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1  22) demonstrated a preponderance of UIP and diffuse alveolar damage.
2  airspaces with protein-rich edema fluid and diffuse alveolar damage.
3 ain histopathologic finding in the lungs was diffuse alveolar damage.
4  demonstrated particle-laden macrophages and diffuse alveolar damage.
5 tivities for MMPs and TIMPs were stronger in diffuse alveolar damage.
6 ostmortem biopsy for 3 patients showed acute diffuse alveolar damage.
7 e respiratory tract disease characterized by diffuse alveolar damage and hyaline membrane formation.
8  histologic features of acute and organizing diffuse alveolar damage and prominent interstitial and a
9 ization of viral antigen in association with diffuse alveolar damage are prominent features of infect
10 n, loss of lung volume, increased shunt, and diffuse alveolar damage-are also present in several crit
11 IP) in 18 of 22 patients (81.8%), organizing diffuse alveolar damage (DAD) in 2, bronchiolitis oblite
12 es of acute respiratory insufficiency due to diffuse alveolar damage (DAD) should suggest the diagnos
13                                              Diffuse alveolar damage (DAD) was the second most common
14 f type II pneumocytes has been identified in diffuse alveolar damage (DAD), is associated with p53 an
15  regulatory proteins such as p53 and WAF1 in diffuse alveolar damage (DAD).
16  4 per etiology) were used for comparison of diffuse alveolar damage, E-cadherin, and molecular biolo
17 ) is a devastating syndrome characterized by diffuse alveolar damage, elevated airspace levels of pro
18        In contrast, severe clinical disease, diffuse alveolar damage, hyaline membrane formation, alv
19 inent histopathological feature observed was diffuse alveolar damage in the lung in all case-patients
20 , the disease is limited to the lungs, where diffuse alveolar damage is accompanied by a disproportio
21                                              Diffuse alveolar damage is the histopathological hallmar
22                                        This "diffuse alveolar damage" is actually patchy in many pati
23 ome, variable ventilation 1) decreased total diffuse alveolar damage (median [interquartile range]: v
24 nts with normal pulmonary histology (n = 3), diffuse alveolar damage (n = 14), and idiopathic pulmona
25 pendent lung regions, reduced the cumulative diffuse alveolar damage score across lungs (median [inte
26 owed focal areas of pulmonary hemorrhage and diffuse alveolar damage, splenic infarct, adrenal necros
27 nt was higher in human lung biopsies showing diffuse alveolar damage than in normal human lung tissue
28       This suggests that the pathogenesis of diffuse alveolar damage that ultimately leads to the chr
29 ped fatal lung inflammation characterized by diffuse alveolar damage with marked peribronchial fibros
30 cause tracheitis, bronchitis, bronchiolitis, diffuse alveolar damage with pulmonary edema and hemorrh
31                 Pathologic findings included diffuse alveolar damage with pulmonary edema and hyaline

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