ライフサイエンスコーパス: dilate

1 the auditory tubes can become pathologically dilated.

2 Adjacent intrahepatic bile ducts were dilated.

3 st, the ventricles are collapsed rather than dilated.

4 sociated with the inability of the vessel to dilate and may predispose to ischemia and abnormal FFR.

5 ependent of arterioles, capillaries actively dilate and regulate blood flow.

6 tA from normoxic or hypoxic pregnant mice to dilate and this dilatation was partially reversed by the

7 ons in FLNC and the development of high-risk dilated and arrhythmogenic cardiomyopathies.

8 Dilated and hypertrophic cardiomyopathies are the most c

9 As expected, patients also had dilated and impaired left atria compared with control su

10 s in FLNC caused an overlapping phenotype of dilated and left-dominant arrhythmogenic cardiomyopathie

11 f the demarcation membrane system, which was dilated and poorly developed.

12 who found FLNC variants among patients with dilated and restrictive cardiomyopathies, pointed to thi

13 tients and families, including hypertrophic, dilated, and arrhythmogenic cardiomyopathies and inherit

14 lumens in ducts and secretory portions were dilated, and blisters and papules formed on the skin sur

15 I) or those with mild to moderate TR with a dilated annulus (>/=40 or >/=21 mm(2), Class IIa).

16 pt knockout mice were viable but with mildly dilated aortas that had a 43% reduction in NAD(+) in the

17 Incompetent bicuspid aortic valves with dilated aortic annuli are also satisfactorily treated wi

18 ts presenting with aortic insufficiency or a dilated aortic annulus or ascending aorta were at greate

19 We studied consecutive patients with a dilated aortic root (>/=4 cm) that underwent echocardiog

20 In patients with dilated aortic root and trileaflet aortic valve, a ratio

21 HODS AND Ascending aortas from patients with dilated aortopathy were immunostained for NAMPT, reveali

22 ied in 28 probands previously diagnosed with dilated, arrhythmogenic, or restrictive cardiomyopathies

23 It dilates arterioles by activating large-conductance Ca(2+

24 In SBS, the remaining SB may dilate as part of intestinal adaptation.

25 In the patient population with dilated ascending aorta, both peak TKE and total TKEsys

26 s associated with bicuspid aortic valves and dilated ascending aortic geometries that are not assessa

27 MRI revealed dilated atypical Virchow-Robin space (VRS).

28 A left hepatectomy was done and dilated bile ducts filled with caseous necrotic material

29 disseminated tuberculosis, and point-of-care dilated binocular indirect ophthalmoscopy eye examinatio

30 Small, round clear spheres in vitreous on dilated biomicroscopic retinal examination.

31 ereoscopic fundus photography (method 1) and dilated biomicroscopy in combination with optical cohere

32 hether patterns are passively, homogeneously dilated by growth or whether they remodel during tissue

33 ignaling is both necessary and sufficient to dilate capillaries.

34 T images showed a striking detailed Y-shaped dilated capillary correlating with the clinical finding.

35 ing of the glomerular basement membrane, and dilated capillary loops, with a similar but later onset

36 ension development predicts hypertrophic and dilated cardiomyopathies in mice associated with essenti

37 c insert are linked to heart disease such as dilated cardiomyopathies.

38 re detected in about one-third of idiopathic dilated cardiomyopathies.

39 Brugada syndrome (7), long QT syndromes (5), dilated cardiomyopathy (2), and hypertrophic cardiomyopa

40 arrhythmias (69% and 52%, respectively), and dilated cardiomyopathy (74% and 14%, respectively).

41 otential of silencing miR-34a in settings of dilated cardiomyopathy (DCM) and atrial fibrillation (AF

42 mains debilitating heart conditions, such as dilated cardiomyopathy (DCM) and hypertrophic cardiomyop

43 ompared them with samples from patients with dilated cardiomyopathy (DCM) and inflammatory cardiomyop

44 phospholamban (PLN) gene are associated with dilated cardiomyopathy (DCM) and severe heart failure.

45 rect targets of FXR1 in human left ventricle dilated cardiomyopathy (DCM) biopsy samples and mouse mo

46 ABSTRACT: Dilated cardiomyopathy (DCM) can be caused by mutations

47 c metabolite quantification in patients with dilated cardiomyopathy (DCM) compared with that at 3 T.

48 Improved understanding of dilated cardiomyopathy (DCM) due to titin truncation (TT

49 Non-ischemic dilated cardiomyopathy (DCM) has been recognized as a he

50 Studies of children with dilated cardiomyopathy (DCM) have suggested that improve

51 esidue 9 in PLN to cysteine (R9C) results in dilated cardiomyopathy (DCM) in humans and transgenic mi

52 Dilated cardiomyopathy (DCM) is a common form of cardiom

53 Dilated cardiomyopathy (DCM) is a condition of abnormal

54 Dilated cardiomyopathy (DCM) is a leading cause of heart

55 Dilated cardiomyopathy (DCM) is an important cause of he

56 Dilated cardiomyopathy (DCM) is best understood as the f

57 The cause of idiopathic dilated cardiomyopathy (DCM) is unknown by definition, b

58 Patients with nonischemic dilated cardiomyopathy (DCM) may be at lower risk for ve

59 ix hypertrophic cardiomyopathy (HCM) and two dilated cardiomyopathy (DCM) mutants were studied by bio

60 Nonischemic dilated cardiomyopathy (DCM) often has a genetic pathoge

61 tudies reveal reduced force generation and a dilated cardiomyopathy (DCM) phenotype.

62 Peripartum cardiomyopathy (PPCM) and dilated cardiomyopathy (DCM) show similarities in clinic

63 g heart and in the heart of a mouse model of dilated cardiomyopathy (DCM) triggered by Serum Response

64 rotein, is often associated with arrhythmia, dilated cardiomyopathy (DCM), and heart failure.

65 rtality) suggest that for many patients with dilated cardiomyopathy (DCM), implantable cardioverter-d

66 mice count among the first mouse models for dilated cardiomyopathy (DCM), yet the exact role of MLP

67 tions of CPET with outcomes in children with dilated cardiomyopathy (DCM).

68 reifuss muscular dystrophy (EDMD) as well as dilated cardiomyopathy (DCM).

69 n-truncating variants (TTNtv) commonly cause dilated cardiomyopathy (DCM).

70 ing variants (TTNtvs) are the major cause of dilated cardiomyopathy (DCM); however, allelic heterogen

71 patients with aortic stenosis (AS, n=9) and dilated cardiomyopathy (DCM, n=6).

72 Inflammatory dilated cardiomyopathy (DCMi) is a major cause of heart

73 loci that are significantly associated with dilated cardiomyopathy (false discovery corrected P</=0.

74 aring the survival of children with familial dilated cardiomyopathy (FDCM) to that of children with i

75 tients (n = 154) with documented chronic and dilated cardiomyopathy (ICM, n = 61; NICM, n = 93) requi

76 y (FDCM) to that of children with idiopathic dilated cardiomyopathy (IDCM) has produced conflicting r

77 nction and myocardial injuries in idiopathic dilated cardiomyopathy (IDCM) using cardiac magnetic res

78 was examined in human explanted hearts with dilated cardiomyopathy (n = 25).

79 Dilated cardiomyopathy (n=27), myocarditis or sarcoidosi

80 findings, patients were classified as having dilated cardiomyopathy (n=40) or iCMP (n=75).

81 tic information in patients with nonischemic dilated cardiomyopathy (NIDCM).

82 hy, few studies exist in chronic nonischemic dilated cardiomyopathy (NIDCM).

83 jection Delivery Effects on Neomyogenesis in Dilated Cardiomyopathy [PoseidonDCM]; NCT01392625).

84 ion using RNA-sequencing in 97 patients with dilated cardiomyopathy and 108 non-diseased controls.

85 .5 years; 64 patients (65.9%) had idiopathic dilated cardiomyopathy and 21 patients (21.6%) had ische

86 wall LGE identifies a group of patients with dilated cardiomyopathy and an LVEF >/=40% at increased r

87 aborted SCD among consecutive referrals with dilated cardiomyopathy and an LVEF >/=40% to our center

88 Here, we studied a family with dilated cardiomyopathy and associated conduction system

89 Mutation in DnaJC19 results in dilated cardiomyopathy and ataxia syndrome, whereas expr

90 tem cells were used to examine a family with dilated cardiomyopathy and atrial and ventricular arrhyt

91 we link a subset of 517 epigenetic loci with dilated cardiomyopathy and cardiac gene expression.

92 compared with subjects with noninflammatory dilated cardiomyopathy and controls.

93 myocardial tissue and blood of patients with dilated cardiomyopathy and controls.

94 obal developmental delay, failure to thrive, dilated cardiomyopathy and epilepsy, ultimately leading

95 l mutations within HSPB7 are associated with dilated cardiomyopathy and heart failure in human patien

96 (2)(+) responsiveness in a model of familial dilated cardiomyopathy and improve cardiac function and

97 nd inflammation in patients with nonischemic dilated cardiomyopathy and inflammatory cardiomyopathy (

98 f Titin truncation variants in patients with dilated cardiomyopathy and population controls.

99 se of ivabradine in paediatric patients with dilated cardiomyopathy and symptomatic chronic heart fai

100 relationship of ivabradine in children with dilated cardiomyopathy and symptomatic chronic HF.

101 In patients with nonischemic dilated cardiomyopathy and VT, endocardial and adjuvant

102 No CEs occurred in patients without dilated cardiomyopathy and/or LGE.

103 ed cardiomyopathies allowed establishment of dilated cardiomyopathy as mostly cytoskeleton, force tra

104 t for either sex in a setting of more severe dilated cardiomyopathy associated with atrial fibrillati

105 these circRNAs are dynamically regulated in dilated cardiomyopathy but not in hypertrophic cardiomyo

106 day 6.5-9.5 developed a phenotype similar to dilated cardiomyopathy by 1 year of age.

107 may be a viable approach to the treatment of dilated cardiomyopathy by not only preventing maladaptiv

108 achycardia (VT) in patients with nonischemic dilated cardiomyopathy can be challenging because of the

109 ncation mutation information from 1714 human dilated cardiomyopathy cases and >69 000 controls and fo

110 Patients with myocarditis or dilated cardiomyopathy develop autoantibodies to SERCA2a

111 in quality control, we used a mouse model of dilated cardiomyopathy driven by cardiac restricted over

112 cardioverter defibrillator in patients with dilated cardiomyopathy for the primary prevention of sud

113 d controls, affecting known as well as novel dilated cardiomyopathy genes.

114 und that eQTL variants are also enriched for dilated cardiomyopathy genome-wide association signals i

115 lume Variability) in pediatric patients with dilated cardiomyopathy has reported reproducibility of s

116 ommon cause of heart failure was nonischemic dilated cardiomyopathy in 27.5% (whites, 19.9%; P<0.001)

117 and metabolic compromise in the etiology of dilated cardiomyopathy in DMD and identify a window of o

118 iated with features of both hypertrophic and dilated cardiomyopathy in mice.

119 ns presented with severe hypertrophic and/or dilated cardiomyopathy in utero, at birth, or in early c

120 Because a common finding in dilated cardiomyopathy is a reduction in the myofilament

121 Up to 40% of dilated cardiomyopathy is associated with inflammation o

122 involved in viral RNA replication.IMPORTANCE Dilated cardiomyopathy is the most common indication for

123 tes into the spectrum of hypertrophic versus dilated cardiomyopathy is unknown.

124 ividuals with either the most severe form of dilated cardiomyopathy or whose mutations demonstrated c

125 We enrolled 409 consecutive ischemic and dilated cardiomyopathy patients (mean age: 64+/-12 years

126 nces of gene expression and splicing between dilated cardiomyopathy patients and controls, affecting

127 ) in ischemic cardiomyopathy and nonischemic dilated cardiomyopathy patients evaluated for primary pr

128 ssion are each important determinants of the dilated cardiomyopathy phenotype and are controlled by g

129 d that the knockout strategy ameliorates the dilated cardiomyopathy phenotype in vitro.

130 humanized" telomere lengths, the devastating dilated cardiomyopathy phenotype seen in patients with D

131 Focusing on the dilated cardiomyopathy phenotype we found that eQTL vari

132 en with myocarditis and 1249 with idiopathic dilated cardiomyopathy received HT.

133 during cardiac contraction in patients with dilated cardiomyopathy relative to controls.

134 etween diastole and systole in patients with dilated cardiomyopathy relative to healthy controls usin

135 d reduced ejection fraction due to ischaemic dilated cardiomyopathy resulted in a significant reducti

136 Myocarditis can cause dilated cardiomyopathy resulting in end-stage heart fail

137 signatures and myocardium from subjects with dilated cardiomyopathy showed excessive Parkin and CHOP

138 ffective in females in a setting of moderate dilated cardiomyopathy than in males.

139 BCL2-associated athanogene 3 (BAG3) develop dilated cardiomyopathy that is associated with a destabi

140 r envelope gene SYNE1 in a child with severe dilated cardiomyopathy that underwent transplant, as wel

141 with ischemic cardiomyopathy or nonischemic dilated cardiomyopathy undergoing cardiovascular magneti

142 -nine patients with ischemic or non-ischemic dilated cardiomyopathy undergoing prophylactic ICD impla

143 y shows that different gene mutations induce dilated cardiomyopathy via diverse cellular pathways.

144 Ang II levels in explanted human hearts with dilated cardiomyopathy were elevated despite ACE inhibit

145 d 59+/-15 years, 80% males) with nonischemic dilated cardiomyopathy who underwent CA.

146 en with myocarditis and 1583 with idiopathic dilated cardiomyopathy who were <18 years old and listed

147 in the mouse heart for the first time during dilated cardiomyopathy with heart failure.

148 magnetic resonance identified patients with dilated cardiomyopathy without severe LV systolic dysfun

149 164 ischemic cardiomyopathy, 150 nonischemic dilated cardiomyopathy), the mean left ventricular eject

150 In patients with dilated cardiomyopathy, a more longitudinal orientation

151 is patients, 11 ischemic heart disease, nine dilated cardiomyopathy, and 11 nonfailing donors.

152 n of function occurs in 20% of children with dilated cardiomyopathy, and 40% die or undergo transplan

153 the regulation of development, ischemic and dilated cardiomyopathy, and myocardial infarction.

154 nd Becker muscular dystrophy (BMD), X-linked dilated cardiomyopathy, as well as DMD and BMD female ca

155 age-dependent cardiac phenotypes, including dilated cardiomyopathy, cardiac conduction disturbance,

156 elanocortin-4 receptor (MC4R) in mice causes dilated cardiomyopathy, characterized by reduced contrac

157 In pediatric patients with dilated cardiomyopathy, compared with dimension and area

158 otypes (hypertrophic cardiomyopathy, HCM and dilated cardiomyopathy, DCM) associated with mutations i

159 c cardiomyopathy, ischemic heart disease, or dilated cardiomyopathy, in comparison to nonfailing hear

160 y bowel disease, infections, cerebral palsy, dilated cardiomyopathy, muscular dystrophy, and schizoph

161 In patients with dilated cardiomyopathy, myocardial Gal-3 expression corr

162 emaining cardiac diagnosis groups, including dilated cardiomyopathy, myocarditis, and ischemic and no

163 if concentric hypertrophy does progress to a dilated cardiomyopathy, such a transition would occur ov

164 ted from a patient suffering from idiopathic dilated cardiomyopathy, suggesting that such mutant viru

165 ere pathologies, such as aseptic meningitis, dilated cardiomyopathy, type I diabetes, paralysis, and

166 V symptomatic heart failure due to ischaemic dilated cardiomyopathy, who had left ventricular ejectio

167 We treated a dilated cardiomyopathy-linked mouse model expressing a m

168 COX and copper deficiency that resulted in a dilated cardiomyopathy.

169 ponin protein stoichiometry in patients with dilated cardiomyopathy.

170 sease remodelling in patients suffering from dilated cardiomyopathy.

171 he most common type of genetic alteration in dilated cardiomyopathy.

172 , and leiomodin-2-knockout mice present with dilated cardiomyopathy.

173 to one that leads to chronic remodeling and dilated cardiomyopathy.

174 venting progression of inflammation-mediated dilated cardiomyopathy.

175 scle cell leads to myocyte instability and a dilated cardiomyopathy.

176 to identify novel mutations causing familial dilated cardiomyopathy.

177 al and adjuvant epicardial CA in nonischemic dilated cardiomyopathy.

178 children who are transplanted for idiopathic dilated cardiomyopathy.

179 heart failure than children with idiopathic dilated cardiomyopathy.

180 on that ultimately results in a severe fatal dilated cardiomyopathy.

181 patients, and those with either ischemic or dilated cardiomyopathy.

182 many cardiac genes, and loss of RBM20 causes dilated cardiomyopathy.

183 the structural alterations in patients with dilated cardiomyopathy.

184 al human disease phenotypes, hypertrophic or dilated cardiomyopathy.

185 recover spontaneously, some develop chronic dilated cardiomyopathy.

186 patients with heart failure due to ischaemic dilated cardiomyopathy.

187 evated in left ventricles from patients with dilated cardiomyopathy.

188 ises heart function and structure, producing dilated cardiomyopathy.

189 ation (Id cDKOs), which develops adult-onset dilated cardiomyopathy.

190 induced hypertrophy and in human hearts with dilated cardiomyopathy.

191 ular arrhythmias in patients with idiopathic dilated cardiomyopathy.

192 t ventricle (LV) ultimately transitions to a dilated cardiomyopathy.

193 ative relatives, and 9 with noncompaction or dilated cardiomyopathy.

194 g heart rate of children with chronic HF and dilated cardiomyopathy.

195 Patients with dilated cardioyopathy had a significantly lower PCr/ATP

196 Chagas disease causes a severe inflammatory dilated chronic cardiomyopathy (CCC).

197 interconnected network of narrow tubules and dilated cisternae that enclose a single lumen.

198 er medical record based on their most recent dilated comprehensive eye examination by a primary eye c

199 roduce a network architecture based on using dilated convolutions to capture features at different im

200 -myosin variants cause hypertrophic (HCM) or dilated (DCM) cardiomyopathy by disrupting sarcomere con

201 To determine the effect of dilating drops on strabismus measurements in adults.

202 CGRP dilated dural, but not pial, vessels and significantly r

203 circulation of healthy aged skin can further dilate during passive heat exposure, despite peripheral

204 hypothesis that the right ventricle is more dilated during resuscitation from cardiac arrest caused

205 The right ventricle was dilated during resuscitation from cardiac arrest caused

206 The right ventricle was more dilated during resuscitation when cardiac arrest was cau

207 ay, resulting in a buildup of virions within dilated ER vesicles.IMPORTANCE In humans, symptoms of RV

208 eported an eye care visit and 21% reported a dilated examination in the past 12 months.

209 in the past 12 months, and ever having had a dilated examination, and odds ratios for factors associa

210 y 25.0% of eyes deemed to be normal based on dilated eye examination by primary eye care physicians h

211 sed as eye care visit in the past 12 months, dilated eye examination in the past 12 months, and ever

212 The odds of reporting receipt of a dilated eye examination in the past year were also highe

213 However, the odds of reported receipt of a dilated eye examination in the past year were lower in c

214 nd Measures: Eye care visit and receipt of a dilated eye examination in the past year.

215 rty-eight percent reported ever having had a dilated eye examination.

216 wer odds of reporting ever having received a dilated eye examination.

217 atients with diabetes reported ever having a dilated eye examination; only 39.62% reported receiving

218 CI] 38.56%-40.67%) reported receiving annual dilated eye examinations, and 90.31% (CI 89.70%-90.91%)

219 ion included Snellen-measured visual acuity, dilated fundus examination, and spectral-domain optical

220 cted visual acuity, slit-lamp biomicroscopy, dilated fundus examination, wide-field photography, fund

221 distance and near visual acuity evaluation; dilated fundus examination; OCT with 12 x 6-mm thickness

222 fraction, clinical findings of slit-lamp and dilated fundus examinations.

223 ent a comprehensive eye examination in which dilated fundus photographs (disc and macula centered) we

224 comprehensive ocular examination, including dilated fundus photography.

225 Dilated funduscopy also showed bone spicule-like pigment

226 d distended duodenum filled with fluid and a dilated gallbladder containing dilute bile with high bic

227 growth of lesions, which exhibited massively dilated glands with a flattened layer of stroma.

228 Computational modeling revealed that the dilated heart results in reduced HA changes compared wit

229 ve never been analysed in human non-ischemic dilated hearts (DCM).

230 The adult mutant mice developed dilated hearts and showed significantly shorter life spa

231 sis revealed CpG hypermethylation within the dilated human thoracic aorta and in SMCs cultured from t

232 Dilated hyporeflective tubular structures within the cho

233 ction of cardiac arrest, the right ventricle dilated in all groups (p < 0.01 for all).

234 The heart either hypertrophies or dilates in response to familial mutations in genes encod

235 mucosa harboring the S631G variant revealed dilated intercellular spaces and reduced desmosomes.

236 g (basal zone hyperplasia, desquamation, and dilated intercellular spaces; P < .0001), lamina propria

237 However, the right ventricle was dilated, irrespective of the cause of arrest, and diagno

238 on the fate of the pore, which may reseal or dilate irreversibly.

239 es in diverse cell types that accumulated as dilated late endosomes.

240 The mice developed hydrocephalus and grossly dilated lateral ventricles, with accumulation of 2-hydro

241 lodged in the aortomesenteric region with a dilated left ovarian vein and pelvic varicose veins.

242 those with an ischemic pathogenesis, a more dilated left ventricle, and a detectable hs-troponin had

243 se with interval cardiovascular events and a dilated LV (increased LV end-diastolic volume [EDV] inde

244 The progression to a dilated LV was uncommon (2%, n=25).

245 as minimal, and few participants developed a dilated LV.

246 e constitutively lacking EC Map4k4 displayed dilated lymphatic capillaries, insufficient lymphatic va

247 Cholangioscopy revealed dilated lymphatic vessels obstructing the bile duct and

248 m in diameter, was inserted to the straight, dilated main duct and plastic stent(s) were inserted to

249 multilocular cysts of the pancreas and a non-dilated main pancreatic duct (<5 mm).

250 e because of mechanical interaction with the dilated main pulmonary artery (MPA).

251 n the majority of participants with branched dilated microvessels (11 of 13) (McNemar Test for equal

252 nce of vascular microhemorrhage and branched dilated microvessels in the tissues lining the clinicall

253 plasma gap could be detected in the branched dilated microvessels in tissues lining the GC.

254 sence of microhemorrhage related to branched dilated microvessels.

255 tive neurons increase their energy supply by dilating nearby arterioles and capillaries.

256 ntraocular pressure measurement, gonioscopy, dilated ophthalmoscopy, and standard automated perimetry

257 ts underwent external ocular examination and dilated ophthalmoscopy.

258 ion in ventricular tissue from patients with dilated or ischemic cardiomyopathy compared to nonfailin

259 an flicker open and closed repeatedly before dilating or resealing irreversibly.

260 electron microscopy of a liver biopsy showed dilated organelles suggestive of the endoplasmic reticul

261 topic sonic hedgehog (Shh) in subsets of non-dilated P7 mutant proximal tubules (likely driving the s

262 ntify white matter lesion load, frequency of dilated perivascular spaces (PVS) and abnormalities in c

263 ransient ('kiss and run') or an irreversibly dilating pore (full fusion).

264 to facilitate early attendance, and careful dilated postoperative examination for retinal tears is r

265 mphatic vessel can individually contract and dilate producing unidirectional flow enforced by intralu

266 In patients with a dilated proximal ascending aorta and trileaflet aortic v

267 In bicuspid aortic valve patients with dilated proximal ascending aorta, ascending aortic area/

268 In patients with bicuspid aortic valve and dilated proximal ascending aorta, we sought to assess (1

269 es, kidneys of newborn mutant mice exhibited dilated proximal tubules and immature glomeruli, and the

270 emokine production (e.g. ectopic Ccl2 in non-dilated proximal tubules), and augmented hedgehog signal

271 Kim1 and noted ectopic expression in P7 non-dilated proximal tubules.

272 valence of LMCA extrinsic compression from a dilated pulmonary artery (PA) in patients with PAH and a

273 ed pupil, and one of active locomotion and a dilated pupil, indicative of heightened attention.

274 d one had exposure keratopathy and traumatic dilated pupil.

275 ncreased NO bioavailability that dynamically dilated resistance vessels in vivo under basal condition

276 ecules and pathways that cause hypertrophic, dilated, restrictive, and arrhythmogenic cardiomyopathie

277 ning for diabetic retinopathy include annual dilated retinal examinations to detect proliferative ret

278 the left uterus with evidence of blood in a dilated retort-shaped left fallopian tube and a normal r

279 dard surgical pulmonary valve replacement in dilated right ventricular outflow tracts, permitting low

280 protogranules with paracrystalline contents, dilated rough endoplasmic reticulum, and electron-lucent

281 eurysm and aortic insufficiency secondary to dilated sinotubular junction and a normal aortic annulus

282 Rbpj from the adult endothelium also led to dilated sinusoids, vascular shunts, and necrosis, albeit

283 surface, abnormal vascular architecture, and dilated sinusoids.

284 ved that immediately afterward, fusion pores dilate spontaneously.

285 n initial small-cation conductive state to a dilated state, which allows the passage of larger synthe

286 ates as either a more closed state or a more dilated state.

287 ter is the intentional fracture of maximally dilated stents, which can be accomplished using ultra-hi

288 Participants underwent dilated stereo-digital fundus photography graded accordi

289 tivated the genioglossus muscle and markedly dilated the pharynx, whereas saline had no effect.

290 Our data suggest that 2-APB directly dilates the pore of open Orai1 channels, both ion size a

291 vesicle membranes; typically, when the pore dilates the vesicle releases its cargo.

292 geometrical effects and surface faceting to dilate thermal stresses at the surface of selectively gr

293 gest that "decisive" and "distilled" moments dilate time in the viewer's mind and might allow the mod

294 s of agonist, and the intrinsic channel pore dilates to allow the passage of fluorescent dyes.

295 lso revealed a left dysplastic kidney with a dilated, tortuous ureter.

296 nes recommend TV repair in the presence of a dilated tricuspid annulus at the time of a left-sided va

297 We further demonstrated that dilated vasculature, loss of vascular integrity resultin

298 PR-OCTA disclosed dilated vessels in the intermediate and deep plexuses in

299 ive hamartomas containing abnormal tortuous, dilated vessels prominent in the forelimbs.

300 y disease, the right coronary artery did not dilate with IHE, and dilation of the IMA was less than t