ライフサイエンスコーパス: dilatation

1 velopment of myogenic tone (28% from maximum dilatation).

2 end-diastolic pressure, and left ventricular dilatation.

3 ignificant elevation of VEGF and ventricular dilatation.

4 scle cells in a process termed flow-mediated dilatation.

5 dilatation and 20% having right ventricular dilatation.

6 ely associated with the degree of esophageal dilatation.

7 dal changes different from a mere sinusoidal dilatation.

8 ish obstruction from other causes of colonic dilatation.

9 calcification and in 25 for limited annular dilatation.

10 ancreatography in the diagnostics of biliary dilatation.

11 ; of these 2 also had right ventricular (RV) dilatation.

12 % of patients and were treated by endoscopic dilatation.

13 sis in VOH to collagen degradation favouring dilatation.

14 9%), without elevated ePASP, had isolated RV dilatation.

15 elial function was assessed by flow-mediated dilatation.

16 scular remodeling and right ventricular (RV) dilatation.

17 tic macular edema without the need for pupil dilatation.

18 AJs, (iii) AJ width, and (iv) acetylcholine dilatation.

19 eatures, including prevalence of aortic root dilatation.

20 l functions, including endothelium-dependent dilatation.

21 aracatinib no longer increased acetylcholine dilatation.

22 ression was used to examine risk factors for dilatation.

23 ade resulted in an increase in flow-mediated dilatation.

24 a media thickness or endothelium-independent dilatation.

25 markedly impaired NO-dependent flow-mediated dilatation.

26 vant pressure gradient compared with balloon dilatation.

27 o the development of intrapulmonary vascular dilatations.

28 by hypoxemia due to intrapulmonary vascular dilatations.

29 Both FNIII1H,8-10 and FNIII1H induced dilatations (12.2 +/- 1.7 mum, n = 12 and 17.2 +/- 2.4 m

30 mural nodularity (16.5%), and biliary ductal dilatation (17.7%).

31 h restrictive evolution with marked biatrial dilatation, (2) early-onset atrial fibrillation and atri

32 binding region significantly diminished the dilatation (3.2 +/- 1.8 mum, n = 10).

33 basal LVOTO (70-120 mm Hg), and left atrial dilatation (44-57 mm).

34 nts, echocardiography revealed severe atrial dilatation (5 cases), mitral/tricuspid regurgitation (5)

35 increased scar thickness, and attenuated LV dilatation 7 days after myocardial infarction.

36 more likely than females to have aortic root dilatation (92% versus 84%), aortic regurgitation (55% v

37 on initial imaging (eg, main pancreatic duct dilatation, a solid component, or mural nodule) require

38 However, E2 vascular actions such as rapid dilatation, acceleration of endothelial repair, and endo

39 ophy (adjusted odds ratio 2.1; P<0.0001), LV dilatation (adjusted odds ratio 2.2; P<0.0001), and righ

40 ratio 2.2; P<0.0001), and right ventricular dilatation (adjusted odds ratio 2.2; P<0.0001).

41 t size, is associated with 3- and 6-month LV dilatation after ST-segment-elevation myocardial infarct

42 ombined modulation of both systems may cause dilatation along the entire bronchial tree.

43 with 26% of patients having left ventricular dilatation and 20% having right ventricular dilatation.

44 n a beating heart swine model and results in dilatation and 3D changes in mitral annular geometry.

45 Atrial dilatation and atrial standstill are etiologically heter

46 stigated using brachial artery flow-mediated dilatation and carotid artery intima-medial hyperplasia.

47 s to spontaneous-onset AF preceded by atrial dilatation and conduction abnormalities.

48 it apoptosis, and attenuate left ventricular dilatation and disease progression.

49 Our novel findings, including T-tubule dilatation and disorganization, associated with defects

50 d collagen quality, thereby blunting cardiac dilatation and dysfunction after MI.

51 r patients with less severe left ventricular dilatation and dysfunction during the years.

52 py proven to attenuate left ventricular (LV) dilatation and dysfunction in volume overload induced by

53 mice exhibited significantly greater cardiac dilatation and dysfunction than wild-type mice after tra

54 rction associated with left ventricular (LV) dilatation and dysfunction, which doubles mortality.

55 nical status and the severity of ventricular dilatation and dysfunction.

56 ssion of NHK alpha1AT caused electron lucent dilatation and expansion of the ER throughout the cell.

57 At present, pneumatic dilatation and Heller myotomy combined with an anti-refl

58 trials comparing this option with pneumatic dilatation and Heller myotomy.

59 cal activity is also associated with cardiac dilatation and hypertrophy in a healthy adult population

60 s and prevented progressive left ventricular dilatation and hypertrophy, whereas adoptive transfer of

61 o inhibit sensory neurotransmission) blocked dilatation and increased constriction during PNS.

62 With perivascular sensory nerve stimulation, dilatation and inhibition of sympathetic vasoconstrictio

63 en secondary in nature and caused by annular dilatation and leaflet tethering from adverse right vent

64 ew and simple composite parameter of both LV dilatation and LV forward flow able to accurately predic

65 Cpt reduced LV dilatation and LV hypertrophy.

66 Extensive changes of dilatation and obstruction in nearly all airway generati

67 Aortic dilatation and other structural cardiac abnormalities we

68 g evidence for the association of sinusoidal dilatation and oxaliplatin-based chemotherapy but not fo

69 Increased microvascular dilatation and permeability is observed during allograft

70 lateral ureteral obstruction-induced tubular dilatation and proliferation, preserved Klf4, and suppre

71 The vessels had no dilatation and rarely had post-cannulation bleeding.

72 ntify and select relevant studies of balloon dilatation and stenting for aortic coarctation based on

73 ess and comparative effectiveness of balloon dilatation and stenting for aortic coarctation.

74 achial artery reactivity (both flow-mediated dilatation and sublingual glycerol trinitrate, markers o

75 icular (LV) cardiomyopathy, manifested by LV dilatation and systolic dysfunction, as well as overexpr

76 (44.1-88.9%) of patients undergoing balloon dilatation, and in 99.5% (97.5-100.0%) and 93.8% (88.5-9

77 n, left ventricular hypertrophy, left atrial dilatation, and interstitial fibrosis.

78 n release, elevated catecholamine levels, RV dilatation, and late gadolinium enhancement on MRI, incr

79 ed to a significant reduction in LV mass, LV dilatation, and neurohormonal activation, and it preserv

80 ys, animals had moderate volume overload, LV dilatation, and reduced ejection fraction (all P<0.01 ve

81 those with early evidence of coronary artery dilatation, and those with extreme abnormalities in labo

82 llowing exercises and manoeuvres, endoscopic dilatations, and for advanced cases, oesophageal reconst

83 s in heartburn scores, patient satisfaction, dilatations, and reoperation rate.

84 These data demonstrate aortic dilatation, aortic medial degeneration, and alterations

85 In benign causes of biliary dilatation, apart from cholelithiasis, MRCP picture is o

86 Benign causes of biliary dilatation, apart from cholelithiasis, were identified i

87 Reduced aortic bioelasticity and aortic root dilatation are present in transposition of the great art

88 diastolic dysfunction and right ventricular dilatation are the echocardiographic variables correlati

89 thological end point: irreversible bronchial dilatation arrived at through diverse etiologies.

90 Heat therapy improved endothelium-dependent dilatation, arterial stiffness, intima media thickness a

91 chieving </=20 mm Hg were lower with balloon dilatation as compared with stenting (odds ratio, 0.105

92 bles reliable diagnosis of causes of biliary dilatation as long as they involve presence of gallbladd

93 hermore, capillaries contribute to metabolic dilatation as they dilate arterioles directly upstream i

94 ricuspid regurgitation and tricuspid annular dilatation, as well as with appreciation of the high ris

95 agulation with heparin alone in reversing RV dilatation at 24 hours, without an increase in bleeding

96 h baseline LVGLS>-15.0% exhibited greater LV dilatation at 3 and 6 months compared with patients with

97 farction is independently associated with LV dilatation at follow-up.

98 function (via brachial artery flow-mediated dilatation) at sea level (344 m) and high altitude (3800

99 function (via brachial artery flow-mediated dilatation) at sea level (344 m) and high altitude (3800

100 IP3-R(2)-/- were similar in terms of chamber dilatation, atrial enlargement, and ventricular wall thi

101 icular remodeling, greater right ventricular dilatation (base, 34+/-7 versus 31+/-6 and 30+/-6 mm, P=

102 e FBA, there was no longer any difference in dilatation between old and young arteries.

103 ficant difference in the rate of aortic-root dilatation between the two treatment groups over a 3-yea

104 t regulator of acute vessel permeability and dilatation but also provide evidence that antagonizing P

105 In contrast, annulus dilatation but not flattening occurred in nonprolapse MR

106 uced right ventricular pressure increase and dilatation, but left ventricular end-diastolic volume im

107 thelial growth factor pathways in sinusoidal dilatation, but the mechanisms linking the activation of

108 xamination including fundus evaluation after dilatation by a cataract surgeon and an ophthalmologist,

109 In addition, pore dilatation by a mutation in the pore-lining region alter

110 lized to assess the influence of ET-1 on the dilatation capacity of vascular smooth muscle cells (sod

111 russide, suggesting that ET-1 diminishes the dilatation capacity of vascular smooth muscle cells.

112 tive axon density) and endothelium-dependent dilatation (carbachol) of the MCA were not different bet

113 attributed to a larger arterial than venous dilatation causing increased fluid filtration.

114 gitudinal systolic function, and left atrial dilatation compared with asymptomatic patients.

115 farction-induced remodeling includes chamber dilatation, contractile dysfunction, and fibrosis.

116 (-1) m(-2), P < 0.02), and improved arterial dilatation Deltabrachial artery flow-mediated dilatation

117 ilatation Deltabrachial artery flow-mediated dilatation/Deltadilation response to glyceryl nitrate (9

118 Anal dilatation did not improve outcome(s).

119 ess, which increases secondary to arteriolar dilatation downstream.

120 heart and induced long-term left ventricular dilatation, dysfunction, and fibrosis in naive recipient

121 e in ARVC/D, independently of history and RV dilatation/dysfunction.

122 n 59%, smooth stenosis in 19%, and segmental dilatation/ectasia in 56%.

123 smooth stenosis, diffuse or focal; segmental dilatation/ectasia; and tortuosity.

124 Endothelium-dependent dilatation (ED) is abnormal in patients with SLE, and en

125 Endothelium-dependent dilatation (EDD), assessed by the maximal dilatation to

126 determined by the ratio of ED to independent dilatation (EI).

127 ony >200 ms, lack of severe left ventricular dilatation (end-systolic dimension index <29 mm/m(2)), a

128 Numerically more patients undergoing balloon dilatation experienced severe complications during admis

129 velop an age-dependent phenotype with atrial dilatation, fibrosis, and atrial fibrillation.

130 rs develop maladaptive RVH, characterized by dilatation, fibrosis, and RV failure.

131 function as assessed by using flow-mediated dilatation (FMD) and arterial compliance as assessed by

132 tion (n = 82) was assessed by flow-meditated dilatation (FMD) at baseline and 6 mo.

133 ED was evaluated by means of flow-mediated dilatation (FMD) of the brachial artery.

134 on assessed by brachial artery flow-mediated dilatation (FMD) was measured before, immediately follow

135 ned with the use of ultrasound flow-mediated dilatation (FMD).

136 to post-exercise reductions in flow-mediated dilatation (FMD).

137 dothelial function measured by flow mediated dilatation (FMD).

138 ntral pulse pressure (cPP) and flow-mediated dilatation (FMD).

139 tion in the brachial artery by flow-mediated dilatation (FMD).

140 acid exposure, esophagitis, heartburn score, dilatation for dysphagia, modified Dakkak dysphagia scor

141 Sinusoidal dilatation found in the absence of an impaired sinusoida

142 eeks of heat therapy increased flow-mediated dilatation from 5.6 +/- 0.3 to 10.9 +/- 1.0% (P < 0.01)

143 Mechanisms mediating the spread of dilatation from local to remote sites have been well stu

144 Patients with RV dilatation had significant (P</= 0.05) displacement of a

145 a new 4-group LVH classification based on LV dilatation (high LV end-diastolic volume [EDV] index) an

146 in both groups developed evidence of oviduct dilatation; however, reduced oviduct dilatation was obse

147 HDL-induced NO production and flow-mediated dilatation improvement by ET was evident.

148 stress-induced cardiac fibrosis and chamber dilatation, improving systolic and diastolic functions.

149 holecystic stranding was seen in 19, biliary dilatation in 12, liver infiltration in 13 and fat in 7

150 the prevalence and predictors of aortic root dilatation in adults with repaired tetralogy of Fallot.

151 prognostic significance of small bowel (SB) dilatation in children with short bowel syndrome (SBS).

152 mmarize the recent data pertaining to aortic dilatation in congenital heart disease (CHD) and to disc

153 copy to monitor intracellular pH and luminal dilatation in enteroids under basal and regulated condit

154 anticoagulation alone in the reversal of RV dilatation in intermediate-risk patients.

155 iency specifically induces right ventricular dilatation in mouse embryos at embryonic day 16.5.

156 Src inhibition did not increase dilatation in old arteries treated with the VE-cadherin

157 ries but did not affect the already impaired dilatation in old arteries.

158 adherens junctions and increased endothelial dilatation in old, but not young, arteries.

159 icant change in diameter of P7 arteries, and dilatation in P21 arteries.

160 has been shown that losartan reduces aortic dilatation in patients with Marfan syndrome.

161 Left atrial dilatation in the population is more common in black and

162 n fibronectin (FN) contributes to functional dilatation in these arterioles.

163 -blocking antibody; FBA) reduced endothelial dilatation in young arteries but did not affect the alre

164 Saracatinib did not affect dilatation in young arteries.

165 perivascular nerve stimulation (PNS) evoked dilatation in Young but not Old MAs while dilatations to

166 rotid arteries and aortae, and flow-mediated dilatations in third-order mesenteric resistance arterie

167 factors predispose this population to aortic dilatation, including underlying genetics, cellular-base

168 Relative amplitudes of dilatation increased with contraction duration and with

169 strain-rate e'-wave ratio, right ventricular dilatation (increased right ventricular end-systolic vol

170 Pore dilatation increases the bicarbonate permeability (P HC

171 Here, we provide evidence that pore dilatation increases the bicarbonate permeability (P HC

172 EC-denuded arterioles failed to produce any dilatation indicating that endothelium was required for

173 ion in his general condition, showed jejunal dilatation, intestinal intramural gas, portomesenteric v

174 Aortic dilatation is a common finding in CHD.

175 Although aortic dilatation is common in CHD, data about adverse long-ter

176 l inefficiency, regional dysfunction, and RV dilatation is common in rTOF children, possibly contribu

177 Sinusoidal dilatation is mainly located in the centrilobular area e

178 final identification of the cause of biliary dilatation is possible when this imagining method is com

179 thrombolysis improves right ventricular (RV) dilatation, is associated with major bleeding, and is wi

180 athletes should know that marked aortic root dilatation likely represents a pathological process and

181 wall, cholelithiasis, infiltration, biliary dilatation, lymph nodes, complications.

182 Sinusoidal dilatation may actually be a nonspecific feature of impa

183 LA dilatation may be mediated by blood pressure control and

184 The dynamic increase in P HC O3/ Cl by pore dilatation may have many physiological and pathophysiolo

185 arker of the LV ejection according to the LV dilatation may predict postoperative LVD and outcome aft

186 Other outcomes (brachial flow-mediated dilatation, microvascular reactive hyperemia index, aort

187 Atrial dilatation, mitral valve regurgitation, myocyte hypertro

188 Vascular aneurysm is an abnormal local dilatation of an artery that can lead to vessel rupture

189 of local blood flow regulation suggests the dilatation of arterioles in response to tissue hypoxia v

190 elevated shear stress secondary to metabolic dilatation of arterioles.

191 aortic left renal vein with gross aneurysmal dilatation of both pre- and retro-aortic part of the ren

192 malities including loss, disorganization and dilatation of cristae.

193 AVH and dilatation of dermal microvessels stimulated by vascular

194 core without gadolinium administration = 1 x dilatation of intrahepatic bile ducts + 2 x dysmorphy +

195 with the presence of intraductal stones and dilatation of intrahepatic ducts.

196 component (P = 0.014), main pancreatic duct dilatation of more than 5 mm (P < 0.001), and jaundice (

197 triction of stenosed epicardial segments and dilatation of normal segments, with trends toward revers

198 mmation, hepatocellular swelling, steatosis, dilatation of portal lymphatics, and periductal fibrosis

199 correction of dialysis fistula - especially dilatation of stenosis.

200 ct in the penile urethra and associated mild dilatation of the anterior urethra ending in a smooth bu

201 ion (14.2% versus 6.7%, P<0.001) and diffuse dilatation of the aortic root and ascending aorta (16.2%

202 stal to the sinotubular junction, or diffuse dilatation of the aortic root and ascending aorta.

203 Dilatation of the aortic root only (type 1) or involving

204 f Valsalva or sinotubular junction, isolated dilatation of the ascending aorta distal to the sinotubu

205 lly, aged Npr2(+/-);Ldlr(-/-) mice developed dilatation of the ascending aortic, with greater aneurys

206 Flow-mediated dilatation of the brachial artery increased in the inter

207 phenotypes are expressed in the kidney with dilatation of the collecting ducts, systemic hypertensio

208 This is partially attributable to dilatation of the gastrojejunostomy (GJ), which diminish

209 Interestingly, Bmpr2(+/-) mice demonstrated dilatation of the hepatic central vein at baseline and p

210 Bmpr2(+/-) mice also showed significant dilatation of the liver sinusoids and an increase in inf

211 metastasis by inducing lymphangiogenesis and dilatation of the lymphatic vasculature, facilitating tu

212 Dilatation of the neoaortic root was common (76%), and r

213 haracterized by severe papillary atrophy and dilatation of the pelvicalyceal system without obvious p

214 Men had more frequently isolated dilatation of the sinus of Valsalva or sinotubular junct

215 opathy configuration was defined as isolated dilatation of the sinus of Valsalva or sinotubular junct

216 intestinal permeability and internal hernia, dilatation of the stomach, gastrointestinal anastomotic

217 SWOPP was diagnosed when a saccular dilatation of the terminal portion of the dorsal pancrea

218 In severe cases dilatation of the ureter, renal pelvis, and calyces migh

219 via alphaARs; with advanced age, attenuated dilatation of upstream branches will restrict muscle blo

220 Coronary artery (CA) dilatation on echocardiography is a criterion for treatm

221 The impact of dilatation on parenteral nutrition (PN) dependence and s

222 hallenge the paradigm that right ventricular dilatation on ultrasound during cardiopulmonary resuscit

223 on the likelihood of subjects having cardiac dilatation or hypertrophy according to standard cardiac

224 nd there is a risk of overdiagnosing cardiac dilatation or hypertrophy in a proportion of active, hea

225 intraventricular hemorrhage with ventricular dilatation or intraparenchymal involvement).

226 antly affect the percentage of flow-mediated dilatation or other measures of vascular function.

227 lability is associated with ascending aortic dilatation, outflow tract malrotation, overriding aorta,

228 everity was associated with gradual vascular dilatation (p = 0.000), and widening of the bifurcating

229 (P<0.05), while A2AR agonism limited aortic dilatation (P<0.05).

230 ificant oversizing group underwent less post-dilatation (P=0.002) but achieved greater stent expansio

231 njury (P=0.048), and more pronounced tubular dilatation (P=0.02) after HMPnoox compared to HMPox100%.

232 uded in the differential diagnosis of atrial dilatation, particularly when associated with atrial wal

233 SB dilatation predicts prolonged PN duration and decreased

234 Dilatation pressure and balloon diameter at the highest

235 d not affect the percentage of flow-mediated dilatation (primary endpoint) or other measures of vascu

236 The local arteriolar dilatation produced by contraction of skeletal muscle is

237 vascular function measured as flow-mediated dilatation (R = -0.3, P < 0.01) or endothelial injury ma

238 gher in rTOF (P<0.05) in association with RV dilatation (r=0.33; P=0.04).

239 ence: 1.27; 95% CI:-0.36 to 2.90; P = 0.13), dilatation rate (1.4% vs 2.8%; RR: 0.60; 95% CI: 0.19-1.

240 , losartan significantly reduced aortic root dilatation rate (no losartan, 1.3+/-1.5 mm/3 years, n=59

241 siderable flame-generated enstrophy, and the dilatation rate and baroclinic torque contributions to t

242 BRZ regimes, with diminishing influences of dilatation rate and baroclinic torque.

243 sartan therapy for inhibition of aortic root dilatation rate compared with dominant negative patients

244 However, losartan reduced only aortic root dilatation rate in haploinsufficient patients (no losart

245 s, and topologies existing for all values of dilatation rate remain significant contributors.

246 ly, flow topologies associated with positive dilatation rate values, contribute significantly to the

247 The 5-year heartburn score, dilatation rate, reoperation rate, PPI use, and patient

248 ersion, heat therapy increased flow-mediated dilatation, reduced arterial stiffness, reduced mean art

249 leocecal valve, both estimates of maximal SB dilatation remained significant independent predictors f

250 raventricular hemorrhage without ventricular dilatation, respectively); and 3) severe intraventricula

251 Whereas women exhibited RV cavity dilatation (RV end-diastolic volume, +1.0 mL per BMI poi

252 to increased afterload and left ventricular dilatation secondary to volume overload.

253 he primary outcome measure was flow-mediated dilatation; secondary outcome measures included fasting

254 rta of 75 subjects: BAV patients with aortic dilatation stratified by leaflet fusion pattern (n=15 RL

255 5 stenting (423 participants) and 12 balloon dilatation studies (361 participants), including patient

256 stenting (3397 participants) and 62 balloon dilatation studies (4331 participants).

257 ervation revealed endoplasmic reticulum (ER) dilatation, suggestive of ER stress, and smaller insulin

258 Cardiac dilatation suggests clinically important changes with bo

259 Acetylcholine caused endothelium-dependent dilatation that was decreased in old compared to young a

260 threshold of aortic root or ascending aortic dilatation that would justify surgical intervention in p

261 Then, by including thermal dilatation, the Vogel-Fulcher-Tammann relation is derive

262 bosis (treated 2 weeks after pPCI by balloon dilatation-this patient stopped all medications after pP

263 (function-blocking antibody, FBA) inhibited dilatation to acetylcholine in young, but not old, arter

264 Venular dilatation to acetylcholine was blunted in OZR vs. LZR d

265 nt dilatation (EDD), assessed by the maximal dilatation to acetylcholine, was approximately 40% lower

266 MAs were lost in Old MAs along with impaired dilatation to calcitonin gene-related peptide (CGRP).

267 dothelial denudation reduced the efficacy of dilatation to CGRP by approximately 30% in Old MAs yet i

268 Endothelial denudation attenuated dilatation to CGRP in Old MAs yet enhanced dilatation to

269 d dilatation to CGRP in Old MAs yet enhanced dilatation to CGRP in Young MAs while abolishing all dil

270 critical for many local responses, including dilatation to skeletal muscle contraction) and in relati

271 cking this signalling sequence decreased the dilatation to skeletal muscle contraction, indicating th

272 acy approximately 15% in Young MAs while all dilatations to ACh were abolished.

273 ed dilatation in Young but not Old MAs while dilatations to ACh were not different between age groups

274 on to CGRP in Young MAs while abolishing all dilatations to ACh.

275 tudied using acetylcholine (ACh), but remote dilatations to contraction of skeletal muscle fibres als

276 OP experts graded 84 images showing vascular dilatation, tortuosity, or both and 251 images showing n

277 1) clinical onset in adulthood; (2) biatrial dilatation up to giant size; (3) early supraventricular

278 A-BRS when overexpanded 1.3 mm above nominal dilatation values ( approximately 48%) and lower number

279 By multivariable analysis, neoaortic root dilatation was associated with worse neoaortic valve reg

280 The definitive cause of biliary dilatation was determined mainly on the basis of MRCP an

281 oviduct dilatation; however, reduced oviduct dilatation was observed for "controllers," i.e., animals

282 or hypoxic pregnant mice to dilate and this dilatation was partially reversed by the NOS inhibitor l

283 Aortic dilatation was present in 29% of the patients, and 26% h

284 After junctional disruption with the FBA, dilatation was similar in young and old arteries.

285 The cause of biliary dilatation was usually (20%) a neoplasm in the group of

286 ith cholecystolithiasis the cause of biliary dilatation was usually (45%) cholelithiasis.

287 entricular hypertrophy, and left ventricular dilatation were associated with the development of QRS p

288 ent and posttreatment with rTMD123 on aortic dilatation were measured using the CaCl2-induced AAA mod

289 Increased LV mass and cavity dilatation were observed with low 25(OH)D and high FGF23

290 toxic effects in the kidney and the gastric dilatation were resolved without interfering with the be

291 Potassium-induced dilatations were unaffected by inhibitors of TRPV4, IK a

292 ophy, concentric remodeling, and left atrial dilatation when corrected for indices of AS severity.

293 P was <12 mm Hg in 11 patients (44%) with LA dilatation, whereas PCWP was <25 mm Hg in 1 patient (4%)

294 Neoaortic root dilatation, which is present in most patients and progre

295 blish a reproducible porcine model of aortic dilatation, which recapitulates the structural and bioch

296 All patients underwent poststent dilatation with a noncompliant balloon.

297 lial cytoplasmatic vacuolization and luminal dilatation with flattening of the epithelium.

298 83, we described 8-years follow-up of atrial dilatation with standstill evolution in 8 patients from

299 r A gene and characterized by extreme atrial dilatation with standstill evolution, thromboembolic ris

300 be effective because this entity has annular dilatation without leaflet deformation.