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1 responsible for the decreased risk seen with dietary calcium.
2 ia and was actually accelerated by increased dietary calcium.
3 ated calciuretic effects were independent of dietary calcium.
4 pically consume below recommended amounts of dietary calcium.
5 her contributes to an anti-obesity effect of dietary calcium.
6 ct of vitamin D status on immune function is dietary calcium.
7 g PIH, may benefit from consuming additional dietary calcium.
8 rs with an etiology that includes inadequate dietary calcium.
9 ganism and isotopically lighter than source (dietary) calcium.
10 dietary fiber, 0.32 (95% CI, 0.11-0.96) for dietary calcium, 0.90 (95% CI, 0.27-2.95) for total fat,
11 airy products are the main natural source of dietary calcium, a diet providing 1500 mg Ca must contai
17 ealth education concerning the importance of dietary calcium and exercise on osteoporosis prevention
25 her potential triggers include deficiency of dietary calcium and repetitive mechanical loading of the
26 y contrast, there was no association between dietary calcium and stone formation in men aged 60 yr or
30 Colonic tumors were prevented by elevating dietary calcium and vitamin D(3) to levels comparable wi
31 may contribute to an anti-obesity effect of dietary calcium, and the mVDR may represent an important
33 indicating that the anti-obesity effects of dietary calcium are mediated by suppression of 1alpha, 2
34 r stone formation in the highest quintile of dietary calcium as compared with the lowest quintile was
35 ve risk was 0.63 (95% CI 0.40-0.98) for high dietary calcium but no supplemental calcium intake and 0
36 creased oxalate absorption in the absence of dietary calcium but not in association with the 300-mg C
37 verall data provide definitive evidence that dietary calcium can reduce oxalate absorption and excret
38 proximately 50 pM; P<0.001), suggesting that dietary calcium could reduce adipocyte mass by suppressi
39 esent in dairy products (the major source of dietary calcium) could be responsible for the decreased
40 suppressing 1alpha,25-(OH)2-D3 by increasing dietary calcium decreases adipocyte intracellular Ca2+ (
41 in aP2-agouti transgenic mice by increasing dietary calcium decreases adipocyte intracellular Ca2+ (
42 y, hypercalciuria in RZ mice is abolished by dietary calcium deprivation, suggesting that the hyperca
44 cium turnover was positively associated with dietary calcium during EP (P < or = 0.01), LP (P < or =
45 In parathyroidectomized rats, an increase in dietary calcium for 10 days increased serum calcium, wit
49 ng, total activity, calories, dietary fiber, dietary calcium, height, parity, recent hormone exposure
50 WC, or waist-hip ratio (WHR) interacts with dietary calcium in relation to subsequent annual change
51 e loss induced in mice by ovariectomy or low dietary calcium, in the latter case in both wild-type an
52 jective was to test the inhibitory effect of dietary calcium, in Western diets with high and low phyt
54 Evidence concerning the relation between dietary calcium intake and development of hypertension i
55 pplements are recommended in settings of low dietary calcium intake and high prevalence of anemia.
59 d not modify the inverse association between dietary calcium intake and the risk of stone formation.
60 ormation in women in the highest quintile of dietary calcium intake compared with women in the lowest
64 wever, differences in study design and a low dietary calcium intake in the populations studied limit
68 period, calcium retention was calculated as dietary calcium intake minus the calcium excreted in the
70 irement for men and women, we determined the dietary calcium intake required to maintain neutral calc
71 ween alcohol consumption and the relation of dietary calcium intake to 10-year incidence of hypertens
73 0.76) among women in the highest quartile of dietary calcium intake versus the lowest (p for trend =
75 r subsequent hip fracture; among women whose dietary calcium intake was less than 400 mg/d, those who
76 ing serum parathyroid hormone, the source of dietary calcium intake was subdivided into milk, which i
77 els and muscle strength, alcohol intake, and dietary calcium intake were associated with higher BMD.
79 r age, sex, weight, total energy intake, and dietary calcium intake) but not in the placebo group.
80 tions between fractional calcium absorption, dietary calcium intake, and risk for fracture have never
81 ause the Gambian women were adapted to a low dietary calcium intake, and/or obesity, high gestational
82 ack-years of smoking, dietary sodium intake, dietary calcium intake, blood lead, tibia lead, and pate
83 PTH levels, 1,25-dihydroxyvitamin D levels, dietary calcium intake, physical activity, and body size
85 mber of fractures since age 45 years and low dietary calcium intake, were associated with increased r
90 ized-order, crossover metabolic study with 3 dietary calcium intakes; the magnesium dietary intake wa
92 r this modulation of adipocyte metabolism by dietary calcium is a direct effect of inhibition of 1alp
93 valent nutritional disorders, and inadequate dietary calcium is a known contributor to the pathophysi
94 do not support the hypothesis that increased dietary calcium is associated with a greater prevalence
97 ects fed BMg, SB decreased the percentage of dietary calcium lost in the urine but increased that per
98 healthy, normal-weight women with intake of dietary calcium < 800 mg/d and energy intake </= 2200 kc
99 Among children in the lowest tertile of dietary calcium (<821 mg/d), fat mass gain was lower in
102 e purpose of this study was to determine how dietary calcium modulates the effects of conjugated lino
109 renal stones consumed almost 250 mg/day less dietary calcium (p < 0.01) than did women without stones
112 lations between %BF and fat mass changes and dietary calcium (r = -0.01, P = 0.9 and r = -0.05, P = 0
118 ic mice (P<0.01), suggesting that increasing dietary calcium stimulates adipose apoptosis and thereby
119 95% CI: 1.02, 1.18; n = 11) per 50 g/d], and dietary calcium [summary RR: 1.05 (95% CI: 1.02, 1.09; n
121 hed reports of trials studying the effect of dietary calcium supplementation on blood pressure were i
126 season on 25(OH)D remained significant when dietary calcium, vitamin D, and physical activity were u
130 stment for potential risk factors, intake of dietary calcium was inversely associated with risk for k
131 d with the form of zinc consumed, but higher dietary calcium was marginally associated with lower zin
132 ty in women (P-trend < 0.01) but not in men; dietary calcium was not associated with all-cause mortal
135 that suppressing 1,25-(OH)2-D by increasing dietary calcium will suppress adipocyte [Ca2+]i, thereby
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