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1 lly active lipids phosphatidic acid (PA) and diglyceride.
2 wn products inositol 1,4,5-trisphosphate and diglyceride.
3 effect on phosphatidic acid, but not that on diglyceride.
4 his lethal process is potently suppressed by diglyceride.
5  as well as for the lateral segregation of a diglyceride.
6 des and in 2-3-fold accumulation of label in diglycerides.
7 al correlate for IL-1-generated ether-linked diglycerides.
8     Here, we report that the 1-O-alkyl ether diglyceride, 1-O-hexadecyl-2-acetyl-sn-glycerol (HAG), l
9        The main hydrolysis products were 1,2-diglycerides, 2-monoglycerides, glycerol and fatty acids
10 lase, and three out of five putative type II diglyceride acyltransferases (DGATs), the enzymes that c
11                           Two ether glucosyl diglyceride analogs were synthesized, and their antiprol
12 ine (ara-C) stimulates the formation of both diglyceride and ceramide in the acute myelogenous leukem
13 g change being observed in the modulation of diglyceride and monoglyceride levels in BAT.
14 of natural ceramides with those of saturated diglycerides and an unsaturated ceramide demonstrates th
15 ers, dimers, oxidized triglyceride monomers, diglycerides and free fatty acids, and induction period
16 tty acids, although small proportions of 1,3-diglycerides and of 1-monoglycerides were also found.
17 centrations of two phosphatidylcholines, two diglycerides and two acyl-carnitines were significantly
18                                          1,2-Diglycerides and vegetable stanols and/or sterols have b
19       Octanol, 1,2-dioctanoyl-sn-glycerol (a diglyceride), and N-hexanoyl-D-erythrosphingosine (a cer
20 vine cardiolipin, chloroplast monogalactosyl-diglyceride, and dioleoyl phosphatidylethanolamine as a
21 fatty acids and their esters, monoglyceride, diglyceride, and triglyceride.
22  such as phosphatidylcholine, sphingomyelin, diglycerides, and triglycerides were detected and identi
23 cates cytokine receptor-induced ether-linked diglycerides as second messengers that inhibit the bioac
24 pases are differentially activated such that diglyceride breakdown is approximately four times faster
25 gment ions corresponding to loss of the full diglyceride chain as well as the remaining headgroup bou
26      Interestingly, hepatic triglyceride and diglyceride contents were normalized to chow-fed levels
27                                              Diglyceride DG(18:1/20:0)-sodium adduct and protonated o
28 ding to TG synthesis and steatosis by way of diglyceride (DG) generation.
29  (PEt) instead of phosphatidic acid (PA) and diglyceride (DG).
30                    In addition, digalactosyl diglyceride (DGDG) 36:4 was shown to be an effective mar
31 t of sea urchin membrane vesicle PtdIns into diglycerides enriched in long chain, polyunsaturated spe
32 n in 1-monoglycerides and an increase in 1,2-diglycerides, especially in the margarines with higher w
33 aracterized by high levels of triglycerides, diglycerides, fatty acids, ceramides, and oxidized fatty
34 atty acids, together with triacylglycerides, diglycerides, free fatty acids and ergosterol in salmon
35 rolysis, mobilization of intracellular Ca2+, diglyceride generation, and redistribution of protein ki
36                                          CDP-diglyceride hydrolase (Cdh) catalyzes the same reaction
37                                          The diglyceride-hydrolyzing lipase was purified from the ext
38 te that production of membrane-destabilizing diglycerides in membranes enriched in PtdIns may facilit
39 and the rate of palmitate incorporation into diglycerides in white muscle was 93% higher in obese rat
40 ut affecting the induced increase in nuclear diglyceride, indicating that the increase in nuclear PLD
41 nterleukin-1 receptor-generated ether-linked diglycerides inhibit immunoprecipitated protein kinase C
42 ramide because, in contrast to the bacterial diglyceride kinase, ceramide is not phosphorylated under
43                  Cell-permeable ether-linked diglycerides mimic the effects of interleukin-1 to induc
44 ol, treating human fibroblasts with octanol, diglyceride, or ceramide stimulated the rapid inactivati
45 phosphorylated under conditions specific for diglyceride phosphorylation.
46 dothelin, selectively generates ether-linked diglyceride species (alkyl, acyl- and alkenyl, acylglyce
47 itory profile of these ether-linked glucosyl diglycerides strengthens the hypothesis that such glycol
48 ctive peptides stimulate distinct species of diglycerides that differentially regulate protein kinase
49                                 The block in diglyceride to triglyceride conversion could not be expl
50 ed defects in FA uptake and in conversion of diglycerides to triglycerides that are similar to those
51  phosphatidic acids but reduced abundance of diglycerides, triglycerides, and phosphatidylglycerol li
52 ated fatty acids beta-sitosterol, sn-1 and 3 diglyceride values.
53 r ones, such as 1- and 2-monoglycerides, 1,2-diglycerides, vegetable stanols and sterols, and sorbic
54 ol (DPG) vesicles (but not with digalactosyl diglyceride vesicles) as demonstrated by flotation in su
55  PLD2 and PAP2b act sequentially to generate diglyceride within this specialized membrane compartment

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