced obesity environment.

184 ke receptor 4 expression was observed in the diet-induced obesity HCC animal model.

185 We developed a diet-induced obesity HCC mouse model and examined TnC ex

186 Diet-induced obesity impaired AT1-ILC killing ability.

187 MH-specific inhibition of TBK-1 in mice with diet-induced obesity impaired glucose metabolism and AKT

188 ggerated the detrimental effects of maternal diet-induced obesity in adipose tissue.

189 Pharmacological inhibition of CBR1 in diet-induced obesity in mice results in more marked gluc

190 and deletion of theNrf2gene protects against diet-induced obesity in mice.

191 on of Id1 causes age-associated and high-fat diet-induced obesity in mice.

192 mide N-methyltransferase (NNMT)-ASO prevents diet-induced obesity in mice.

193 Maternal diet-induced obesity increased miR-126 expression howeve

194 In mice with a GSNOR deletion, diet-induced obesity increases lysosomal nitrosative str

195 According to our previous results, diet-induced obesity induces epigenetic modifications to

196 y, microbial and dietary factors incurred by diet-induced obesity influence underlying innate and ada

197 Here, we demonstrate that resistance to diet-induced obesity inNrf2(-/-)mice is associated with

198 High-fat diet-induced obesity is a major risk factor for osteoart

199 Diet-induced obesity is associated with strong circadian

200 Previous studies have shown that maternal diet-induced obesity leads to increased risk of type 2 d

201 suggest that the memory-impairing effects of diet-induced obesity may potentially be mediated by neur

202 ce but increased over time in overweight and diet-induced obesity mice, suggesting CR obviates epigen

203 e metabonomics data and microbiome data in a diet-induced obesity model using C57BL/6 mice.

204 oreover, betaine administration to mice with diet-induced obesity prevents the development of impaire

205 by increased intestinal permeability during diet-induced obesity promotes insulin resistance in mice

206 zed to receive a CR, overweight-inducing, or diet-induced obesity regimen (n = 27/group).

207 Relative to the overweight and diet-induced obesity regimens, CR decreased body weight,

208 Recent studies revealed that diet-induced obesity suppressed guanylin and uroguanylin

209 the current study, we used a mouse model of diet-induced obesity to identify putative cellular mecha

210 use models of a regular diet and of high-fat-diet-induced obesity to investigate the role of dietary

211 mice have increased food intake and greater diet-induced obesity when fed high-fat diet.

212 ator (SRA) (SRAKO) are resistant to high fat diet-induced obesity with a phenotype that includes impr

213 Our findings suggest that the combination of diet-induced obesity with other risk factors may increas

214 duces WAT lipolysis in vivo but also reduces diet-induced obesity without affecting LPL activity.

215 ting) and excessive energy storage (high-fat diet-induced obesity) blunt the effect of leptin.

216 5(-/-) (double knock-out (DKO)) mice show HF diet-induced obesity, adipocyte hypertrophy, and present

217 a therapeutic target for insulin resistance, diet-induced obesity, and associated metabolic dysfuncti

218 proves insulin sensitivity, protects against diet-induced obesity, and elicits the browning of white

219 at they were protected from atherosclerosis, diet-induced obesity, and insulin resistance.

220 Following diet-induced obesity, biochemical analysis of livers rev

221 ansfer in mice suppressed the development of diet-induced obesity, but did not affect pre-existing ad

222 om brain cells is known to protect mice from diet-induced obesity, but the effects on HCD-induced inf

223 ient mice have been shown to be resistant to diet-induced obesity, but the mechanism behind this rema

224 In diet-induced obesity, cellular Ca(2+) handling propertie

225 lays a protective role in the progression of diet-induced obesity, hepatosteatosis, and atheroscleros

226 nfirmed in both serum and liver of mice with diet-induced obesity, implying that such a metabolic alt

227 nvestigated the effects of Lcn2 depletion on diet-induced obesity, inflammation, and PDAC development

228 w that intestinal PPARdelta protects against diet-induced obesity, insulin resistance and dyslipidemi

229 tion, transgenic mice were protected against diet-induced obesity, insulin resistance, and inflammati

230 Additionally, in mice with diet-induced obesity, INT-767 prevented mitochondrial dy

231 Our study indicates that, in diet-induced obesity, these circadian fluctuations in ga

232 Maternal diet-induced obesity, together with androgen excess, aff

233 he consequences of hepatic BIM deficiency in diet-induced obesity, we generated liver-specific BIM-kn

234 In mice, diet-induced obesity, which decreases insulin sensitivit

235 ssions in the mutant pedigrees and mice with diet-induced obesity, which showed that each obesity mou

236 esults implicate a new class of compounds in diet-induced obesity-C18 epoxide and diol oxylipins.

237 lin and leptin receptors in animal models of diet-induced obesity.

238 ic reticulum contacts in POMC neurons during diet-induced obesity.

239 ylcholinesterase (BChE), in a mouse model of diet-induced obesity.

240 s control pancreatic dysfunction in high-fat-diet-induced obesity.

241 f Bmal1 also worsens chronic inflammation in diet-induced obesity.

242 deletion exerts a protective effect against diet-induced obesity.

243 increased oxygen consumption during high-fat-diet-induced obesity.

244 nd appetitive motivation under conditions of diet-induced obesity.

245 tervention in dealing with the prevalence of diet-induced obesity.

246 is in the beta-cell compensatory response to diet-induced obesity.

247 tion prior to high calorie feeding prevented diet-induced obesity.

248 des in adipocytes and protected animals from diet-induced obesity.

249 ation of hepatic Dpp4 in young mice prone to diet-induced obesity.

250 n resistance, particularly in the context of diet-induced obesity.

251 f alternative splicing to the development of diet-induced obesity.

252 electrophysiological properties observed in diet-induced obesity.

253 adipose remodeling and adipocyte survival in diet-induced obesity.

254 tained glucose tolerance and did not develop diet-induced obesity.

255 stance and as a novel therapeutic target for diet-induced obesity.

256 iR-155 (-5p and -3p) in female mice prevents diet-induced obesity.

257 7 also prevented kidney disease in mice with diet-induced obesity.

258 hat mediate the resistance of female mice to diet-induced obesity.

259 rvention protects offspring against high-fat diet-induced obesity.

260 al white adipose tissue (WAT) in response to diet-induced obesity.

261 a marked increase in their susceptibility to diet-induced obesity.

262 2 diabetes, and C57BL/6J mice with high-fat diet-induced obesity.

263 d insulin resistance despite protection from diet-induced obesity.

264 This study evaluates the role of cafeteria diet-induced obesity/hyperlipidemia (CAF) on alveolar bo

265 our unique gut microbiota with resistance to diet-induced-obesity-mediated alteration of the gut micr

266 According to our statistical approach, diet-induced overweight/obesity increased the risk of al

267 and carbohydrates consumption) combined with diet-induced overweight/obesity on the risk of periodont

268 Here, we show that diet-induced paternal overweight around the time of conc

269 Diet-induced postprandial hyperglycemia and fasting hype

270 transplanted islets of Langerhans throughout diet-induced progression from normal glucose homeostasis

271 treatment with cGAMP significantly decreases diet-induced proinflammatory responses in liver and adip

272 Both diets induced severe hepatic steatosis in the LTKO mice

273 In ileum, formula diet induced significant up-regulation of AMCFII, IL-8,

274 Mice with high-fat diet-induced simple hepatic steatosis and lipid-loaded H

275 The results show that high-fat diet-induced steatohepatitis aggravates the inflammation

276 Herein, we investigated the impact of diet-induced steatohepatitis on the severity of imiquimo

277 r; LCR) displayed susceptibility to high fat diet-induced steatosis in association with reduced hepat

278 iated depletion of COP1 ameliorates high-fat diet-induced steatosis in mouse liver and improves liver

279 noparticles confers hepatoprotection against diet-induced steatosis in murine models and extends life

280 y to acute and chronic high-fat/high-sucrose diet-induced steatosis, without observed increases in li

281 cell mass that occur in response to age- and diet-induced stress.

282 bolic rate, physical activity thermogenesis, diet-induced thermogenesis, and energy intake) were meas

283 Meal tests were performed to investigate diet-induced-thermogenesis (DIT) and appetite sensation.

284 The addition of L. rhamnosus in the diets, induced transcriptional reduction of orexigenic g

285 stress in mice completely abolishes high fat diet-induced upregulation of TRIP-Br2 in BAT.

286 , Il5 (Tg) /CD300f (-/-) were protected from diet-induced weight gain and glucose intolerance.

287 Mutant mice are resistant to age- and diet-induced weight gain and insulin resistance, by mech

288 weight, which was associated with decreased diet-induced weight gain and insulin resistance.

289 ased insulin secretion and reversed high-fat-diet-induced weight gain and insulin resistance.

290 genic EE, which protected mice from high-fat diet-induced weight gain at ambient temperature (23 degr

291 EPO treatment reduced diet-induced weight gain from 9.6 +/- 1.5 to 4.2 +/- 1.4

292 particles either increases susceptibility to diet-induced weight gain in adulthood or increases insul

293 ble difference in resistance to high caloric diet-induced weight gain of the dab2-deleted mice was pr

294 total cholesterol levels and suppression of diet-induced weight gain.

295 pported a link between air pollution and non-diet-induced weight increases.

296 tive of this study was to examine effects of diet-induced weight loss on various vascular function ma

297 As in 8 obese human subjects after a 12-week diet-induced weight loss program.

298 plementation, adjunct to those achieved with diet-induced weight loss, on heptic injury and liver fat

299 her lymphatic dysfunction is reversible with diet-induced weight loss.

300 r into the developmental origins of obesity, dieting-induced weight gain, and anorexia nervosa.