ライフサイエンスコーパス: dietary intervention

1 0.65; 95% CI, 0.45 to 0.94; P = .02) by the dietary intervention.

2 tion and taking into account the effect of a dietary intervention.

3 asma insulin and inflammatory markers before dietary intervention.

4 individual disease penetrance as well as the dietary intervention.

5 cid metabolism, most of which are treated by dietary intervention.

6 l real-life metabolomic changes, following a dietary intervention.

7 otentially be used as a motivational tool in dietary intervention.

8 sensitivity) changed significantly after the dietary intervention.

9 loss in obese subjects who complete a 12-wk dietary intervention.

10 a randomized, crossover design after 6 d of dietary intervention.

11 which lethal cardiomyopathy is mitigated by dietary intervention.

12 olism influences the response to weight-loss dietary intervention.

13 ll animals were sacrificed at 24 weeks after dietary intervention.

14 le-isotope tracers were performed after each dietary intervention.

15 acement complete dentures through a tailored dietary intervention.

16 lications for reducing breast cancer risk by dietary intervention.

17 ated with the plasma lipoprotein response to dietary intervention.

18 her nutritional adequacy was altered by this dietary intervention.

19 st 5 days), during which they were given one dietary intervention.

20 roprotective effects of CR without requiring dietary intervention.

21 ches for skin barrier diseases, including by dietary intervention.

22 ate after a 5-y follow-up of a Mediterranean dietary intervention.

23 lt-onset MetS, and are partially amenable to dietary intervention.

24 ic patients within a multicenter, controlled dietary intervention.

25 rmination of exogenous metabolites following dietary interventions.

26 d) intervention, and 4) collectively for all dietary interventions.

27 re often the proximal target of family-based dietary interventions.

28 e possibility of modulating these effects by dietary interventions.

29 a wide range of energy (calorie)-restricted dietary interventions.

30 as missing or if they did not complete their dietary interventions.

31 ariability and better predict the success of dietary interventions.

32 atment options, particularly weight loss and dietary interventions.

33 dies are smaller, especially those including dietary interventions.

34 05) in response to Step II but not to Step I dietary interventions.

35 raneous variation, and predict the effect of dietary interventions.

36 ted for microbiota analyses before and after dietary interventions.

37 e increased by genetic, pharmacological, and dietary interventions.

38 ctors for chronic disease more than do other dietary interventions.

39 nd abdominal fat distribution in response to dietary interventions.

40 5(OH)D concentrations in comparison to other dietary interventions.

41 low-carbohydrate diets, and other higher-fat dietary interventions.

42 tions for this disorder have been limited to dietary interventions.

43 ed, controlled, single-blind, parallel-group dietary intervention, 195 men and women aged 21-60 y fro

44 A total of 67 patients completed the dietary intervention (33 completed the diet low in FODMA

45 6 months' DJBL treatment in combination with dietary intervention (34 successfully implanted, 31 comp

46 rations of esterified CLA in total lipids, a dietary intervention (6 wk) was conducted with six men a

47 In a longitudinal, double-blind, randomized dietary intervention, 6 volunteers were fed breakfast do

48 o significant differences in remission after dietary interventions (67.2% vs 63.3%).

49 Unexpectedly, acute dietary intervention aimed at modulating systemic nitrat

50 e mechanisms of EPA and DHA to better design dietary interventions aimed at reducing MetS.

51 al trial examined the feasibility of low-fat dietary interventions among postmenopausal women of dive

52 nts influencing individuals' BP responses to dietary intervention and cold pressor test.

53 r breast cancer during 8.5 years (median) of dietary intervention and cumulatively for all breast can

54 CD-DIET Study (Celiac Disease and Diabetes - Dietary Intervention and Evaluation Trial) is a multicen

55 rved within 3 wk after the initiation of the dietary intervention and in both casual seated and ambul

56 increased in several LSDs, is responsive to dietary intervention and is reduced in MPSVII and MPSI m

57 sh the relationship between an infancy-onset dietary intervention and risk of having MetS between 15

58 growth in SGA term infants can be altered by dietary intervention and to examine whether there is a c

59 r 12 wk of moderate aerobic exercise without dietary intervention and weight loss.

60 Dietary interventions and interventions to reduce neurop

61 ed fluid intake, many among those with other dietary interventions and more among those who received

62 These findings may be important in crafting dietary interventions and predicting adherence to these

63 stent lack of effect for low-fat, high-fiber dietary interventions and risk of colorectal cancer.

64 All dietary interventions and those focused on macronutrient

65 change in abdominal adiposity in response to dietary interventions, and the effects of the rs16147 si

66 ewer pharmacologic adjuncts; the efficacy of dietary interventions; and current inroads into the trea

67 Two weeks of dietary intervention ( approximately 4.3% weight loss) r

68 the Women's Health Initiative (WHI) low-fat dietary intervention are unknown.

69 While dietary interventions are beneficial, more stringent die

70 Dietary interventions are effective in producing histolo

71 onsequence, while not always evidence-based, dietary interventions are enjoying a renaissance in irri

72 l therapeutic targets, and several drugs and dietary interventions are now in development to treat mu

73 omized controlled trials that test different dietary interventions are required to identify maternal

74 o increase dietary compliance with long-term dietary interventions are warranted.

75 Results were reported by dietary intervention as follows: 1) counseling, 2) food

76 iew assessed the effectiveness of telehealth dietary interventions at facilitating dietary change in

77 uals with MetS may benefit from personalized dietary interventions based on APOE genotype.

78 Specific dietary interventions based on genetic predisposition in

79 These findings indicate that early dietary interventions based on n-3 FA may represent an a

80 We synthesized effects of dietary interventions before or during pregnancy on neon

81 nt in glucose concentrations after the 10-wk dietary intervention (beta +/- SE: 0.05 +/- 0.02 mg/dL;

82 There is insufficient data that any dietary intervention beyond 4-6 months of age has any pr

83 delayed physical activity) had the identical dietary intervention but with physical activity delayed

84 s and investigators were not masked from the dietary intervention, but investigators analysing the da

85 The separate and combined effects of these dietary interventions by baseline blood pressure (BP) ha

86 We tested the hypothesis that dietary intervention can inhibit the development of recu

87 ness studies show that individually targeted dietary interventions can be effective and cost-effectiv

88 The study determined that low-fat dietary interventions can influence body weight and decr

89 We conclude that dietary interventions can partially offset genetic susce

90 Tailored dietary intervention contemporaneous with replacement de

91 ention (diet group, n = 43) or HAART without dietary intervention (control group, n = 40) for 12 mont

92 ci (ACF) in C57BL/6N female mice fed various dietary interventions (control, energy restricted and hi

93 pleted the study), 39 patients received only dietary intervention (controls, 35 completed the study).

94 fferences in the interindividual response to dietary intervention could be modified by genetic variat

95 Unlike a drug, a dietary intervention could be quickly transitioned to th

96 fect the interpretation of epidemiologic and dietary intervention data and the design of food-content

97 es at baseline and longitudinally by using a dietary intervention design.

98 ce from a randomized controlled trial that a dietary intervention designed to elicit weight loss redu

99 Ultimately, novel dietary interventions designed to reactivate BAT could b

100 mpliance and CVD risk factor response to two dietary interventions designed to treat hypertension, dy

101 ned before and at the end of weight-reducing dietary intervention (DI) in 23 obese women.

102 by weight regain after an energy-restricted dietary intervention (DI).

103 HAART, were randomly assigned to HAART with dietary intervention (diet group, n = 43) or HAART witho

104 le studies are warranted to evaluate whether dietary intervention during pregnancy aimed at lowering

105 isk of developing obesity can be modified by dietary intervention early in life.

106 structured weight loss program that includes dietary intervention, exercise therapy, and behavior mod

107 Our dietary intervention experiments revealed that diet affe

108 ss liner (DJBL) treatment in comparison with dietary intervention for obesity and type 2 diabetes mel

109 diet (LGD) has been proposed as an effective dietary intervention for pediatric obesity, but to our k

110 ) study, which is a randomized Mediterranean dietary intervention for primary cardiovascular preventi

111 Efficacy of dietary intervention for treatment and prevention of HIV

112 wever, little is known about how a long-term dietary intervention for weight loss affects circulating

113 Participants followed the 3 weight-stable dietary interventions for 4 wk with glucose, insulin, an

114 Ts does not support low-fat diets over other dietary interventions for long-term weight loss.

115 Data supporting the efficacy of dietary interventions for preventing or treating high bl

116 ht to review the latest literature regarding dietary interventions for prostate cancer with a special

117 These data can guide dietary interventions for the prevention of diverticulit

118 In developing dietary interventions for this population, one goal may

119 sleep may compromise the efficacy of typical dietary interventions for weight loss and related metabo

120 We compared the dietary intervention group with a group who received fiv

121 ng their third trimester to participate in 3 dietary intervention groups: a control group, who were e

122 Various dietary interventions have been used to treat patients w

123 Step I and Step II dietary interventions have multiple beneficial effects o

124 Recent studies indicate that dietary interventions have the potential to prevent and

125 cinogenesis, and might have implications for dietary intervention in human familial adenomatous polyp

126 ermining biochemical modifications following dietary intervention in humans.

127 new data and its potential implications for dietary intervention in the treatment of inflammatory di

128 C phenotype) without the requirement for any dietary intervention in their suckling period.

129 nvelopes that each contained one of the four dietary interventions in a random order.

130 controlled trials evaluating multifactorial dietary interventions in adults with chronic disease tha

131 dings underscore the potential importance of dietary interventions in improving amino acid profiles (

132 Dietary interventions in low-income countries and underw

133 We also discuss more recent dietary interventions in rodents, monkeys, and humans, w

134 Short-term intensive dietary interventions in selected populations increase f

135 n pubertal children, sexual maturation, BMI, dietary intervention (in girls), and dietary cholesterol

136 gn that consisted of a 14-d fully controlled dietary intervention involving 37 subjects [mean +/- SD

137 icrobiota respond consistently to prescribed dietary interventions, irrespective of prior dietary pra

138 ly, this improved metabolic status after 2DG dietary intervention is associated with markedly reduced

139 undernutrition has been assessed, successful dietary intervention is contingent on an accurate estima

140 Repeated infancy-onset dietary intervention is effective in the prevention of M

141 little dietary improvement, suggesting that dietary intervention is necessary.

142 tion, which is easily modifiable by means of dietary intervention, is an important risk factor for hi

143 Caloric restriction (CR) is a dietary intervention known to enhance cardiovascular hea

144 However, the addition of exercise to dietary intervention led to greater improvements in leve

145 ulates crypt hyperplasia and/or colitis, and dietary intervention may be a novel strategy to modulate

146 In addition, a Mediterranean dietary intervention may mitigate potential deleterious

147 sented support the possibility that maternal dietary interventions may be an effective way to promote

148 Additionally, because adherence to dietary interventions may be particularly problematic am

149 Intensive dietary interventions may be safe, with modest short-ter

150 Moreover, the data suggest that early dietary interventions may reduce the severity of some fe

151 After 5 weeks of dietary intervention, mice were challenged with Citrobac

152 to 1998 were randomly assigned to a low-fat dietary intervention (n = 19,541) or comparison (n = 29,

153 supplementation (n = 372), and four RCTs for dietary intervention (n = 201) were meta-analysed using

154 Certain DPs may impair responses to dietary interventions, necessitating the introduction of

155 English-language studies with a dietary intervention of > or =4200 kJ/d (1000 kcal/d), w

156 When compared with dietary interventions of similar intensity, evidence fro

157 f this study was to evaluate the efficacy of dietary intervention on blood lipids of human immunodefi

158 regression models to examine the effect of a dietary intervention on colorectal adenoma recurrence in

159 al abnormalities and the potential impact of dietary intervention on outcome.

160 as to analyze the effects of the WHI low-fat dietary intervention on serum glucose and insulin and in

161 No effect of dietary interventions on anthropometric measurements and

162 terol Education Program's Step I and Step II dietary interventions on major cardiovascular disease ri

163 comparing the effects of specific long-term dietary interventions on pericardial fat tissue mobiliza

164 Although the effects of acute dietary interventions on the human metabolome have been

165 effect (>/=1 year) of low-fat and higher-fat dietary interventions on weight loss by searching MEDLIN

166 These transporter proteins are targetable by dietary interventions, opening the avenue to a precision

167 stearoyl-coenzyme A desaturase 1 activity by dietary intervention or genetic manipulation strongly in

168 (RCTs) with blood lipid outcomes, involving dietary intervention or supplementation for the treatmen

169 ls but had the opposite effect in ad libitum dietary interventions or long-term trials (>/=1 y).

170 andomized studies using dietary supplements, dietary interventions, or both.

171 receive or not receive estrogen without any dietary intervention other than calcium and multivitamin

172 , high sodium and high sodium plus potassium dietary intervention periods.

173 LA intake increased significantly during the dietary intervention, plasma CLA concentrations were not

174 The effect of a dietary intervention plus n-3 (omega-3) fatty acid suppl

175 interest and to design, target, and evaluate dietary intervention programs.

176 Both omega-3 supplementation and dietary intervention reduced triglyceride level, with th

177 Dietary intervention reduced triglyceride levels by -0.4

178 es for studies investigating the efficacy of dietary interventions (reducing infiltration by immune c

179 rated list of random numbers to one of three dietary interventions: replacement calories using an iso

180 We included 107 articles describing 94 dietary interventions reporting data for 3268 participan

181 ed to usual prenatal care (control group) or dietary intervention, self-monitoring of blood glucose,

182 Dietary interventions should focus on decreasing energy

183 Step I and Step II dietary interventions significantly decreased plasma lip

184 d, crossover design of three 1-mo controlled dietary interventions: soy protein isolate enriched with

185 double-blind, placebo-controlled trial (the Dietary Intervention, Stem cells and Colorectal Cancer (

186 Dietary intervention studies are needed to prove that co

187 Dietary intervention studies have shown that enteral the

188 MEDLINE was used to select 37 dietary intervention studies in free-living subjects pub

189 Numerous dietary intervention studies in humans and animals indic

190 Evidence from dietary intervention studies shows that the intake of fl

191 Dietary intervention studies support the concepts that r

192 upport the need for further experimental and dietary intervention studies that target circulating tra

193 Two human dietary intervention studies were used to translate the

194 Dietary intervention studies with soy will provide infor

195 l emphasis is placed on CHD risk assessment, dietary intervention studies, diet-gene interactions, an

196 lipoprotein responses have been variable in dietary intervention studies.

197 for biomarkers of meat and fish intake in a dietary intervention study and in free-living subjects f

198 hundred fifty-nine adolescent females in the Dietary Intervention Study in Children (age range: 14-18

199 The Dietary Intervention Study in Children (DISC) is a multi

200 Our preliminary studies in the Dietary Intervention Study in Children suggest a signifi

201 d, randomized, double-masked, 2-parallel-arm dietary intervention study in which healthy participants

202 This dietary intervention study provides evidence that regula

203 se-dependent, parallel randomized controlled dietary intervention study was conducted.

204 ive of this prospective, single-site, 2-year dietary intervention study was to evaluate the effects o

205 ) recruited into the Food4Me pan-European PN dietary intervention study were randomly assigned to 4 t

206 ouble-blinded, randomized 2 x 5-wk crossover dietary intervention study with a 14-d run-in period dur

207 In a randomized parallel-group dietary intervention study, 316 overweight and obese par

208 nto Cancer and Nutrition (EPIC) study.In the dietary intervention study, 4 groups of 10 subjects cons

209 In this single-blind, parallel, controlled, dietary intervention study, MetS subjects (n = 472) from

210 randomized, double-blind, placebo-controlled dietary-intervention study involving 944 children who we

211 Our data suggest that dietary interventions such as FR, or treatment that mimi

212 Telehealth-delivered dietary interventions targeting whole foods and/or dieta

213 Calorie restriction (CR) is a dietary intervention that extends lifespan and healthspa

214 Our findings suggest a dietary intervention that may suppress the disease proce

215 Over a mean of 8.1 years, a dietary intervention that reduced total fat intake and i

216 nd 1994 to test the feasibility of a low-fat dietary intervention that targeted low-income, black, an

217 To identify a dietary intervention that would effect GIT cell prolifer

218 Here we investigated the impact of dietary interventions that affect systemic glucose and l

219 These findings might be relevant to dietary interventions that include noncaloric beverages,

220 the lack of randomized, prospective trials, dietary interventions that modify iron intake and bioava

221 ment that promote hyperglycemia and identify dietary interventions that reverse these effects.

222 /E3, n = 44 E3/E4) and followed a sequential dietary intervention (the SATgenepsilon study) in which

223 After 6 weeks of dietary intervention, the left iliac artery of each anim

224 cipants, the composition and duration of the dietary interventions, the nutrients studied and dietary

225 As such, clinical and dietary intervention to affect this process - on the gut

226 gies using study data collected as part of a dietary intervention to improve health and which elicits

227 ttle evidence to support the hypothesis that dietary intervention to modify fat composition during pr

228 lts encourage healthy life style choices and dietary intervention to modify this risk.

229 2000 in a multicenter, controlled trial of a dietary intervention to prevent additional breast cancer

230 gned to examine the efficacy and safety of a dietary intervention to reduce serum LDL cholesterol (LD

231 Dietary interventions to increase F&V intake may be a pr

232 strategies aimed at developing lifestyle and dietary interventions to maintain adequate energy intake

233 cations of obesity have prompted interest in dietary interventions to reduce weight, including low-ca

234 evention will be key, and this could include dietary interventions to stimulate bone formation.

235 rates in situ, to guide physical activity or dietary interventions toward efficient weight loss, are

236 een 1996 and 1998, a multicenter, randomized dietary intervention trial among breast cancer survivors

237 ed dietary intake over time as shown in this dietary intervention trial being conducted among breast

238 4 US states who participated in a randomized dietary intervention trial in 1995-2006, with maximum fo

239 We conducted a dietary intervention trial to study these effects in par

240 abochip in a long-term randomized controlled dietary intervention trial, the Special Turku Coronary R

241 Dietary intervention trials aim to change dietary patter

242 iomarkers to assess compliance in short-term dietary intervention trials and habitual diets in observ

243 etabolic studies, epidemiologic studies, and dietary intervention trials of diet and CHD.

244 atty acid biomarkers to assess compliance in dietary intervention trials with their application in ep

245 d by nitrate at concentrations achievable by dietary intervention under normoxic and hypoxic conditio

246 e subjected to the following 2 intensive 3-d dietary interventions under isocaloric conditions: 1) a

247 The dietary intervention was associated with a change in the

248 randomized, double-blind, placebo-controlled dietary intervention was carried out in 119 healthy men

249 The dietary intervention was conducted among 1,906 participa

250 An 8-wk dietary intervention was conducted to test the hypothesi

251 The telehealth dietary intervention was effective at improving diet qua

252 endoscopic biopsy specimens associated with dietary intervention was identified.

253 insulin AUC did not improve, supporting that dietary intervention was not sufficient to recover gluco

254 After a 2-y dietary intervention, we showed significant interactions

255 Dietary interventions were: donated banked breastmilk ve

256 gnificantly among participants receiving the dietary intervention, while increases among control wome

257 ese studies provide hope that evidence-based dietary interventions will significantly impact the risk

258 children aged 18 years or younger, comparing dietary intervention with a diet plus exercise program o

259 A low-intensity dietary intervention with an LGI diet compared with an H

260 pted guidelines, we tested whether a 1-month dietary intervention with flavanol-containing cocoa lead

261 were increased in mice and humans following dietary intervention with GF/Bb and correlated with redu

262 1993 to 1998 at 40 US clinical centers to a dietary intervention with goals of a reduction of fat in

263 We tested the hypothesis that a 12-wk dietary intervention with high-glucoraphanin (HG) brocco

264 In NOD mice, dietary intervention with omega-3 PUFAs sharply reduced

265 d to investigate metabolic changes following dietary intervention with soy isoflavones in healthy pre

266 tudies and human clinical trials showed that dietary intervention with these dietary oligosaccharides

267 on of cardiovascular disease risk factors by dietary intervention with whole grains, which included l

268 We developed four dietary interventions with a stepwise variance in concor

269 ries; these results support the inclusion of dietary interventions with plant sources of beta-caroten

270 ere, we have summarized the main findings of dietary interventions with these specific oligosaccharid

271 During the 8.5-year dietary intervention, with 1,764 incident breast cancers

272 We hypothesized that a dietary intervention would demonstrate the importance of

273 CR has been shown to be a potent dietary intervention, yet the mechanisms by which the CR