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1 alue in the occasional patient intolerant of dietary protein.
2 - 0.2 kg body weight during ED regardless of dietary protein.
3 h different glycemic indexes and contents of dietary protein.
4 ted by insufficient or ineffectual intake of dietary protein.
5 nephron HCO(3) secretion that is induced by dietary protein.
6 ession of S phase proteins in the absence of dietary protein.
7 ortant gustatory stimulus believed to signal dietary protein.
8 was high in liver and varied with changes in dietary protein.
9 ts appeared to be due to the lower amount of dietary protein.
10 se signals represent meal-based responses to dietary protein.
11 tion of daily energy, but not necessarily of dietary protein.
12 re pancreatic serine proteinases that digest dietary proteins.
13 o experiments to track the metabolic fate of dietary proteins.
14 derived from pathogens, microbial flora, or dietary proteins.
15 role in promoting tolerance of the flora and dietary proteins.
16 ly of resident salivary proteins but also of dietary proteins.
17 ge described for complexes of EGCG and other dietary proteins.
18 d be used as a supplement to poorly balanced dietary proteins.
19 to evaluate the effect of graded intakes of dietary protein (0.7, 0.8, 0.9, and 1.0 g/kg) on calcium
21 cid intake that was higher by 4.72% of total dietary protein (2 SD) were -1.5 to -3.0 mm Hg systolic
23 d across membranes as an efficient route for dietary protein absorption and for maintaining cellular
25 study shows that the quantity and source of dietary proteins act as regulators of gut microbiota met
26 acterized a transient feeding preference for dietary protein after modest starvation in the fruit fly
27 e decreases suggests that the safe amount of dietary protein, although sufficient for maintenance of
30 d longitudinal associations between baseline dietary protein and bone mineral density (BMD) among 560
32 a finite element analysis, and we evaluated dietary protein and calcium with the use of a validated
35 udy was to determine the association between dietary protein and changes in total LM and nonbone appe
37 licited by chronically inadequate intakes of dietary protein and energy are affected by the protein c
39 d it is likely that the observed decrease in dietary protein and energy intake plays an important rol
42 dent, inverse dose-response relation between dietary protein and In tHcy (P = 0.002) and a positive,
47 ous non-carbonic acid production (NEAP) from dietary protein and potassium content enables exploratio
48 In 2 studies, we examined the ability of dietary protein and potassium to predict markers of bone
50 ctive was to examine the association between dietary protein and risk of IHD in a prospective study o
51 n have suggested an inverse relation between dietary protein and risk of ischemic heart disease (IHD)
52 bjective was to examine the relation between dietary protein and risk of stroke in men who participat
55 t growth and proliferation in the absence of dietary protein and thus uncouple neuroblasts from syste
57 se findings question the respective roles of dietary proteins and endogenous sources in generating si
58 ast growth factor 21 (FGF21) is regulated by dietary proteins and not, as has been heretofore assumed
61 est that CPO cleaves acidic amino acids from dietary proteins and peptides, thus complementing the ac
67 proteolysis, independent of energy status or dietary protein, as the activities of the 26S proteasome
74 ids affect the digestion and assimilation of dietary proteins by accelerating hydrolysis by pancreati
75 ody protein turnover and the contribution of dietary protein can be quantified in critically ill pati
76 nsume adequate or moderately high amounts of dietary protein can use RT to improve body composition,
77 cterial richness tended to decrease when the dietary protein concentration reduced from 16% to 10%.
78 mous effects: mouse strain (A/J or C57BL/6), dietary protein content (14% protein and 0.3% L-cysteine
80 sions were efficiently corrected by lowering dietary protein content, and this was associated with tr
86 )) augments the postprandial availability of dietary protein-derived amino acids in the circulation a
87 )) augments the postprandial availability of dietary protein-derived amino acids in the circulation a
88 represent an important factor that modulates dietary protein digestion and absorption kinetics and th
90 e assessed the effect of meat texture on the dietary protein digestion rate, amino acid availability,
95 xamined the prospective association of novel dietary protein food clusters (derived from established
98 simulated substitution of total and type of dietary protein for carbohydrate and of vegetable for an
99 his introduced alga as a potential source of dietary protein for human consumption in New Zealand.
102 ased oral exposure to chemicals compete with dietary proteins for the development of oral tolerance,
105 le-genome profiling revealed that increasing dietary protein from 5 to 40% increased duodenal transcr
106 nder practical dietary conditions, increased dietary protein from animal sources was not detrimental
109 s, which provided approximately 50% of total dietary protein from meat sources (beef, poultry, pork,
110 his study examined the gastric hydrolysis of dietary protein from raw and roasted almonds in the grow
111 varied in main protein sources (60% of total dietary proteins from lean-seafood or nonseafood sources
112 postmenopausal women, a moderate increase in dietary protein, from 10% to 20% of energy, slightly imp
114 The response of this metabolic pathway to dietary protein (i.e., meal threshold) declines with adv
115 such as low glycemic index carbohydrates and dietary protein impact appetite, calorie intake and meta
118 ng the first 6 mo postpartum while consuming dietary protein in amounts that exceeded those of their
119 sceptibility to infections; yet, the role of dietary protein in immune memory homeostasis remains poo
124 The findings show a metabolic requirement of dietary protein in sustaining functional CD8 memory and
128 nderlying negative responses to an excess of dietary protein, including the causes of the wasting syn
129 atically enhanced the proteolysis of several dietary proteins, including beta-lactoglobulin, bovine s
138 showed a significant association with higher dietary protein intake (effect per allele = 0.08 [0.06,
140 and meta-analysis evaluating the effects of dietary protein intake alone and with calcium with or wi
146 -increasing allele of FTO variant and higher dietary protein intake and offer insight into potential
147 uthors aimed to examine the relation between dietary protein intake and risk of cholecystectomy among
149 certain whether lower dietary acidity (lower dietary protein intake but higher potassium intake-ie, l
151 ether regular physical activity and adequate dietary protein intake can attenuate the loss of skeleta
154 hat plasma glutathione turnover decreases as dietary protein intake decreases suggests that the safe
156 nciple trial to test whether manipulation of dietary protein intake during a marked energy deficit in
157 We examined the relation between maternal dietary protein intake during pregnancy and offspring an
158 l studies suggest that specifically maternal dietary protein intake during pregnancy influences child
163 ential benefits and challenges of optimizing dietary protein intake in older adults continues to evol
170 tudies have shown that more than half of the dietary protein intake is used by the gut and that a lar
174 y and suggest that interventions to optimize dietary protein intake may improve vaccine efficacy in m
175 controlling serum phosphorus by restricting dietary protein intake may outweigh the benefit of contr
178 e is currently no consensus on the effect of dietary protein intake on the skeleton, but there is som
179 cle mass persisted after standing height and dietary protein intake per kilogram body mass was contro
185 hat the maintenance of physical activity and dietary protein intake would attenuate the age-related d
186 tary intake (particularly in caloric intake, dietary protein intake, dietary fiber intake, and micron
187 n proteasome system (UPS) response to varied dietary protein intake, energy deficit (ED), and consump
189 and stiffness of the peripheral skeleton and dietary protein intake, which is mainly related to chang
193 intakes (P-trend = 0.003), higher calibrated dietary protein intakes (P-trend = 0.03), higher aMED sc
194 load and stiffness, bone microstructure, and dietary protein intakes from various origins (animal, di
195 ed by areal bone mineral density (aBMD), and dietary protein intakes, particularly from specific diet
196 density (BMD) are positively correlated with dietary protein intakes, which account for 1-8% of BMC a
198 ood intake suggesting the perceived value of dietary protein is a critical determinant of its effect
200 odels.In a protein-replete cohort of adults, dietary protein is associated with ALM and QS but not wi
202 sults suggest that the requirement for total dietary protein is not different for healthy older adult
204 ntial amino acid or protein degradation when dietary protein is reduced by anorexia, negative nitroge
205 a function of drinking water, bulk diet, and dietary protein isotope ratios, explains >85% of the obs
206 males and females and juveniles derive their dietary protein largely from daily fruit and seasonal nu
208 tudy was conducted to investigate impacts of dietary protein levels on gut bacterial community and gu
209 ism by evaluating the effect of 2 amounts of dietary protein (low: 0.7 g/kg; and high: 2.1 g/kg) on f
212 ort the notion that modifying the sources of dietary protein may be potentially applied to prevent T2
215 .e. the precise macronutrient composition of dietary protein) may impact the effectiveness of weight
216 moderate increases in both cereal fibers and dietary protein (Mix diet) on insulin sensitivity, as me
219 stone disease are sparse, and the effects of dietary protein of different origins are not clear.
221 fication effects for intervention varying in dietary protein on 2-year changes in fat-free mass, whol
222 o determine the effects of varying levels of dietary protein on body composition and muscle protein s
224 despite intense investigation, the impact of dietary protein on calcium metabolism and bone balance r
226 The objective was to test the effect of dietary protein on calcium retention at low and high int
227 aluated the short-term effects of increasing dietary protein on Fe absorption and expression of genes
230 te the effects of the quantity and source of dietary protein on microbiota composition, bacterial met
232 protein intake level and the food sources of dietary protein on the risk of ESRD in the general popul
233 yrosine intake (alone or as a constituent of dietary protein) on the production of the brain neurotra
236 for the first half of gestation of maternal dietary protein, or of total calorific intake on isolate
237 to our knowledge, we evaluate the effects of dietary protein (P), carbohydrate (C), fat (F), and ener
238 nificant negative correlations between total dietary protein (per kg) and markers of bone turnover (P
239 renal acid load (with an algorithm including dietary protein, phosphorous, potassium, magnesium, and
242 ifferences in the time of ingestion of daily dietary protein providing sulfate are related to the dev
245 studies using gnotobiotic mice revealed that dietary protein reduces the in vivo microbial metabolism
249 This study was designed to determine the dietary protein requirement of elderly women by using th
251 ose a framework for approaching the variable dietary protein requirements in patients with CKD or end
253 minated when urea excretion was decreased by dietary protein restriction (4% by weight), consistent w
254 d methylmalonic acidemia (MMA) is managed by dietary protein restriction and medical food supplementa
257 RNA levels of all fibrinogen genes caused by dietary protein restriction in rats after weaning (alpha
258 hepatic, genomic, and metabolic responses to dietary protein restriction in the non-pregnant Sprague-
263 expenditure is a primary metabolic effect of dietary protein restriction, and requires both UCP1 and
264 that hepatic FGF21 expression is induced by dietary protein restriction, but not energy restriction.
269 nsiderable attention has recently focused on dietary protein's role in the mature skeleton, prompted
270 ts of "high versus low" protein intake or 2) dietary protein's synergistic effect with Ca+/-D intake
274 s experiencing increasing global demand as a dietary protein source and constituent of livestock feed
278 imate of the study site, adult age, sex, and dietary protein source on individual requirements; and t
281 as to elucidate the potentials of the 2 main dietary protein sources lean seafood and nonseafood to m
282 hensive and in-depth assessment of different dietary protein sources related to type 2 diabetes (T2D)
283 zones of Bangladesh) of six prioritized key dietary protein sources: Oryza sativa (rice), Triticum a
286 subjects were randomly assigned to 12 wk of dietary protein supplementation (42 g/d) with either a m
287 ed trials (RCTs) investigating the effect of dietary protein supplementation during prolonged (>6 wk)
289 prudent to advise the intake of high-quality dietary protein to ensure adequate intakes of a number o
290 The recommendation to intentionally restrict dietary protein to improve bone health is unwarranted, a
291 c sulfur to urinary sulfate excretion and of dietary protein to urinary sulfate and nitrogen excretio
292 was developed to predict the potential of 72 dietary proteins to act as a source of dipeptidyl peptid
295 of nematodes by either limiting use of their dietary protein uptake from the crop or by preventing ro
297 cently, a more even mealtime distribution of dietary protein was positively associated with muscle ma
298 udy site, adult age class, sex, or source of dietary protein were observed, although there was an ind
299 mmunity-dwelling adults consume insufficient dietary protein, which may contribute to the age-related
300 topic and meta-analysis studies suggest that dietary protein works synergistically with calcium to im
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