ライフサイエンスコーパス: diethylamine

1 r, which leads to the elimination of neutral diethylamine.

2 Each complex was reacted with diethylamine.

3 amine functionalities of 2,2'-(ethylenedioxy)diethylamine.

4 s similarly to 1 for allylic aminations with diethylamine.

5 ile with very electron-rich species, such as diethylamine.

6 The most potent compounds such as the diethylamine 44 (K(i) = 60 nM) contain a basic group at

7 Oxime carbamates derived from the volatile diethylamine afforded aryliminyl radicals that proved co

8 -order rate constants for reaction of 5 with diethylamine and 1,3-cyclohexadiene were determined to b

9 ts 2-lithio-N,N-dimethylbenzylamine (1), the diethylamine and diisopropylamino analogues (2, 3), and

10 mine derivative 2 (anion 1, where R = Et) to diethylamine and NO; (b) results for the zwitterionic pi

11 is considered here to be a rigid analogue of diethylamine, and thus, the target compounds are all con

12 ucts originated from the photolysis of 8a in diethylamine are characterized by analytical techniques,

13 rated on a weak convective interaction media diethylamine (CIM DEAE) monolithic chromatographic colum

14 nitroso-1-propylhydrazine (PAPA) NONOate and diethylamine (DEA) NONOate) that do not target thiol res

15 Diethylamine (DEA) was chosen as the amine source due to

16 t systolic depression induced by an NO donor diethylamine(DEA)NO or nitroglycerin (NTG).

17 ), amines (including dimethylamine, DMA, and diethylamine, DEA), alkyl nitrates (RONO(2)) and nitrous

18 indicate a transition in structure, and for diethylamine (DeltaGB = 40 kJ/mol), the dominant structu

19 ssociation of the otherwise unfunctionalized diethylamine derivative 2 (anion 1, where R = Et) to die

20 ssing sGC resulted in a 30-50% inhibition of diethylamine diazeniumdiolate-NO-stimulated sGC activity

21 koff to Markovnikoff addition occurs without diethylamine dissociation.

22 enyl)[1-Me2Si((t)BuN)TiCl2]-3-C2H4-[N,N-bis((diethylamine)ethyl)-amine ]CrCl3 (Ti-C2-Cr(NNN)), are sy

23 > triethylamine > ethylamine > methylamine > diethylamine > tert-butylamine > ammonia.

24 apped stereoselectively by acrylonitrile and diethylamine in high yields.

25 r, a primary site of action of lysergic acid diethylamine (LSD), appears to dominate efavirenz's beha

26 Consistent with this, an NO donor, diethylamine nitric oxide (DEANO), induced TGF-beta1 gen

27 NO or NO released from NO donors such as the diethylamine/nitric oxide complex (DEA/NO) and SNAP.

28 The NO donor, diethylamine NO complex sodium salt (10-100 microM), act

29 icrovascular endothelial cells, the NO donor diethylamine-NO (DETA-NO, 100 microM) reduced VCAM-1 gen

30 rified DbetaH was inhibited by the NO donor, diethylamine/NO (DEA/NO), with an IC(50) of 1 mm.

31 of human neuroblastoma cells (SK-N-MC) with diethylamine/NO decreased cellular DbetaH activity witho

32 These data contrasted with the pure NO donor diethylamine/NO, which induced a negligible inotropic re

33 NO generated from diethylamine NONOate (DEA-NONOate) or spermine NONOate (

34 In this study, diethylamine NONOate (DEA/NO) was used to characterize t

35 taneous treatment with LPS and the NO donor, diethylamine NONOate (DEA/NO), enhanced and prolonged JN

36 that various nitric oxide donors, including diethylamine NONOate (DEA/NO), stimulated large increase

37 The NO donor diethylamine NONOate (DEA/NO, 10 microM) significantly i

38 and determined the abilities of an NO donor, diethylamine NONOate (DEANO), and a single dose of HU to

39 hen the pathway was stimulated by NO donors (diethylamine NONOate or sodium nitroprusside) or the cyc

40 on of either ANGII (500 fmol) or a NO donor (diethylamine nonoate, 500 pmol) both depressed barorefle

41 Three NO donors, diethylamine NONOate, spermine NONOate, and S-nitrosoglu

42 tors S-nitroso-N-acetyl-D,L-pencillamine and diethylamine NONOate, the absorbance bands of the [2Fe-2

43 In the presence of the NO.-generator diethylamine nonoate, the electron spin resonance spectr

44 s prevented by coperfusion with the NO-donor diethylamine NONOate.

45 inoma cells exposed to the chemical NO donor diethylamine-NONOate showed increased immunoreactivity o

46 nd intracavernosal injection of the NO donor diethylamine-NONOate were augmented in eNOS-/- and nNOS-

47 elated peptide, whereas neither the NO donor diethylamine/NONOate nor the nitrovasodilator nitroglyce

48 ly, plasma cGMP was increased by infusion of diethylamine/NONOate or nitroglycerin but was unaffected

49 For the smaller alkenes, the diethylamine nucleophilically attacks the coordinated al

50 presentative Re-BFPCA 8a in a model solvent, diethylamine, proceeded to give a high yield of intermol

51 xide (NO) donors sodium nitroprusside (SNP), diethylamine sodium (DEA), 3-morpholinosydnonimine (SIN-

52 oreover, regeneration of the LPG surface via diethylamine treatment resulted in approximately 80% rem

53 icities and nucleophilicities: n-butylamine, diethylamine, triethylamine, N-methylpyrrolidine, and tr

54 nitrile, methanol, and water containing 0.1% diethylamine (v/v), at a flow rate of 2.5 ml/min, a colu

55 nitrile, methanol, and water containing 0.1% diethylamine (v/v).

56 Vascular smooth-muscle relaxation to diethylamine was enhanced in endothelium-denuded HC vess

57 l (HNO), a product of the reaction of 5 with diethylamine, was also observed.

58 mixing (baseline noise 0.8 nS/cm for 27 muM diethylamine) with low band dispersion (as small as 30 m