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1 rgroup phase 3 trial in recurrent/refractory diffuse large cell lymphoma.
2 se to treatment similar to that reported for diffuse large cell lymphoma.
3 intermediate- or high-grade lymphoma, 13 had diffuse large cell lymphoma.
4 or is frequently translocated and mutated in diffuse large cell lymphoma.
5 to its ability to transform B lymphocytes in diffuse large cell lymphoma.
6 ion of chronic lymphocytic leukemia (CLL) to diffuse large cell lymphoma.
7 re seen, prominently follicular lymphoma and diffuse large-cell lymphoma.
8 s in 24% (8 out of 34) activated B cell-like diffuse large cell lymphoma (ABC-DLBCL), but not in GC B
9 rmation of MALT lymphoma to activated B-cell diffuse large-cell lymphoma (ABC-DLBCL).
10                                Virtually all diffuse large cell lymphomas and a significant fraction
11 me 1q21 are among the most common lesions in diffuse large-cell lymphoma and have been associated wit
12                The BCL-6 gene is involved in diffuse large-cell lymphoma and overexpressed in other t
13                         Approximately 40% of diffuse large cell lymphoma are associated with chromoso
14                                       B cell diffuse large cell lymphoma (B-DLCL) is a heterogeneous
15  CL was morphologically more high grade (50% diffuse large cell lymphoma) compared with primary CL (3
16                             In patients with diffuse large-cell lymphoma (DLBCL), we observed higher
17 nd large-cell lymphomas (DMxs), and 16 of 84 diffuse large-cell lymphomas (DLCCs); the difference bet
18 y chain disease case (MAL) and KI-1 positive diffuse large cell lymphoma (DLCL) cell line (KIS-1).
19  target of aberrant somatic hypermutation in diffuse large cell lymphoma (DLCL), the most common form
20 pression data set based on 160 patients with diffuse large cell lymphoma (DLCL).
21 o [OR] = 0.57; 0.34-0.94), particularly with diffuse large cell lymphoma (DLCL; OR = 0.29; 0.10-0.82)
22                      In approximately 40% of diffuse large cell lymphomas (DLCL) and approximately 14
23  of all NHL and of two predominant subtypes, diffuse large-cell lymphoma (DLCL) (n = 233) and follicu
24 otypic features of 71 cases of primary CD30+ diffuse large-cell lymphomas (DLCL) and 128 cases of Hod
25  and various other partners are recurrent in diffuse large-cell lymphomas (DLCL).
26 ning for CPP32 was present in 10 of 12 (83%) diffuse large cell lymphomas (DLCLs) and 2 of 3 diffuse
27 hoH/TTF (ARHH) and PAX5, in more than 50% of diffuse large-cell lymphomas (DLCLs), which are tumours
28 s of FL showed morphologic transformation to diffuse large-cell lymphoma (DLL) in the second biopsy.
29 gated the molecular features of EBV-positive diffuse large cell lymphomas in 2 patients with RA.
30  lymphoma in patients with secondary CL were diffuse large cell lymphoma (n = 5, 28%), chronic lympho
31 icular lymphomas (37 of 40 cases) as well as diffuse large-cell lymphomas of B-cell type (35 cases) d
32 eaved lymphoma (P =.0005) and an increase in diffuse large cell lymphoma (P <.0001).
33 cluding 16 relapsed CLL and 9 RT (all proven diffuse large cell lymphoma) patients were enrolled, and
34                             In patients with diffuse large-cell lymphoma, serum interleukin-6 levels
35 gkin's lymphoma and, in a subset of cases of diffuse large cell lymphoma, the mechanism of Bcl-6 over
36 eported cases of the non-Hodgkin's lymphoma, diffuse large cell lymphoma, there are translocations of
37 f 13 follicular small cleaved and five of 13 diffuse large cell lymphomas were KAI1 negative.
38                  118 untreated patients with diffuse large-cell lymphoma who were enrolled in frontli

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