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1 e aorta and use it to assess the response to dietary modification.
2 easily achieved in the general population by dietary modification.
3 to influence cardiovascular disease risk by dietary modification.
4 including active therapeutic approaches and dietary modification.
5 ithelial TLR4 that could be reversed through dietary modification.
6 increased significantly from baseline during dietary modification.
7 cal conditions, myopathy may be mitigated by dietary modifications.
8 ound the world for patients nonresponsive to dietary modifications.
9 ng early development in response to maternal dietary modifications.
11 in atherosclerotic plaques, and confirm that dietary modification and statin therapy reduce MMP activ
12 hibitor (MPI) and to determine the effect of dietary modification and statin treatment on MMP activit
13 jor categories of interventions are covered: dietary modification and supplementation, herbal product
16 iation of new subspecialist care, medication/dietary modifications, and furthering life-saving proced
17 ials of the effects of specific nutrients or dietary modifications are not always feasible, they prov
20 ion trials are testing whether postdiagnosis dietary modification can influence breast cancer recurre
21 changes such as weight loss in obese men and dietary modification can lessen urgency, nocturia, and i
26 nd iNOS expression, and that weight loss via dietary modification effectively reverses these deleteri
29 tical review of the most recent evidence for dietary modifications, food supplements, and herbs in pr
30 n MP density were obtained within 4 weeks of dietary modification for most, but not all, subjects.
33 loss in obese men with diabetes and LUTS and dietary modification has also been shown to be effective
37 opathy and warrant further investigations of dietary modification in the management of these conditio
38 gned to lower plasma cholesterol by means of dietary modifications in accordance with recommendations
39 novel index [Women's Health Initiative (WHI) Dietary Modification Index (DMI)] of diet quality was as
40 Participants were randomly assigned to the dietary modification intervention (n = 19,541; 40%) or t
42 ry fat intake was significantly lower in the dietary modification intervention group compared with th
46 the effect of the Women's Health Initiative Dietary Modification low-fat and increased fruit, vegeta
47 tries, but epidemiological data suggest that dietary modification might reduce these by as much as 90
52 alculations of body-weight change and of the dietary modifications required for weight-loss maintenan
53 examines the current indications for various dietary modification strategies in patients with CKD (eg
55 ced bronchoconstriction (EIB) may respond to dietary modification, thereby reducing the need for phar
58 ed lactose intolerance may result in adverse dietary modifications; thus, more studies are needed to
59 en enrolled in the Women's Health Initiative dietary modification trial comparison group and 59,105 w
60 articipants in the Women's Health Initiative Dietary Modification Trial completed a doubly labeled wa
61 Purpose Earlier Women's Health Initiative Dietary Modification trial findings suggested that a low
62 led onto the Women's Health Initiative (WHI) Dietary Modification trial from 1993 to 1998 were random
64 PARTICIPANTS: The Women's Health Initiative Dietary Modification Trial, a randomized controlled tria
65 mone therapy trial, and randomization to the dietary modification trial, the predictive model include
67 tion phases of the Women's Health Initiative Dietary Modification Trial.Participants comprised 48,835
68 articipated in the Women's Health Initiative Dietary Modification Trial; 40% (19,541) were randomized
70 pinach and corn, three types of responses to dietary modification were identified: Eight "retinal res
71 p new knowledge about specific nutrients and dietary modifications within a framework of interaction
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