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1 muscle cells and serves as a skeletal muscle differentiation marker.
2 roblast platelet-derived growth factor alpha differentiation marker.
3 y, and increase the expression of hepatocyte differentiation markers.
4 t-like structures expressing region-specific differentiation markers.
5 igration, and a decline in the expression of differentiation markers.
6 eroxide levels and reduced the expression of differentiation markers.
7 ells and ectopic expression of squamous-like differentiation markers.
8 did not correlate with the expression of the differentiation markers.
9  or progenitor markers and low expression of differentiation markers.
10 a-catenin signaling express higher levels of differentiation markers.
11 s, and increased the expression of epidermal differentiation markers.
12 escued the decreased mRNA levels of terminal differentiation markers.
13  coexpression of PLS3 with a panel of T-cell differentiation markers.
14 apoptosis, and the expression of Paneth cell differentiation markers.
15 ibroblasts was able to restore expression of differentiation markers.
16 , thus revealing altered expression of these differentiation markers.
17 aspase-3 activity and expression of terminal differentiation markers.
18 ce and upregulated the expression of myeloid differentiation markers.
19 ancers that can present with tissue-specific differentiation markers.
20  Ca(2+)(o)-stimulated expression of terminal differentiation markers.
21 ficantly restored expression of Runx2 and OB differentiation markers.
22 ohistochemistry with specific antibodies for differentiation markers.
23 imbal stem cell (LSC) and corneal epithelial differentiation markers.
24 ith changes in hyperplasia, infiltrates, and differentiation markers.
25  were different and distinct from changes in differentiation markers.
26 scade controlling the expression of terminal differentiation markers.
27 lthough it could not completely suppress all differentiation markers.
28 iber region, lens fibers express appropriate differentiation markers.
29 s immature neural crest cell markers but not differentiation markers.
30  IP3, and Cai; and induction of keratinocyte differentiation markers.
31 n persist in mouse brain and retain neuronal differentiation markers.
32 d a reciprocal gain in some lineage-specific differentiation markers.
33 tion, and in concomitant induction of neural differentiation markers.
34 eratrol, enhanced expression of keratinocyte differentiation markers.
35 on, and promoting the robust induction of KC differentiation markers.
36 -1, they did not co-express DCAMKL-1 or cell differentiation markers.
37 ation of BAF155 leads to the upregulation of differentiation markers.
38 morphology, mucin features and expression of differentiation markers.
39 d delaying expression of neural identity and differentiation markers.
40  a variety of prostate-specific and terminal differentiation markers.
41 , correlating closely with expression of VSM differentiation markers.
42 rker expression but suppressed certain glial differentiation markers.
43 dermal maturation with reduced expression of differentiation markers.
44  killer cell Ig-like receptors or other late-differentiation markers.
45  and proliferation genes, and an increase in differentiation markers.
46  by premature ectopic expression of neuronal differentiation markers.
47 gulates chromatin and expression of neuronal differentiation markers.
48  cellular markers and through alterations in differentiation markers.
49 e induced to form tubules expressing nephron differentiation markers.
50 ision (cytokinesis), associated with loss of differentiation markers.
51 etraenoic acid attenuated expression of late differentiation markers.
52 olecular profile lacking most progenitor and differentiation markers.
53 orally affects calcium-induced expression of differentiation markers.
54 C small interfering RNA inhibited osteoblast differentiation marker alkaline phosphatase activity, wh
55 o increased the levels of smooth muscle cell differentiation markers alpha-smooth muscle actin and ca
56 onstrate that butyrate induction of the cell differentiation marker ALPi is mediated through KLF5 and
57 of E-cadherin and the dental epithelial cell differentiation marker amelogenin.
58 d highlights a concomitant increase of beige differentiation marker and a decrease in extracellular m
59      In addition, mRNA levels of the neuroD1 differentiation marker and BDNF, a neurotrophin required
60                                    Cytokine, differentiation marker and transcription factor mRNA exp
61 or cell markers with increased expression of differentiation markers and cell cycle exit.
62 ively correlate with expression of epidermal differentiation markers and components of the Notch1 pat
63 icient keratinocytes do not express terminal differentiation markers and continue to proliferate even
64 ion, as indicated by decreased expression of differentiation markers and decreased translocation of E
65 docytes increased the expression of podocyte differentiation markers and enhanced cell motility; howe
66 fferentiation by promoting the expression of differentiation markers and enhanced colonic barrier fun
67     Here we show that podocytes rapidly lose differentiation markers and enter the cell cycle in adul
68 the expression of mRNAs encoding chondrocyte differentiation markers and growth factors.
69                     This subpopulation lacks differentiation markers and HLA class I (HLAI) antigens,
70 ding rapid cell-cycle exit, re-expression of differentiation markers and improved filtration barrier
71     This was associated with a rapid loss of differentiation markers and increased expression of CSC
72                   The expression of terminal differentiation markers and key enzymes mediating epider
73  Pak2 display delayed expression of myogenic differentiation markers and myotube formation.
74 tment with glucocorticoids restores podocyte differentiation markers and normal ultrastructure and im
75 delayed, along with reduced expression of HF differentiation markers and of transcriptional regulator
76 , associated with down-regulation of thyroid differentiation markers and ongoing apoptosis.
77 al transition (MET), increased expression of differentiation markers and presence of partially reprog
78 ritical role in regulating expression of SMC differentiation markers and proliferation of SMCs in viv
79 ted in the reexpression of breast epithelial differentiation markers and repression of EMT transcript
80                   All EFNAs induce epidermal differentiation markers and suppress cell adhesion genes
81        Expression levels of the chondrogenic differentiation markers and transcriptional regulators S
82 phology, hyperplasia, aberrant expression of differentiation markers and transcriptional regulators,
83                                   Osteoblast differentiation markers and Wnt target gene expression w
84 n proliferation and migration, expression of differentiation markers, and activation of neurotrophin
85 tro differentiation protocols, expression of differentiation markers, and assessment of the ability o
86 ll densities, cell proliferation, osteoblast differentiation markers, and capillaries in human iliac
87 inflammation and epidermal proliferation and differentiation markers, and it has been unclear whether
88 s RA-induced RARalpha binding, activation of differentiation markers, and the repression of pluripote
89 vealed by the premature expression of muscle differentiation markers, and, especially, by a reduced e
90 ed relative to the wild type, and osteoclast differentiation markers are expressed at earlier time po
91 is dependent on culture conditions, and many differentiation markers are usually absent.
92 th a reduction in the expression of podocyte differentiation markers as compared with the wild-type t
93 sociated with podocyte apoptosis and loss of differentiation markers as well as a faster decline in a
94 nment, and expression of smooth muscle (SMC) differentiation markers, as those have been associated w
95  was able to increase the gene expression of differentiation markers, as well as the activity of MMP-
96 induced differentiation as indicated by cell differentiation markers associated with early (CD38 and
97 etween microRNA expression and messenger RNA differentiation markers BMP-4, CK8 and CK14 were analyze
98               P2ry12-/- OCs exhibited intact differentiation markers, but diminished resorptive funct
99 pithelial cells show increased expression of differentiation markers, but loss of progenitor cell mar
100 hermore, ERK inhibition and the induction of differentiation markers by DSG1 required both Erbin and
101 e thus provide evidence that certain sets of differentiation markers can be enhanced, whereas others
102 of mutant mice, there was a reduction of the differentiation marker, carbonic anhydrase-1, and failur
103 n) and inhibited the expression of adipocyte differentiation markers (CCAAT/enhancer-binding protein
104 both CD-45/KRT markers (and for the monocyte differentiation marker CD-14).
105 e been shown to overexpress the vasculogenic differentiation markers CD144 (VE-cadherin) and TIE1 and
106 1 and HOXC11 ChIPseq analysis identified the differentiation marker, CD24, and the apoptotic protein,
107 molecules CCR7 and L-selectin as well as the differentiation marker CD27, a phenotype consistent with
108 28 costimulation exhibit lower expression of differentiation markers CD27 and CD122 (IL-15Rbeta).
109  proportions of CD56dim cells expressing the differentiation marker CD57 and expansion of the NKG2C+
110 8 cells less commonly expressed the terminal differentiation marker CD57, a finding consistent with a
111 ed by decreased expression of megakaryocytic differentiation marker CD61 and cell cycle behavior.
112 pressed intestinal transcription factors and differentiation markers (Cdx2, GATA4, HNF1alpha, villin
113 led to stratify and did not express terminal differentiation markers characteristic of basal, interme
114 ulate proinflammatory genes or down-regulate differentiation markers characteristic of RAS-expressing
115 DAC4 reverses miR-1 induction of chondrocyte differentiation markers Col X and Ihh.
116 inhibited the premature expression of muscle differentiation markers, corrected the cytoskeletal abno
117 roliferation, and expression of stem cell or differentiation markers demonstrates that rx1 maintains
118                                     Although differentiation markers Dj and Fzo are expressed in late
119          The expression of Klf5, odontoblast-differentiation markers, Dspp and Dmp1 was co-localized
120         In contrast, a homolog of the neural differentiation marker elav, CapI-elav1, is restricted t
121 ced expression of the distal lung epithelial differentiation markers Erm, Napsa and Sftpc, and type I
122 mation in NC is marked by loss of follicular differentiation markers, expansion of keratin-15-positiv
123  cells, nor does it affect the expression of differentiation markers expressed in lens fibers, althou
124  AT2R knockdown by siRNA suppressed myoblast differentiation marker expression and myoblast different
125 ratinocytes, overexpressed YY1 also inhibits differentiation marker expression induced by calcium, su
126                                The defect in differentiation marker expression was independent of apo
127 icantly inhibited AQP3 re-expression-induced differentiation marker expression with calcium elevation
128 d Notch1 processing, decreased cell size and differentiation marker expression, and increased prolife
129     Hypoxia-induced changes in stem cell and differentiation marker expression, clone-forming potenti
130 ne deacetylase inhibitor, partially restored differentiation marker expression, suggesting a potentia
131 roughput screen of neutrophil CD (cluster of differentiation) marker expression and a thorough litera
132 T-PEMs have a lower expression of macrophage differentiation markers F4/80, CD68, CD115, and CD11b, w
133  supported by a lower mRNA expression of the differentiation markers; fatty acid binding protein 4, p
134 latum also induced expression of key barrier differentiation markers (filaggrin and loricrin), increa
135 e identification of cell surface proteins as differentiation markers, flow cytometry requires suitabl
136 on of Jak3 resulted in reduced expression of differentiation markers for the cells of both enterocyti
137 3, FoxM1b also represses the mammary luminal differentiation marker FoxA1 by promoter-methylation, an
138 e which encoded the expression of functional differentiation markers from the ATRA-inducible transcri
139 d that TGF-beta-mediated upregulation of SMC differentiation marker gene expression in 10T1/2 cells w
140 esults suggest, for the first time, that SMC differentiation marker gene expression is regulated by H
141  MAPK/ERK and Akt signaling, suppresses VSMC differentiation marker gene expression.
142 n the regulation of smooth muscle cell (SMC) differentiation marker gene expression.
143 on at the CArG-containing regions of the SMC differentiation marker gene promoters.
144 There was increased expression of osteoblast differentiation marker genes and reduced expression of g
145 ing whether oxPLs regulate expression of SMC differentiation marker genes and the molecular mechanism
146     Pitx2 induces expression of multiple SMC differentiation marker genes by binding to a TAATC(C/T)
147 r regions of the p21(WAF1/Cip1) gene and SMC differentiation marker genes following vascular injury.
148 , another homeodomain protein, regulates SMC differentiation marker genes in fully differentiated SMC
149 ppression of Pitx2 reduces expression of SMC differentiation marker genes in the early stages of SMC
150                                         VSMC differentiation marker genes such as SM alpha-actin, cal
151 tion was blocked and down-regulation of VSMC differentiation marker genes was enhanced.
152                                     The VSMC differentiation marker genes, including alphaSMA, SM22,
153     In DGCR8 cKO embryos and knockout VSMCs, differentiation marker genes, including alphaSMA, SM22,
154 detected by reduced expression of osteoblast differentiation marker genes.
155 ching of VSMCs, including suppression of SMC differentiation marker genes.
156 etylation levels within the promoters of SMC differentiation marker genes.
157 cyte marker gene PPP1R14a and other neuronal differentiation marker genes.
158                       However, expression of differentiation markers human chorionic gonadotrophin an
159 xpression for p27(kip1), Atoh1 and hair cell differentiation markers implicating notch signaling in t
160 ntly permits detection of only up to a dozen differentiation markers in a single measurement.
161  nonmalignant pulmonary tissues (n = 285) as differentiation markers in an analysis of DNA methylatio
162 se expression correlates highly with that of differentiation markers in both the bladder and skin, in
163 expression of KLF15 stimulated expression of differentiation markers in both wild-type and HIV-1-infe
164 elayed down-regulation of smooth muscle cell differentiation markers in carotid arteries following in
165  Kruppel-like factor 4 (Klf4) suppressed SMC differentiation markers in cultured SMCs.
166 alyses confirmed the decreased expression of differentiation markers in D2J osteoblasts.
167 n factor, is required for restoring podocyte differentiation markers in mice and human podocytes unde
168 pression of MAF promoted expression of glial differentiation markers in MPNST cells in vitro, decreas
169 D117, and CD34) and myeloid (CD115 and CD14) differentiation markers in parallel with increased phago
170 ssion, DNA synthesis, expression of neuronal differentiation markers in PC12 cells, and Ras-induced f
171 elates with impaired induction of osteoclast differentiation markers in response to RANKL stimulation
172 ecule RNA FISH to measure mRNA expression of differentiation markers in single cells reveals that sis
173 se embryos exhibit impaired induction of SMC differentiation markers in the dorsal aorta and branchia
174 uces the expression of keratinocyte terminal differentiation markers in the duct luminal cells, which
175                            The expression of differentiation markers in these organotypic cultures we
176 temness markers and activation of early cell differentiation markers in treated embryonic stem cells.
177 rphology or expression of smooth muscle cell differentiation markers in vessels of SM22alpha-CreKI(+)
178 els correlate with the expression of various differentiation markers in vitro in response to differen
179  on lipid accumulation and the expression of differentiation markers, in vitro adipogenesis increased
180 es exhibit increased expression of foam cell differentiation markers including 15-lipoxygenase and le
181  reverses miR-365 stimulation of chondrocyte differentiation markers including Ihh, Col X, and Runx2.
182 y determines the expression of smooth muscle differentiation markers including SMA.
183 erentiation as indicated by reduced terminal differentiation markers, including alkaline phosphatase,
184 gnificantly higher mRNA levels of osteoblast differentiation markers, including COL1A1, ALP, and OC,
185 senger ribonucleic acid levels of osteogenic differentiation markers, including collagen 1alpha1, alk
186 ed proliferation and increased expression of differentiation markers, including ERVW-1.
187 iated with a profound repression of terminal differentiation markers, including filaggrin, an essenti
188  architecture, and downregulation of several differentiation markers, including filaggrin.
189          Transcripts for parietal endodermal differentiation markers, including laminin, J6(Hsp 47),
190          Using a panel of different terminal differentiation markers, including neurotransmitter synt
191            This study demonstrated important differentiation markers, including pigmentation and West
192                                    Epidermal differentiation markers, including small proline-rich pr
193                     Multiple tissue-specific differentiation markers, including the tightly regulated
194 nflammatory phenotype including a decline in differentiation markers, increased cytokine production,
195  ZBTB16 induced the expression of osteogenic differentiation markers independently of RUNX2.
196 nduced expression and enzyme activity of the differentiation marker intestinal alkaline phosphatase (
197 contact exhibited enhanced expression of the differentiation markers involucrin and keratin 10 compar
198 dify the expression profile of the epidermal differentiation markers involucrin, keratin 10, and fila
199 phic, and has altered expression of terminal differentiation markers involucrin, loricrin, and filagg
200                       Expression of neuronal differentiation markers is ablated in both KIF1Bbeta-def
201 and are rescued by DKK1, whereas the loss of differentiation markers is CaMKII dependent.
202                     The expression of T cell differentiation markers is known to increase during Myco
203 sphoprotein (DSPP), an important odontoblast differentiation marker, is necessary for tooth developme
204                                              Differentiation markers (K1, K10, filaggrin, loricrin, c
205 evel of N-cadherin, but a lower level of the differentiation marker K12.
206 USP6 and decreased expression of the corneal differentiation marker K12.
207 of the basal layer marker keratin 14 and the differentiation marker keratin 1 was evident in Aurora-A
208 al cell markers keratin 5 and 14 but not the differentiation marker keratin 4.
209    We examined the requirement of the T cell differentiation marker killer cell lectin-like receptor
210  cardiac progenitor cell markers and reduced differentiation marker levels.
211 hron formation, induces tubule formation and differentiation markers Lim1 and E-cadherin in MM cells,
212 tubule-derived cystic segments that lost the differentiation marker lotus tetragonolobus lectin.
213 NPs decreases the expression of the neuronal differentiation markers MAP2 and NeuN and downregulates
214    Expression and localization of the acinar differentiation marker MIST1 were altered in Irf6-null s
215 h1 ligand, Jagged1, with increased levels of differentiation marker mRNAs and decreased colony-formin
216 A/Io increased expression of the goblet cell differentiation marker MUC2; these changes were attenuat
217  increased the expression of the goblet cell differentiation marker mucin 2 (MUC2).
218 ly members MAL, MAL2, and myeloid-associated differentiation marker (MYADM) regulate the function and
219 and decreased expression of SC proliferation/differentiation markers (MyoD, myogenin, and active-Notc
220 ing myofibers and expression of the myoblast differentiation markers myogenin and embryonic myosin he
221 nterference reduced the expression levels of differentiation markers (myogenin, myosin heavy chain, t
222  in maintaining the expression level of a CE differentiation marker, N-cadherin, and the hexagonal ce
223 Hes5, with a reduction of the spermatogonial differentiation marker, Neurog3 expression in the mutant
224 olecular markers including the early cardiac differentiation marker Nkx2.5.
225       Dentin sialophosphoprotein (Dspp) as a differentiation marker of odontoblasts is regulated by B
226 luding the expression of prestin, a terminal differentiation marker of outer hair cells, although man
227 d for a variety of transcription factors and differentiation markers of esophageal squamous epitheliu
228 ent does not affect expression of macrophage differentiation markers or macrophage biological functio
229   Progressive loss of the myoepithelial cell differentiation markers p63, calponin, and alpha-smooth
230 expressed squamous transcription factors and differentiation markers (p63, Sox2, CK14 and CK4) in all
231  found that expression of the late adipocyte differentiation marker peroxisome proliferator-activated
232 sociated with HCs, including the terminal HC differentiation marker prestin.
233 glandular infolding through 24 weeks of age, differentiation markers probasin, PSP-94, and Nkx3.1 wer
234                 The expression of well-known differentiation markers profilaggrin, loricrin, and kera
235     Our study included analysis of germ cell differentiation markers, proliferation markers, and cell
236  populations marked by varying levels of the differentiation marker prostate-specific antigen (PSA).
237 egregated with the template DNA, whereas the differentiation markers prosurfactant protein-C and pan-
238 tress fibers and expression of myofibroblast differentiation marker proteins.
239   It was shown that one of the most powerful differentiation markers proved to be Mn content.
240 h (PSA(+)) and low (PSA(-/lo)) levels of the differentiation marker PSA.
241 ted at 7 T in six C57BL/6 mice and in immune differentiation marker (recombination activation gene [R
242             However, while the apoptosis and differentiation markers remained unchanged at this age,
243  and immunohistochemistry of sebaceous gland differentiation markers revealed reduced peroxisome prol
244  stimulated the production of the osteoblast differentiation markers runt-related transcription facto
245 ant reductions in the expression of the lung differentiation markers Sftpa, Sftpb, Sftpc, and Abca3.
246 he Gata4-expressing cells eventually express differentiation markers showing commitment to liver deve
247  3 (Slfn3) correlated with the levels of the differentiation markers SI, Dpp4, Glut2, and villin.
248 M population did not upregulate the terminal differentiation marker sialic acid-binding immunoglobuli
249 this specifically prevents expression of the differentiation marker smooth muscle alpha-actin.
250 , and their switched expression up-regulates differentiation marker Sox6.
251 essed genes in KCOT were squamous epithelial differentiation markers SPRR1A, KRTDAP, and KRT4, as wel
252                            However, terminal differentiation markers such as beta- or gamma-crystalli
253 s stem cell markers such as Prom1 as well as differentiation markers such as Ck19 and Hnf4a.
254 DMEM, induced dark pigmentation and promoted differentiation markers such as CRALBP and MerTK.
255  (N3) transcription, inducing HES5 and early differentiation markers such as involucrin (IVL) and cyt
256 ced mRNA and protein expression of epidermal differentiation markers such as keratin 1, keratin 10, a
257 ndent manner that is similar to induction of differentiation markers such as keratin 10 and involucri
258  CD271 expression concomitantly with loss of differentiation markers such as melan-A and tyrosinase,
259          The gene expression for odontogenic differentiation markers such as osteocalcin, bone sialop
260 apoptosis was observed but the expression of differentiation markers, such as filaggrin, involucrin,
261 Foxi3 cKO there was an early upregulation of differentiation markers, such as p21, Fgf15 and Sfrp5.
262 ermal architecture with proper expression of differentiation markers, suggesting that although kerati
263 nin correlated with crypt progenitor but not differentiation markers, suggesting that the Wnt/beta-ca
264 s, with an increase in apoptosis and thyroid differentiation markers, suggesting that these cell line
265                During EMT, the expression of differentiation markers switches from cell-cell junction
266  podocytes, ET-1 caused loss of the podocyte differentiation marker synaptopodin and acquisition of t
267 Delta/-) ESCs re-initiates the expression of differentiation markers T and Gata-6.
268 tive Klf9 target genes include proliferation/differentiation markers that also show circadian express
269 on of panels of N-glycoproteins as potential differentiation markers that are currently not accessibl
270 d by the absence of major corneal epithelial differentiation markers, that is, K3 and K12 keratins, i
271 ing the expression of airway epithelial cell differentiation markers, the MUC2, MUC5AC, and MUC5B gen
272  directly modulate expression of Paneth cell differentiation markers through its effects on TCF4/beta
273  spatial distribution of GJIC and osteogenic differentiation markers throughout 3D constructs.
274 d thyrocytes increased expression of thyroid differentiation markers, thyroglobulin, thyroid-stimulat
275                    Analysis of stem cell and differentiation marker transcripts, as well as Oct 3/4 p
276 A induced the expression of lineage-specific differentiation markers Tuj1 and GFAP and reduced the ex
277 messenger RNA expression of PDL osteoblastic differentiation markers: type I collagen, alkaline phosp
278 ng the injury downregulate the expression of differentiation markers, upregulate markers of the pre-o
279 ositive cells had lost expression of the end-differentiation marker, urocortin-3, or appeared to co-e
280 ers, putative stem cell markers and cellular differentiation markers using an immunohistochemical app
281 n to assay the expression of the enterocytic differentiation markers villin, sucrase-isomaltase, gluc
282     The expression of epidermal keratinocyte differentiation markers was affected in the Klf4CN corne
283                      Transcription of dental differentiation markers was as follows: Dentin matrix pr
284                            The expression of differentiation markers was evaluated by quantitative PC
285 ll differentiation by calcium, expression of differentiation markers was impaired in both DeltaNp63al
286                            The expression of differentiation markers was quantified by enzyme-linked
287             Injury-induced repression of SMC differentiation markers was transiently delayed in Klf4-
288            A panel of putative stem cell and differentiation markers was used to analyze the epitheli
289 idase IV (DPP-IV), two well known intestinal differentiation markers, was examined.
290                                  Among those differentiation markers, we show that Sprr2a transcripti
291 stasis, chondrocyte maturation, and terminal differentiation markers were all up-regulated versus iso
292       Gene expression of leptin and selected differentiation markers were analyzed during preadipocyt
293 erentiation and the expression of osteogenic differentiation markers were assessed by real-time rever
294 nd Paneth cell lysozyme, whereas enterocytic differentiation markers were reduced.
295 on concurrent with decreased expression of a differentiation marker when compared with untreated cell
296 cient precursors normally express osteoclast differentiation markers when exposed to M-CSF and recept
297 inamide inhibited expression of keratinocyte differentiation markers, whereas a SIRT1 activator, resv
298 ir expression of mRNA for several osteoclast differentiation markers, whereas ES significantly reduce
299 roxide levels and promoted the expression of differentiation markers, whereas overexpression decrease
300 6 rescued the reduced expression of terminal differentiation markers, whereas transfection of AnxA6-/

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